Search results for "Blood–brain barrier"

showing 10 items of 134 documents

Fingolimod (FTY720-P) Does Not Stabilize the Blood–Brain Barrier under Inflammatory Conditions in an in Vitro Model

2015

Breakdown of the blood-brain barrier (BBB) is an early hallmark of multiple sclerosis (MS), a progressive inflammatory disease of the central nervous system. Cell adhesion in the BBB is modulated by sphingosine-1-phosphate (S1P), a signaling protein, via S1P receptors (S1P\(_1\)). Fingolimod phosphate (FTY720-P) a functional S1P\(_1\) antagonist has been shown to improve the relapse rate in relapsing-remitting MS by preventing the egress of lymphocytes from lymph nodes. However, its role in modulating BBB permeabilityin particular, on the tight junction proteins occludin, claudin 5 and ZO-1has not been well elucidated to date. In the present study, FTY720-P did not change the transendotheli…

Pathologytight junctionsDrug Evaluation PreclinicalApoptosisVascular permeabilityOccludinlcsh:ChemistryMedicinelcsh:QH301-705.5Cells CulturedSpectroscopyTight junctionrat brain microvascular endothelial cell cultureGeneral MedicineFingolimodComputer Science ApplicationsCell biologyEndothelial stem cellmedicine.anatomical_structureMatrix Metalloproteinase 2Immunosuppressive AgentsFTY720-P; blood-brain barrier; rat brain microvascular endothelial cell culture; inflammation; tight junctionsmedicine.drugmedicine.medical_specialtyMultiple SclerosisMAP Kinase Signaling SystemBlood–brain barrierArticleCatalysisCapillary PermeabilityInorganic ChemistryOccludinFingolimod HydrochlorideAnimalsFTY720-Pddc:610Physical and Theoretical ChemistryClaudinMolecular BiologyFingolimod Hydrochloridebusiness.industryOrganic ChemistryEndothelial Cellsblood-brain barrierRatslcsh:Biology (General)lcsh:QD1-999inflammationMicrovesselsbusinessInternational Journal of Molecular Sciences
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FROM THE BLOOD- BRAIN BARRIER TO BEHAVIOR

2017

Research in the field of neuropsychoimmunology has enabled the researchers to show that cytokines target the brain to organize a "sickness response," which is fever, activation of hypothalamuspituitary- adrenal axis and behavioural alterations that develop in sick individuals. perypheral cytokines do not act directly on the brain; they trigger the production of cytokines in the brain parenchima itself, with a possible relay at the interface between internal milieeu and the brain, which are endothelial cells and circumven- tricular organs. The affective and behavioural changes that develop during in- fluenza are the product of a transient brain inflammatory response induced by the same proin…

Perypheral cytokineNeuropsychology and Physiological Psychologymedicine.anatomical_structureBehavioural changeChemistryNeuropsychoimmunologymedicineBlood–brain barrierNeuroscienceApplied PsychologyActa Neuropsychologica
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mPEG-PLGA Nanoparticles Labelled with Loaded or Conjugated Rhodamine-B for Potential Nose-to-Brain Delivery

2021

Nowdays, neurodegenerative diseases represent a great challenge from both the therapeutic and diagnostic points of view. Indeed, several physiological barriers of the body, including the blood brain barrier (BBB), nasal, dermal, and intestinal barriers, interpose between the development of new drugs and their effective administration to reach the target organ or target cells at therapeutic concentrations. Currently, the nose-to-brain delivery with nanoformulations specifically designed for intranasal administration is a strategy widely investigated with the goal to reach the brain while bypassing the BBB. To produce nanosystems suitable to study both in vitro and/or in vivo cells traffickin…

Pharmaceutical Scienceolfactory ensheathing cellsBlood–brain barrierArticlefluorescent dye olfactory ensheathing cells PC12 cell line co-polymers nanomedicine imagingchemistry.chemical_compoundPharmacy and materia medicaIn vivomedicineRhodamine BPC12 cell lineCytotoxicityfluorescent dye; olfactory ensheathing cells; PC12 cell line; co-polymers; nanomedicine; imagingChemistrytechnology industry and agricultureimagingnanomedicineRS1-441medicine.anatomical_structureSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoBiophysicsNanomedicineco-polymersNasal administrationfluorescent dyeDrug carrierEthylene glycolPharmaceutics
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Effects of repeated social defeat on adolescent mice on cocaine-induced CPP and self-administration in adulthood: integrity of the blood-brain barrier

2015

Social stress in adulthood enhances cocaine self-administration, an effect that has been related with an increase in extracellular signal-regulated kinase and p38α mitogen-activated protein kinase phosphorylation. A detrimental effect of cocaine on blood-brain barrier (BBB) integrity has also been reported. This study evaluates the effects of repeated social defeat (RSD) during adolescence on the reinforcing and motivational effects of cocaine in adult mice and the changes induced by RSD on BBB permeability. Cocaine self-administration, conditioned place preference and quantitative analysis of claudin-5, laminin, collagen-IV and IgG immunoreactivity took place 3 weeks after RSD. Mice social…

PharmacologySocial stressmedicine.medical_specialtyMedicine (miscellaneous)HippocampusNucleus accumbensBlood–brain barrierConditioned place preference030227 psychiatrySocial defeat03 medical and health sciencesPsychiatry and Mental health0302 clinical medicinemedicine.anatomical_structureEndocrinologyInternal medicinemedicineSelf-administrationProtein kinase APsychologyNeuroscience030217 neurology & neurosurgeryAddiction Biology
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Formation of N-methylnicotinamide in the brain from a dihydropyridine-type prodrug

1999

The enhancement of brain choline levels is a possible therapeutic option in neurodegenerative diseases; however, brain choline levels are held within narrow limits by homeostatic mechanisms including the rapid clearance of excess choline from the brain. The present study tests whether N-methylnicotinamide (NMN), an inhibitor of the outward transport of choline from the brain, can elevate brain choline levels in vivo. As NMN does not cross the blood-brain barrier, we synthesized and administered the brain-permeable prodrug, 1,4-dihydro-N-methyl-nicotinamide (DNMN), and tested its effect on the levels of NMN and choline in brain extracellular fluid, using the microdialysis procedure. Administ…

Pharmacologymedicine.medical_specialtyMicrodialysisProdrugBlood–brain barrierBiochemistrychemistry.chemical_compoundmedicine.anatomical_structureCerebrospinal fluidEndocrinologychemistryBiochemistryInternal medicineExtracellular fluidBlood plasmamedicineExtracellularCholineBiochemical Pharmacology
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Image-Guided Synthesis Reveals Potent Blood-Brain Barrier Permeable Histone Deacetylase Inhibitors

2014

Recent studies have revealed that several histone deacetylase (HDAC) inhibitors, which are used to study/treat brain diseases, show low blood-brain barrier (BBB) penetration. In addition to low HDAC potency and selectivity observed, poor brain penetrance may account for the high doses needed to achieve therapeutic efficacy. Here we report the development and evaluation of highly potent and blood-brain barrier permeable HDAC inhibitors for CNS applications based on an image-guided approach involving the parallel synthesis and radiolabeling of a series of compounds based on the benzamide HDAC inhibitor, MS-275 as a template. BBB penetration was optimized by rapid carbon-11 labeling and PET im…

PhysiologyCognitive NeuroscienceHistone Deacetylase 2Vascular permeabilityHistone Deacetylase 1Blood–brain barrierBiochemistrylaw.inventionCapillary Permeabilitychemistry.chemical_compoundlawmedicineAnimalsHumansCarbon RadioisotopesBenzamideHistone deacetylase 2BrainCell BiologyGeneral MedicinePenetration (firestop)Papio anubisHDAC1Recombinant ProteinsHistone Deacetylase Inhibitorsmedicine.anatomical_structurechemistryBiochemistryBlood-Brain BarrierPositron-Emission TomographyBenzamidesRecombinant DNABiophysicsDrug EvaluationFemaleHistone deacetylaseRadiopharmaceuticals
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Na+ -dependent neutral amino acid transporters A, ASC, and N of the blood-brain barrier: mechanisms for neutral amino acid removal.

2004

Four Na+-dependent transporters of neutral amino acids (NAA) are known to exist in the abluminal membranes (brain side) of the blood-brain barrier (BBB). This article describes the kinetic characteristics of systems A, ASC, and N that, together with the recently described Na+-dependent system for large NAA (Na+-LNAA), provide a basis for understanding the functional organization of the BBB. The data demonstrate that system A is voltage dependent (3 positive charges accompany each molecule of substrate). Systems ASC and N are not voltage dependent. Each NAA is a putative substrate for at least one system, and several NAA are transported by as many as three. System A transports Pro, Ala, His,…

PhysiologyEndocrinology Diabetes and MetabolismSodiumKineticschemistry.chemical_elementNerve Tissue ProteinsIn Vitro TechniquesLithiumBlood–brain barrierMembrane PotentialsPhysiology (medical)mental disordersExtracellular fluidmedicineAnimalsMembrane potentialchemistry.chemical_classificationMembranesTransporterExtracellular FluidAmino acidKineticsmedicine.anatomical_structureMembraneAmino Acid Transport Systems NeutralAmino Acids Neutralnervous systemchemistryBiochemistryBlood-Brain BarrierCattleAlgorithmsAmerican journal of physiology. Endocrinology and metabolism
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Uptake of polymeric nanoparticles in a human induced pluripotent stem cell-based blood-brain barrier model: Impact of size, material, and protein cor…

2021

The blood–brain barrier (BBB) maintains the homeostasis of the central nervous system, which is one of the reasons for the treatments of brain disorders being challenging in nature. Nanoparticles (NPs) have been seen as potential drug delivery systems to the brain overcoming the tight barrier of endothelial cells. Using a BBB model system based on human induced pluripotent stem cells (iPSCs), the impact of polymeric nanoparticles has been studied in relation to nanoparticle size, material, and protein corona. PLGA [poly(lactic-co-glycolic acid)] and PLLA [poly(d,l-lactide)] nanoparticles stabilized with Tween® 80 were synthesized (50 and 100 nm). iPSCs were differentiated into human brain m…

PolymersInduced Pluripotent Stem CellsStatic ElectricityGeneral Physics and AstronomyNanoparticleProtein Corona02 engineering and technology010402 general chemistryBlood–brain barrier01 natural sciencesModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyBiomaterialschemistry.chemical_compoundPolylactic Acid-Polyglycolic Acid CopolymermedicineElectric ImpedanceHumansGeneral Materials ScienceParticle SizeInduced pluripotent stem cellEndothelial CellsCell DifferentiationGeneral ChemistryHuman brain021001 nanoscience & nanotechnologyDynamic Light ScatteringFractionation Field Flow0104 chemical sciencesPLGAmedicine.anatomical_structurechemistryBlood-Brain BarrierSelective adsorptionDrug deliveryCalibrationBiophysicsNanoparticlesPolystyrenesProtein Corona0210 nano-technologyBiointerphases
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Polysorbate-80 coating enhances uptake of polybutylcyanoacrylate (PBCA)-nanoparticles by human and bovine primary brain capillary endothelial cells

2000

Certain drugs such as dalargin, loperamide or tubocurarine are not transported across the blood-brain barrier (BBB) and therefore exhibit no effects on the central nervous system. However, effects on the central nervous system can be observed when these drugs are loaded onto polybutylcyanoacrylate (PBCA)-nanoparticles and coated with polysorbate 80. The mechanism by which these complexed nanoparticles cross the BBB and exhibit their effects has not been elucidated. Cultured microvessel brain endothelial cells of human and bovine origin were used as an in vitro model for the BBB to gain further insight into the mechanism of uptake of nanoparticles. With cells from these species we were able …

PolysorbateEndotheliumGeneral NeuroscienceConfocalDrug delivery to the brainBiologyBlood–brain barrierchemistry.chemical_compoundmedicine.anatomical_structurechemistryTargeted drug deliveryNanoparticles for drug delivery to the brainImmunologyBiophysicsmedicineMicrovesselEuropean Journal of Neuroscience
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Imaging P-Glycoprotein Induction at the Blood–Brain Barrier of a β-Amyloidosis Mouse Model with 11C-Metoclopramide PET

2019

P-glycoprotein (ABC subfamily B member 1, ABCB1) plays an important role at the blood–brain barrier (BBB) in promoting clearance of neurotoxic β-amyloid (Aβ) peptides from the brain into the blood. ABCB1 expression and activity were found to be decreased in the brains of Alzheimer disease patients. Treatment with drugs that induce cerebral ABCB1 activity may be a promising approach to delay the build-up of Aβ deposits in the brain by enhancing clearance of Aβ peptides from the brain. The aim of this study was to investigate whether PET with the weak ABCB1 substrate radiotracer 11C-metoclopramide can measure ABCB1 induction at the BBB in a β-amyloidosis mouse model (APP/PS1-21 mice) and in w…

Pregnane X receptorMetoclopramidebiologybusiness.industryActivator (genetics)AmyloidosisPharmacologyBlood–brain barriermedicine.disease03 medical and health sciences0302 clinical medicinemedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinMedicineImmunohistochemistryRadiology Nuclear Medicine and imagingAlzheimer's diseasebusiness030217 neurology & neurosurgerymedicine.drugP-glycoproteinJournal of Nuclear Medicine
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