Search results for "BrdU"

showing 5 items of 5 documents

Hypocellularity in the murine model for Down Syndrome Ts65Dn is not affected by adult neurogenesis

2016

Down syndrome (DS) is caused by the presence of an extra copy of the chromosome 21 and it is the most common aneuploidy producing intellectual disability. Neural mechanisms underlying this alteration may include defects in the formation of neuronal networks, information processing and brain plasticity. The murine model for DS, Ts65Dn, presents reduced adult neurogenesis. This reduction has been suggested to underlie the hypocellularity of the hippocampus as well as the deficit in olfactory learning in the Ts65Dn mice. Similar alterations have also been observed in individuals with DS. To determine whether the impairment in adult neurogenesis is, in fact, responsible for the hypocellularity …

0301 basic medicineanimal diseasesHippocampusSubventricular zoneBiotecnologiaHippocampusSubgranular zonelcsh:RC321-57103 medical and health sciences0302 clinical medicinedoublecortinNeuroplasticitymental disordersmedicineBrdUlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchbiologyGeneral NeuroscienceNeurogenesisOlfactory BulbOlfactory bulbDoublecortinCell biologyadult neurogenesisTs65Dn mice030104 developmental biologymedicine.anatomical_structureHypocellularityPsicobiologianervous systembiology.proteinDown SyndromeKi67Neuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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Neuronal cell cycle: the neuron itself and its circumstances.

2015

Neurons are usually regarded as postmitotic cells that undergo apoptosis in response to cell cycle reactivation. Nevertheless, recent evidence indicates the existence of a defined developmental program that induces DNA replication in specific populations of neurons, which remain in a tetraploid state for the rest of their adult life. Similarly, de novo neuronal tetraploidization has also been described in the adult brain as an early hallmark of neurodegeneration. The aim of this review is to integrate these recent developments in the context of cell cycle regulation and apoptotic cell death in neurons. We conclude that a variety of mechanisms exists in neuronal cells for G1/S and G2/M check…

ApoptosisBrdU 5-bromo-2′-deoxyuridineReviewp75NTR neurotrophin receptor p75Nervous SystemG0 quiescent stateCKI Cdk-inhibitorNeuronsCell DeathNeurodegenerationCell CycleapoptosisNeurodegenerative DiseasesCell cycleCell biologymedicine.anatomical_structureInk inhibitor of kinaseBDNF brain-derived neurotrophic factorp38MAPK p38 mitogen-activated protein kinaseG2 growth phase 2Programmed cell deathS-phasePD Parkinson diseaseRb RetinoblastomaMcm2 minichromosome maintenance 2PCNA proliferating cell nuclear antigenMitosisContext (language use)BiologyCdk cyclin-dependent kinaseCNS central nervous systemS-phase synthesis phase.Cip/Kip cyclin inhibitor protein/kinase inhibitor proteinmedicineAnimalsHumansMolecular BiologyMitosisTetraploidAD Alzheimer diseasecell cycle re-entryDNA replicationCell BiologyNeuronmedicine.diseaseG1 growth phase 1neuronRGCs retinal ganglion cellsCell cycle re-entrytetraploidnervous systemApoptosisNeuronDevelopmental BiologyCell cycle (Georgetown, Tex.)
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The origin of postembryonic neuroblasts in the ventral nerve cord of Drosophila melanogaster.

1991

ABSTRACT Embryonic and postembryonic neuroblasts in the thoracic ventral nerve cord of Drosophila melanogaster have the same origin. We have traced the development of threefold-labelled single precursor cells from the early gastrula stage to late larval stages. The technique allows in the same individual monitoring of progeny cells at embryonic stages (in vivo) and differentially staining embryonic and postembryonic progeny within the resulting neural clone at late postembryonic stages. The analysis reveals that postembryonic cells always appear together with embryonic cells in one clone. Further-more, BrdU labelling suggests that the embryonic neuroblast itself rather than one of its proge…

Central Nervous Systemanimal structuresNeurogenesisClone (cell biology)BiologyNeuroblastNeuroblasts/dk/atira/pure/subjectarea/asjc/2700/2702AnimalsBrdUMolecular BiologyCell lineageNeuroblast proliferationStem CellsfungiEmbryogenesisCell BiologyAnatomyGastrulaEmbryonic stem cellCell biologyGastrulationDrosophila melanogasterBromodeoxyuridineVentral nerve cordDrosophilaAnatomy/dk/atira/pure/subjectarea/asjc/1300/1307Ganglion mother cellDevelopmental BiologyDevelopment (Cambridge, England)
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Differential alterations in the small intestine epithelial cell turnover during acute and chronic infection with Echinostoma caproni (Trematoda)

2015

Background The intestinal epithelium plays a multifactorial role in mucosal defense. In this sense, augmented epithelial cell turnover appears as a potential effector mechanism for the rejection of intestinal-dwelling helminths. Methods A BrdU pulse-chase experiment was conducted to investigate the infection-induced alterations on epithelial cell kinetics in hosts of high (mouse) and low (rat) compatibility with the intestinal trematode Echinostoma caproni. Results High levels of crypt-cell proliferation and tissue hyperplasia were observed in the ileum of infected mice, coinciding with the establishment of chronic infections. In contrast, the cell migration rate was about two times higher …

MaleProliferationEchinostoma caproniIleumBiologyMiceCell MovementEchinostomaIntestine SmallmedicineAnimalsHumansBrdUExpulsionIntestinal MucosaRats WistarCell ProliferationEchinostomiasisMice Inbred ICRCell growthResearchCell migrationHyperplasiamedicine.diseaseIntestinal epitheliumEpitheliumSmall intestineIntestineRatsCell biologyChronic infectionInfectious Diseasesmedicine.anatomical_structureCell turnoverAcute DiseaseChronic DiseaseImmunologyChronicityParasitologyParasites & Vectors
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Fibroblast growth factor-2 and its receptor expression in proliferating precursor cells of the subventricular zone in the adult rat brain

2008

Several findings have suggested the existence in the subventricular zone (SVZ) from sagittal sections of adult rat brain of a trophic mechanism, mediated by fibroblast growth factor-2 (FGF-2) and its multiple high-affinity FGF receptors (FGFRs), regulating neurogenesis mainly by controlling precursor cell proliferation. However, no clear data are available on the expression of FGF-2 and FGFRs in proliferating precursor cells of the SVZ. To address these questions we examined FGF-2 mRNA and its FGFR mRNA expression in proliferating precursor cells of the SVZ by using a double labeling procedure, combining in situ hybridization for FGF-2 and its FGFR mRNA with immunohistochemistry for bromode…

Maleanimal diseasesReceptor expressionGene ExpressionFGF-2Subventricular zoneSVZBiologySettore BIO/09 - FisiologiaCerebral Ventricleschemistry.chemical_compoundPrecursor cellmedicineAnimalsReceptor Fibroblast Growth Factor Type 3RNA MessengerReceptor Fibroblast Growth Factor Type 1Rats WistarReceptor Fibroblast Growth Factor Type 2BrdUCell ProliferationFGF receptorGeneral NeuroscienceFibroblast growth factor receptor 1NeurogenesisBrainPrecursor cellsFibroblast growth factor receptor 4RatsCell biologyAdult Stem CellsFGF receptorsmedicine.anatomical_structureBromodeoxyuridinenervous systemchemistryFibroblast growth factor receptorFibroblast Growth Factor 2NeuroscienceBromodeoxyuridineBrdU; FGF receptors; FGF-2Neuroscience Letters
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