Search results for "Bumetanide"

showing 8 items of 8 documents

Bumetanide prevents brain trauma-induced depressive-like behavior

2019

AbstractBrain trauma triggers a cascade of deleterious events leading to enhanced incidence of drug resistant epilepsies, depression and cognitive dysfunctions. The underlying mechanisms leading to these alterations are poorly understood and treatment that attenuates those sequels not available. Using controlled-cortical impact (CCI) as experimental model of brain trauma in adult mouse we found a strong suppressive effect of the sodium-potassium-chloride importer (NKCC1) specific antagonist bumetanide on appearance of depression-like behavior. We demonstrate that this alteration in behavior is associated with a block of CCI-induced decrease in parvalbumin-positive interneurons and impairmen…

0301 basic medicineDOWN-REGULATIONpotassium chloride cotransporter 2 (KCC2)[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHippocampusUP-REGULATION0302 clinical medicineMedicineCOTRANSPORTER KCC2NEURAL STEM-CELLBrain traumaDepression (differential diagnoses)Original Research0303 health sciencesNeurogenesisDepolarizationNeural stem cell3. Good healthADULT HIPPOCAMPAL NEUROGENESISneurogenesis[SDV.SP.PHARMA] Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologydepressionBumetanidemedicine.druginterneuron cell deathpsychiatric diseaseINHIBITIONbumetanidelcsh:RC321-571Cellular and Molecular Neuroscience03 medical and health sciencesINJURYlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryMolecular Biology030304 developmental biologybusiness.industryMechanism (biology)GRANULE CELLSDentate gyrusAntagonist3112 Neurosciences[SDV.NEU.NB] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology030104 developmental biologyDENTATE GYRUSDIURETIC BUMETANIDE[SDV.SP.PHARMA]Life Sciences [q-bio]/Pharmaceutical sciences/PharmacologybusinessNeuroscience030217 neurology & neurosurgeryNeuroscience
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Pathology-selective antiepileptic effects in the focal freeze-lesion rat model of malformation of cortical development

2021

Malformations of cortical development (MCD) represent a group of rare diseases with severe clinical presentation as epileptic and pharmacoresistant encephalopathies. Morphological studies in tissue from MCD patients have revealed reduced GABAergic efficacy and increased intracellular chloride concentration in neuronal cells as important pathophysiological mechanisms in MCD. Also, in various animal models, alterations of GABAergic inhibition have been postulated as a predominant factor contributing to perilesional hyperexcitability. Along with this line, the NKCC1 inhibitor bumetanide has been postulated as a potential drug for treatment of epilepsy, mediating its antiepileptic effect by red…

Male0301 basic medicinePathologymedicine.medical_specialtyZonisamideInhibitory postsynaptic potentialCryosurgeryLesion03 medical and health sciencesEpilepsyOrgan Culture Techniques0302 clinical medicineSodium Potassium Chloride Symporter InhibitorsDevelopmental NeuroscienceSeizuresmedicineAnimals4-AminopyridineRats WistarBumetanideCerebral Cortexbusiness.industryCarbamazepinemedicine.diseaseRatsMicrogyrusMalformations of Cortical Development030104 developmental biologyNeurologyGABAergicAnticonvulsantsmedicine.symptombusiness030217 neurology & neurosurgeryBumetanidemedicine.drugExperimental Neurology
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Kinetic Properties of Cl−Uptake Mediated by Na+-Dependent K+-2Cl−Cotransport in Immature Rat Neocortical Neurons

2007

GABA, the main inhibitory neurotransmitter in the adult nervous system, evokes depolarizing membrane responses in immature neurons, which are crucial for the generation of early network activity. Although it is well accepted that depolarizing GABA actions are caused by an elevated intracellular Cl−concentration ([Cl−]i), the mechanisms of Cl−accumulation in immature neurons are still a matter of debate. Using patch-clamp, microfluorimetric, immunohistochemical, and molecular biological approaches, we studied the mechanism of Cl−uptake in Cajal-Retzius (CR) cells of immature [postnatal day 0 (P0) to P3] rat neocortex. Gramicidin-perforated patch-clamp and 6-methoxy-N-ethylquinolinium-microfl…

Sodium-Potassium-Chloride SymportersNeocortexStimulationBiologyChloridesmedicineAnimalsSolute Carrier Family 12 Member 2Rats Wistargamma-Aminobutyric AcidNeuronsNeocortexGeneral NeuroscienceExcitatory Postsynaptic PotentialsCell DifferentiationDepolarizationArticlesRatsKineticsmedicine.anatomical_structureAnimals NewbornBiochemistryExcitatory postsynaptic potentialBiophysicsGABAergicCotransporterIntracellularBumetanidemedicine.drugThe Journal of Neuroscience
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Activation of glycine receptors modulates spontaneous epileptiform activity in the immature rat hippocampus

2014

While the expression of glycine receptors in the immature hippocampus has been shown, no information about the role of glycine receptors in controlling the excitability in the immature CNS is available. Therefore, we examined the effect of glycinergic agonists and antagonists in the CA3 region of an intact corticohippocampal preparation of the immature (postnatal days 4-7) rat using field potential recordings. Bath application of 100 μM taurine or 10 μM glycine enhanced the occurrence of recurrent epileptiform activity induced by 20 μM 4-aminopyridine in low Mg(2+) solution. This proconvulsive effect was prevented by 3 μM strychnine or after incubation with the loop diuretic bumetanide (10 …

TaurinePhysiologyTaurineGlycinePharmacologyHippocampuschemistry.chemical_compoundNeuroscience: Development/Plasticity/RepairReceptors GlycinemedicineAnimalsRats WistarReceptorGlycine receptorCells CulturedEpilepsyChemistryGABAA receptorStrychnineRatsBiochemistrynervous systemAnimals NewbornGlycineGabazineAnticonvulsantsBumetanidemedicine.drug
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Analysis of Diuretics in Urine by Column-Switching Chromatography and Fluorescence Detection

1997

The potential of column-switching chromatography and fluorescence detection for the analysis of diuretics in urine is evaluated. Sample cleanup and chromatographic parameters have been optimized to achieve maximum sensitivity for the detection and quantification of some relevant diuretics. On the basis of these studies, an on-line procedure for the simultaneous determination of amiloride, furosemide, bumetanide and triamterene is presented. The linearity, precision, accuracy and sensitivity of the method are discussed. The utility of the described approach has been tested by analysing urine samples obtained after administration of bumetanide.

TriamtereneChromatographyChemistrymedicine.drug_classmedicine.medical_treatmentClinical BiochemistryPharmaceutical ScienceUrineBiochemistryHigh-performance liquid chromatographyFluorescence spectroscopyAnalytical ChemistryPotassium-sparing diureticmedicineDiureticQuantitative analysis (chemistry)Bumetanidemedicine.drugJournal of Liquid Chromatography & Related Technologies
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Chromatographic determination of diuretics in urine samples using hybrid micellar mobile phases with fluorimetric detection

1998

Abstract A rapid and simple chromatographic procedure, that uses hybrid micellar mobile phases of sodium dodecyl sulphate (SDS), propanol and phosphate buffer at pH 3, and fluorimetric detection ( λ exc =270 nm, λ em =430 nm), is reported for the determination of mixtures of seven diuretics of diverse efficacy (amiloride, bendroflumethiazide, bumetanide, furosemide, hydroflumethiazide, piretanide and triamterene), in urine samples. The application of a sequential optimization procedure for the resolution of the mixtures of the diuretics was problematic. However, their separation was achieved through the use of an interpretive optimization procedure based on the accurate description of the r…

TriamtereneDetection limitHydroflumethiazideChromatographyChemistrymedicine.medical_treatmentPiretanideBiochemistryAnalytical Chemistrychemistry.chemical_compoundMicellar liquid chromatographymedicineEnvironmental ChemistryBendroflumethiazideDiureticSpectroscopyBumetanidemedicine.drugAnalytica Chimica Acta
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Model-specific effects of bumetanide on epileptiform activity in the in-vitro intact hippocampus of the newborn mouse.

2007

The immature brain has a higher susceptibility to develop seizures, which often respond poorly to classical pharmacological treatment. It has been recently suggested that bumetanide, which blocks Na(+)-dependent K(+)-Cl(-)-cotransporter isoform 1 (NKCC1) and thus attenuates depolarizing GABAergic responses, could soothe epileptiform activity in immature nervous systems. To evaluate whether bumetanide consistently attenuates epileptiform activity, we investigated the effect of 10 microM bumetanide in five different in-vitro epilepsy models using field potential recordings in the CA3 region of intact mouse hippocampal preparations at postnatal day 4-7. Bumetanide reduced amplitude and frequen…

medicine.medical_specialtySodium-Potassium-Chloride SymportersHippocampusKainate receptorHippocampal formationIn Vitro TechniquesHippocampusMembrane PotentialsCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsyMiceChloride ChannelsInternal medicinemedicineAnimalsSolute Carrier Family 12 Member 2MagnesiumBumetanidePharmacologyEpilepsyDepolarizationStrychninemedicine.diseaseDisease Models AnimalEndocrinologychemistryAnimals NewbornGabazinePotassiumBumetanidemedicine.drugNeuropharmacology
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Hemodynamic Changes in Patients with Essential Hypertension after Therapy with Muzolimine

1987

Muzolimine is a new nonsulfonamide diuretic belonging to the pyrazolinone group (1).It is a potent high-ceiling diuretic agent like furosemide and bumetanide with long duration of action like thiazides (1, 2).

medicine.medical_specialtybusiness.industrymedicine.medical_treatmentCardiac indexFurosemideHemodynamicsEssential hypertensionmedicine.diseasechemistry.chemical_compoundchemistryInternal medicinemedicineCardiologyMuzolimineIn patientDiureticbusinessBumetanidemedicine.drug
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