Search results for "C3"

showing 10 items of 1295 documents

HLA and killer cell immunoglobulin-like receptor (KIRs) genotyping in patients with acute ischemic stroke

2019

Abstract Introduction In humans, a major component of natural killer (NK) and T cell target recognition depends on the surveillance of human leukocyte antigen (HLA) class I molecules by killer immunoglobulin-like receptors (KIRs). Aims To implement the knowledge about the immunological genetic background of acute ischemic stroke susceptibility in relation to the frequency of the KIR genes and HLA alleles. Methods Subjects with acute ischemic stroke and subjects without stroke were genotyped for the presence of KIR genes and of the three major KIR ligand groups, HLA-C1, HLA-C2, and HLA-Bw4, both HLA-B and HLA-A loci. Results Between November 2013 and February 2016, consecutive patients with …

MaleKiller immunoglobulin-like receptors (KIRs)0301 basic medicinemedicine.medical_specialtySettore MED/09 - Medicina InternaNeurologyGenotypeT cellKIR LigandImmunologyKiller-cell immunoglobulin-like receptorchemical and pharmacologic phenomenaHuman leukocyte antigenlcsh:RC346-429Proinflammatory cytokine03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineReceptors KIRotorhinolaryngologic diseasesHumansMedicineGenetic Predisposition to DiseaseReceptorStrokelcsh:Neurology. Diseases of the nervous systemAgedSettore MED/04 - Patologia GeneraleNeuroscience (all)business.industryResearchGeneral NeuroscienceHistocompatibility Antigens Class IMiddle Agedmedicine.diseaseStrokeHLACross-Sectional Studies030104 developmental biologymedicine.anatomical_structureNeurologyImmunologyFemalebusiness030217 neurology & neurosurgeryJournal of Neuroinflammation
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Biochemical properties of MHC class II molecules endogenously synthesized and expressed by mouse Langerhans cells

1991

The cell surface expression and biosynthesis of Langerhans cells (LC)-derived major histocompatibility complex (MHC) class II molecules from epidermal cells (EC) prepared freshly and cultured for up to 3 days was investigated. Based on the constitutive expression of MHC class II determinants by LC, a panning and magnetic bead selection procedure was employed, yielding 65% and 86% of I-A+ cells, respectively. Phenotypical and cytochemical examinations revealed that the two LC preparations were free of contaminating macrophages as well as B and T cells. Freshly prepared enriched LC were highly efficient in the stimulation of protein antigen-specific T cell clones, while LC purified from short…

MaleLangerhans cellT cellImmunologyCellMajor histocompatibility complexFlow cytometryIodine RadioisotopesMicemedicineAnimalsImmunology and AllergyCells CulturedLymphokinesMice Inbred BALB CMice Inbred C3HMHC class IIEpidermis (botany)medicine.diagnostic_testbiologyHistocompatibility Antigens Class IIFlow CytometryMolecular biologyIn vitroPhenotypemedicine.anatomical_structureLangerhans CellsImmunologybiology.proteinFemaleEuropean Journal of Immunology
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Oncogene transformation can induce tolerogenicity in murine macrophages after down-regulation of immunogenicity without altering major histocompatibi…

1993

In vitro studies on cell lines may allow analyses of the mechanisms of immunogenicity and tolerogenicity in cells. We used a model of oncogenic transformation of an established murine macrophage cell line and report here that one v-mos-transformed clone expressing unaltered high amounts of MHC class I and II antigens does not induce proliferation of unprimed T cells in primary mixed lymphocyte reactions, in sharp contrast to its non-transformed parental cells. Interestingly, this clone induces specific unresponsiveness, as revealed by the lack of responsiveness of MHC-specific T cells when subsequently exposed to the pertinent MHC alloantigens in immunogenic form but unaltered MHC-third par…

MaleLymphocyteT-LymphocytesImmunologyClone (cell biology)Down-RegulationBiologyMajor histocompatibility complexImmune toleranceCell LineMiceTransformation GeneticAntigenHistocompatibility AntigensMHC class ImedicineImmune ToleranceAnimalsRNA MessengerGenes mosMice Inbred BALB CMice Inbred C3HImmunogenicityMacrophagesGeneral MedicineCell biologyClone CellsMice Inbred C57BLmedicine.anatomical_structureCell cultureImmunologybiology.proteinFemaleLymphocyte Culture Test MixedCell DivisionInterleukin-1Scandinavian journal of immunology
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Circulating CSF-1 Promotes Monocyte and Macrophage Phenotypes that Enhance Lupus Nephritis

2009

Macrophages mediate kidney disease and are prominent in a mouse model (MRL- Fas lpr ) of lupus nephritis. Colony stimulating factor-1 (CSF-1) is the primary growth factor for macrophages, and CSF-1 deficiency protects MRL- Fas lpr mice from kidney disease and systemic illness. Whether this renoprotection derives from a reduction of macrophages and whether systemic CSF-1, as opposed to intrarenal CSF-1, promotes macrophage-dependent lupus nephritis remain unclear. Here, we found that increasing systemic CSF-1 hastened the onset of lupus nephritis in MRL- Fas lpr mice. Using mutant MRL- Fas lpr strains that express high, moderate, or no systemic CSF-1, we detected a much higher tempo of kidne…

MaleMacrophage colony-stimulating factorMice Inbred MRL lprLupus nephritisMice TransgenicInflammationKidneyMonocytesMiceimmune system diseasesmedicineAnimalsHumansskin and connective tissue diseasesCell ProliferationInflammationMice Inbred BALB CMice Inbred C3HSystemic lupus erythematosusbiologyCD68business.industryMacrophage Colony-Stimulating FactorMacrophagesMonocyteGeneral MedicineMonocyte proliferationmedicine.diseaseLupus NephritisMice Inbred C57BLDisease Models AnimalBasic ResearchPhenotypemedicine.anatomical_structureIntegrin alpha MNephrologyImmunologybiology.proteinFemalemedicine.symptombusinessJournal of the American Society of Nephrology
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Prevention of the post-chemotherapy relapse of tuberculous infection by combined immunotherapy

2008

Summary We report that a recently developed combined immunotherapy (CIT) has the capacity to prevent a spontaneous relapse of replicating Mycobacterium tuberculosis bacilli in the lungs of BALB/c, C57Bl/6 or C3H/HeJ strains of mice, following 4 weeks of non-sterilising treatment with isoniazid and rifampicin. The CIT regimen, represented by recombinant IFNγ, anti-α crystalline monoclonal IgA antibody and IL-4 neutralizing polyclonal antibody, reduced the 8-week relapse of viable bacterial counts in the lungs most significantly, when CIT was inoculated during the 5th week post infection, i.e. during the 3rd week of chemotherapy. Although CIT enhanced lung granuloma area, nitric oxide, cytoki…

MaleMicrobiology (medical)TuberculosisTuberculosiAntibodiemedicine.medical_treatmentImmunologyAntitubercular AgentsColony Count MicrobialMicrobiologyAntibodiesMycobacterium tuberculosisInterferon-gammaMiceAdjuvants ImmunologicRecurrencemedicineAnimalsalpha-CrystallinsRelapseTuberculosis PulmonaryCytokineMice Inbred BALB CMice Inbred C3HChemotherapyLungbiologybusiness.industryTuberculosis; Cytokines; Antibodies; Immunotherapy; RelapseIsoniazidMycobacterium tuberculosisImmunotherapybiology.organism_classificationmedicine.diseaseCombined Modality TherapyRecombinant ProteinsImmunoglobulin AMice Inbred C57BLRegimenInfectious Diseasesmedicine.anatomical_structureModels AnimalImmunologyInterleukin-4ImmunotherapybusinessRifampicinmedicine.drugTuberculosis
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Dysfunction of attention switching networks in amyotrophic lateral sclerosis

2019

Objective To localise and characterise changes in cognitive networks in Amyotrophic Lateral Sclerosis (ALS) using source analysis of mismatch negativity (MMN) waveforms. Rationale The MMN waveform has an increased average delay in ALS. MMN has been attributed to change detection and involuntary attention switching. This therefore indicates pathological impairment of the neural network components which generate these functions. Source localisation can mitigate the poor spatial resolution of sensor-level EEG analysis by associating the sensor-level signals to the contributing brain sources. The functional activity in each generating source can therefore be individually measured and investigat…

MaleMismatch negativitySource localisationEEG ElectroencephalographyMismatch negativityNetworkElectroencephalographylcsh:RC346-429PET Positron emission tomographyCognition0302 clinical medicineC9orf72AttentionEEGAUROC Area under receiver operating characteristic curveAmyotrophic lateral sclerosisAged 80 and overmedicine.diagnostic_test05 social sciencesCognitive flexibilityBrainRegular ArticleElectroencephalographyCognitionMiddle AgedSTG Superior temporal gyrusNeurologyMTG Mid temporal gyrusDLPFC Dorsolateral prefrontal cortexlcsh:R858-859.7FemaleLCMV Linearly constrained minimum varianceIFG Inferior frontal gyrusAdultCognitive Neurosciencelcsh:Computer applications to medicine. Medical informatics050105 experimental psychologyCWIT Colour-word interference test03 medical and health sciencesfMRI Functional magnetic resonance imagingMEG MagnetoencephalographymedicineMMN Mismatch negativityHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingLS Amyotrophic Lateral SclerosisAAL Automated Anatomical Labellinglcsh:Neurology. Diseases of the nervous systemAEP Auditory evoked potentialAgedbusiness.industryAmyotrophic Lateral SclerosisIQR Interquartile rangeNeurophysiologyqEEG Quantitative EEGmedicine.diseaseNeurology (clinical)Nerve NetFunctional magnetic resonance imagingbusinessNeuroscience030217 neurology & neurosurgeryeLORETA Exact low-resolution brain electromagnetic tomographyNeuroImage: Clinical
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Tower of London test: a comparison between conventional statistic approach and modelling based on artificial neural network in differentiating fronto…

2011

The early differentiation of Alzheimer’s disease (AD) from frontotemporal dementia (FTD) may be difficult. The Tower of London (ToL), thought to assess executive functions such as planning and visuo-spatial working memory, could help in this purpose. Twentytwo Dementia Centers consecutively recruited patients with early FTD or AD. ToL performances of these groups were analyzed using both the conventional statistical approaches and the Artificial Neural Networks (ANNs) modelling. Ninety-four non aphasic FTD and 160 AD patients were recruited. ToL Accuracy Score (AS) significantly (p < 0.05) The use of hidden information contained in the different items of ToL and the non linear processing of…

MaleModels NeurologicalneuropsychologyNeurosciences. Biological psychiatry. NeuropsychiatryTower of LondonNeuropsychological Testsfrontotemporal dementiaSensitivity and SpecificityDiagnosis DifferentialExecutive FunctionAlzheimer's disease frontotemporal dementia Tower of London neuropsychology executive functionsAlzheimer Diseasemental disordersHumansAgedMED/26 - NEUROLOGIAAged 80 and overGeneral MedicineMiddle Agedexecutive functionsNeuropsychology and Physiological PsychologyNeurologyNonlinear Dynamicstest torre di Londra neuropsicologia demenzaFemaleSettore MED/26 - NeurologiaNeurology (clinical)Neural Networks ComputerAlzheimer’s diseaseRC321-571Research Article
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Functional networks of motor inhibition in conversion disorder patients and feigning subjects

2016

The neural correlates of motor inhibition leading to paresis in conversion disorder are not well known. The key question is whether they are different of those of normal subjects feigning the symptoms. Thirteen conversion disorder patients with hemiparesis and twelve healthy controls were investigated using functional magnetic resonance tomography under conditions of passive motor stimulation of the paretic/feigned paretic and the non-paretic hand. Healthy controls were also investigated in a non-feigning condition. During passive movement of the affected right hand conversion disorder patients exhibited activations in the bilateral triangular part of the inferior frontal gyri (IFG), with a…

MaleMotor DisordersAudiologylcsh:RC346-4290302 clinical medicineddc:150Neural PathwaysImage Processing Computer-AssistedYoung adultPrefrontal cortexFeigningParesismedicine.diagnostic_test05 social sciencesfMRIRegular ArticleMiddle AgedMagnetic Resonance ImagingInhibition PsychologicalTreatment OutcomeNeurologyMotor inhibitionlcsh:R858-859.7Femalemedicine.symptomPsychologyAdultmedicine.medical_specialtyImagery PsychotherapyCognitive Neurosciencelcsh:Computer applications to medicine. Medical informatics050105 experimental psychologyFunctional networks03 medical and health sciencesYoung AdultmedicineHumans0501 psychology and cognitive sciencesRadiology Nuclear Medicine and imagingIn patientConversion disorderlcsh:Neurology. Diseases of the nervous systemMagnetic resonance imagingmedicine.diseaseOxygenHemiparesisConversion disorderMotor paresisNeurology (clinical)NeuroscienceConversion disorder ; Motor inhibition ; Feigning ; fMRI ; Motor paresis030217 neurology & neurosurgery
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Reinstatement of Morphine-Induced Conditioned Place Preference in Mice by Priming Injections

2004

To construct a model of relapse of drug abuse in mice, the induction, we evaluated the extinction and reinstatement of morphine-induced place preference. In Experiment 1, we examined the effects of morphine (0, 2, 3, 5, 10, 20 and 40 mg/kg) in the conditioned place preference (CPP) paradigm. Mice showed CPP with 5, 10, 20 and 40 mg/kg. In Experiment 2, we evaluated the effects of two different extinction procedures. After conditioning with 40 mg/kg of morphine, the mice underwent daily extinction sessions of 60 or 15 min of duration. CPP was extinguished after seven and nine sessions, respectively. In Experiment 3, we tested the reinstating effects of several priming doses of morphine. Mice…

MaleNarcoticsReinforcement SchedulePharmacologyArticleExtinction Psychologicallcsh:RC321-571MiceRewardmedicineAnimalslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryDose-Response Relationship DrugMorphineExtinction (psychology)Conditioned place preferenceDose–response relationshipNeurologyAnesthesiaMorphineConditioning OperantConditioningNeurology (clinical)PsychologyReinforcement PsychologyPriming (psychology)Injections Intraperitonealmedicine.drugNeural Plasticity
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Validation of a New Cognitive Screening Method for Stroke Patients

2019

Objective. Two million adults under fifty years of age have a cerebral stroke every year worldwide. Neuropsychological assessment is the best way to identify poststroke cognitive dysfunction, but it is often time-consuming and can be tiring for the patient, and hospitals vary in their availability of neuropsychological expertise. A valid and reliable cognitive screening method could be advantageous in identifying patients who need comprehensive neuropsychological examination. Our purpose in this study was to validate a newly developed cognitive screening method as an identifier of cognitive dysfunction after stroke in working-aged patients. Methods. We analyzed new cognitive screening metho…

MaleNeuropsychological TestsaivohalvauspotilaatCognition0302 clinical medicineMass ScreeningMedicine030212 general & internal medicineNeuropsychological assessmentCognitive declineStrokevalidationmedicine.diagnostic_testNeuropsychologyCognitionGeneral MedicineMiddle Agedkognitiiviset prosessitMental Status and Dementia TestsStrokeNeuropsychology and Physiological PsychologyNeurologyvalidointiFemaleRC321-571Research ArticleAdultmedicine.medical_specialtyPsychometricsArticle SubjecttoimintahäiriötConcurrent validityNeurosciences. Biological psychiatry. NeuropsychiatrySensitivity and Specificity03 medical and health sciencesCronbach's alphaHumansCognitive DysfunctionAgedseulontatutkimusReceiver operating characteristicbusiness.industryReproducibility of Resultsstroke patientsmedicine.diseasecognitive screening methodROC CurvePhysical therapyNeurology (clinical)Cognition Disordersbusiness030217 neurology & neurosurgeryBehavioural Neurology
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