Search results for "C3"

showing 10 items of 1295 documents

Modeling Vestibular Compensation: Neural Plasticity Upon Thalamic Lesion.

2020

The present study in rats was conducted to identify brain regions affected by the interruption of vestibular transmission and to explore selected aspects of their functional connections. We analyzed, by positron emission tomography (PET), the regional cerebral glucose metabolism (rCGM) of cortical, and subcortical cerebral regions processing vestibular signals after an experimental lesion of the left laterodorsal thalamic nucleus, a relay station for vestibular input en route to the cortical circuitry. PET scans upon galvanic vestibular stimulation (GVS) were conducted in each animal prior to lesion and at post-lesion days (PLD) 1, 3, 7, and 20, and voxel-wise statistical analysis of rCGM a…

Thalamusneuronal tracingSensory systemSomatosensory systemInsular cortexlcsh:RC346-429Lesionlesion03 medical and health sciences0302 clinical medicinethalamusMedicinePET-imagingimmunofluorescenceGalvanic vestibular stimulationlcsh:Neurology. Diseases of the nervous system030304 developmental biologyOriginal ResearchVestibular system0303 health sciencesbusiness.industrymedicine.anatomical_structureNeurologyCerebral cortexcerebral cortexNeurology (clinical)medicine.symptombusinessNeuroscience030217 neurology & neurosurgeryFrontiers in neurology
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Automaton (Semi)groups (Basic Concepts)

2018

In this paper, we give an introduction to basic concepts of automaton semigroups. While we must note that this paper does not contain new results, it is focused on extended introduction in the subject and detailed examples.

TheoryofComputation_MATHEMATICALLOGICANDFORMALLANGUAGESTheoryofComputation_COMPUTATIONBYABSTRACTDEVICES20M35 68Q70 20B35 20C32Mathematics - Group Theory
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The designer cytokine hyper-interleukin-6 is a potent activator of STAT3-dependent gene transcription in vivo and in vitro.

1999

Interleukin-6 (IL-6) triggers pivotal pathways in vivo. The designer protein hyper-IL-6 (H-IL-6) fuses the soluble IL-6 receptor (sIL-6R) through an intermediate linker with IL-6. The intracellular pathways that are triggered by H-IL-6 are not defined yet. Therefore, we studied the molecular mechanisms leading to H-IL-6-dependent gene activation. H-IL-6 stimulates haptoglobin mRNA expression in HepG2 cells, which is transcriptionally mediated as assessed by run-off experiments. The increase in haptoglobin gene transcription correlates with higher nuclear translocation of tyrosine-phosphorylated STAT3 and its DNA binding. As H-IL-6 stimulates STAT3-dependent gene transcription, we compared t…

Therapeutic gene modulationSTAT3 Transcription FactorTranscriptional ActivationTranscription GeneticRecombinant Fusion ProteinsResponse elementE-boxBiologyTransfectionBiochemistryCell LineMiceSp3 transcription factorAntigens CDCytokine Receptor gp130E2F1AnimalsHumansRNA MessengerPhosphorylationMolecular BiologyCell NucleusATF3Sp1 transcription factorMice Inbred C3HMembrane GlycoproteinsHaptoglobinsInterleukin-6Liver receptor homolog-1Biological TransportCell BiologyDNAReceptors InterleukinMolecular biologyReceptors Interleukin-6DNA-Binding ProteinsGene Expression RegulationTrans-ActivatorsTyrosineThe Journal of biological chemistry
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JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun

2003

The AP-1 transcription factor c-Jun is a prototypical nuclear effector of the JNK signal transduction pathway. The integrity of JNK phosphorylation sites at serines 63/73 and at threonines 91/93 in c-Jun is essential for signal-dependent target gene activation. We show that c-Jun phosphorylation mediates dissociation of an inhibitory complex, which is associated with histone deacetylase 3 (HDAC3). The subsequent events that ultimately cause increased mRNA synthesis are independent of c-Jun phosphorylation and its interaction with JNK. These findings provide an 'activation by de-repression' model as an explanation for the stimulatory function of JNK on c-Jun.

ThreonineTranscriptional ActivationTranscription GeneticMAP Kinase Kinase 4Proto-Oncogene Proteins c-junRecombinant Fusion ProteinsMitogen-activated protein kinase kinaseHistone DeacetylasesGeneral Biochemistry Genetics and Molecular BiologyCell LinePhosphorylation cascadeMiceSuppression GeneticGenes ReporterSerineAnimalsHumansRNA MessengerPhosphorylationMolecular BiologyTranscription factorSequence DeletionMitogen-Activated Protein Kinase KinasesGeneral Immunology and MicrobiologybiologyGeneral Neurosciencec-junJNK Mitogen-Activated Protein KinasesArticles3T3 CellsHDAC3Molecular biologyProtein Structure TertiaryMitogen-activated protein kinaseMutationMutagenesis Site-Directedbiology.proteinPhosphorylationSignal transductionProtein BindingThe EMBO Journal
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Dendritic cell aggresome-like-induced structure formation and delayed antigen presentation coincide in influenza virus-infected dendritic cells.

2005

Abstract Influenza virus infection induces maturation of murine dendritic cells (DCs), which is most important for the initiation of an immune response. However, in contrast to EL-4 and MC57 cells, DCs present viral CTL epitopes with a delay of up to 10 h. This delay in Ag presentation coincides with the up-regulation of MHC class I molecules as well as costimulatory molecules on the cell surface and the accumulation of newly synthesized ubiquitinated proteins in large cytosolic structures, called DC aggresome-like-induced structures (DALIS). These structures were observed previously after LPS-induced maturation of DCs, and it was speculated that they play a role in the regulation of MHC cl…

Time FactorsImmunologyAntigen presentationCellAntigen-Presenting CellsEpitopes T-Lymphocytechemical and pharmacologic phenomenaBone Marrow CellsVirusCell LineMiceImmune systemCell Line TumorMHC class ImedicineImmunology and AllergyAnimalsHumansReceptors ImmunologicCells CulturedAntigen PresentationMice Inbred C3HbiologyUbiquitinViral Core ProteinsRNA-Binding ProteinsCell DifferentiationDendritic cellDendritic CellsNucleocapsid ProteinsVirologyToll-Like Receptor 2Cell biologyNucleoproteinMice Inbred C57BLToll-Like Receptor 4Aggresomemedicine.anatomical_structureNucleoproteinsInfluenza A virusbiology.proteinCytoplasmic StructuresT-Lymphocytes CytotoxicJournal of immunology (Baltimore, Md. : 1950)
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French firms’ strategies for protecting their intellectual property

2012

In attempting to protect their innovations, firms can choose from a range of mechanisms, which may be either non-statutory (trade secrets, design complexity, and lead-time advantage over competitors) or statutory (patent, design registration, trademark, copyright). Yet, little is known about how firms do actually make their choices from among these different appropriability mechanisms. The aim of this paper is to determine how French firms’ use of intellectual property protection mechanisms relates to the type of innovation, the characteristics of the market sector in which they operate, the firms’ characteristics, and their human resources strategies. Our empirical model draws on four Fren…

Trademarkbusiness.industryStrategy and Managementjel:C35Competitor analysisManagement Science and Operations ResearchIntellectual propertyMarket sectorIntellectual property rights; Multivariate probit; Appropriability; Innovation; Human resources strategies;Statutory lawManagement of Technology and Innovationjel:O34Economicsjel:O32MarketingHuman resourcesbusinessIndustrial organization
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Interleukin-4 induces secretion of CSF for granulocytes and CSF for macrophages by peripheral blood monocytes.

1989

Abstract T cells are known to interact cooperatively with monocytes to produce Colony-Stimulating Factors (CSF), although T cell-mediated signals leading to CSF secretion by monocytes are not completely understood. We have made use of Northern blot hybridization and specific bioassays to study the effects of the T cell product interleukin-4 (IL-4) on monocyte CSF expression. The results suggest a previously unrecognized role of IL-4 as a CSF inducer since exposure of monocytes to IL-4 resulted in accumulation of transcripts for granulocyte-CSF (G-CSF) and macrophage-CSF (M-CSF). Consequently, IL-4-activated monocytes released factors in their culture supernatants biologically and antigenica…

Transcription GeneticT cellImmunologyBiologyBiochemistryMonocytesColony-Forming Units AssayMiceColony-Stimulating FactorsGranulocyte Colony-Stimulating FactormedicineBioassayAnimalsHumansInducerSecretionNorthern blotInterleukin 4Mice Inbred C3HMonocyteInterleukinsMacrophage Colony-Stimulating FactorMacrophagesCell BiologyHematologyMolecular biologyPeripheral bloodRecombinant Proteinsmedicine.anatomical_structureImmunologyInterleukin-4GranulocytesBlood
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Hyperventilation in Adult TBI Patients: How to Approach It?

2021

Hyperventilation is a commonly used therapy to treat intracranial hypertension (ICTH) in traumatic brain injury patients (TBI). Hyperventilation promotes hypocapnia, which causes vasoconstriction in the cerebral arterioles and thus reduces cerebral blood flow and, to a lesser extent, cerebral blood volume effectively, decreasing temporarily intracranial pressure. However, hyperventilation can have serious systemic and cerebral deleterious effects, such as ventilator-induced lung injury or cerebral ischemia. The routine use of this therapy is therefore not recommended. Conversely, in specific conditions, such as refractory ICHT and imminent brain herniation, it can be an effective life-savin…

Traumatic brain injurybusiness.industrytraumatic brain injuryIschemiahyperventilationReviewLung injurymedicine.diseaseBrain herniationcerebral ischemialcsh:RC346-429hypocapniaHypocapniaCerebral blood flowNeurologyAnesthesiaintracranial hypertensionHyperventilationmedicineNeurology (clinical)medicine.symptombusinesslcsh:Neurology. Diseases of the nervous systemIntracranial pressureFrontiers in neurology
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BDNF-induced nitric oxide signals in cultured rat hippocampal neurons: time course, mechanism of generation, and effect on neurotrophin secretion

2014

BDNF and nitric oxide signaling both contribute to plasticity at glutamatergic synapses. However, the role of combined signaling of both pathways at the same synapse is largely unknown. Using NO imaging with diaminofluoresceine in cultured hippocampal neurons we analyzed the time course of neurotrophin-induced NO signals. Application of exogenous BDNF, NT-4, and NT-3 (but not NGF) induced NO signals in the soma and in proximal dendrites of hippocampal neurons that were sensitive to NO synthase activity, TrkB signaling, and intracellular calcium elevation. The effect of NO signaling on neurotrophin secretion was analyzed in BDNF-GFP, and NT-3-GFP transfected hippocampal neurons. Exogenous ap…

Tropomyosin receptor kinase BBiologyHippocampal formationneurotrophinsNitric oxidelcsh:RC321-571SynapseCellular and Molecular Neurosciencechemistry.chemical_compoundnitric oxideOriginal Research Articlelcsh:Neurosciences. Biological psychiatry. Neuropsychiatrypeptide secretionsynaptic plasticityTrkBPSD95DepolarizationPeptide secretionBDNFchemistrynervous systemSynaptic plasticitybiology.proteinNeuroscienceNeurotrophinNeuroscienceFrontiers in Cellular Neuroscience
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European evidence-based recommendations for clinical assessment of upper limb in neurorehabilitation (CAULIN): data synthesis from systematic reviews…

2021

Abstract Background Technology-supported rehabilitation can help alleviate the increasing need for cost-effective rehabilitation of neurological conditions, but use in clinical practice remains limited. Agreement on a core set of reliable, valid and accessible outcome measures to assess rehabilitation outcomes is needed to generate strong evidence about effectiveness of rehabilitation approaches, including technologies. This paper collates and synthesizes a core set from multiple sources; combining existing evidence, clinical practice guidelines and expert consensus into European recommendations for Clinical Assessment of Upper Limb In Neurorehabilitation (CAULIN). Methods Data from systema…

UT-Gold-Dassessmentmedicine.medical_treatmentDelphi methodmultiple sclerosisOutcome measuresaivohalvausOutcome measureTraumatic brain injuryInternational Classification of Functioning Disability and HealthMS-tautiupper extremityMedicineUpper limblääkinnällinen kuntoutusRehabilitationtraumatic brain injuryRehabilitationStroke RehabilitationstrokeTest (assessment)StrokeSystematic reviewRC321-571medicine.medical_specialtyEvidence-based practiceConsensusUpper extremity616.8: Neurologie und Krankheiten des NervensystemsNeurosciences. Biological psychiatry. NeuropsychiatryHealth Informaticsupper limbkädetSpinal cord injuryAssessmenthermoston tauditkäsivarretaivovammatrehabilitationMultiple sclerosisoutcome measuresPhysical medicine and rehabilitationOutcome measures; Stroke; Traumatic brain injury; Spinal cord injury;HumansUpper limb; Upper extremity; Assessment; Rehabilitation; Therapy;Set (psychology)Neurorehabilitationselkäydinvammattherapybusiness.industryResearchReproducibility of ResultsRecovery of FunctionHandspinal cord injuryTherapybusiness
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