Search results for "CARBOHYDRATE METABOLISM"

showing 10 items of 188 documents

Ketogenic diet and microbiota: Friends or enemies?

2019

Over the last years, a growing body of evidence suggests that gut microbial communities play a fundamental role in many aspects of human health and diseases. The gut microbiota is a very dynamic entity influenced by environment and nutritional behaviors. Considering the influence of such a microbial community on human health and its multiple mechanisms of action as the production of bioactive compounds, pathogens protection, energy homeostasis, nutrients metabolism and regulation of immunity, establishing the influences of different nutritional approach is of pivotal importance. The very low carbohydrate ketogenic diet is a very popular dietary approach used for different aims: from weight …

0301 basic medicinelcsh:QH426-470medicine.medical_treatmentReviewGut microbiotaBiologyGut floraBioinformaticsEnergy homeostasis03 medical and health sciencesHuman health0302 clinical medicineWeight lossGeneticsmedicineAnimalsHumansLow carbohydrateGenetics (clinical)Gut microbiomeBiological Variation IndividualMicrobiotaKetogenic diet and fatKetosisKetogenic dietbiology.organism_classificationGut microbiomeIntestinal microbiomeGastrointestinal Microbiomelcsh:Genetics030104 developmental biologyIntestinal MicrobiomeCarbohydrate Metabolismgut microbiome; gut microbiota; intestinal microbiome; ketogenic diet; ketogenic diet and fatmedicine.symptomDiet Ketogenic030217 neurology & neurosurgeryKetogenic diet
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The Mitochondria-Targeted Antioxidant MitoQ Modulates Mitochondrial Function and Endoplasmic Reticulum Stress in Pancreatic β Cells Exposed to Hyperg…

2019

Background/aims Mitochondria-targeted antioxidants such as mitoquinone (MitoQ) have demonstrated protective effects against oxidative damage in several diseases. The increase in reactive oxygen species (ROS) production during glucose metabolism in β cells can be exacerbated under hyperglycaemic conditions such as type 2 diabetes (T2D), thus contributing to β cell function impairment. In the present work, we aimed to evaluate the effect of MitoQ on insulin secretion, oxidative stress, endoplasmic reticulum (ER) stress and nuclear factor kappa B (NFκB) signalling in a pancreatic β cell line under normoglycaemic (NG, 11.1 mM glucose), hyperglycaemic (HG, 25 mM glucose) and lipidic (palmitic ac…

0301 basic medicinemedicine.medical_specialtyPhysiologyUbiquinoneCarbohydrate metabolismmedicine.disease_causeMitoQlcsh:PhysiologyPancreatic β cellsAntioxidantsProinflammatory cytokinelcsh:Biochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOrganophosphorus CompoundsInternal medicineCell Line TumorInsulin-Secreting CellsmedicineAnimalslcsh:QD415-436chemistry.chemical_classificationReactive oxygen speciesMitoQlcsh:QP1-981Endoplasmic reticulumGlutathioneEndoplasmic Reticulum StressType 2 DiabetesMitochondriaRatsOxidative Stress030104 developmental biologyEndocrinologyGlucosechemistry030220 oncology & carcinogenesisHyperglycemiaUnfolded protein responseER stressMitochondrial dysfunctionReactive Oxygen SpeciesOxidative stressSignal Transduction
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Insulin resistance and diabetes in hyperthyroidism: a possible role for oxygen and nitrogen reactive species.

2019

In addition to insulin, glycemic control involves thyroid hormones. However, an excess of thyroid hormone can disturb the blood glucose equilibrium, leading to alterations of carbohydrate metabolism and, eventually, diabetes. Indeed, experimental and clinical hyperthyroidism is often accompanied by abnormal glucose tolerance. A common characteristic of hyperthyroidism and type 2 diabetes is the altered mitochondrial efficiency caused by the enhanced production of reactive oxygen and nitrogen species. It is known that an excess of thyroid hormone leads to increased oxidant production and mitochondrial oxidative damage. It can be hypothesised that these species represent the link between hype…

0301 basic medicinemedicine.medical_specialtyendocrine systemendocrine system diseasesmedicine.medical_treatmentDiabetes hyperthyroidism insulin resistance insulin secretion reactive nitrogen species (RNS) reactive oxygen species (ROS)Type 2 diabetesCarbohydrate metabolismBiochemistryHyperthyroidism03 medical and health sciencesInsulin resistanceDiabetes mellitusInternal medicinemedicineHumansGlycemic030102 biochemistry & molecular biologyChemistryInsulinThyroidGeneral Medicinemedicine.diseaseReactive Nitrogen SpeciesOxygen030104 developmental biologymedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 2Insulin ResistanceReactive Oxygen SpeciesHormoneFree radical research
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Srebf2 Locus Overexpression Reduces Body Weight, Total Cholesterol and Glucose Levels in Mice Fed with Two Different Diets

2020

Macronutrients represent risk factors for hyperlipidemia or diabetes. Lipid alterations and type 2 diabetes mellitus are global health problems. Overexpression of sterol regulatory element-binding factor (Srebf2) in transgenic animals is linked to elevated cholesterol levels and diabetes development. We investigated the impact of increased Srebf2 locus expression and the effects of control and high-fat, high-sucrose (HFHS) diets on body weight, glucose and lipid metabolisms in transgenic mice (S-mice). Wild type (WT) and S-mice were fed with both diets for 16 weeks. Plasma glucose, insulin and lipids were assessed (n = 25). Immunostainings were performed in liver, pancreas and fat (N = 10).…

0301 basic medicinemedicine.medical_specialtymedicine.medical_treatment030209 endocrinology & metabolismlcsh:TX341-641Carbohydrate metabolismtransgenic miceArticle03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInternal medicineAdipocyteDiabetes mellitusHyperlipidemialipid metabolismmedicinecarbohydrate metabolismhigh-sucrose diethigh-fatNutrition and DieteticsCholesterolInsulinType 2 Diabetes MellituscholesterolLipid metabolismmedicine.diseaselipoproteins030104 developmental biologyEndocrinologychemistrylipids (amino acids peptides and proteins)atherosclerosissterol regulatory element-binding protein 2 (SREBP-2)lcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Safflower Yellow and Its Main Component HSYA Alleviate Diet-Induced Obesity in Mice: Possible Involvement of the Increased Antioxidant Enzymes in Liv…

2020

PurposeOxidative stress plays an important role in the pathogenesis of obesity and its associated disorders. Safflower yellow (SY) and hydroxysafflor yellow A (HSYA), the natural compounds isolated from Carthamus tinctorius L., has been found to possess antioxidative and anti-obesity properties. The purpose of the present study is to investigate whether SY and its main component HSYA alleviate obesity by the antioxidant effects.MethodsDiet-induced obese (DIO) mice were treated with 200 mg/kg/d SY or HSYA for 10 weeks. Body weight, fat mass, serum biochemical parameters and superoxide dismutase (SOD) activities were measured. Glucose and insulin tolerance tests were performed. The expression…

0301 basic medicinemedicine.medical_specialtyobesityAntioxidantmedicine.medical_treatmentAdipose tissueCarbohydrate metabolismmedicine.disease_causeliverSuperoxide dismutase03 medical and health sciences0302 clinical medicineantioxidant enzymesInternal medicinemedicinePharmacology (medical)Original ResearchPharmacologysafflower yellow (SY)biologyChemistryCarthamuslcsh:RM1-950Metabolismbiology.organism_classificationadipose tissue030104 developmental biologyEndocrinologylcsh:Therapeutics. Pharmacology030220 oncology & carcinogenesishydroxysafflor yellow A (HSYA)biology.proteinLiver functionOxidative stressFrontiers in Pharmacology
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Activation of AMPK by medicinal plants and natural products: Its role in type 2 diabetes mellitus

2019

Type-2 Diabetes (T2D) is a metabolic disease characterized by permanent hyperglycemia, whose development can be prevented or delayed by using therapeutic agents and implementing lifestyle changes. Some therapeutic alternatives include regulation of glycemia through modulation of different mediators and enzymes, such as AMP-activated protein kinase (AMPK), a highly relevant cellular energy sensor for metabolic homeostasis regulation, with particular relevance in the modulation of liver and muscle insulin sensitivity. This makes it a potential therapeutic target for antidiabetic drugs. In fact, some of them are standard drugs used for treatment of T2D, such as biguanides and thiazoli-dindione…

AMPKCIENCIAS MÉDICAS Y DE LA SALUDBiguanidesAMP-Activated Protein KinasesPharmacologyResveratrolCarbohydrate metabolismNATURAL PRODUCTS03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBerberineDiabetes mellitusDrug DiscoveryHumansHypoglycemic AgentsMedicineTYPE 2 DIABETES MELLITUSProtein kinase A030304 developmental biologyPharmacologychemistry.chemical_classificationBiological Products0303 health sciencesPlants Medicinalbusiness.industryType 2 Diabetes MellitusAMPKGeneral MedicineBioquímica y Biología Molecularmedicine.diseaseANTIDIABETIC DRUGSMedicina BásicaGlucoseEnzymeDiabetes Mellitus Type 2chemistry030220 oncology & carcinogenesisThiazolidinedionesbusinessMEDICINAL PLANTS
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Flying insects: model systems in exercise physiology

1996

Insect flight is the most energy-demanding exercise known. It requires very effective coupling of adenosine triphosphate (ATP) hydrolysis and regeneration in the working flight muscles.31P nuclear magnetic resonance (NMR) spectroscopy of locust flight muscle in vivo has shown that flight causes only a small decrease in the content of ATP, whereas the free concentrations of inorganic phosphate (P i ), adenosine diphosphate (ADP) and adenosine monophosphate (AMP) were estimated to increase by about 3-, 5- and 27-fold, respectively. These metabolites are potent activators of glycogen phosphorylase and phosphofructokinase (PFK). Activation of glycolysis by AMP and P i is reinforced synergistica…

Adenosine monophosphateInsectaMagnetic Resonance SpectroscopyPhysical ExertionGrasshoppersCarbohydrate metabolismBiologyModels BiologicalPhosphatesCellular and Molecular Neurosciencechemistry.chemical_compoundGlycogen phosphorylaseAnimalsGlycolysisMolecular BiologyPharmacologyAdenine NucleotidesCell BiologyAdenosine diphosphateFructose 26-bisphosphatechemistryBiochemistryFlight AnimalMolecular MedicineEnergy MetabolismGlycolysisAdenosine triphosphateMuscle ContractionPhosphofructokinaseExperientia
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A computational model of postprandial adipose tissue lipid metabolism derived using human arteriovenous stable isotope tracer data

2019

Given the association of disturbances in non-esterified fatty acid (NEFA) metabolism with the development of Type 2 Diabetes and Non-Alcoholic Fatty Liver Disease, computational models of glucose-insulin dynamics have been extended to account for the interplay with NEFA. In this study, we use arteriovenous measurement across the subcutaneous adipose tissue during a mixed meal challenge test to evaluate the performance and underlying assumptions of three existing models of adipose tissue metabolism and construct a new, refined model of adipose tissue metabolism. Our model introduces new terms, explicitly accounting for the conversion of glucose to glyceraldehye-3-phosphate, the postprandial …

Adipose Tissue/metabolismDIETARY FATTY-ACIDSmedicine.medical_treatmentFatty Acids NonesterifiedBiochemistry0302 clinical medicineEndocrinologyModelsInsulinGlucose/metabolismBiology (General)Organic CompoundsFatty AcidsChemical ReactionsPostprandial Period/physiologyPostprandial PeriodLipidsPostprandialBloodComputational Theory and MathematicsAdipose TissueModeling and SimulationPhysical Sciencesmedicine.medical_specialtyQH301-705.5LipolysisCarbohydratesLIPOPROTEIN-LIPASECarbohydrate metabolism03 medical and health sciencesNEFASDG 3 - Good Health and Well-beingGeneticsLipolysisHumansComputer SimulationMolecular BiologyEcology Evolution Behavior and SystematicsBlood Glucose/metabolismArteriovenous AnastomosisChemical CompoundsBiology and Life SciencesComputational BiologyComputational Biology/methodsmedicine.diseaseLipid MetabolismBiologicalHormonesLipid Metabolism/physiology030104 developmental biologyEndocrinologyBiological TissueGlucoseMOBILIZATION030217 neurology & neurosurgery0301 basic medicineGlycerolBlood GlucosePhysiologyPATHOGENESISAdipose tissueLipids/physiologySDG 3 – Goede gezondheid en welzijnVoeding Metabolisme en GenomicaGlucose MetabolismIsotopesMedicine and Health SciencesMetabolitesINSULIN-RESISTANCEEcologyChemistryHydrolysisMonomersMonosaccharidesArteriovenous Anastomosis/metabolismMetabolism and GenomicsBody FluidsChemistryFatty Acids/metabolismMetabolisme en GenomicaCarbohydrate MetabolismNutrition Metabolism and GenomicsFatty Acids Nonesterified/metabolismAnatomyResearch ArticleInsulin/metabolismINHIBITIONWEIGHT-LOSSModels BiologicalBlood PlasmaMECHANISMSCellular and Molecular NeuroscienceInsulin resistanceVoedingInternal medicinemedicineLife ScienceNonesterified/metabolismNutritionDiabetic EndocrinologyInsulinOrganic ChemistryLipid metabolismECTOPIC FATPolymer ChemistryMetabolism
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Serum osteocalcin is not associated with glucose but is inversely associated with leptin across generations of nondiabetic women.

2012

The skeleton is recognized as an important player in energy metabolism through its interactions with other tissues. Whether the association of osteocalcin with glucose metabolism is age dependent has not been fully addressed.The objective of the study was to examine the age-specific association between different forms of osteocalcin and glucose and adipokines.This was a family-based study across three generations.The study was conducted at a university laboratory.Sixty-four daughter-premenopausal mother-maternal grandmother trios participated in the study.Fasting plasma glucose and insulin concentrations, serum total (tOC), carboxylated (cOC), and uncarboxylated (ucOC = tOC - cOC) osteocalc…

AdultBlood GlucoseLeptinmedicine.medical_specialtyAdolescentEndocrinology Diabetes and Metabolismmedicine.medical_treatmentClinical BiochemistryOsteocalcinAdipokineContext (language use)Carbohydrate metabolismBiochemistryBone and BonesEndocrinologyInsulin resistanceInternal medicinemedicineHumansInsulinAgedbiologyAdiponectinLeptinInsulinBiochemistry (medical)Age FactorsMiddle Agedmedicine.diseaseEndocrinologyPremenopauseOsteocalcinbiology.proteinFemaleAdiponectinInsulin ResistanceThe Journal of clinical endocrinology and metabolism
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Erratum: Effect of magnesium supplementation on glucose metabolism in people with or at risk of diabetes: a systematic review and meta-analysis of do…

2016

Although higher dietary intakes of magnesium (Mg) seem to correspond to lower diabetes incidence, research concerning Mg supplementation in people with or at risk of diabetes is limited. Thus, we aimed to investigate the effect of oral Mg supplementation on glucose and insulin-sensitivity parameters in participants with diabetes or at high risk of diabetes compared with placebo. A literature search in PubMed, EMBASE, SCOPUS, Cochrane Central Register of Controlled Trials and Clinicaltrials.gov without language restriction, was undertaken. Eligible studies were randomized controlled trials (RCTs) investigating the effect of oral Mg supplementation vs placebo in patients with diabetes or at h…

AdultBlood GlucoseMale0301 basic medicinemedicine.medical_specialtyCalorieMagnesium supplementationMedicine (miscellaneous)030209 endocrinology & metabolismCarbohydrate metabolismlaw.inventionDouble blind03 medical and health sciences0302 clinical medicineDouble-Blind MethodRandomized controlled triallawInternal medicineDiabetes mellitusDiabetes MellitusmedicineHumansMagnesiumAgedRandomized Controlled Trials as TopicGlucose tolerance test030109 nutrition & dieteticsNutrition and Dieteticsmedicine.diagnostic_testbusiness.industryMedicine (all)Medicine (miscellaneous); Medicine (all); Nutrition and DieteticsFastingGlucose Tolerance TestMiddle Agedmedicine.diseaseObesityEndocrinologyMeta-analysisDietary Supplementsmagnesium Nutrition diabetes Dietetics glucose metabolismFemalebusinessBody mass indexEuropean Journal of Clinical Nutrition
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