Search results for "CARDIOTOXICITY"

showing 10 items of 104 documents

Use of myocardial work for multiparametric detection of subclinical anthracycline cardiotoxicity in breast cancer patients

2021

Abstract Funding Acknowledgements Type of funding sources: None. Background The aim of our study was to assess subclinical cardiac effects of anthracyclines (ANTs) in women treated for breast cancer (BC). Methods We enrolled 46 female patients with BC undergoing adjuvant treatment with anthracycline-containing chemotherapy (CT) followed by taxane (paclitaxel/docetaxel).  Patients underwent physical examination, electrocardiogram (ECG) and standard transthoracic echocardiography (TTE) including evaluation of diastolic and systolic function, measured as left ventricular ejection fraction (LVEF), left ventricular global longitudinal strain (GLS) and myocardial work (MW) expressed as global wor…

Oncologymedicine.medical_specialtyCardiotoxicityEjection fractionAnthracyclinebusiness.industryGeneral Medicinemedicine.diseaseChemotherapy regimenBreast cancerDocetaxelInternal medicinemedicineRadiology Nuclear Medicine and imagingCardiology and Cardiovascular MedicineAdverse effectbusinessSubclinical infectionmedicine.drugEuropean Heart Journal - Cardiovascular Imaging
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PKP-020 Impact of nadph oxidase functional polymorphisms in acute myeloid leukaemia induction chemotherapy

2016

Background NADPH oxidase, a key mediator of oxidative cardiac damage and remodelling, modulates anthracycline clinical cardiotoxicity. Purpose Single nucleotide polymorphisms (SNPs) of NADPH oxidase genes could lead to interindividual differences in treatment outcome in acute myeloid leukaemia (AML) patients. Material and methods The main three NADPH oxidase polymorphisms (CYBA:rs4673, NCF4:rs1883112 and RAC2:rs13058338) were evaluated in 225 adult patients at the initial diagnosis of AML using a mass spectrometry based multiplex genotyping assay (Sequenom). All patients received induction chemotherapy consisting of idarubicin plus cytarabine (PETHEMA 99, 2007 and 2010 trials). The efficacy…

Oncologymedicine.medical_specialtyCardiotoxicityNADPH oxidasebiologyAnthracyclinebusiness.industryInduction chemotherapySingle-nucleotide polymorphismInternal medicineGenotypeImmunologymedicinebiology.proteinCytarabineIdarubicinGeneral Pharmacology Toxicology and Pharmaceuticsbusinessmedicine.drugEuropean Journal of Hospital Pharmacy
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Early Detection of Cardiac Damage

2018

Early detection and quantification of cardiac damage in cancer patients are essential to readily intervene with cardioprotective strategies and avoid the need of the discontinuation of antineoplastic treatment.

Oncologymedicine.medical_specialtyCardiotoxicitybusiness.industryInternal medicinemedicineEarly detectionCancerbusinessmedicine.diseaseDiscontinuation
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Cardiovascular Damage Induced by Anti-BCR-ABL TKIs

2018

Anti-BCR-ABL TKIs (tyrosine kinase inhibitors) are drugs that inhibit BCR ABL tyrosine. They are used especially in the treatment of hematological cancer and gastrointestinal stromal tumors (GIST). Anti-BCR-ABL TKIs include first (imatinib), second (nilotinib, dasatinib, bosutinib) and third-generation drugs (ponatinib). Especially second- and third-generation drugs can cause cardiovascular complications such as arterial thrombosis, myocardial ischemia, peripheral arterial diseases, QTc prolongation, and pulmonary hypertension. Nilotinib and ponatinib can cause thrombotic arterial events with various mechanisms. Particularly dasatinib can cause pulmonary hypertension. Compared to convention…

Oncologymedicine.medical_specialtyCardiotoxicitybusiness.industryPonatinibImatinibDasatinibchemistry.chemical_compoundchemistryNilotinibhemic and lymphatic diseasesInternal medicinemedicineAdverse effectbusinessBosutinibTyrosine kinasemedicine.drug
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Redox imbalances in ageing and metabolic alterations: Implications in cancer and cardiac diseases. An overview from the working group of cardiotoxici…

2020

Metabolic syndrome (MetS) is a well established risk factor for cardiovascular (CV) diseases. In addition, several studies indicate that MetS correlates with the increased risk of cancer in adults. The mechanisms linking MetS and cancer are not fully understood. Several risk factors involved in MetS are also cancer risk factors, such as the consumption of high calorie-food or high fat intake, low fibre intake, and sedentary lifestyle. Other common aspects of both cancer and MetS are oxidative stress and inflammation. In addition, some anticancer treatments can induce cardiotoxicity, including, for instance, left ventricular (LV) dysfunction and heart failure (HF), endothelial dysfunction an…

Oncologymedicine.medical_specialtyPhysiologyClinical BiochemistryReview030204 cardiovascular system & hematologymedicine.disease_causeBiochemistry03 medical and health sciences0302 clinical medicineInternal medicineAgeing; Cancer; Cardiovascular disease; Cardiovascular toxicity from anticancer drugs; Metabolic syndromemedicineEndothelial dysfunctionRisk factorMolecular BiologySedentary lifestyleCancerCardiotoxicitybusiness.industrylcsh:RM1-950CancerCell Biologymedicine.diseaseCardiovascular diseaseMetabolic syndromeAgeingCardiovascular toxicity from anticancer drugslcsh:Therapeutics. PharmacologyCardiovascular toxicity from anticancer drug030220 oncology & carcinogenesisHeart failureMetabolic syndromebusinessOxidative stress
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Early diagnosis, clinical management, and follow-up of cardiovascular events with ponatinib

2020

Chronic myeloid leukemia (CML) is a myeloproliferative disorder characterized by neoplastic transformation of pluripotent cells due to a typical cytogenetic and molecular mutation known as Philadelphia (Ph) chromosome. In 2001, the introduction of the tyrosine kinasis inhibitor (TKI) imatinib as a therapeutic strategy for CML with PH chromosome mutation represented an important step towards treatment of these patients, and nowadays, this drug represents the gold therapeutic standard in this clinical setting. A second generation of TKIs (dasatinib, nilotinib, and bosutinib) showed an effective action in all patients with mutations resistant to imatinib. Ponatinib is a third-generation TKI an…

Oncologymedicine.medical_specialtyTyrosine kinase inhibitorReview030204 cardiovascular system & hematology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsLeukemia Myelogenous Chronic BCR-ABL Positivehemic and lymphatic diseasesInternal medicinemedicineHumansNeoplastic transformation030212 general & internal medicineProtein Kinase InhibitorsTyrosine kinase inhibitorsCardiotoxicitybusiness.industryPonatinibChronic myeloid leukemiaImidazolesDisease ManagementMyeloid leukemiaImatinibPyridazinesDasatinibCardio-oncologyEarly DiagnosisNilotinibchemistryCardiovascular DiseasesPonatinibPonatinib . Tyrosine kinase inhibitors . Chronic myeloid leukemia . Cardio-oncology . ReviewCardiology and Cardiovascular MedicinebusinessBosutinibFollow-Up Studiesmedicine.drug
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Polycyclic Aromatic Hydrocarbons Phenanthrene and Retene Modify the Action Potential via Multiple Ion Currents in Rainbow Trout Oncorhynchus mykiss C…

2019

Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous contaminants in aqueous environments. They affect cardiovascular development and function in fishes. The 3-ring PAH phenanthrene has recently been shown to impair cardiac excitation-contraction coupling by inhibiting Ca2+ and K+ currents in marine warm-water scombrid fishes. To see if similar events take place in a boreal freshwater fish, we studied whether the PAHs phenanthrene and retene (an alkylated phenanthrene) modify the action potential (AP) via effects on Na+ (INa ), Ca2+ (ICaL ), or K+ (IKr , IK1 ) currents in the ventricular myocytes of the rainbow trout (Oncorhynchus mykiss) heart. Electrophysiological characteristics of myo…

PAH-yhdisteetHealth Toxicology and MutagenesiscardiotoxicityAction Potentialstoksikologia010501 environmental sciences01 natural sciences03 medical and health scienceschemistry.chemical_compoundmode of actionEnvironmental ChemistryMyocyteAnimalsaquatic toxicologyMyocytes CardiacPatch clampPolycyclic Aromatic HydrocarbonsMode of actionIon channelkalat030304 developmental biology0105 earth and related environmental sciencesvesistöt0303 health sciencesRetenebiologyPhenanthrenePhenanthrenesAryl hydrocarbon receptorhiilivedytchemistryReceptors Aryl Hydrocarbonpolycyclic aromatic hydrocarbons (PAHs)Oncorhynchus mykissbiology.proteinBiophysicsRainbow troutWater Pollutants ChemicalEnvironmental toxicology and chemistry
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Late Cardiac Events after Childhood Cancer: Methodological Aspects of the Pan-European Study PanCareSurFup.

2016

Background and AimChildhood cancer survivors are at high risk of long-term adverse effects of cancer and its treatment, including cardiac events. The pan-European PanCareSurFup study determined the incidence and risk factors for cardiac events among childhood cancer survivors. The aim of this article is to describe the methodology of the cardiac cohort and nested case-control study within PanCareSurFup.MethodsEight data providers in Europe participating in PanCareSurFup identified and validated symptomatic cardiac events in their cohorts of childhood cancer survivors. Data on symptomatic heart failure, ischemia, pericarditis, valvular disease and arrhythmia were collected and graded accordi…

PediatricsCritical Care and Emergency MedicineCancer Treatmentlcsh:MedicineADULT SURVIVORSCHILDREN030204 cardiovascular system & hematologyDOXORUBICIN THERAPYPediatricsCohort Studies0302 clinical medicineRisk FactorsNeoplasmsMedicine and Health SciencesSurvivorsYoung adultChildlcsh:Scienceeducation.field_of_studyMultidisciplinaryGUIDELINE HARMONIZATION GROUPCancer Risk Factors3. Good healthEuropeOncologyHelminth Infections030220 oncology & carcinogenesisChild PreschoolCohortHEALTH OUTCOMESRecord linkageCohort studyResearch ArticleNeglected Tropical DiseasesAdultCARDIOTOXICITYmedicine.medical_specialtyAdolescentHeart DiseasesLONG-TERMPopulationCardiology610 Medicine & health03 medical and health sciencesYoung Adult360 Social problems & social servicesDiagnostic MedicineEchinococcosismedicineCancer Detection and DiagnosisParasitic DiseasesHumanseducationAdverse effectCONGESTIVE-HEART-FAILUREbusiness.industryMORTALITYlcsh:RCase-control studyCancerInfantmedicine.diseaseTropical DiseasesPediatric OncologyCase-Control StudiesRISK-FACTORSlcsh:QbusinessPLoS ONE
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Isolated myocardiocytes and DNA synthesis: a possible experimental model to predict the in vivo cardiotoxicity

1989

PharmacologyCardiotoxicityHeart DiseasesDNA synthesisBiological modelingbusiness.industryExperimental modelMyocardiumDNAPharmacologyModels BiologicalRatsMiceDoxorubicinIn vivoToxicityAnimalsMedicineMitoxantronebusinessPharmacological Research
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Dissimilar effects of doxorubicin and isoproterenol on morphology, H2O2 content and catalase activity in mouse heart

1988

PharmacologyCardiotoxicityMorphology (linguistics)biologyCatalaseChemistrybiology.proteinmedicineDoxorubicinPharmacologyMouse Heartmedicine.drugPharmacological Research Communications
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