Search results for "CB(1)"

showing 10 items of 146 documents

Assessment of the abuse potential of MDMA in the conditioned place preference paradigm: Role of CB1 receptors

2013

Numerous reports have highlighted the role of the endocannabinoid system in the addictive potential of MDMA (3,4-methylenedioxy-methamphetamine). A previous report showed that CB1 knockout (KOCB1) mice do not acquire MDMA self-administration, despite developing conditioned place preference (CPP). This contradiction could be due to the particular procedure of place conditioning used. The present work compares MDMA-induced CPP in KOCB1 mice using unbiased and biased procedures of place conditioning. In the unbiased procedure, MDMA induced CPP and reinstatement of the extinguished preference in wild type (WT) mice, but not in KOCB1 mice. In contrast, in a biased protocol of CPP, MDMA produced …

MaleElevated plus mazeTime FactorsSubstance-Related Disordersmedicine.drug_classDopamineN-Methyl-34-methylenedioxyamphetamineNucleus accumbensPharmacologyAnxiolyticDevelopmental psychologyMiceNeurochemicalReceptor Cannabinoid CB1mental disordersmedicineAnimalsMaze LearningBiological PsychiatryMice KnockoutPharmacologyAnalysis of VarianceDose-Response Relationship DrugBrainHomovanillic AcidMDMAConditioned place preferenceDisease Models AnimalMonoamine neurotransmitternervous systemHallucinogens34-Dihydroxyphenylacetic AcidConditioning OperantSerotoninPsychologyReinforcement Psychologypsychological phenomena and processesmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Peripheral and central CB1 cannabinoid receptors control stress-induced impairment of memory consolidation

2016

Stressful events can generate emotional memories linked to the traumatic incident, but they also can impair the formation of nonemotional memories. Although the impact of stress on emotional memories is well studied, much less is known about the influence of the emotional state on the formation of nonemotional memories. We used the novel object-recognition task as a model of nonemotional memory in mice to investigate the underlying mechanism of the deleterious effect of stress on memory consolidation. Systemic, hippocampal, and peripheral blockade of cannabinoid type-1 (CB1) receptors abolished the stress-induced memory impairment. Genetic deletion and rescue of CB1 receptors in specific ce…

MaleEstrès0301 basic medicineIndolesCannabinoid receptormedicine.medical_treatmentPopulationDopamine beta-HydroxylaseHippocampal formation03 medical and health sciences0302 clinical medicinePiperidinesReceptor Cannabinoid CB1Cannabinoides -- ReceptorsmedicineAnimalsMemory impairmentReceptoreducationMemory ConsolidationMice KnockoutNeuronsElectroshockMemory Disorderseducation.field_of_studyMultidisciplinaryBiological SciencesEndocannabinoid system3. Good health030104 developmental biologyHindlimb SuspensionPyrazolesMemory consolidationCannabinoidRimonabantPsychologyNeuroscienceAnisomycinStress Psychological030217 neurology & neurosurgeryMemòriaProceedings of the National Academy of Sciences
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Loss of striatal type 1 cannabinoid receptors is a key pathogenic factor in Huntington's disease.

2010

Endocannabinoids act as neuromodulatory and neuroprotective cues by engaging type 1 cannabinoid receptors. These receptors are highly abundant in the basal ganglia and play a pivotal role in the control of motor behaviour. An early downregulation of type 1 cannabinoid receptors has been documented in the basal ganglia of patients with Huntington's disease and animal models. However, the pathophysiological impact of this loss of receptors in Huntington's disease is as yet unknown. Here, we generated a double-mutant mouse model that expresses human mutant huntingtin exon 1 in a type 1 cannabinoid receptor-null background, and found that receptor deletion aggravates the symptoms, neuropatholog…

MaleHuntingtinCannabinoid receptorCell Survivalmedicine.medical_treatmentBlotting WesternMice TransgenicBiologyMotor ActivityGrowth Hormone-Releasing HormoneMiceReceptor Cannabinoid CB1medicineCannabinoid receptor type 2AnimalsDronabinolReceptorBrain-derived neurotrophic factorNeuronsAnalysis of VarianceReverse Transcriptase Polymerase Chain ReactionEndocannabinoid systemMagnetic Resonance ImagingCorpus StriatumHuntington DiseaseRotarod Performance TestGPR18Neurology (clinical)CannabinoidNeuroscienceBrain : a journal of neurology
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Understanding Cannabinoid Psychoactivity with Mouse Genetic Models

2007

Marijuana and its main psychotropic ingredient Δ9-tetrahydrocannabinol (THC) exert a plethora of psychoactive effects through the activation of the neuronal cannabinoid receptor type 1 (CB1), which is expressed by different neuronal subpopulations in the central nervous system. The exact neuroanatomical substrates underlying each effect of THC are, however, not known. We tested locomotor, hypothermic, analgesic, and cataleptic effects of THC in conditional knockout mouse lines, which lack the expression of CB1 in different neuronal subpopulations, including principal brain neurons, GABAergic neurons (those that release γ aminobutyric acid), cortical glutamatergic neurons, and neurons expres…

MaleMESH: Body TemperatureCannabinoid receptormedicine.medical_treatmentGene ExpressionMESH: Receptor Cannabinoid CB1NeocortexMESH: gamma-Aminobutyric AcidMESH: CatalepsyPharmacologyHippocampusMESH: Mice KnockoutMESH: Corpus StriatumBody TemperatureMESH: Autonomic Nervous SystemMESH: NeocortexMice0302 clinical medicineReceptor Cannabinoid CB1MESH: Behavior AnimalCannabinoid receptor type 1MESH: AnimalsMESH: Gene SilencingDronabinolMESH: NociceptorsBiology (General)gamma-Aminobutyric AcidMice Knockout0303 health sciencesBehavior Animalmusculoskeletal neural and ocular physiologyGeneral NeuroscienceMESH: Pain ThresholdNociceptorsMESH: Glutamic AcidMESH: InterneuronsMESH: Motor Activity3. Good healthGABAergicMESH: TetrahydrocannabinolGeneral Agricultural and Biological SciencesResearch Articlemedicine.drugPain ThresholdMESH: Gene ExpressionMESH: Psychotropic DrugsQH301-705.5Glutamic AcidMotor ActivityBiologyAutonomic Nervous SystemGeneral Biochemistry Genetics and Molecular Biologygamma-Aminobutyric acid03 medical and health sciencesGlutamatergicDopamine receptor D1InterneuronsCannabinoid Receptor Modulatorsmental disorders[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsGenetic Predisposition to Disease[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyGene SilencingTetrahydrocannabinolMESH: MiceAnesthesiology and Pain Management030304 developmental biologyPharmacologyCatalepsyPsychotropic DrugsModels GeneticGeneral Immunology and MicrobiologyCannabinoidsIllicit Drugsorganic chemicalsMESH: MaleCorpus StriatumPrimerDisease Models Animalnervous systemCannabinoidNervous System Diseases030217 neurology & neurosurgeryNeurosciencePLoS Biology
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Prenatal exposure to the CB1 receptor agonist WIN 55,212-2 causes learning disruption associated with impaired cortical NMDA receptor function and em…

2005

The aim of this study was to investigate whether prenatal exposure to the cannabinoid CB1 receptor agonist WIN 55,212-2 (WIN) at a daily dose devoid of overt signs of toxicity and/or gross malformations (0.5 mg/kg, gestation days 5-20), influences cortical glutamatergic neurotransmission, learning and emotional reactivity in rat offspring. Basal and K+-evoked extracellular glutamate levels were significantly lower in cortical cell cultures obtained from pups exposed to WIN during gestation with respect to those measured in cultures obtained from neonates born from vehicle-treated dams. The addition of NMDA to cortical cell cultures from neonates born from vehicle-treated dams concentration-…

MaleMarijuana AbuseCannabinoid receptoractive avoidance behaviour; basal and K+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationEmotionsReceptor Cannabinoid CB1Pregnancyactive avoidance behaviourWIN 55212-2Cells CulturedCerebral CortexBehavior AnimalGlutamate receptorBraincortical cell culturesCalcium Channel Blockersactive avoidance behaviour; basal and k plus -evoked glutamate levels; basal and k+-evoked glutamate levels; cortical cell cultures; homing behaviour; maternal marijuana consumption; ultrasonic vocalizationPrenatal Exposure Delayed EffectsChloratesNMDA receptorbasal and K+-evoked glutamate levelsFemaleMicrotubule-Associated Proteinsmedicine.drugAgonistmedicine.medical_specialtyOffspringmedicine.drug_classCognitive NeuroscienceMorpholinesGlutamic Acidmaternal marijuana consumptionNeurotransmissionBiologyNaphthalenesReceptors N-Methyl-D-AspartateCellular and Molecular NeuroscienceGlutamatergicInternal medicinemedicineAvoidance LearningAnimalsRats WistarBenzoxazinesRatsultrasonic vocalizationEndocrinologyAnimals Newbornhoming behaviourVocalization AnimalExtracellular SpaceNeuroscience
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The use of the Emotional-Object Recognition as an assay to assess learning and memory associated to an aversive stimulus in rodents

2016

Abstract Background Emotionally salient experiences induce the formation of explicit memory traces, besides eliciting automatic or implicit emotional memory in rodents. This study aims at investigating the implementation of a novel task for studying the formation of limbic memory engrams as a result of the acquisition- and retrieval- of fear-conditioning – biased declarative memory traces, measured by animal discrimination of an “emotional-object”. Moreover, by using this new method we investigated the potential interactions between stimulation of cannabinoid transmission and integration of emotional information and cognitive functioning. New method The Emotional-Object Recognition task is …

MaleNociceptionLimbic memory engramCB1 receptorMorpholinesConditioning ClassicalExplicit emotional memoryNaphthalenesSpatial memory03 medical and health sciences0302 clinical medicineExplicit memoryAvoidance LearningSemantic memoryAnimalsVisual short-term memoryRats WistarMaze LearningGeneralLiterature_REFERENCE(e.g.dictionariesencyclopediasglossaries)Episodic memoryMethods used to study memoryAnalgesicsAnalysis of VarianceNeuroscience (all)Long-term memoryGeneral NeuroscienceRecognition PsychologyFearElectric Stimulation030227 psychiatryBenzoxazinesRatsObject discriminationExploratory BehaviorMemory consolidationCuesPsychology030217 neurology & neurosurgeryLocomotionCognitive psychology
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The endocannabinoid system controls food intake via olfactory processes

2014

Comment in Sensory systems: the hungry sense. [Nat Rev Neurosci. 2014] Inhaling: endocannabinoids and food intake. [Nat Neurosci. 2014]; International audience; Hunger arouses sensory perception, eventually leading to an increase in food intake, but the underlying mechanisms remain poorly understood. We found that cannabinoid type-1 (CB1) receptors promote food intake in fasted mice by increasing odor detection. CB1 receptors were abundantly expressed on axon terminals of centrifugal cortical glutamatergic neurons that project to inhibitory granule cells of the main olfactory bulb (MOB). Local pharmacological and genetic manipulations revealed that endocannabinoids and exogenous cannabinoid…

MaleOlfactory systemMESH: Olfactory PerceptionCannabinoid receptorMESH: Feedback Physiological[SDV]Life Sciences [q-bio]medicine.medical_treatmentMESH: Cannabinoid Receptor AgonistsMESH: EndocannabinoidsMESH: Receptor Cannabinoid CB1Synaptic TransmissionMESH: Mice KnockoutMESH: EatingEatingMiceOlfactory bulbReceptor Cannabinoid CB1MESH: AnimalsFeedback PhysiologicalMice Knockoutmusculoskeletal neural and ocular physiologyGeneral Neurosciencedigestive oral and skin physiologyOlfactory PathwaysEndocannabinoid systemMESH: Feeding Behaviorlipids (amino acids peptides and proteins)psychological phenomena and processesMESH: Olfactory BulbBiologyInhibitory postsynaptic potentialGlutamatergicMESH: Mice Inbred C57BLMESH: Synaptic TransmissionmedicineAnimalsMESH: MiceCannabinoid Receptor AgonistsFeeding BehaviorOlfactory PerceptionMESH: MaleOlfactory bulbMice Inbred C57BLnervous systemOdorFeeding behaviourCannabinoid[SDV.AEN]Life Sciences [q-bio]/Food and NutritionNeuroscienceMESH: Olfactory PathwaysEndocannabinoidsNature Neuroscience
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A high cannabinoid CB1 receptor immunoreactivity is associated with disease severity and outcome in prostate cancer

2008

In the light of findings indicating that cannabinoids can affect the proliferation of a number of cancer cell types and that cannabinoid receptor expression is higher in prostate cancer cell lines than in non-malignant cells, we investigated whether the level of cannabinoid 1 receptor immunoreactivity (CB(1)IR) in prostate cancer tissues is associated with disease severity and outcome. Formalin-fixed paraffin-embedded non-malignant and tumour tissue samples from patients who were diagnosed with prostate cancer at a transurethral resection for voiding problems were used. CB(1)IR, which was scored in a total of 399 cases, was associated with the epithelial cell membranes, with little staining…

MaleOncologyCancer Researchmedicine.medical_specialtyPathologyPrognostic variableCannabinoid receptormedicine.medical_treatmentAdenocarcinomaProstate cancerReceptor Cannabinoid CB1Internal medicineBiomarkers TumormedicineHumansSurvival rateAgedCell ProliferationProportional Hazards Modelsbusiness.industryProportional hazards modelCell MembraneProstatic NeoplasmsEpithelial Cellsmedicine.diseaseImmunohistochemistrySurvival RateOncologyArea Under CurveCancer cellImmunohistochemistryCannabinoidbusinessEuropean Journal of Cancer
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The Cannabinoid Receptor CB1 Interacts with the WAVE1 Complex and Plays a Role in Actin Dynamics and Structural Plasticity in Neurons.

2015

The molecular composition of the cannabinoid type 1 (CB1) receptor complex beyond the classical G-protein signaling components is not known. Using proteomics on mouse cortex in vivo, we pulled down proteins interacting with CB1 in neurons and show that the CB1 receptor assembles with multiple members of the WAVE1 complex and the RhoGTPase Rac1 and modulates their activity. Activation levels of CB1 receptor directly impacted on actin polymerization and stability via WAVE1 in growth cones of developing neurons, leading to their collapse, as well as in synaptic spines of mature neurons, leading to their retraction. In adult mice, CB1 receptor agonists attenuated activity-dependent remodeling o…

MaleReceptor complexCannabinoid receptorDendritic spineQH301-705.5medicine.medical_treatmentDendritic SpinesNeurogenesisRecombinant Fusion ProteinsGrowth ConesWiskott-Aldrich Syndrome Protein NeuronalNerve Tissue ProteinsBiologyCannabinoidergicGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesActin remodeling of neurons0302 clinical medicineReceptor Cannabinoid CB1Parietal LobeChlorocebus aethiopsmedicineAnimalsBiology (General)Cells Cultured030304 developmental biologyMice KnockoutNeurons0303 health sciencesNeuronal PlasticityGeneral Immunology and MicrobiologyCannabinoidsGeneral NeuroscienceNeurogenesisActin cytoskeletonEmbryo MammalianCell biologyFrontal LobeMice Inbred C57BLActin CytoskeletonLuminescent Proteinsnervous systemCOS Cellslipids (amino acids peptides and proteins)CannabinoidGeneral Agricultural and Biological Sciences030217 neurology & neurosurgeryResearch ArticlePLoS Biology
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Serotonin, the Prefrontal Cortex, and the Antidepressant-Like Effect of Cannabinoids

2007

Preclinical and clinical studies show that cannabis modulates mood and possesses antidepressant-like properties, mediated by the agonistic activity of cannabinoids on central CB1 receptors (CB1Rs). The action of CB1R agonists on the serotonin (5-HT) system, the major transmitter system involved in mood control and implicated in the mechanism of action of antidepressants, remains however poorly understood. In this study, we demonstrated that, at low doses, the CB1R agonist WIN55,212-2 [R(+)-[2,3-dihydro-5-methyl-3-[(morpholinyl)]pyrrolo[1,2,3-de]-1,4-benzoxazinyl]-(1-naphthalenyl) methanone mesylate] exerts potent antidepressant-like properties in the rat forced-swim test (FST). This effect …

MaleSerotoninJournal ClubMorpholinesmedicine.medical_treatmentPrefrontal CortexNaphthalenesPharmacologyEuphoriantAntidepressant likeRats Sprague-Dawleychemistry.chemical_compoundReceptor Cannabinoid CB1mental disordersAnimalsEthanolamideMedicineReceptorPrefrontal cortexNeuronsCannabinoidsDepressionbusiness.industryorganic chemicalsGeneral NeuroscienceAnandamideAntidepressive AgentsBenzoxazinesRatschemistryCannabinoidSerotoninbusinessNeuroscienceThe Journal of Neuroscience
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