Search results for "CB2"

showing 6 items of 26 documents

The yeast inositol monophosphatase is a lithium- and sodium-sensitive enzyme encoded by a non-essential gene pair

1999

Inositol monophosphatases (IMPases) are lithium-sensitive enzymes that participate in the inositol cycle of calcium signalling and in inositol biosynthesis. Two open reading frames (YHR046c and YDR287w) with homology to animal and plant IMPases are present in the yeast genome. The two recombinant purified proteins were shown to catalyse inositol-1-phosphate hydrolysis sensitive to lithium and sodium. A double gene disruption had no apparent growth defect and was not auxotroph for inositol. Therefore, lithium effects in yeast cannot be explained by inhibition of IMPases and inositol depletion, as suggested for animal systems. Overexpression of yeast IMPases increased lithium and sodium toler…

PLCB1ATPaseGenes FungalMolecular Sequence DataPLCB2PLCB3Inositol monophosphataseSaccharomyces cerevisiaeLithiumMicrobiologychemistry.chemical_compoundInositolAmino Acid SequenceCloning MolecularMolecular BiologybiologySodiumPhosphoric Monoester HydrolasesRecombinant ProteinsYeastchemistryBiochemistrybiology.proteinCalciumGene DeletionInositolIntracellularPlasmidsMolecular Microbiology
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Impact du système endocannabinoïdien sur la physiologie de l'obésité : effets de l'antagonisme des récepteurs CB1 sur le métabolisme glucido-lipidiqu…

2010

The endocannabinoïd system (ECS) is involved in many biological functions such as regulation of energy metabolism. Recently, several studies have shown an association between obesity and ECS overactivity. In addition, specific CB1R antagonists such as Rimonabant (SR141716) improved metabolic parameters in obese patients essentially through inactivation of central CB1R. However, peripheral CB1R inactivation could also contribute to the improvement of these parameters and it is this notion that we have studied. To this purpose, we tested the effects of SR141716 on obese mice in order to establish relationships between the ECS activity and lipid metabolism by looking more specifically to its r…

[SDV.SA]Life Sciences [q-bio]/Agricultural sciences[SDV.SA] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyStéatoseTissu adipeuxAnandamideMétabolisme glucido-lipidiqueEndocannabinoïde[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathologyCb1rNo english keywordsCb2rSystème endocannabinoïdienAdiponectineSr141716Foie[ SDV.SA ] Life Sciences [q-bio]/Agricultural sciences[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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A Low Dose of Pure Cannabidiol Is Sufficient to Stimulate the Cytotoxic Function of CIK Cells without Exerting the Downstream Mediators in Pancreatic…

2022

Despite numerous studies conducted over the past decade, the exact role of the cannabinoid system in cancer development remains unclear. Though research has focused on two cannabinoid receptors (CB1, CB2) activated by most cannabinoids, CB2 holds greater attention due to its expression in cells of the immune system. In particular, cytokine-induced killer cells (CIKs), which are pivotal cytotoxic immunological effector cells, express a high-level of CB2 receptors. Herein, we sought to investigate whether inducing CIK cells with cannabidiol can enhance their cytotoxicity and if there are any possible counter effects in its downstream cascade of phosphorylated p38 and CREB using a pancreatic d…

cytokine-induced killer cells; cannabidiol; pancreatic cancerpancreatic cancer.CannabinoidsOrganic ChemistryGeneral MedicineCatalysisComputer Science ApplicationsInorganic ChemistryPancreatic NeoplasmsReceptor Cannabinoid CB2Cytokine-Induced Killer CellsReceptor Cannabinoid CB1CannabidiolHumanslipids (amino acids peptides and proteins)Physical and Theoretical Chemistrycytokine-induced killer cellMolecular BiologySpectroscopyInternational Journal of Molecular Sciences; Volume 23; Issue 7; Pages: 3783
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Cannabinoid 1/2 Receptor Activation Induces Strain-Dependent Behavioral and Neurochemical Changes in Genetic Absence Epilepsy Rats From Strasbourg an…

2022

Childhood absence epilepsy (CAE) is characterized by absence seizures, which are episodes of lack of consciousness accompanied by electrographic spike-wave discharges. About 60% of children and adolescents with absence seizures are affected by major neuropsychological comorbidities, including anxiety. Endocannabinoids and monoamines are likely involved in the pathophysiology of these CAE psychiatric comorbidities. Here, we show that the synthetic cannabinoid receptor type 1/2 (CB1/2R) agonist WIN 55,212-2 (2 mg/kg) has a strain-dependent effect on anxiety-like and motor behavior when assess in the hole board test and cerebral monoaminergic levels in Genetic Absence Epilepsy Rats from Strasb…

high-pressure liquid chromatographyGABACellular and Molecular Neurosciencecannabinoid receptorsstrain-dependent effectsthalamusglutamateCB1Settore BIO/09 - FisiologiaCB2Frontiers in Cellular Neuroscience
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Impaired border zone formation and adverse remodeling after reperfused myocardial infarction in cannabinoid CB2 receptor deficient mice.

2014

Abstract Aims Reperfusion of myocardial infarction is associated with inflammatory reaction and subsequent myocardial remodeling with a rapid scar formation in mice. The cannabinoid receptor CB2 has been associated with cardioprotection and regulation of macrophage function. We investigated its role in remodeling of reperfused infarction. Main methods One hour LAD-occlusion was followed by reperfusion over 6 h and 1, 3 and 7 days in wild-type C57/BL6J (WT) and CB2 receptor-deficient (Cnr2 −/− ) mice (n = 8/group). Hearts were processed for functional, morphological and mRNA/protein analysis, and tissue concentration of endocannabinoids was determined using liquid chromatography-multiple rea…

medicine.medical_specialtyIschemiaMyocardial InfarctionInfarctionMyocardial Reperfusion InjuryGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceInternal medicinemedicineCannabinoid receptor type 2AnimalsMyocytes CardiacMyocardial infarctionGeneral Pharmacology Toxicology and PharmaceuticsCardioprotectionInflammationMice KnockoutbiologyChemistryMyocardiumTenascin CHemodynamicsGranulation tissueGeneral Medicinemedicine.diseaseEndocannabinoid systemMice Inbred C57BLEndocrinologymedicine.anatomical_structureCardiologybiology.proteinGranulation TissueCytokinesLife sciences
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The identification of peroxisome proliferator-activated receptor alpha-independent effects of oleoylethanolamide on intestinal transit in mice

2009

Oleoylethanolamide (OEA) is an endogenous lipid produced in the intestine that mediates satiety by activation of peroxisome proliferator-activated receptor alpha (PPARalpha). OEA inhibits gastric emptying and intestinal motility, but the mechanism of action remains to be determined. We investigated whether OEA inhibits intestinal motility by activation of PPARalpha. PPARalpha immunoreactivity was examined in whole mount preparations of mouse gastrointestinal (GI) tract. The effect of OEA on motility was assessed in wildtype, PPARalpha, cannabinoid CB(1) receptor and CB(2) receptor gene-deficient mice and in a model of accelerated GI transit. In addition, the effect of OEA on motility was as…

medicine.medical_specialtyPhysiologymedicine.medical_treatmentTRPV Cation ChannelsMotilityOleic AcidsBiologydigestive systemReceptor Cannabinoid CB2MiceOleoylethanolamidechemistry.chemical_compoundReceptor Cannabinoid CB1Glucagon-Like Peptide 1Internal medicinemedicineAnimalsPPAR alphaReceptorMice KnockoutGastric emptyingEndocrine and Autonomic Systemsdigestive oral and skin physiologyGastroenterologyImmunohistochemistryEndocannabinoid systemEndocrinologyMechanism of actionchemistrylipids (amino acids peptides and proteins)CannabinoidPeroxisome proliferator-activated receptor alphamedicine.symptomGastrointestinal MotilityEndocannabinoids
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