Search results for "CD11"

showing 10 items of 78 documents

Profile of leukocyte-endothelial cell interactions induced in venules and arterioles by nucleoside reverse-transcriptase inhibitors in vivo.

2013

Background There is controversy regarding cardiovascular (CV) toxicity of the nucleoside reverse-transcriptase inhibitors used to treat human immunodeficiency virus infection. Methods We evaluated the effects of nucleoside reverse-transcriptase inhibitors on leukocyte-endothelium interactions, a hallmark of CV diseases, in rat mesenteric vessels using intravital microscopy and in human arterial cells using a flow chamber system. Results Abacavir and didanosine increased rolling, adhesion and emigration in rat vessels. These effects were reversed with antibodies against Macrophage-1 antigen (Mac-1) or intercellular adhesion molecule 1 and were reproduced in human cells. Lamivudine, zidovudin…

Anti-HIV AgentsNeutrophilsIntercellular Adhesion Molecule-1Cell CommunicationPharmacologyEmtricitabineNucleoside Reverse Transcriptase InhibitorRats Sprague-DawleyVenulesAbacavirmedicineCell AdhesionImmunology and AllergyAnimalsHumansLeukocyte RollingDidanosineCells CulturedCD11b AntigenChemistryLamivudineEndothelial CellsIntercellular Adhesion Molecule-1VirologyDideoxynucleosidesRatsArteriolesDidanosineInfectious DiseasesCD18 AntigensLeukocytes MononuclearReverse Transcriptase InhibitorsEndothelium VascularNucleosideIntravital microscopymedicine.drugThe Journal of infectious diseases
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Microglial involvement in neuroplastic changes following focal brain ischemia in rats.

2009

The pathogenesis of ischemic stroke is a complex sequence of events including inflammatory reaction, for which the microglia appears to be a major cellular contributor. However, whether post-ischemic activation of microglial cells has beneficial or detrimental effects remains to be elucidated, in particular on long term brain plasticity events. The objective of our study was to determine, through modulation of post-stroke inflammatory response, to what extent microglial cells are involved in some specific events of neuronal plasticity, neurite outgrowth and synaptogenesis. Since microglia is a source of neurotrophic factors, the identification of the brain-derived neurophic factor (BDNF) as…

Brain InfarctionMaleTime FactorsNeuriteSciencePoly (ADP-Ribose) Polymerase-1SynaptophysinSynaptogenesisCell CountEnzyme-Linked Immunosorbent AssayNerve Tissue ProteinsBrain damageBiologyBrain IschemiaProinflammatory cytokineBrain ischemiaGAP-43 ProteinNeurotrophic factorsNeuroscience/Neuronal Signaling MechanismsmedicineAnimalsRats WistarCD11b AntigenNeuronal PlasticityMultidisciplinaryMicrogliaNeuroscience/Neuronal and Glial Cell BiologyBrain-Derived Neurotrophic FactorQRNeurological Disorders/Cerebrovascular DiseaseAntigens NuclearMacrophage Activationmedicine.diseaseImmunohistochemistryNeuroregenerationRatsEnzyme ActivationProtein Transportmedicine.anatomical_structureBenzamidesImmunologyMedicineMicrogliaPoly(ADP-ribose) Polymerasesmedicine.symptomNeuroscienceResearch ArticleNeurosciencePLoS ONE
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Interleukin 1α Promotes Th1 Differentiation and Inhibits Disease Progression in Leishmania major–susceptible BALB/c Mice

2003

Protective immunity against pathogens such as Leishmania major is mediated by interleukin (IL)-12–dependent Th1-immunity. We have shown previously that skin-dendritic cells (DCs) from both resistant C57BL/6 and susceptible BALB/c mice release IL-12 when infected with L. major, and infected BALB/c DCs effectively vaccinate against leishmaniasis. To determine if cytokines other than IL-12 might influence disease outcome, we surveyed DCs from both strains for production of a variety of cytokines. Skin-DCs produced significantly less IL-1α in response to lipopolysaccharide/interferon γ or L. major when expanded from BALB/c as compared with C57BL/6 mice. In addition, IL-1α mRNA accumulation in l…

CD4-Positive T-LymphocytesLipopolysaccharidedendritic cellT helper cell type 1/T helper cell type 2 immune responsemedicine.medical_treatmentImmunologyLeishmaniasis CutaneousMice Inbred StrainsLymphocyte ActivationArticleBALB/cMicechemistry.chemical_compoundCutaneous leishmaniasismedicineAnimalsImmunology and AllergyLeishmania majorLeishmania majorMice Inbred BALB CCD11b AntigenbiologyIL-1InterleukinDendritic Cellsbiology.organism_classificationmedicine.diseaseLeishmaniainfectionDisease Models AnimalCytokinechemistryImmunologyLymphInterleukin-1Journal of Experimental Medicine
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Preservation of dendritic cell function upon labeling with amino functionalized polymeric nanoparticles.

2010

Dendritic cells (DCs) are key players in eliciting immunity against antigens, therefore making them the focus of many investigations on immune responses in infections, cancer and autoimmune diseases. Nanosized materials have just recently been investigated for their use as carriers of antigens and as labeling agents for DCs. For this later use nanoparticles should be non-toxic and should most importantly not alter the physiological functions of DCs. Here we demonstrate that by the use of polymeric fluorescent nanoparticles as synthesized by the miniemulsion process immature DCs (iDCs) can be efficiently labeled intracellularly. Amino functionalized nanoparticles are more effective than carb…

CD4-Positive T-LymphocytesMaterials scienceBiophysicsCD11cchemical and pharmacologic phenomenaBioengineeringCD8-Positive T-LymphocytesFlow cytometryBiomaterialsCell therapyImmune systemAntigenmedicineHumansCells CulturedMicroscopy Confocalmedicine.diagnostic_testELISPOThemic and immune systemsDendritic cellDendritic CellsFlow CytometryCell biologyMechanics of MaterialsImmunologyCeramics and CompositesNanoparticlesPolystyrenesCD80Biomaterials
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β-Catenin Signaling Drives Differentiation and Proinflammatory Function of IRF8-Dependent Dendritic Cells

2014

Abstract β-Catenin signaling has recently been tied to the emergence of tolerogenic dendritic cells (DCs). In this article, we demonstrate a novel role for β-catenin in directing DC subset development through IFN regulatory factor 8 (IRF8) activation. We found that splenic DC precursors express β-catenin, and DCs from mice with CD11c-specific constitutive β-catenin activation upregulated IRF8 through targeting of the Irf8 promoter, leading to in vivo expansion of IRF8-dependent CD8α+, plasmacytoid, and CD103+CD11b− DCs. β-Catenin–stabilized CD8α+ DCs secreted elevated IL-12 upon in vitro microbial stimulation, and pharmacological β-catenin inhibition blocked this response in wild-type cells…

CD8 AntigensCellular differentiationImmunologyReceptors Cell SurfaceVaccinia virusPyrimidinonesCD8-Positive T-LymphocytesBiologyParasite LoadArticleProinflammatory cytokineMiceAntigens CDVacciniaAnimalsImmunology and AllergyPromoter Regions Geneticbeta CateninInflammationMice KnockoutCell DifferentiationDendritic CellsT lymphocyteTh1 CellsBridged Bicyclo Compounds HeterocyclicInterleukin-12CD11c AntigenCell biologyEnzyme ActivationMice Inbred C57BLInterferon Regulatory FactorsInterleukin 12FemaleIRF8Signal transductionIntegrin alpha ChainsToxoplasmaSpleenToxoplasmosisCD8Signal TransductionInterferon regulatory factorsThe Journal of Immunology
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Efficient Targeting of Protein Antigen to the Dendritic Cell Receptor DEC-205 in the Steady State Leads to Antigen Presentation on Major Histocompati…

2002

To identify endocytic receptors that allow dendritic cells (DCs) to capture and present antigens on major histocompatibility complex (MHC) class I products in vivo, we evaluated DEC-205, which is abundant on DCs in lymphoid tissues. Ovalbumin (OVA) protein, when chemically coupled to monoclonal alphaDEC-205 antibody, was presented by CD11c+ lymph node DCs, but not by CD11c- cells, to OVA-specific, CD4+ and CD8+ T cells. Receptor-mediated presentation was at least 400 times more efficient than unconjugated OVA and, for MHC class I, the DCs had to express transporter of antigenic peptides (TAP) transporters. When alphaDEC-205:OVA was injected subcutaneously, OVA protein was identified over a …

CD8-Positive T-LymphocytesMice0302 clinical medicineImmunology and AllergyCytotoxic T cellMice KnockoutAntigen Presentation0303 health sciencesMembrane GlycoproteinstoleranceAntibodies MonoclonalDEC-205 receptorrespiratory systemFlow CytometryEndocytosismedicine.anatomical_structureMHC class IFemaleOvalbuminT cellImmunologyAntigen presentationReceptors Cell Surfacechemical and pharmacologic phenomenaBiologyMajor histocompatibility complexArticleMinor Histocompatibility Antigens03 medical and health sciencesAntigenAntigens CDMHC class IImmune TolerancemedicineAnimalsLectins C-Typedendritic cellsAntigensCD40 Antigens030304 developmental biologyHistocompatibility Antigens Class IDendritic cellMolecular biologyCD11c AntigenMice Inbred C57BLCD8 T cellbiology.proteinLymph NodesCarrier ProteinsCD8030215 immunologyJournal of Experimental Medicine
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Extracorporeal shock waves increase markers of cellular proliferation in primary bronchial fibroblasts of COPD patients

2019

COPD is due to a remodeling of small airways and destruction of the lung parenchyma with loss of the alveolar attachment as a result of pulmonary emphysema. Treatment of connective tissue fibroblasts with extracorporeal shock waves (ESW) increases their cellular proliferation and differentiation. To date no studies are available on ESW treatment of human primary bronchial fibroblasts from COPD and control subjects. Primary bronchial fibroblasts were obtained from bronchial biopsies from three patients with mild/moderate COPD and 3 control smokers with normal lung function. After stabilization for 24h, 1 ml of cell suspension (106 cells) from COPD and controls was treated in 2 ml tubes with …

COPDPathologymedicine.medical_specialtybiologybusiness.industryCell growthCD117Connective tissuemedicine.diseaseExtracorporealrespiratory tract diseasesProliferating cell nuclear antigenmedicine.anatomical_structureParenchymabiology.proteinMedicineCD90businessAirway pharmacology and treatment
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Glioblastoma cells induce differential glutamatergic gene expressions in human tumor-associated microglia/macrophages and monocyte-derived macrophages

2015

Glioblastoma cells produce and release high amounts of glutamate into the extracellular milieu and subsequently can trigger seizure in patients. Tumor-associated microglia/macrophages (TAMs), consisting of both parenchymal microglia and monocytes-derived macrophages (MDMs) recruited from the blood, are known to populate up to 1/3 of the glioblastoma tumor environment and exhibit an alternative, tumor-promoting and supporting phenotype. However, it is unknown how TAMs respond to the excess extracellular glutamate in the glioblastoma microenvironment. We investigated the expressions of genes related to glutamate transport and metabolism in human TAMs freshly isolated from glioblastoma resecti…

Cancer ResearchAntigens Differentiation MyelomonocyticGlutamic AcidglutamateAMPA receptorSLC7A11Antigens CDTumor Cells CulturedExtracellularmedicineHumansReceptors AMPAGRIA2PharmacologyCD11b AntigenbiologyMicrogliaBrain NeoplasmsMacrophagesmonocyte-derived macrophagesCalcium-Binding ProteinsMicrofilament Proteinsglioblastomatumor-associated microglia/macrophagesGlutamate receptorSLC1A2Coculture TechniquesDNA-Binding ProteinsGlutaminemedicine.anatomical_structureGene Expression RegulationOncologyAstrocytesImmunologybiology.proteinCancer researchLeukocyte Common AntigensMolecular MedicineMicrogliaResearch PaperCancer Biology & Therapy
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Mast Cell–deficient KitW-sh “Sash” Mutant Mice Display Aberrant Myelopoiesis Leading to the Accumulation of Splenocytes That Act as Myeloid-Derived S…

2013

Abstract Mast cell-deficient KitW-sh “sash” mice are widely used to investigate mast cell functions. However, mutations of c-Kit also affect additional cells of hematopoietic and nonimmune origin. In this study, we demonstrate that KitW-sh causes aberrant extramedullary myelopoiesis characterized by the expansion of immature lineage-negative cells, common myeloid progenitors, and granulocyte/macrophage progenitors in the spleen. A consistent feature shared by these cell types is the reduced expression of c-Kit. Populations expressing intermediate and high levels of Ly6G, a component of the myeloid differentiation Ag Gr-1, are also highly expanded in the spleen of sash mice. These cells are …

Cell typeMyeloidT cellImmunologyBiologyImmunophenotypingMice03 medical and health sciences0302 clinical medicineNeoplasmsmedicineAnimalsAntigens LyImmunology and AllergyMyeloid CellsMast CellsProgenitor cell030304 developmental biologyMice KnockoutMyelopoiesis0303 health sciencesCD11b AntigenMast cellAdoptive Transfer3. Good healthCell biologyProto-Oncogene Proteins c-kitHaematopoiesismedicine.anatomical_structureHematopoiesis ExtramedullaryMutationImmunologyMyeloid-derived Suppressor CellFemaleMyelopoiesisNeoplasm TransplantationSpleen030215 immunologyThe Journal of Immunology
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Circulating specific antibodies enhance systemic cross-priming by delivery of complexed antigen to dendritic cells in vivo

2012

Increasing evidence suggests that antibodies can have stimulatory effects on T-cell immunity. However, the contribution of circulating antigen-specific antibodies on MHC class I cross-priming in vivo has not been conclusively established. Here, we defined the role of circulating antibodies in cross-presentation of antigen to CD8(+) T cells. Mice with hapten-specific circulating antibodies, but naϊve for the T-cell antigen, were infused with haptenated antigen and CD8(+) T-cell induction was measured. Mice with circulating hapten-specific antibodies showed significantly enhanced cross-presentation of the injected antigen compared with mice that lacked these antibodies. The enhanced cross-pre…

Cellular immunityOvalbuminImmunologyMice Transgenicchemical and pharmacologic phenomenaAntigen-Antibody ComplexCD8-Positive T-LymphocytesBiologyDendritic cellsAntibodiesMiceCross-PrimingImmune systemAntigenAntigens NeoplasmMHC class ITcellsAnimalsImmunology and AllergyImmunity CellularB cellsCross-presentationHistocompatibility Antigens Class ICross-presentationSerum Albumin BovineFlow CytometryCD11c AntigenMice Inbred C57BLMacrophage-1 antigenHumoral immunityImmunologybiology.proteinAntibodyHaptensEuropean Journal of Immunology
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