Search results for "CD1"

showing 10 items of 333 documents

CD1a expression by Barrett's metaplasia of gastric type may help to predict its evolution towards cancer

2005

As emerging in the recent literature, CD1a has been regarded as a molecule whose expression may reflect tumour evolution. The aim of the present work was to investigate the expression of CD1a in a series of Barrett's metaplasia (BM), gastric type (GTBM), with and without follow-up, in order to analyse whether its expression may help to diagnose this disease and to address the outcome. Indeed, GTBM may be confused sometimes with islets of ectopic gastric mucosa and its evolution towards dysplasia (Dy) or carcinoma (Ca) could not be foreseen. We showed a significant higher expression of CD1a in GTBM than in both Dy and Ca; nevertheless, the number of positive GTBM was significantly lower in t…

Cancer Researchmedicine.medical_specialtyDiseaseBiologyCD1aGastroenterologyAntigens CD1Barrett EsophagusStomach NeoplasmsMetaplasiaInternal medicinemedicineCarcinomaGastric mucosaHumansMolecular DiagnosticsRetrospective StudiesMetaplasiaintegumentary systemStomachCancerDendritic CellsBarrett’s metaplasiamedicine.diseaseImmunohistochemistrymedicine.anatomical_structureCell Transformation NeoplasticOncologyDysplasiaGastric MucosaCancer researchBiomarker (medicine)Barrett’s metaplasia; CD1a; carcinogenesismedicine.symptomBarrett's metaplasiacarcinogenesis
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Pharmacogenomics: a tool to prevent and cure coronary heart disease.

2007

Inflammation and genetics play an important role in the pathogenesis of coronary heart disease (CHD). This is supported by epidemiological studies which have thoroughly investigated the association between CHD and gene polymorphisms of the inflammatory molecules. Moreover, efforts to find elective therapy have not been rewarding and, despite the increasing appreciation of the role of genetics in CHD and myocardial infarction (MI) pathogenesis, pharmacogenomic approaches to uncover drug target have not been extensively explored. A critical search of published literature has suggested few inflammatory genes directly involved in the risk to develop CHD and MI. The selected genes are, the pro- …

Candidate genepharmacogenomicLipoxygenaseLipopolysaccharide ReceptorsMyocardial InfarctionCoronary DiseaseDiseaseBioinformaticsRisk AssessmentPathogenesisRisk FactorsDrug DiscoverymedicinecytokineHumansGenetic Predisposition to DiseaseMyocardial infarctionTLR4PharmacologyInflammationPolymorphism Geneticbusiness.industryPatient SelectionCase-control studyCOXLOXmedicine.diseaseAtherosclerosisToll-Like Receptor 4Treatment OutcomePharmacogeneticsProstaglandin-Endoperoxide SynthasesPharmacogenomicsCase-Control StudiesImmunologyCytokinesReceptors ChemokineChemokinesbusinessRisk assessmentCD14CCR5PharmacogeneticsCurrent pharmaceutical design
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Microparticles: Modulators and biomarkers of liver disease

2012

Division of Molecular and Translational Medicine,Dept. of Medicine I, Johannes Gutenberg University, Mainz, GermanyEmerging role of microparticlesMicroparticles (MP) have gained increasing attention as biomark-ers for various diseases. First described as platelet dust, MP wereregarded as unspecific debris [1]. However, it has become appar-ent that cell derived MP or ectosomes represent a novel route ofhorizontal communication between cells. MP are between 100–1000 nm in size and generated through cell membrane shedding(ectocytosis), a process that can be triggered by the activation ofthe complement C5b-9 complex, as shown for platelet derivedMP, or by inhibition of flippase activitythroughCa

Cell signalingCD14+MacrophageT cellCellApoptosisCell CommunicationBiologyMonocyteExosomeAnnexin VCD4+Cell membraneCell-Derived MicroparticlesmedicineHumansMacrophageEctosomeCD41+InflammationHepatologyLiver DiseasesMonocytePlateletNASHT cellBiomarkerDendritic cellCD8+FibrosisHepatitis CCell biologyExosomemedicine.anatomical_structureLiverMicroparticleBiochemistryNAFLiNKTBiomarkersDendritic cellJournal of Hepatology
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Mast Cell–deficient KitW-sh “Sash” Mutant Mice Display Aberrant Myelopoiesis Leading to the Accumulation of Splenocytes That Act as Myeloid-Derived S…

2013

Abstract Mast cell-deficient KitW-sh “sash” mice are widely used to investigate mast cell functions. However, mutations of c-Kit also affect additional cells of hematopoietic and nonimmune origin. In this study, we demonstrate that KitW-sh causes aberrant extramedullary myelopoiesis characterized by the expansion of immature lineage-negative cells, common myeloid progenitors, and granulocyte/macrophage progenitors in the spleen. A consistent feature shared by these cell types is the reduced expression of c-Kit. Populations expressing intermediate and high levels of Ly6G, a component of the myeloid differentiation Ag Gr-1, are also highly expanded in the spleen of sash mice. These cells are …

Cell typeMyeloidT cellImmunologyBiologyImmunophenotypingMice03 medical and health sciences0302 clinical medicineNeoplasmsmedicineAnimalsAntigens LyImmunology and AllergyMyeloid CellsMast CellsProgenitor cell030304 developmental biologyMice KnockoutMyelopoiesis0303 health sciencesCD11b AntigenMast cellAdoptive Transfer3. Good healthCell biologyProto-Oncogene Proteins c-kitHaematopoiesismedicine.anatomical_structureHematopoiesis ExtramedullaryMutationImmunologyMyeloid-derived Suppressor CellFemaleMyelopoiesisNeoplasm TransplantationSpleen030215 immunologyThe Journal of Immunology
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Endothelial Nitric Oxide Synthase

2007

Endothelial nitric oxide synthase (eNOS; also referred to as NOS3 or NOSIII), a low output enzyme is the prototypical isoform being found in endothelial cells. This isoform (like nNOS) synthesizes NO in a short-lasting, pulsatile, Ca++/calmodulin-activated manner. Endothelium-derived NO is a physiologically significant vasodilator and inhibitor of platelet aggregation and adhesion. In addition, vascular NO can prevent leukocyte adhesion to the endothelium by down-regulating the leukocyte adhesion glycoprotein complex CD11/CD18. Finally, endothelial NO has also been shown to inhibit the proliferation of vascular smooth muscle cells. Therefore, endothelial NO is likely to represent a protecti…

Cell typeVascular smooth muscleEndotheliumbiologyCD18Vasodilationbiology.organism_classificationCell biologymedicine.anatomical_structureBiochemistryEnosGlycoprotein complexmedicinePlatelet
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Invariant natural killer T cells act as an extravascular cytotoxic barrier for joint-invading Lyme Borrelia

2014

CXCR6-GFP(+) cells, which encompass 70% invariant natural killer T cells (iNKT cells), have been found primarily patrolling inside blood vessels in the liver. Although the iNKT cells fail to interact with live pathogens, they do respond to bacterial glycolipids presented by CD1d on liver macrophage that have caught the microbe. In contrast, in this study using dual laser multichannel spinning-disk intravital microscopy of joints, the CXCR6-GFP, which also made up 60-70% iNKT cells, were not found in the vasculature but rather closely apposed to and surrounding the outside of blood vessels, and to a lesser extent throughout the extravascular space. These iNKT cells also differed in behavior,…

CellMice TransgenicSpleenjoint iNKT cellsGranzymesMicegranzyme BmedicineAnimalsHumansCytotoxic T cellBorrelia burgdorferiImmunity CellularLyme DiseaseMice Inbred BALB CMultidisciplinarybiologyLyme arthritisNatural killer T cellbiology.organism_classificationGranzyme Bmedicine.anatomical_structureLiverGranzymeOrgan SpecificityBorrelia burgdorferiCD1DImmunologybiology.proteinNatural Killer T-CellsJointsJoint DiseasesSpleen
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Circulating specific antibodies enhance systemic cross-priming by delivery of complexed antigen to dendritic cells in vivo

2012

Increasing evidence suggests that antibodies can have stimulatory effects on T-cell immunity. However, the contribution of circulating antigen-specific antibodies on MHC class I cross-priming in vivo has not been conclusively established. Here, we defined the role of circulating antibodies in cross-presentation of antigen to CD8(+) T cells. Mice with hapten-specific circulating antibodies, but naϊve for the T-cell antigen, were infused with haptenated antigen and CD8(+) T-cell induction was measured. Mice with circulating hapten-specific antibodies showed significantly enhanced cross-presentation of the injected antigen compared with mice that lacked these antibodies. The enhanced cross-pre…

Cellular immunityOvalbuminImmunologyMice Transgenicchemical and pharmacologic phenomenaAntigen-Antibody ComplexCD8-Positive T-LymphocytesBiologyDendritic cellsAntibodiesMiceCross-PrimingImmune systemAntigenAntigens NeoplasmMHC class ITcellsAnimalsImmunology and AllergyImmunity CellularB cellsCross-presentationHistocompatibility Antigens Class ICross-presentationSerum Albumin BovineFlow CytometryCD11c AntigenMice Inbred C57BLMacrophage-1 antigenHumoral immunityImmunologybiology.proteinAntibodyHaptensEuropean Journal of Immunology
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IL-23-mediated mononuclear phagocyte crosstalk protects mice from Citrobacter rodentium-induced colon immunopathology.

2014

Gut homeostasis and mucosal immune defense rely on the differential contributions of dendritic cells (DC) and macrophages. Here we show that colonic CX3CR1+ mononuclear phagocytes are critical inducers of the innate response to Citrobacter rodentium infection. Specifically, the absence of IL-23 expression in macrophages or CD11b+ DC results in the impairment of IL-22 production and in acute lethality. Highlighting immunopathology as a death cause, infected animals are rescued by the neutralization of IL-12 or IFNγ. Moreover, mice are also protected when the CD103+ CD11b− DC compartment is rendered deficient for IL-12 production. We show that IL-12 production by colonic CD103+ CD11b− DC is r…

ChemokineColonCX3C Chemokine Receptor 1General Physics and Astronomychemical and pharmacologic phenomenaMice TransgenicInterleukin-23General Biochemistry Genetics and Molecular BiologyMonocytesArticleMicrobiologyInterferon-gammaMiceIntestinal mucosaAntigens CDImmunopathologyCitrobacter rodentiummedicineAnimalsHomeostasisInterferon gammaIntestinal MucosaImmunity MucosalMultidisciplinaryCD11b AntigenbiologyInterleukinsMacrophagesEnterobacteriaceae InfectionsGeneral ChemistryMononuclear phagocyte systemDendritic CellsInterleukin-12Survival AnalysisImmunity InnateIntegrin alpha MGene Expression RegulationImmunologyInterleukin 12biology.proteinCitrobacter rodentiumTh17 CellsReceptors ChemokineIntegrin alpha Chainsmedicine.drugSignal TransductionNature communications
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Thy-1 (CD90) regulates the extravasation of leukocytes during inflammation.

2010

Human Thy-1 (CD90) has been shown to mediate adhesion of inflammatory cells to activated microvascular endothelial cells via interaction with Mac-1 (CD11b/CD18) in vitro. Since there are no data showing the physiological relevance of Thy-1 for the recruitment of inflammatory cells in vivo, different inflammation models were investigated in Thy-1-deficient mice and littermate controls. In thioglycollate-induced peritonitis, the number of neutrophils and monocytes was significantly diminished in Thy-1-deficient mice. During acute lung inflammation, the extravasation of eosinophils and monocytes into the lung was significantly reduced in Thy-1-deficient mice. Moreover, during chronic lung infl…

ChemokineMice 129 StrainNeutrophilsmedicine.medical_treatmentT-LymphocytesImmunologyInflammationCD18In Vitro TechniquesPeritonitisMonocytesMiceCell MovementCell AdhesionLeukocytesImmunology and AllergyMedicineAnimalsHumansCD90Thy-1InflammationMice KnockoutTransplantation Chimerabiologybusiness.industryInterleukinsEndothelial CellsPneumoniaExtravasationTransplantationEosinophilsMice Inbred C57BLCytokineIntegrin alpha MImmunologybiology.proteinThy-1 Antigensmedicine.symptomChemokinesbusinessextravasationPeptide HydrolasesEuropean journal of immunology
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Atherosclerosis as an inflammatory disease.

2011

In many ways, atherosclerosis is a chronic inflammatory disorder and this issue is confirmed by recent investigations of that have focused on inflammation, providing new insight into mechanisms of disease. Several recent studies have addressed the role of chemokines in leukocyte accumulation in atherosclerosis, extending our knowledge and understanding of the complex and cell type-specific functions of chemokines in atherosclerosis. Activated T-lymphocytes within the atherosclerotic vessel wall express the CD40 ligand surface molecule, known to play a major role in several immunological pathways. In addition to activated T-lymphocytes, functional CD40 and CD40L are coexpressed by human vasc…

ChemokineSettore MED/09 - Medicina InternaEndotheliumT-LymphocytesInflammationDrug DiscoverymedicineLeukocytesAnimalsHumansCD154PharmacologyInflammationCD40Innate immune systembiologyCell adhesion moleculeMacrophagesAtherosclerosis inflammationAcquired immune systemAtherosclerosisPlaque Atheroscleroticmedicine.anatomical_structureDrug DesignImmunologybiology.proteinCancer researchCytokinesEndothelium Vascularmedicine.symptomChemokinesCurrent pharmaceutical design
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