Search results for "CD1"

showing 10 items of 333 documents

A role for Toll-like receptor mediated signals in neutrophils in the pathogenesis of the anti-phospholipid syndrome.

2012

The anti-phospholipid syndrome (APS) is characterized by recurrent thrombosis and occurrence of anti-phospholipid antibodies (aPL). aPL are necessary, but not sufficient for the clinical manifestations of APS. Growing evidence suggests a role of innate immune cells, in particular polymorphonuclear neutrophils (PMN) and Toll-like receptors (TLR) to be additionally involved. aPL activate endothelial cells and monocytes through a TLR4-dependent signalling pathway. Whether this is also relevant for PMN in a similar way is currently not known. To address this issue, we used purified PMN from healthy donors and stimulated them in the presence or absence of human monoclonal aPL and the TLR4 agonis…

LipopolysaccharidesNeutrophilsImmunology610 MedizinImmunoglobulinslcsh:MedicineInflammationApoptosisImmunopathologyBiologyNeutrophil ActivationAutoimmune DiseasesPhagocytosisimmune system diseases610 Medical sciencesmedicineHumansInterleukin 8L-SelectinReceptorlcsh:ScienceBiologyImmune ResponseneoplasmsRespiratory BurstInflammationToll-like receptorMultidisciplinaryInnate immune systemCD11b AntigenCoagulation DisordersEffectorInterleukin-8lcsh:RImmunityHematologyAntiphospholipid SyndromeFlow CytometryInnate ImmunityRespiratory burstToll-Like Receptor 4ImmunologyTLR4MedicineClinical Immunologylcsh:Qmedicine.symptomResearch ArticleSignal TransductionPLoS ONE
researchProduct

Dendritic cells in liver injury and fibrosis: shortcomings and promises.

2013

SummaryThe phenotype and function of liver dendritic cells (LDCs) are poorly understood. This Snapshot summarizes our current knowledge on LDCs in the healthy and injured liver, and their role in fibrosis progression and reversal. It also draws attention to various pitfalls in the current experimental design and conclusions based on available data.

Liver CirrhosisLiver dendritic cellsPlasmacytoid dendritic cellBiologyCCL2MiceFMS-like tyrosine kinase 3 ligandFibrosismedicineAnimalsHumansAntigen-presenting cellLiver injuryHepatologyFlt3LDendritic Cellsmedicine.diseaseCD11c-DTRDisease Models Animalmedicine.anatomical_structurePhenotypeLiverImmunologyHepatic stellate cellDisease ProgressionBone marrowToleranceJournal of hepatology
researchProduct

Coincident airway exposure to low-potency allergen and cytomegalovirus sensitizes for allergic airway disease by viral activation of migratory dendri…

2019

Despite a broad cell-type tropism, cytomegalovirus (CMV) is an evidentially pulmonary pathogen. Predilection for the lungs is of medical relevance in immunocompromised recipients of hematopoietic cell transplantation, in whom interstitial CMV pneumonia is a frequent and, if left untreated, fatal clinical manifestation of human CMV infection. A conceivable contribution of CMV to airway diseases of other etiology is an issue that so far attracted little medical attention. As the route of primary CMV infection upon host-to-host transmission in early childhood involves airway mucosa, coincidence of CMV airway infection and exposure to airborne environmental antigens is almost unavoidable. For i…

Lung DiseasesPulmonologyMedizinCytomegalovirusImmunoglobulin EPathology and Laboratory MedicineWhite Blood CellsMiceAnimal CellsMedicine and Health SciencesCytotoxic T cellBiology (General)Enzyme-Linked ImmunoassaysImmune ResponseLung0303 health sciencesAntigen PresentationbiologyT Cells030302 biochemistry & molecular biologyAnimal Modelsrespiratory systemExperimental Organism SystemsFemalemedicine.symptomCellular TypesResearch ArticleQH301-705.5OvalbuminImmune CellsAntigen presentationImmunologyInflammationCytotoxic T cellsMouse ModelsResearch and Analysis MethodsMicrobiology03 medical and health sciencesSigns and SymptomsModel OrganismsTh2 CellsAntigenDiagnostic MedicineVirologyGeneticsmedicineHypersensitivityAnimalsT Helper CellsMolecular Biology TechniquesImmunoassaysMolecular Biology030304 developmental biologyInflammationBlood Cellsbusiness.industryCD11 AntigensBiology and Life SciencesCell BiologyDendritic CellsRC581-607Allergensrespiratory tract diseasesTransplantationMice Inbred C57BLOvalbuminDisease Models AnimalImmunologyRespiratory Infectionsbiology.proteinAnimal StudiesImmunologic TechniquesParasitologyVirus ActivationImmunologic diseases. AllergybusinessCD8CloningPLoS Pathogens
researchProduct

Evaluation of soluble CD 14 and neopterin as serum parameters of the inflammatory activity of pulmonary sarcoidosis.

1992

CD14 represents the most specific marker for monocytes/macrophages. It has been demonstrated in vitro that monocytes/macrophages lose this antigen upon activation. Results of studies investigating the expression of membrane-bound CD14 on the surface of monocytes/macrophages in sarcoidosis patients are controversial. To investigate whether the soluble form of CD14 reflects monocyte/macrophage activation in sarcoidosis, serum levels of soluble CD14 were determined concurrently with other serum markers of monocyte/macrophage activation (neopterin, angiotensin-converting enzyme) in 50 consecutive patients with bioptically confirmed sarcoidosis. The patients were allocated to three groups accord…

Lung Diseasesmedicine.medical_specialtySarcoidosisCD14CD4-CD8 RatioLipopolysaccharide ReceptorsAntigens Differentiation MyelomonocyticPeptidyl-Dipeptidase ANeopterinSensitivity and SpecificityMonocyteschemistry.chemical_compoundImmune systemAntigenAntigens CDInternal medicineDrug DiscoverymedicineMacrophageHumansGenetics (clinical)Inflammationmedicine.diagnostic_testMonocyteNeopterinGeneral MedicineMacrophage Activationmedicine.diseaseBiopterinBronchoalveolar lavageEndocrinologymedicine.anatomical_structurechemistrySolubilityImmunologyMolecular MedicineInterleukin-2SarcoidosisBronchoalveolar Lavage FluidBiomarkersThe Clinical investigator
researchProduct

Global distributions of diazotrophs Gamma-A nifH genes abundance - Depth integrated values computed from a collection of source datasets - Contributi…

2013

The MAREDAT atlas covers 11 types of plankton, ranging in size from bacteria to jellyfish. Together, these plankton groups determine the health and productivity of the global ocean and play a vital role in the global carbon cycle. Working within a uniform and consistent spatial and depth grid (map) of the global ocean, the researchers compiled thousands and tens of thousands of data points to identify regions of plankton abundance and scarcity as well as areas of data abundance and scarcity. At many of the grid points, the MAREDAT team accomplished the difficult conversion from abundance (numbers of organisms) to biomass (carbon mass of organisms). The MAREDAT atlas provides an unprecedente…

M60/5SalinityChlorophyll aDiazotrophs total biomass as carbonUniform resource locator link to source data fileNitrateCTD/RosetteLatitude of eventNiskinM55 1Temperature waterCalothrix abundance expressed in number of nifH gene copiesratio expressed in mass of carbon per amount of nifH gene copiesCalculatedtop minUnicellular cyanobacteria-B biological trait ratio expressed in mass of carbon per amount of nifH gene copiesCD132biomass as carbonTrichodesmium biomass as carbonM55/1bottom maxCTD SeabirdTemperatureDepth top/minCTD RosetteSeabirdRichelia biological trait ratio expressed in mass of carbon per amount of nifH gene copiesCalothrixSO187 2Unicellular cyanobacteria-B abundance expressed in number of nifH gene copiesTrichodesmiumEarth System ResearchMARine Ecosystem Model Intercomparison Project MAREMIPDiazotrophsLongitude of eventRichelia associated speciesSample methodCalothrix biological trait ratio expressed in mass of carbon per amount of nifH gene copiesIronBottle NiskinwaterIn situ pumpMARine Ecosystem Model Intercomparison Project (MAREMIP)Unicellular cyanobacteria-C abundance expressed in number of nifH gene copiesPhosphateWater sampleSample commentUnicellular cyanobacteria biomassUniform resource locator/link to source data filetotal biomass as carbonHeterocyst biomassUnicellular cyanobacteriaProteobacteriaDate/Time of eventMeteor 1986Richelia abundance expressed in number of nifH gene copiesUnicellular cyanobacteria CUnicellular cyanobacteria Bbiological traitSO187/2RicheliaUnicellular cyanobacteria ADEPTH waterbiomassTrichodesmium abundance expressed in number of nifH gene copiesMeteor (1986)BottleDepthEvent labelDate Time of eventTrichodesmium biological trait ratio expressed in mass of carbon per amount of nifH gene copiesUnicellular cyanobacteria-C biological trait ratio expressed in mass of carbon per amount of nifH gene copiesMeasured at sea surfaceCTDCalothrix associated speciesCharles DarwinSonneabundance expressed in number of nifH gene copiesM60 5Depth bottom/maxUnicellular cyanobacteria-A abundance expressed in number of nifH gene copiesassociated speciesProteobacteria abundance expressed in number of nifH gene copiesHeterocyst
researchProduct

Coupling of Contact Sensitizers to Thiol Groups is a Key Event for the Activation of Monocytes and Monocyte-Derived Dendritic Cells

2003

Strong contact sensitizers are able to induce distinct signal transduction mechanisms in antigen-presenting cells by coupling to cell proteins. The predominant target structures of haptens are thought to be thiol and amino groups in cysteine and lysine residues. We studied whether coupling of small reactive chemicals to thiol or amino groups might be responsible for the activation of monocytes and mature monocyte-derived dendritic cells. Human peripheral blood mononuclear cells were stimulated in vitro with subtoxic concentrations of the strong haptens 5-chloro-2-methylisothiazolinone plus 2-methylisothiazolinone and 2, 4, 6-trinitrochlorobenzene, the thiol-reactive reagents N-hydroxymaleim…

MAP Kinase Signaling SystemCD14SuccinimidesPicryl ChlorideDermatologyAcetatesPeripheral blood mononuclear cellBiochemistryamino groupsAntioxidantsMonocytesMaleimideschemistry.chemical_compoundAnti-Infective AgentsmedicineHumansCysteineSulfhydryl CompoundsPhosphorylationAntigen-presenting cellMolecular Biologythiol groupsChemistryMonocyteLysineSulfhydryl ReagentsTyrosine phosphorylationDendritic cellDendritic CellsCell BiologyThiazolesmedicine.anatomical_structureBiochemistryEthylmaleimidehaptenTyrosineSignal transductionsignal transductionCysteineInterleukin-1Journal of Investigative Dermatology
researchProduct

The Late Endosomal Adaptor Molecule p14 (LAMTOR2) Regulates TGFβ1-Mediated Homeostasis of Langerhans Cells

2014

Langerhans cells (LCs), a sub-population of dendritic cells (DCs) in the skin, participate in the regulation of immunity and peripheral tolerance. The adaptor molecule p14 is part of the late endosomal/lysosomal adaptor and mitogen-activated protein kinase and mammalian target of rapamycin (mTOR) activator/regulator (LAMTOR) complex, which mediates the activation of lysosome-associated extracellular signaling regulated kinase (ERK) and the mTOR cascade. In previous work, we demonstrated that CD11c-specific deficiency of p14 disrupts LC homeostasis by affecting the LAMTOR-mediated ERK and mTOR signaling. In this study, we extended our analysis on p14 deficiency specifically in LCs. Langerin-…

MAPK/ERK pathwayMaleMAP Kinase Signaling SystemReceptor Transforming Growth Factor-beta Type IDown-Regulationchemical and pharmacologic phenomenaEndosomesDermatologyBiologyProtein Serine-Threonine KinasesDermatitis ContactBiochemistryArticleImmune toleranceImmunophenotypingTransforming Growth Factor beta103 medical and health sciences0302 clinical medicineDownregulation and upregulationCell MovementImmune ToleranceAnimalsHomeostasisProtein kinase AMolecular BiologyPI3K/AKT/mTOR pathway030304 developmental biologySkin0303 health sciencesintegumentary systemKinaseReceptor Transforming Growth Factor-beta Type IIPeripheral toleranceProteinshemic and immune systemsCell BiologyMice Mutant StrainsCell biologyCD11c AntigenLangerhans CellsFemaleReceptors Transforming Growth Factor beta030215 immunologyTransforming growth factorJournal of Investigative Dermatology
researchProduct

Major histocompatibility complex class I-restricted activation of cloned T cells by a soluble protein in the absence of accessory cells.

1989

A T-cell clone, 10BK.1, was established from the draining lymph nodes of (B10 x B10.BR)F1 mice immunized with ovalbumin (OVA) according to standard protocols. Upon coculture with the antigen, 10BK.1 cells reacted by production of lymphokines and by proliferation despite the absence of additional antigen-presenting cells. These T cells do not express major histocompatibility complex (MHC) class II molecules on the cell surface as assessed on the basis of several criteria: by cytofluorometric analysis I-A and I-E determinants were not detectable; 10BK.1 cells could not act as antigen-presenting cells for long-term-cultured MHC class II-restricted T-cell clones; and monoclonal antibodies direc…

MHC class IIMultidisciplinarybiologyOvalbuminT-LymphocytesHistocompatibility Antigens Class IAntigen presentationCD1chemical and pharmacologic phenomenaMHC restrictionLymphocyte ActivationVirologyMolecular biologyAntibodiesCell LineClone CellsMiceAntigenMHC class Ibiology.proteinAnimalsCytotoxic T cellAntigen-presenting cellResearch ArticleProceedings of the National Academy of Sciences
researchProduct

Alveolar macrophage dynamics in murine lung regeneration

2012

In most mammalian species, the removal of one lung results in dramatic compensatory growth of the remaining lung. To investigate the contribution of alveolar macrophages (AMs) to murine post-pneumonectomy lung growth, we studied bronchoalveolar lavage (BAL)-derived AM on 3, 7, 14 and 21 days after left pneumonectomy. BAL demonstrated a 3.0-fold increase in AM (CD45(+), CD11b(-), CD11c(+), F4/80(+), Gr-1(-)) by 14 days after pneumonectomy. Cell cycle flow cytometry of the BAL-derived cells demonstrated an increase in S + G2 phase cells on days 3 (11.3 ± 2.7%) and 7 (12.1 ± 1.8%) after pneumonectomy. Correspondingly, AM demonstrated increased expression of VEGFR1 and MHC class II between days…

MHC class IIeducation.field_of_studyLungbiologymedicine.diagnostic_testPhysiologymedicine.medical_treatmentClinical BiochemistryPopulationCD11cCell Biologyrespiratory systemFlow cytometryAndrologyPneumonectomyBronchoalveolar lavagemedicine.anatomical_structureImmunologyAlveolar macrophagebiology.proteinmedicineeducationJournal of Cellular Physiology
researchProduct

A role for miR-142-3p in colony-stimulating factor 1-induced monocyte differentiation into macrophages

2013

AbstractThe differentiation of human peripheral blood monocytes into macrophages can be reproduced ex vivo by culturing the cells in the presence of colony-stimulating factor 1 (CSF1). Using microarray profiling to explore the role of microRNAs (miRNAs), we identified a dramatic decrease in the expression of the hematopoietic specific miR-142-3p. Up- and down-regulation of this miRNA in primary human monocytes altered CSF1-induced differentiation of monocytes, as demonstrated by changes in the expression of the cell surface markers CD16 and CD163. One of the genes whose expression is repressed by miR-142-3p encodes the transcription factor Early Growth Response 2 (Egr2). In turn, Egr2 assoc…

Macrophage colony-stimulating factorAntigens Differentiation MyelomonocyticDown-RegulationChronic myelomonocytic leukemiaReceptors Cell SurfaceCD16BiologyGPI-Linked ProteinsMonocyte–macrophage differentiationMonocytesChronic myelomonocytic leukemiaAntigens CDCell Line TumorMiR-142-3pmedicineHumansTranscription factorMolecular BiologyEarly Growth Response Protein 2Early Growth Response Protein 1Cluster of differentiationMolecular circuitryMacrophage Colony-Stimulating FactorMacrophagesReceptors IgGCell DifferentiationLeukemia Myelomonocytic ChronicCell Biologymedicine.diseaseUp-RegulationRepressor ProteinsMicroRNAsHaematopoiesisMonocyte differentiationCancer researchEgr2K562 CellsK562 cellsBiochimica et Biophysica Acta (BBA) - Molecular Cell Research
researchProduct