Search results for "CELL PROLIFERATION"

showing 10 items of 1056 documents

Curcumin inhibits in vitro and in vivo chronic myelogenous leukemia cells growth : a possible role for exosomal disposal of miR-21

2015

// Simona Taverna 1 , Marco Giallombardo 1 , Marzia Pucci 1 , Anna Flugy 1 , Mauro Manno 2 , Samuele Raccosta 2 , Christian Rolfo 3 , Giacomo De Leo 1 , Riccardo Alessandro 1, 4 1 Dipartimento di Biopatologia e Metodologie Biomediche, Sezione di Biologia e Genetica, Universita di Palermo, Italy 2 Istituto di Biofisica, CNR, Palermo, Italy 3 Phase I - Early Clinical Trials Unit Oncology Department and Center of Oncological Research (CORE), University Hospital Antwerp & Antwerp University, Belgium 4 Istituto di Biomedicina e Immunologia Molecolare (IBIM), Consiglio Nazionale delle Ricerche, Palermo, Italy Correspondence to: Riccardo Alessandro, e-mail: riccardo.alessandro@unipa.it Keywords: e…

MaleCurcuminexosomes microRNAs CML curcumin miR-21exosomesMice SCIDBiologyTransfectionMiceRandom Allocationchemistry.chemical_compoundDownregulation and upregulationLeukemia Myelogenous Chronic BCR-ABL Positivehemic and lymphatic diseasesmedicineAnimalsHumansCMLBiologyCell ProliferationCell growthTransfectionmedicine.diseaseXenograft Model Antitumor AssaysMolecular biologyMicrovesiclesmicroRNAsOncologychemistryCancer cellCurcuminmiR-21Human medicineK562 CellsResearch PaperChronic myelogenous leukemiaK562 cellsOncotarget
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Evaluation of cell proliferation rate in non-dysplastic leukoplakias

2008

Objective: Analyze whether the most frequent cases of non-dysplastic leukoplakias, hyperkeratosis (H), acanthosis (A), and hyperkeratosis with acanthosis (HA) have similar cell proliferation rates and to compare them with epithelial dysplastic (ED) leukoplakias and normal oral epithelium (NOE).Study design: The sample comprised 10 cases of normal oral epithelium, 10 cases of hyperkeratosis, 10 cases of acanthosis, 10 cases of hyperkeratosis with acanthosis and 10 cases of epithelial dysplasia. The mean number of AgNORs per nucleus (mAgNOR) and the mean percentage of cells with 1, 2, 3 and 4 or more AgNORs per nucleus (pAgNOR) were recorded. Results: The results of mAgNOR showed differences …

MaleEpithelial dysplasiaPathologymedicine.medical_specialtyHyperkeratosisAcanthosisHyperkeratosisEpithelial dysplasiaAcanthosisBasal (phylogenetics)Biological profileProliferation rateOral and maxillofacial pathologyHumansMedicineLeucoplasia bucalGeneral DentistryCell proliferationCell ProliferationLeukoplakiabusiness.industryAntigens NuclearProliferação celularMiddle Agedmedicine.disease:CIENCIAS MÉDICAS [UNESCO]Doencas [Mucosa bucal]AgNORstomatognathic diseasesOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASPatologia bucalFemaleSurgeryLeukoplakia OralbusinessLeukoplakia
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Uptake and cytotoxicity of citrate-coated gold nanospheres : comparative studies on human endothelial and epithelial cells

2012

Abstract Background The use of gold nanoparticles (AuNPs) for diagnostic applications and for drug and gene-delivery is currently under intensive investigation. For such applications, biocompatibility and the absence of cytotoxicity of AuNPs is essential. Although generally considered as highly biocompatible, previous in vitro studies have shown that cytotoxicity of AuNPs in certain human epithelial cells was observed. In particular, the degree of purification of AuNPs (presence of sodium citrate residues on the particles) was shown to affect the proliferation and induce cytotoxicity in these cells. To expand these studies, we have examined if the effects are related to nanoparticle size (1…

MaleHealth Toxicology and Mutagenesis610 MedizinMetal Nanoparticles02 engineering and technologyToxicology01 natural scienceschemistry.chemical_compoundCoated Materials Biocompatible610 Medical sciencesQDCitratesCytotoxicityGeneral Medicine021001 nanoscience & nanotechnologyEndothelial stem cellmedicine.anatomical_structureColloidal goldBlood-Brain Barrier0210 nano-technologyNanospheresMaterials scienceEndotheliumCell SurvivalForeskinlcsh:Industrial hygiene. Industrial welfare010402 general chemistrySodium CitrateCell LineMicroscopy Electron Transmissionlcsh:RA1190-1270Sodium citratemedicineHumansViability assayParticle Sizelcsh:Toxicology. PoisonsCell ProliferationResearchCytoplasmic VesiclesEpithelial CellsQPIn vitro0104 chemical scienceschemistryCell culture[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieImmunologyBiophysics[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieEndothelium VascularGoldlcsh:HD7260-7780.8
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Posttranslational modifications by ADAM10 shape myeloid antigen-presenting cell homeostasis in the splenic marginal zone

2021

The spleen contains phenotypically and functionally distinct conventional dendritic cell (cDC) subpopulations, termed cDC1 and cDC2, which each can be divided into several smaller and less well-characterized subsets. Despite advances in understanding the complexity of cDC ontogeny by transcriptional programming, the significance of posttranslational modifications in controlling tissue-specific cDC subset immunobiology remains elusive. Here, we identified the cell-surface–expressed A-disintegrin-and-metalloproteinase 10 (ADAM10) as an essential regulator of cDC1 and cDC2 homeostasis in the splenic marginal zone (MZ). Mice with a CD11c-specific deletion of ADAM10 (ADAM10(ΔCD11c)) exhibited a …

MaleLangerinLymphoid TissueNotch signaling pathwayAntigen-Presenting CellsCD11cSpleenADAM10 ProteinMicePhosphatidylinositol 3-KinasesmedicineAnimalsHomeostasisMyeloid CellsProtein kinase BPI3K/AKT/mTOR pathwayCell ProliferationMultidisciplinarybiologyMacrophagesMembrane ProteinsCell DifferentiationDendritic CellsBiological SciencesCD11c AntigenCell biologyMice Inbred C57BLmedicine.anatomical_structurebiology.proteinFemaleAmyloid Precursor Protein SecretasesSignal transductionProtein Processing Post-TranslationalSpleenConventional Dendritic CellSignal TransductionProceedings of the National Academy of Sciences
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Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Toxicological profile of cereulide, the Bacillus cereus emetic toxin, in functional assays with human, animal and bacterial cells

2007

International audience; Some strains of the endospore-forming bacterium Bacillus cereus produce a heat-stable ionophoric peptide, cereulide, of high human toxicity. We assessed cell toxicity of cereulide by measuring the toxicities of crude extracts of cereulide producing and non-producing strains of B. cereus, and of pure cereulide, using cells of human, animal and bacterial origins. Hepatic cell lines and boar sperm, with cytotoxicity and sperm motility, respectively, as the end points, were inhibited by <= 1 nM of cereulide present as B. cereus extract. RNA synthesis and cell proliferation in HepG2 cells was inhibited by 2 nM of cereulide. These toxic effects were explainable by the acti…

MaleLuminescenceSwineCytotoxicityBacillus cereusCYP1A1Toxicologymedicine.disease_causeHepa-1Ames testPotassium carrierchemistry.chemical_compoundMiceDepsipeptidesBioassayRNA Neoplasm0303 health sciencesbiologyMotilityAliivibrio fischeriSpermatozoaAmes testCereusBiochemistry[SDV.TOX]Life Sciences [q-bio]/ToxicologySperm MotilityBiological AssayERODBioluminescenceHepG2CereulideCell SurvivalBacterial ToxinsVibrio fischeriHEp-2Microbiology03 medical and health sciencesBacillus cereusCell Line TumorIonophoremedicineAnimalsHumansRNA synthesis030304 developmental biologyCell ProliferationDose-Response Relationship Drug030306 microbiologyToxinMutagenicity TestsfungiMicronucleus assayCereulidecomet test (SCG)biology.organism_classificationComet assaychemistryHepatocytesbacteriaBoar spermGenotoxicityGenotoxicity
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Roles of the Raf/MEK/ERK and PI3K/PTEN/Akt/mtor pathways in controlling growth and sensitivity to therapy-implications for cancer and aging

2011

Dysregulated signaling through the Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR pathways is often the result of genetic alterations in critical components in these pathways or upstream activators. Unrestricted cellular proliferation and decreased sensitivity to apoptotic-inducing agents are typically associated with activation of these pro-survival pathways. This review discusses the functions these pathways have in normal and neoplastic tissue growth and how they contribute to resistance to apoptotic stimuli. Crosstalk and commonly identified mutations that occur within these pathways that contribute to abnormal activation and cancer growth will also be addressed. Finally the recently described …

MaleMAPK/ERK pathwayAgingMAP Kinase Signaling SystemCancer aging RAF MEK mTORApoptosisReviewBiologyPI3KModels BiologicalApoptosis; Cancer; Kinases; MEK; MTOR; PI3K; Protein phosphorylation; RAF; Signal transductionMicePhosphatidylinositol 3-Kinases03 medical and health sciences0302 clinical medicineCancer stem cellNeoplasmscancerAnimalsHumansPTENProtein kinase BCellular SenescencePI3K/AKT/mTOR pathwayCell Proliferation030304 developmental biology0303 health sciencesKinaseTOR Serine-Threonine KinasesapoptosisPTEN PhosphohydrolaseRafCell BiologyMEKprotein phosphorylation3. Good healthCell biologyCrosstalk (biology)kinases030220 oncology & carcinogenesisMutationmTORCancer researchbiology.proteinFemaleraf KinasesProto-Oncogene Proteins c-aktCell agingsignal transduction
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Plasticity-related gene-1 inhibits lysophosphatidic acid-induced vascular smooth muscle cell migration and proliferation and prevents neointima forma…

2012

International audience; Plasticity-related gene-1 (PRG-1) protects neuronal cells from lysophosphatidic acid (LPA) effects. In vascular smooth muscle cells (VSMCs), LPA was shown to induce phenotypic modulation in vitro and vascular remodeling in vivo. Thus we explored the role of PRG-1 in modulating VSMC response to LPA. PCR, Western blot, and immunofluorescence experiments showed that PRG-1 is expressed in rat and human vascular media. PRG-1 expression was strongly inhibited in proliferating compared with quiescent VSMCs both in vitro and in vivo (medial vs. neointimal VSMCs), suggesting that PRG-1 expression is dependent on the cell phenotype. In vitro, adenovirus-mediated overexpression…

MaleMAPK/ERK pathwayNeointimaVascular smooth musclePhysiologyPhenotypic modulation[SDV]Life Sciences [q-bio]Genetic VectorsBiologyPlasticityMuscle Smooth VascularAdenoviridaechemistry.chemical_compoundCell MovementNeointimaLysophosphatidic acidAnimalsHumansRats WistarCells CulturedCell ProliferationCell BiologyLipid-phosphate phosphatasePhosphoric Monoester HydrolasesIn vitroRatsCell biologyGene Expression RegulationchemistryBiochemistryCalmodulin-Binding ProteinsLysophospholipidsAmerican Journal of Physiology-Cell Physiology
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MUC16 Is Overexpressed in Idiopathic Pulmonary Fibrosis and Induces Fibrotic Responses Mediated by Transforming Growth Factor-β1 Canonical Pathway

2021

Several transmembrane mucins have demonstrated that they contribute intracellularly to induce fibrotic processes. The extracellular domain of MUC16 is considered as a biomarker for disease progression and death in IPF patients. However, there is no evidence regarding the signalling capabilities of MUC16 that contribute to IPF development. Here, we demonstrate that MUC16 was overexpressed in the lung tissue of IPF patients (n = 20) compared with healthy subjects (n = 17) and localised in fibroblasts and hyperplastic alveolar type II cells. Repression of MUC16 expression by siRNA-MUC16 transfection inhibited the TGF-β1-induced fibrotic processes such as mesenchymal/ myofibroblast transformati…

MaleMUC16Gene ExpressionIdiopathic pulmonary fibrosis0302 clinical medicineBiology (General)PhosphorylationMyofibroblastsLungSpectroscopytransforming growth factor betabiologyChemistryGeneral MedicineTransfectionMiddle Agedrespiratory systemidiopathic pulmonary fibrosisImmunohistochemistryRespiratory Function TestsComputer Science ApplicationsChemistrymedicine.anatomical_structure030220 oncology & carcinogenesisFemaleDisease SusceptibilityMyofibroblastSignal TransductionQH301-705.5Models BiologicalArticleCatalysisCell LineTransforming Growth Factor beta1Inorganic Chemistry03 medical and health sciencesmedicineHumansPhysical and Theoretical ChemistryFibroblastQD1-999Molecular BiologyAgedCell ProliferationA549 cellOrganic ChemistryMesenchymal stem cellMembrane ProteinsTransforming growth factor betaFibroblastsmedicine.diseaserespiratory tract diseases030228 respiratory systemCA-125 AntigenCase-Control StudiesCancer researchbiology.proteinIdiopathic Pulmonary Fibrosis ; Muc16 ; Transforming Growth Factor BetaBiomarkersTransforming growth factorInternational Journal of Molecular Sciences
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Circulating CSF-1 Promotes Monocyte and Macrophage Phenotypes that Enhance Lupus Nephritis

2009

Macrophages mediate kidney disease and are prominent in a mouse model (MRL- Fas lpr ) of lupus nephritis. Colony stimulating factor-1 (CSF-1) is the primary growth factor for macrophages, and CSF-1 deficiency protects MRL- Fas lpr mice from kidney disease and systemic illness. Whether this renoprotection derives from a reduction of macrophages and whether systemic CSF-1, as opposed to intrarenal CSF-1, promotes macrophage-dependent lupus nephritis remain unclear. Here, we found that increasing systemic CSF-1 hastened the onset of lupus nephritis in MRL- Fas lpr mice. Using mutant MRL- Fas lpr strains that express high, moderate, or no systemic CSF-1, we detected a much higher tempo of kidne…

MaleMacrophage colony-stimulating factorMice Inbred MRL lprLupus nephritisMice TransgenicInflammationKidneyMonocytesMiceimmune system diseasesmedicineAnimalsHumansskin and connective tissue diseasesCell ProliferationInflammationMice Inbred BALB CMice Inbred C3HSystemic lupus erythematosusbiologyCD68business.industryMacrophage Colony-Stimulating FactorMacrophagesMonocyteGeneral MedicineMonocyte proliferationmedicine.diseaseLupus NephritisMice Inbred C57BLDisease Models AnimalBasic ResearchPhenotypemedicine.anatomical_structureIntegrin alpha MNephrologyImmunologybiology.proteinFemalemedicine.symptombusinessJournal of the American Society of Nephrology
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