Search results for "CHEMOPREVENTION"

showing 10 items of 49 documents

Post-initiation modulating effects of allyl sulfides in rat hepatocarcinogenesis

2004

Effects of administration of diallyl sulfide (DAS) and diallyl disulfide (DADS) on the promotion stage of hepatocarcinogenesis were investigated in rats using the Ito model. They were compared with those of phenobarbital (PB), a well-known liver promoter in rats. Initiation was induced by a single dose of N-nitrosodiethylamine (NDEA) and 3 weeks later, a partial hepatectomy was conducted. Two weeks after the NDEA injection, rats received either 0.05% allyl sulfides, PB or both in their diet for 8 weeks. Feeding with DAS increased the number of liver preneoplastic foci by 63% with respect to the untreated group. However, rats fed DAS showed a lower foci development than rats fed PB. The DADS…

Malemedicine.medical_treatment[SDV]Life Sciences [q-bio]Toxicologymedicine.disease_causechemistry.chemical_compound0302 clinical medicineLiver Neoplasms ExperimentalDiethylnitrosamineDrug InteractionsDisulfidesComputingMilieux_MISCELLANEOUS0303 health sciencesDiallyl disulfideGeneral MedicineCANCER3. Good healthSpecific Pathogen-Free OrganismsAllyl Compounds[SDV] Life Sciences [q-bio]BiochemistryDiallyl sulfide030220 oncology & carcinogenesismedicine.drugmedicine.medical_specialtySulfidesDIALLYL DISULFITEChemopreventionPreneoplastic foci03 medical and health sciencesInternal medicinemedicineAnimalsAnticarcinogenic AgentsHepatectomyDIALLYL SULFITERats Wistar030304 developmental biologyRatsEndocrinologychemistryRat liverCarcinogensRATPhenobarbitalHepatectomyCarcinogenesisAllyl SulfidePrecancerous ConditionsFood Science
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Risk factors for breakthrough invasive fungal infection during secondary prophylaxis.

2008

BACKGROUND: Intensive chemotherapy with severe neutropenia is associated with invasive fungal infections (IFIs) leading to high mortality rates. During leukaemia induction chemotherapy, IFI often prohibited further curative treatment, thus predisposing for leukaemia relapse. Continuing myelosuppressive chemotherapy after diagnosis of IFI has become feasible with the now expanding arsenal of safe and effective antifungals. Secondary prophylaxis of IFI is widely administered, but reliable data on outcome and risk factors for recurrent IFI during subsequent chemotherapy are not available. This study determines risk factors for recurrent IFI in leukaemia patients. METHODS: From 25 European canc…

Microbiology (medical)AdultMalemedicine.medical_specialtyAntifungal AgentsAdolescentNeutropeniaChemopreventionRecurrenceRisk FactorsInternal medicinemedicineHumansPharmacology (medical)Risk factorChildAir filterAgedPharmacologyAged 80 and overbusiness.industryInduction chemotherapyOdds ratioMiddle Agedmedicine.diseaseChemotherapy regimenSurgeryLeukemia Myeloid AcuteInfectious DiseasesLogistic ModelsTreatment OutcomeMycosesChild PreschoolChemoprophylaxisCytarabineFemalebusinessmedicine.drugThe Journal of antimicrobial chemotherapy
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Cytomegalovirus Infection Management in Allogeneic Stem Cell Transplant Recipients: a National Survey in Spain.

2015

ABSTRACT This study gathered information about current practices of cytomegalovirus (CMV) infection management in allogeneic stem cell transplant recipients at Spanish centers. A wide variety of preemptive antiviral therapy strategies for CMV infection guided by real-time PCR assays was found, yet the incidence of CMV disease was low (<3%).

Microbiology (medical)AdultPcr assayCongenital cytomegalovirus infectionReal-Time Polymerase Chain ReactionAntiviral AgentsChemopreventionVirologyMedicineHumansTransplantation Homologousbusiness.industryIncidence (epidemiology)Antiviral therapyvirus diseasesCase managementmedicine.diseaseCytomegalovirus infectionTransplantationMolecular Diagnostic TechniquesSpainImmunologyCytomegalovirus InfectionsStem cellbusinessCase ManagementStem Cell Transplantation
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Reduction of nevirapine-driven HIV mutations by carbamazepine is modulated by CYP3A activity

2014

Item does not contain fulltext OBJECTIVES: The reduction in mother-to-child transmission of HIV-1 by single-dose nevirapine given at birth onset is achieved at the expense of de novo HIV-1 resistance mutations. In the VITA1 study, single-dose carbamazepine accelerated nevirapine elimination, but the accompanying trend towards fewer de novo HIV-1 mutations was statistically non-significant. METHODS: We investigated if the effect of carbamazepine was confounded by the individual variability in nevirapine metabolism and transport. RESULTS: Nine of 34 (26%) single-dose nevirapine-treated women had one or more nevirapine-associated resistance mutations, compared with 3 of 34 (9%) in the single-d…

Microbiology (medical)NevirapineCYP3AAnti-HIV AgentsHuman immunodeficiency virus (HIV)Mutation MissenseEndogenyHIV InfectionsPharmacologyBiologymedicine.disease_causeChemopreventionPregnancyDrug Resistance ViralmedicineClinical endpointCytochrome P-450 CYP3AHumansPharmacology (medical)NevirapinePharmacologyMutationCYP3A4Cytochrome P-450 CYP3A InducersCarbamazepinelnfectious Diseases and Global Health Radboud Institute for Health Sciences [Radboudumc 4]Infectious DiseasesCarbamazepineTreatment OutcomeHIV-1Femalemedicine.drug
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Anti-16-kilodalton mycobacterial protein immunoglobulin M levels in healthy but purified protein derivative-reactive children decrease after chemopro…

2007

ABSTRACT Serum responses against Mycobacterium tuberculosis HSP16 were determined for children with tuberculosis (TB) and for healthy purified protein derivative (PPD)-positive and PPD-negative children. Immunoglobulin G (IgG) and IgM responses were higher for TB patients than for other groups. After chemotherapy, IgM and IgG responses decreased for TB patients and PPD-positive subjects. Monitoring of anti- M. tuberculosis HSP16 responses could assist in the management of pediatric TB.

Microbiology (medical)TuberculosisAdolescentChaperoninsmedicine.medical_treatmentClinical BiochemistryImmunologyAntitubercular AgentsTuberculinEnzyme-Linked Immunosorbent AssayTuberculinChemopreventionImmunoglobulin GMicrobiologyKilodaltonMycobacterium tuberculosisBacterial ProteinsmedicineHumansTuberculosisImmunology and AllergyChildChemotherapyMycobacterium tuberculosis IgMpurified protein derivative chemoprophylaxisbiologybusiness.industryClinical and Diagnostic Laboratory ImmunologyMycobacterium tuberculosisbiology.organism_classificationmedicine.diseaseImmunoglobulin MImmunoglobulin MChild PreschoolImmunoglobulin GImmunologyChemoprophylaxisbiology.proteinbusiness
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Lipid profiling following intake of the Omega 3 fatty acid DHA identifies the peroxidized metabolites F-4-Neuroprostanes as the best predictors of at…

2014

International audience; The anti-atherogenic effects of omega 3 fatty acids, namely eicosapentaenoic (EPA) and docosahexaenoic acids (DHA) are well recognized but the impact of dietary intake on bioactive lipid mediat or profiles remains unclear. Such a profiling effort may offer novel targets for future studies into the mechanism of action of omega 3 fatty acids. The present study aimed to determine the impact of DHA supplementation on the profiles of polyunsaturated fatty acids (PUFA) oxygenated metabolites and to investigate their contribution to therosclerosis prevention. A special emphasis was given to the non-enzymatic metabolites knowing the high susceptibility of DHA to free radical…

MouseBlood PressureCardiovascularBiochemistryMice0302 clinical medicineTandem Mass SpectrometryAetiologylcsh:Sciencechemistry.chemical_classificationLiquid0303 health sciences[CHIM.ORGA]Chemical Sciences/Organic chemistryFatty Acidsanti-atherogenic;omega 3 fatty acids;epa;dha;bioactive lipid;atherosclerosis;pufas;effects;molecularLipids3. Good healthFatty Acids UnsaturatedMedicinemedicine.medical_specialtyanti-atherogenicKnockoutAortic DiseasesMédecine humaine et pathologieGas Chromatography-Mass SpectrometryLDLDose-Response RelationshipLipid Mediators03 medical and health sciencesomega 3 fatty acids[ CHIM.ORGA ] Chemical Sciences/Organic chemistryacide gras n 3Complementary and Integrative HealtheffectsBiologybioactive lipidDose-Response Relationship DrugPreventionlcsh:RathéroscléroseEPALipid MetabolismPrevention of disease and conditionsmedicine.diseaseEndocrinologychemistryNeuroprostaneslcsh:QHuman health and pathologyBiomarkersand promotion of well-beinglcsh:Medicine030204 cardiovascular system & hematologymedicine.disease_causeOral and gastrointestinalHeart RateReceptorsBlood plasmaCluster Analysis2.1 Biological and endogenous factorsMice KnockoutUnsaturatedChromatographyMultidisciplinaryFatty liverAnimal ModelsDHALiverBiochemistryDocosahexaenoic acidlipids (amino acids peptides and proteins)DrugResearch ArticlePolyunsaturated fatty acidDocosahexaenoic AcidsClinical Research DesignGeneral Science & TechnologyBiologyModel OrganismsInternal medicinemedicineAnimalsNeuroprostanesAnimal Models of Diseasemolecular3.3 Nutrition and chemopreventionOmega 3 fatty acidNutrition030304 developmental biologyAnalysis of Varianceacide docosahexaénoiqueLipid metabolismAtherosclerosis[SDV.AEN] Life Sciences [q-bio]/Food and NutritionReceptors LDLPUFAs[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOxidative stressChromatography Liquid
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Effects of Oleuropein on colon cancer progression in vitro. A preliminary report

2019

Introduction. Accumulating evidence highlights that Oleuropein (OLE), one of the main bioactive phenolic compound present in olives, olive oil and olive leaves, appears to exert chemo-preventive effects against several human malignancies including gastrointestinal tumors. As the cellular mechanisms underlying this phenomenon are still not fully elucidated, we have undertaken some in vitro studies to examine the effects of OLE on the growth, adhesion and invasion of HTC116 and SW480 human colon cancer cells and the influence of this molecule on the production of certain proteins that appear to be relevant to cancer progression namely, matrix metalloproteinase-2 (MMP-2), matrix metalloprotein…

Oleuropeinnatural productColon CancerSettore BIO/14 - Farmacologiatumor progressionChemoprevention
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Vitamin D in cancer chemoprevention

2015

Context: There is increasing evidence that Vitamin D (Vit D) and its metabolites, besides their well-known calcium-related functions, may also exert antiproliferative, pro-differentiating, and immune modulatory effects on tumor cells in vitro and may also delay tumor growth in vivo. Objective: The aim of this review is to provide fresh insight into the most recent advances on the role of Vit D and its analogues as chemopreventive drugs in cancer therapy. Methods: A systematic review of experimental and clinical studies on Vit D and cancer was undertaken by using the major electronic health database including ISI Web of Science, Medline, PubMed, Scopus and Google Scholar. Results and conclus…

OncologyMalemedicine.medical_specialtyColorectal cancerPharmaceutical ScienceAntineoplastic AgentsBreast NeoplasmsPharmacologyChemopreventionMalignant transformationProstate cancerImmune systemBreast cancerInternal medicineNeoplasmsDrug DiscoverymedicineVitamin D and neurologyAnimalsHumansImmunologic FactorsVitamin DPharmacologyClinical Trials as TopicCancer preventionbusiness.industryCancerProstatic NeoplasmsGeneral Medicinemedicine.diseaseCancer cancer prevention calcitriol vitamin D vitamin D analoguesGrowth InhibitorsComplementary and alternative medicineSettore BIO/14 - FarmacologiaMolecular MedicineFemalebusinessColorectal Neoplasms
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Does chemotherapy prevent HCV-related hepatocellular carcinoma? Cons.

2010

The accuracy and the reliability of well-recognized clinical, virologic, histologic, and molecular risk factors for hepatocellular carcinoma (HCC) are still insufficient. Thus, accurate risk prediction of cancer development in individual patients with the aim of selecting high risk cohorts of patients for HCC chemoprevention programs remains an elusive goal. Future directions in chemoprevention of HCC will be in the development of molecular risk models and of new chemopreventive agents. Studies examining multiple genes and proteins (genomics and proteomics) in the same HCCs will be required to evaluate this possibility thoroughly. A strategy aiming at preventing chronic liver disease of any…

Oncologymedicine.medical_specialtyCirrhosisCarcinoma Hepatocellularmedicine.medical_treatmentInterferon alpha-2Chronic liver diseaseAntiviral AgentsChemopreventionlaw.inventionPolyethylene GlycolsRandomized controlled triallawInternal medicinemedicineHumansHCCChemotherapyHepatologybusiness.industryIncidence (epidemiology)Patient SelectionLiver NeoplasmsGastroenterologyInterferon-alphaHepatitis C Chronicmedicine.diseasedigestive system diseasesRecombinant ProteinsHepatocellular carcinomaImmunologyEtiologyCancer developmentbusinessDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
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From clinical description, to in vitro and animal studies, and backward to patients: Oxidative stress and mitochondrial dysfunction in Fanconi anemia

2013

Fanconi anemia (FA) is a rare genetic disease associated with deficiencies in DNA repair pathways. A body of literature points to a pro-oxidant state in FA patients, along with evidence for oxidative stress (OS) in the FA phenotype reported by in vitro, molecular, and animal studies. A highlight arises from the detection of mitochondrial dysfunction (MDF) in FA cell lines of complementation groups A, C, D2, and G. As yet lacking, in vivo studies should focus on FA-associated MDF, which may help in the understanding of the mitochondrial basis of OS detected in cells and body fluids from FA patients. Beyond the in vitro and animal databases, the available analytical devices may prompt the dir…

Pathologymedicine.medical_specialtyDNA RepairFree RadicalsDNA repairmitochondrial nutrientsCell Cycle ProteinsFree radicalsDiseaseBiologymedicine.disease_causeBioinformaticsBiochemistryChemopreventionPathogenesis03 medical and health sciencesMice0302 clinical medicineIn vivoFanconi anemiaPhysiology (medical)medicineAnimalsHumans030304 developmental biology0303 health sciencesMitochondrial nutrientNuclear ProteinsFanconi anemia Mitochondrial dysfunction Mitochondrial nutrients Chemoprevention Free radicalsmedicine.diseasePhenotype3. Good healthMitochondriaOxidative StressFanconi Anemia030220 oncology & carcinogenesisFanconi anemiaAnimal studiesReactive Oxygen SpeciesMitochondrial dysfunctionOxidative stress
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