Search results for "CHON"

showing 10 items of 1866 documents

Cytotoxic Compounds from the Fruits of Uapaca togoensis towards Multifactorial Drug-Resistant Cancer Cells

2014

Cancer cells may rapidly acquire multidrug resistance, mainly due to the presence of adenosine triphosphate-binding cassette transporters, epidermal growth factor receptor, or mutations in the p53 tumor suppressor gene. This work was designed to assess the cytotoxicity of the methanol crude extracts and compounds from the fruits of Uapaca togoensis, namely, β-amyryl acetate (1), 11-oxo-α-amyryl acetate (2), lupeol (3), pomolic acid (4), futokadsurin B (5), arborinin (6), and 3-O-β-D-glucopyranosyl sitosterol (7) against nine drug sensitive and multidrug-resistant cancer cell lines. The resazurin reduction assay was used to evaluate the cytotoxicity of the fruits of U. togoensis and compound…

Pharmaceutical ScienceBiologyLignansAnalytical ChemistryInhibitory Concentration 50chemistry.chemical_compoundCell Line TumorDrug DiscoveryHumansCytotoxic T cellOleanolic AcidCytotoxicityLupeolMembrane Potential MitochondrialPharmacologyMolecular StructurePlant ExtractsAlkaloidOrganic ChemistryEuphorbiaceaeCell cycleAntineoplastic Agents PhytogenicDrug Resistance MultipleTriterpenesComplementary and alternative medicinechemistryBiochemistryDoxorubicinDrug Resistance NeoplasmApoptosisCell cultureCancer cellAcridinesMolecular MedicineDrug Screening Assays AntitumorPentacyclic TriterpenesReactive Oxygen SpeciesPlanta Medica
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Effects of malvidin, cyanidin and delphinidin on human adipose mesenchymal stem cell differentiation into adipocytes, chondrocytes and osteocytes.

2019

Abstract Background Anthocyanidins are plant phytochemicals found at high concentrations in berries, vegetables and flowers. Anthocyanidins have been extensively investigated due to their antioxidative, antidiabetic and anti-inflammatory effects. Few studies show that anthocyanidins decrease obesity and improve bone density. However, the effects of anthocyanidins on tissue regeneration have not been sufficiently clarified. Human mesenchymal stem cells (MSCs) are multipotent adult stem cells responsible for the regeneration of fat, bone and cartilage. Although MSCs are often used for screening of biologically active compounds, so far, the effect of anthocyanidins on MSC differentiation has n…

Pharmaceutical ScienceOsteocytesAnthocyanins03 medical and health scienceschemistry.chemical_compound0302 clinical medicineChondrocytesOsteogenesisDrug DiscoveryAdipocytesHumansAggrecansCells Cultured030304 developmental biologyAnthocyanidinPharmacology0303 health sciencesAdipogenesisMesenchymal stem cellfood and beveragesCell DifferentiationMesenchymal Stem CellsChondrogenesisMalvidinCell biologyAnthocyanidinsComplementary and alternative medicinechemistryAdipose TissueGene Expression RegulationAdipogenesis030220 oncology & carcinogenesisMolecular MedicineMesenchymal stem cell differentiationAnti-Obesity AgentsDelphinidinChondrogenesisPhytomedicine : international journal of phytotherapy and phytopharmacology
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A new class of organic nitrates: investigations on bioactivation, tolerance and cross-tolerance phenomena

2009

Background and purpose:  The chronic use of organic nitrates is limited by serious side effects including oxidative stress, nitrate tolerance and/or endothelial dysfunction. The side effects and potency of nitroglycerine depend on mitochondrial aldehyde dehydrogenase (ALDH-2). We sought to determine whether this concept can be extended to a new class of organic nitrates with amino moieties (aminoalkyl nitrates). Experimental approach:  Vasodilator potency of the organic nitrates, in vitro tolerance and in vivo tolerance (after continuous infusion for 3 days) were assessed in wild-type and ALDH-2 knockout mice by isometric tension studies. Mitochondrial oxidative stress was analysed by L-012…

PharmacologyChemistryMetabolismMitochondrionPharmacologymedicine.disease_causeNitric oxideCross-tolerancechemistry.chemical_compoundBiochemistryIn vivomedicinePotencyAcetylcholineOxidative stressmedicine.drugBritish Journal of Pharmacology
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Mitochondrial Toxicity in HAART: An Overview of In Vitro Evidence

2011

The combined antiretroviral therapeutic approach currently employed for the treatment of HIV infection, known as Higly Active Antiretroviral Therapy (HAART), has dramatically reduced AIDS-related morbidity and mortality. However, the adverse reactions associated with the long term use of this therapy have now become a major issue and researchers have focused on understanding the cellular mechanisms underlying these drug-induced detrimental effects which englobe a large list of different events including rash and hypersensibility reactions, hepatotoxicity, metabolic disturbances including lipodystrophy, and other metabolic syndrome-like disturbances such as hyperlactatemia, hyperlipedimia, i…

PharmacologyMitochondrial DNAAnti-HIV AgentsMitochondrionBiologyPharmacologymedicine.diseaseDNA MitochondrialReverse transcriptaseMitochondriaMitochondrial toxicityInsulin resistancePharmacotherapyAntiretroviral Therapy Highly ActiveDrug DiscoverymedicineHumansDrug Therapy CombinationHyperlactatemiaLipodystrophyCurrent Pharmaceutical Design
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Mitochondrial biogenesis in health and disease. Molecular and therapeutic approaches.

2014

Mitochondrial biogenesis (MB) is the essential mechanism by which cells control the number of mitochondria. Cells respond to different physiologic, metabolic, and pathologic changes by regulating this organelle with high morphological and functional adaptability. A considerable number of proteins, transcription factors, upstream regulatory proteins and secondary mechanisms are involved in MB and the stabilization of new mitochondrial DNA. These MB activators and regulators, including the main participating proteins (e.g. PGC-1α and mtTFA), are candidates for therapeutic intervention in diverse diseases, like neurodegenerative disorders, metabolic syndrome, sarcopenia, cardiac pathophysiolo…

PharmacologyMitochondrial DNAMitochondrial DiseasesMechanism (biology)Health StatusDiseaseBiologyTFAMMitochondrionBioinformaticsmedicine.diseaseCell biologyMitochondriaMitochondrial biogenesisSarcopeniaDrug DiscoverymedicineAnimalsHumansEnergy MetabolismTranscription factorSignal TransductionTranscription FactorsCurrent pharmaceutical design
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Enhanced oxidative stress and increased mitochondrial mass during Efavirenz-induced apoptosis in human hepatic cells

2010

BACKGROUND AND PURPOSE Efavirenz (EFV) is widely used in the treatment of HIV-1 infection. Though highly efficient, there is growing concern about EFV-related side effects, the molecular basis of which remains elusive. EXPERIMENTAL APPROACH In vitro studies were performed to address the effect of clinically relevant concentrations of EFV (10, 25 and 50 µM) on human hepatic cells. KEY RESULTS Cellular proliferation and viability were reduced in a concentration-dependent manner. Analyses of the cell cycle and several cell death parameters (chromatin condensation, phosphatidylserine exteriorization, mitochondrial proapoptotic protein translocation and caspase activation) revealed that EFV trig…

PharmacologyMitochondrial DNAProgrammed cell deathMitochondrionBiologymedicine.diseasemedicine.disease_causeCell biologyMitochondrial toxicitychemistry.chemical_compoundchemistryBiochemistryApoptosismedicineCardiolipinOxidative stressMitochondrial DNA replicationBritish Journal of Pharmacology
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Mitochondrial function and energy metabolism in neuronal HT22 cells resistant to oxidative stress

2014

Background and Purpose The hippocampal cell line HT22 is an excellent model for studying the consequences of endogenous oxidative stress. Extracellular glutamate depletes cellular glutathione by blocking the glutamate/cystine antiporter system xc−. Glutathione depletion induces a well-defined programme of cell death characterized by an increase in reactive oxygen species and mitochondrial dysfunction. Experimental Approach We compared the mitochondrial shape, the abundance of mitochondrial complexes and the mitochondrial respiration of HT22 cells, selected based on their resistance to glutamate, with those of the glutamate-sensitive parental cell line. Key Results Glutamate-resistant mitoch…

PharmacologyOligomycinATP synthaseCellular respirationOxidative phosphorylationMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundMitochondrial permeability transition poreBiochemistrychemistrymedicinebiology.proteinATP–ADP translocaseOxidative stressBritish Journal of Pharmacology
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Antigen-distribution in rat liver mitochondria.

1962

I mitocondri di fegato di ratto possiedono almeno tre gruppi di antigeni, dei quali uno e esclusivamente legato alle membrane mitocondriali, uno e presente nella frazione solubile in 0,3% desossicolato di sodio ed un altro, che, sebbene principalmente localizzato nelle membrane, e anche dimostrabile nella frazione desossicolato solubile.

PharmacologyRat liver mitochondriaMitochondria LiverCell BiologyMitochondrionBiologyMitochondriaRatsCellular and Molecular NeuroscienceAntigenBiochemistryLiverMolecular MedicineDistribution (pharmacology)AnimalsAntigensMolecular BiologyExperientia
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Beneficial effects of l-carnitine in myoblastic C2C12 cells

2003

L-Carnitine is a key molecule in the transfer of fatty acid across mitochondrial membranes. Bioavailable L-carnitine is either provided by an endogeneous biosynthesis or after intestinal absorption of dietary items containing L-carnitine. After intestinal absorption or hepatic biosynthesis, L-carnitine is transferred to organs whose metabolism is dependent upon fatty acid oxidation, such as skeletal muscle. To cross the muscle plasma membrane, there are several transporters involved. Among those transporters, OCTN2 is actually the only one to have been clearly characterized. Zidovudine is a commonly used inhibitor of human immunodeficiency virus (HIV) replication. Zidovudine has many side e…

PharmacologySkeletal muscleBiologyMitochondrionPharmacologyBiochemistryIntestinal absorptionZidovudinemedicine.anatomical_structureBiochemistrymedicineMyocyteCarnitinemedicine.symptomMyopathyBeta oxidationmedicine.drugBiochemical Pharmacology
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Mitochondrial (dys)function - a factor underlying the variability of efavirenz-induced hepatotoxicity?

2015

Background and Purpose The non-nucleoside analogue reverse transcriptase inhibitor efavirenz is associated with hepatic toxicity and metabolic disturbances. Although the mechanisms involved are not clear, recent evidence has pinpointed a specific mitochondrial action of efavirenz accompanied by the induction of an endoplasmic reticulum (ER) stress/unfolded protein response in human hepatic cells. The aim of this study was to further investigate the involvement of this organelle by evaluating efavirenz's effects in cells lacking functional mitochondria (rho°) and comparing them with those of the typical mitotoxic agent rotenone, a standard complex I inhibitor, and the ER stress inducer thaps…

PharmacologyThapsigarginEfavirenzReverse-transcriptase inhibitorEndoplasmic reticulumRotenoneBiologyMitochondrionPharmacologychemistry.chemical_compoundchemistryUnfolded protein responseHepatic stellate cellmedicinemedicine.drugBritish Journal of Pharmacology
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