Search results for "CHROMOSOME"

showing 10 items of 1175 documents

WHO-defined ‘myelodysplastic syndrome with isolated del(5q)’ in 88 consecutive patients: survival data, leukemic transformation rates and prevalence …

2010

The 2008 World Health Organization (WHO) criteria were used to identify 88 consecutive Mayo Clinic patients with 'myelodysplastic syndrome with isolated del(5q)' (median age 74 years; 60 females). In all, 60 (68%) patients were followed up to the time of their death. Overall median survival was 66 months; leukemic transformation was documented in five (5.7%) cases. Multivariable analysis identified age >or=70 years (P=0.01), transfusion need at diagnosis (P=0.04) and dysgranulopoiesis (P=0.02) as independent predictors of shortened survival; the presence of zero (low risk), one (intermediate risk) or >or=2 (high risk) risk factors corresponded to median survivals of 102, 52 and 27 months, r…

AdultMaleCancer Researchmedicine.medical_specialtyIDH1Biology5q-World Health OrganizationPolymerase Chain ReactionGastroenterologyIDH2ironInternal medicineMyelodysplastic Syndrome with Isolated del(5q)medicineHumansSurvival rateAgedAged 80 and overThrombopoietin receptorHematologyMyelodysplastic syndromesferritinHematologyJanus Kinase 2Middle AgedPrognosismedicine.diseaseIsocitrate DehydrogenaseSurvival RateLeukemiaCell Transformation NeoplasticOncologyMyelodysplastic SyndromesMutationImmunologyChromosomes Human Pair 5Original ArticleFemaleChromosome DeletionReceptors ThrombopoietinLeukemia
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Cytogenetic findings in secondary acute nonlymphocytic leukemia

1992

Abstract We here report the results of cytogenetic studies carried out in eight patients with acute nonlymphocytic leukemia developed after primary neoplasias. In seven of the reported cases, clonal chromosome aberrations were found, some being specific of de novo acute nonlymphocytic leukemia (ANLL). Numerical abnormalities were detected, such as the total monosomy of chromosomes 5, 7, 21, trisomy of chromosomes 8, 11, 15, and duplication of chromosome Y. Structural changes were also observed: a del(12)(p12), a del(16)(q22), the translocations t(3;5)(p21;q35),t(3;7)(p21;q35), and t(12;14)(p12;q32) and other changes involving chromosome 8. The finding of a hypertetraploid karyotype with com…

AdultMaleCancer Researchmedicine.medical_specialtyMonosomyChromosomal translocationBiologyTranslocation GeneticPolyploidyMonosomyhemic and lymphatic diseasesGeneticsmedicineHumansMolecular BiologyAgedChromosome AberrationsCytogeneticsChromosomeNeoplasms Second PrimaryKaryotypeMiddle Agedmedicine.diseaseLymphomaLeukemia Myeloid AcuteLeukemiaImmunologyCancer researchChromosomes Human Pair 5FemaleTrisomyChromosomes Human Pair 7Cancer Genetics and Cytogenetics
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Chromosomal abnormalities in Waldenström's macroglobulinemia

1992

We report the results of cytogenetic studies of direct bone marrow (BM) preparations and of short-term BM and peripheral blood (PB) cultures from 17 patients with Waldenström's macroglobulinemia. We noted clonal chromosome changes in 10 patients. Abnormalities affected chromosomes X, Y, 2, 4, 5, 15, 16, 18, 19, 20, 21, and 22; in particular, chromosomes 2, 4, and 5 were involved in structural changes: a homogeneously staining region [hsr(2)], a der(4)t(4;?)(q32;?), and a 5q+. The other chromosomes were involved in numerical abnormalities, such as pseudodiploidy (a 46,X,-X,+15 clone), loss of chromosome Y, and monosomy of chromosomes 16, 18, 19, 20, 21, and 22. Nonclonal chromosome rearrange…

AdultMaleCancer Researchmedicine.medical_specialtyMonosomyClone (cell biology)Chromosome rearrangements Waldenström's macroglobulinemiaBiologyGeneticsmedicineHumanseducationMolecular BiologyHomogeneously Staining RegionAgedGeneticsAged 80 and overChromosome Aberrationseducation.field_of_studyCytogeneticsMacroglobulinemiaWaldenstrom macroglobulinemiaChromosomeKaryotypeMiddle Agedmedicine.diseaseMolecular biologySettore BIO/18 - GeneticaChromosomes Human Pair 2FemaleWaldenstrom Macroglobulinemia
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The 2q37-deletion syndrome: an update of the clinical spectrum including overweight, brachydactyly and behavioural features in 14 new patients

2012

International audience; The 2q37 locus is one of the most commonly deleted subtelomeric regions. Such a deletion has been identified in >100 patients by telomeric fluorescence in situ hybridization (FISH) analysis and, less frequently, by array-based comparative genomic hybridization (array-CGH). A recognizable ‘2q37-deletion syndrome’ or Albright’s hereditary osteodystrophy-like syndrome has been previously described. To better map the deletion and further refine this deletional syndrome, we formed a collaboration with the Association of French Language Cytogeneticists to collect 14 new intellectually deficient patients with a distal or interstitial 2q37 deletion characterized by FISH and …

AdultMaleCandidate geneAdolescentDNA Copy Number Variations[SDV]Life Sciences [q-bio]Chromosome DisordersLocus (genetics)BiologyFibrous Dysplasia PolyostoticBioinformaticsArticleYoung Adult03 medical and health sciences0302 clinical medicineIntellectual DisabilityGeneticsmedicineHumansChildGenetic Association StudiesGenetics (clinical)030304 developmental biologyKIF1AGeneticsBehaviorComparative Genomic Hybridization0303 health sciences[ SDV ] Life Sciences [q-bio]medicine.diagnostic_testBrachydactylyBrachydactylyChromosome MappingOverweightSubtelomeremedicine.disease[SDV] Life Sciences [q-bio]Child PreschoolChromosomes Human Pair 2AutismFemaleChromosome Deletion030217 neurology & neurosurgeryComparative genomic hybridizationFluorescence in situ hybridizationEuropean Journal of Human Genetics
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Structure, chromosomal localization, and brain expression of human Cx36 gene

1999

Rat connexin-36 (Cx36) is the first gap junction protein shown to be expressed predominantly in neuronal cells of the mammalian central nervous system. As a prerequisite for studies devoted to the investigation of the possible role of this connexin in human neurological diseases, we report the cloning and sequencing of the human Cx36 gene, its chromosomal localization, and its pattern of expression in the human brain analyzed by radioactive in situ hybridization. The determination of the human gene sequence revealed that the coding sequence of Cx36 is highly conserved (98% identity at the protein level with the mouse and rat Cx36 and 80% with the ortholog perch and skate Cx35), and that the…

AdultMaleCandidate geneAdolescentgenetic structuresMolecular Sequence DataIn situ hybridizationBiologyHippocampal formationPolymerase Chain ReactionConnexinsMiceCellular and Molecular NeurosciencemedicineAnimalsHumansCoding regionAmino Acid SequenceSkates FishCloning MolecularEye ProteinsPeptide Chain Initiation TranslationalGeneIn Situ Hybridization FluorescenceChromosomes Human Pair 15Genomic LibrarySequence Homology Amino Acidmedicine.diagnostic_testBrainChromosome MappingHuman brainMiddle AgedMolecular biologyIntronsRatsmedicine.anatomical_structureSpinal CordOrgan SpecificityPerchesCerebellar cortexFemalesense organsSequence AlignmentFluorescence in situ hybridizationJournal of Neuroscience Research
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Genetics and Beyond – The Transcriptome of Human Monocytes and Disease Susceptibility

2010

BACKGROUND: Variability of gene expression in human may link gene sequence variability and phenotypes; however, non-genetic variations, alone or in combination with genetics, may also influence expression traits and have a critical role in physiological and disease processes. METHODOLOGY/PRINCIPAL FINDINGS: To get better insight into the overall variability of gene expression, we assessed the transcriptome of circulating monocytes, a key cell involved in immunity-related diseases and atherosclerosis, in 1,490 unrelated individuals and investigated its association with >675,000 SNPs and 10 common cardiovascular risk factors. Out of 12,808 expressed genes, 2,745 expression quantitative trait …

AdultMaleChromosomes Human Pair 21Cardiovascular DisordersQuantitative Trait Locilcsh:MedicineGenome-wide association studyGenetics and Genomics/Complex TraitsBiologyPolymorphism Single NucleotideMonocytesTranscriptomeQuantitative Trait HeritableCell MovementRisk FactorsHumansGenetic Predisposition to DiseaseGenetics and Genomics/GenomicsAllelelcsh:ScienceGeneAgedGeneticsRegulation of gene expressionMultidisciplinaryBase SequenceGenome HumanGene Expression ProfilingSmokinglcsh:RImmunityGenetic VariationGenetics and GenomicsGenetics and Genomics/Gene ExpressionMiddle AgedAtherosclerosisPhenotypeHuman geneticsGene expression profilingPhenotypeGene Expression RegulationCardiovascular and Metabolic DiseasesFemalelcsh:QDNA ProbesGenome-Wide Association StudyResearch ArticlePLoS ONE
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Microduplications at 22q11.21 are associated with non-syndromic classic bladder exstrophy

2009

The exstrophy-epispadias complex (EEC) comprises a spectrum of urogenital anomalies in which part or all of the distal urinary tract fails to close. The present study aimed to identify microaberrations characterized by loss or gain of genomic material that contribute to the EEC at a genome-wide level. Molecular karyotyping, utilizing 549,839 single nucleotide polymorphisms (SNPs) with an average spacing of 5.7 kilobases, was performed to screen an initial cohort of 16 patients with non-syndromic EEC. A de novo microduplication involving chromosomal region 22q11.21 was identified in one patient with classic exstrophy of the bladder (CBE). Subsequent multiplex ligation-dependent probe amplifi…

AdultMaleChromosomes Human Pair 22MedizinMolecular Probe TechniquesSingle-nucleotide polymorphismBiologyBioinformaticsPolymorphism Single NucleotideChromosomesGene DuplicationDiGeorge syndromeGene duplicationGeneticsmedicineHumansGenetic Predisposition to DiseaseMultiplex ligation-dependent probe amplificationChildGenetics (clinical)GeneticsGene Expression ProfilingBladder ExstrophyGeneral Medicinemedicine.diseasePenetranceBladder exstrophyPhenotypeKaryotypingChromosomal regionFemaleSNP arrayEuropean Journal of Medical Genetics
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Biodosimetry Based on γ-H2AX Quantification and Cytogenetics after Partial- and Total-Body Irradiation during Fractionated Radiotherapy

2015

The aim of this current study was to quantitatively describe radiation-induced DNA damage and its distribution in leukocytes of cancer patients after fractionated partial- or total-body radiotherapy. Specifically, the impact of exposed anatomic region and administered dose was investigated in breast and prostate cancer patients receiving partial-body radiotherapy. DNA double-strand breaks (DSBs) were quantified by γ-H2AX immunostaining. The frequency of unstable chromosomal aberrations in stimulated lymphocytes was also determined and compared with the frequency of DNA DSBs in the same samples. The frequency of radiation-induced DNA damage was converted into dose, using ex vivo generated ca…

AdultMaleDNA damagemedicine.medical_treatmentBiophysicsBiologyRadiation ToleranceHistonesYoung AdultProstate cancerBiodosimetryNeoplasmsmedicineHumansRadiology Nuclear Medicine and imagingLymphocytesRadiation InjuriesRadiometryAgedAged 80 and overChromosome AberrationsRadiationbusiness.industryRadiotherapy Planning Computer-AssistedCancerDose-Response Relationship RadiationMiddle AgedTotal body irradiationmedicine.diseaseRadiation therapyCalibrationCytogenetic AnalysisFemaleDose Fractionation RadiationNuclear medicinebusinessWhole-Body IrradiationImmunostainingEx vivoDNA DamageRadiation Research
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Screening of subtelomeric rearrangements in autistic disorder: identification of a partial trisomy of 13q34 in a patient bearing a 13q;21p translocat…

2006

Within the framework of a FISH screening protocol to detect cryptic subtelomeric rearrangements in autistic disorder (AD), a patient bearing three copies of the subtelomeric portion of the q arm of chromosome 13 has been identified. Beside AD, the patient also has severe mental retardation and displays several dysmorphic features. Further FISH analyses revealed that the trisomy was caused by the translocation of a 13q subtelomeric fragment to the acrocentric tip of one chromosome 21 [46,XY.ish der(21) t(13;21) (q34;p13)(D13S1825+)]. Gene dosage experiments carried out with three multiallelic polymorphisms of the subtelomeric region of chromosome 13q showed that the putative length of the tr…

AdultMaleDerivative chromosomeAdolescentGene DosageautismChromosomal translocationTrisomyBiologyGene dosagePolymerase Chain ReactionTranslocation GeneticCellular and Molecular NeurosciencemedicineHumansAutistic DisorderChildGenetics (clinical)In Situ Hybridization FluorescenceChromosome 13GeneticsChromosomes Human Pair 13ChromosomeTelomereSubtelomeremedicine.diseasePsychiatry and Mental healthfrontal bossingFemaleTrisomyChromosome 21American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics
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Genome-wide analysis for micro-aberrations in familial exstrophy of the bladder using array-based comparative genomic hybridization

2007

OBJECTIVE: Exstrophy of the bladder (EB) is part of the bladder-exstrophy-epispadias complex (BEEC). Because familial occurrence of BEEC is rare, exogenous factors are thought to play a major role in the etiology of most BEEC cases. We aimed to investigate a possible genetic basis of BEEC in a consanguineous kindred of Moroccan origin with three members showing the same phenotypic expression of BEEC. PATIENTS AND METHODS: The three affected males (two cousins and their maternal uncle) all presenting with nonsyndromic classic EB, were born in Morocco or The Netherlands. One Moroccan patient had an open bladder surface for 22 years due to late surgical reconstruction, avoided upright posture …

AdultMaleEpispadiasAdolescentUrologyClone (cell biology)GenomeMedicineHumansAbnormalities MultipleGenetic Predisposition to DiseaseGeneIn Situ Hybridization FluorescenceOligonucleotide Array Sequence AnalysisGeneticsChromosome AberrationsGenomebusiness.industryBladder ExstrophyNucleic Acid HybridizationKaryotypeDNAmedicine.diseasePhenotypePedigreeBladder exstrophyMoroccoEtiologybusinessComparative genomic hybridizationBJU International
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