Search results for "CIDI"

showing 10 items of 778 documents

Caution on the assessment of intestinal parasitic load in studying parasite-mediated sexual selection: the case of Blackbirds coccidiosis.

2009

6 pages; International audience; The parasite-mediated sexual selection (PMSS) theory has led to an increasing number of experimental studies essentially focussed on blood parasites. Currently, more research is being carried out on intestinal parasites in relationship to this theory. Before testing the theory with gastrointestinal parasites, it is important: (i) to determine an optimal research methodology to obtain an accurate assessment of parasite burden and (ii) to have information about life-history traits of the parasite to interpret data appropriately. In this study, we present data on oocyst output of Isosporaturdi in the faeces of blackbirds (Turdus merula) that illustrate the impo…

Male0106 biological sciences[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyIsosporiasisZoologyParasite-mediated sexual selection010603 evolutionary biology01 natural sciences030308 mycology & parasitologySongbirdsFeces03 medical and health sciencesCoccidiaParasite Egg Countmedicine[ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisAnimalsParasite hosting[SDV.MP.PAR]Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyTurdus merulaParasite Egg CountParasitic load0303 health sciences[ SDE.BE ] Environmental Sciences/Biodiversity and EcologyParasitic loadIsosporabiologyBird Diseases[SDV.BID.EVO]Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]Isosporiasismedicine.diseasebiology.organism_classification3. Good healthCoccidiaTemporal variationIsospora turdiIntestinesCoccidiosisInfectious Diseases[ SDV.BID.EVO ] Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]ParasitologySexual selectionBlackbirdsImmunologyFemaleParasitology[SDE.BE]Environmental Sciences/Biodiversity and Ecology[SDV.EE.IEO]Life Sciences [q-bio]/Ecology environment/Symbiosis
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Bi- and uniciliated ependymal cells define continuous floor-plate-derived tanycytic territories

2017

Multiciliated ependymal (E1) cells line the brain ventricles and are essential for brain homeostasis. We previously identified in the lateral ventricles a rare ependymal subpopulation (E2) with only two cilia and unique basal bodies. Here we show that E2 cells form a distinct biciliated epithelium extending along the ventral third into the fourth ventricle. In the third ventricle floor, apical profiles with only primary cilia define an additional uniciliated (E3) epithelium. E2 and E3 cells' ultrastructure, marker expression and basal processes indicate that they correspond to subtypes of tanycytes. Using sonic hedgehog lineage tracing, we show that the third and fourth ventricle E2 and E3 …

Male0301 basic medicineEpendymal CellScienceEpendymoglial CellsGene ExpressionGeneral Physics and AstronomyMice TransgenicS100 Calcium Binding Protein beta SubunitFourth ventricleArticleGeneral Biochemistry Genetics and Molecular BiologyNestinMice03 medical and health sciencesLateral ventriclesEpendymaGlial Fibrillary Acidic ProteinmedicineAnimalsHumansVimentinCell LineageHedgehog ProteinsCiliaSonic hedgehogAgedBrain VentricleFloor plateBrain MappingMultidisciplinaryThird ventriclebiologyQCD24 AntigenCell DifferentiationGeneral ChemistryAnatomyMiddle Aged030104 developmental biologymedicine.anatomical_structureCell Trackingbiology.proteinFemaleNerve NetEpendymaBiomarkersNature Communications
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Comparative Study of the Effects Exerted by N-Valproyl-L-Phenylalanine and N-valproyl-L-tryptophan on CA1 Hippocampal Epileptiform Activity in Rat

2018

Background: The research on the improvement of epilepsy therapy is constantly growing. Valproyl-LPhenylalanine (VPA-Phen) and N-valproyl-L-tryptophan (VPA-Tryp) were synthesized to increase the antiepileptic efficacy of valproic acid. Methods: VPA-Phen and VPA-Tryp were comparatively tested on CA1 hippocampal epileptiform bursting activity obtained by increasing potassium and lowering calcium and magnesium concentrations in the fluid perfusing rat brain slices. Each slice was treated with a single concentration (0.2, 0.5, 1 mM) of VPA-Phen or VPA-Tryp. Both burst duration and interburst frequency, during and after treatment, were off-line compared with baseline values. For both parameters,…

Male0301 basic medicinePhenylalaninePotassiumchemistry.chemical_elementPharmacologyHippocampal formationCalciumInhibitory postsynaptic potentialHippocampusSettore BIO/09 - Fisiologia03 medical and health sciencesantiepileptic drug0302 clinical medicineDrug DiscoveryN-valproyl-L-tryptophanvalproic acid.medicineAnimalshippocampal epilepsyRats WistarPharmacologyValproic AcidEpilepsyValproyl-L-Phenylalanine (VPA-Phen)Dipeptidesinterictal burstRat brainAmino-acidic derivativeRats030104 developmental biologychemistryAnticonvulsantslipids (amino acids peptides and proteins)030217 neurology & neurosurgerymedicine.drugN-valproyl-L-phenylalanineCurrent Pharmaceutical Design
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Fungal Dysbiosis and Intestinal Inflammation in Children With Beta-Cell Autoimmunity

2020

Although gut bacterial dysbiosis is recognized as a regulator of beta-cell autoimmunity, no data is available on fungal dysbiosis in the children at the risk of type 1 diabetes (T1D). We hypothesized that the co-occurrence of fungal and bacterial dysbiosis contributes to the intestinal inflammation and autoimmune destruction of insulin-producing beta-cells in T1D. Fecal and blood samples were collected from 26 children tested positive for at least one diabetes-associated autoantibody (IAA, GADA, IA-2A or ICA) and matched autoantibody-negative children with HLA-conferred susceptibility to T1D (matched for HLA-DQB1 haplotype, age, gender and early childhood nutrition). Bacterial 16S and funga…

Male0301 basic medicinebeta-Defensinstype 1 diabetessuolistomikrobistoAutoimmunityGut floramedicine.disease_causeautoimmuniteettiAutoimmunityFeces0302 clinical medicineautoimmuunisairaudetInsulin-Secreting CellsHLA-DQ beta-ChainsImmunology and AllergyMedicineChildFinlandOriginal ResearchCandida2. Zero hungerRISKMUCOSAtulehdusbiologyGUT MICROBIOTAdysbiosisFungal antigen3. Good healthChild PreschoolgutCATHELICIDIN LL-37Femalemedicine.symptomlcsh:Immunologic diseases. AllergyAdolescentImmunologyInflammationIMMUNITY03 medical and health sciencesmycobiomeSaccharomycesSEROCONVERSIONHumansPERMEABILITYAntibodies FungalTYPE-1AutoantibodiesType 1 diabetesbusiness.industrynuoruustyypin diabetesAutoantibodymedicine.diseasebiology.organism_classificationDiabetes Mellitus Type 1030104 developmental biologyMycoseshiivasienetinflammation3121 General medicine internal medicine and other clinical medicineImmunologyANTIBODIESONSET3111 BiomedicineCalprotectinbusinesslcsh:RC581-607Dysbiosis030215 immunology
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Immunity, Inflammation and Heart Failure. Their Role on Cardiac Function and Iron Status

2019

Aims: Heart failure is a clinical syndrome characterized by subclinical systemic inflammation and immune system activation associated with iron deficiency. No data exist on the various activations of immune-mediated mechanisms of inflammation in heart failure patients with reduced/preserved ejection fraction. We aimed to (1) investigate possible differences in inflammatory parameters and oxidative stress, and (2) detect a different iron status between groups. Materials and Methods: We enrolled 50 consecutive Caucasian outpatients with heart failure. All patients underwent echocardiographic measurements, laboratory determinations, evaluation of iron status and Toll-like receptors, and NF-κB …

Male0301 basic medicineheart failureSystemic inflammationGastroenterologyVentricular Function LeftElectrocardiographychemistry.chemical_compound0302 clinical medicineiron deficiencyImmunology and Allergyejection fraction; heart failure; inflammation; iron deficiency; toll-like receptorejection fractionOriginal ResearchAged 80 and overEjection fractionbiologymedicine.diagnostic_testToll-Like ReceptorsIron deficiencyMiddle AgedHeart Function TestsSerum ironCytokinesFemaleDisease SusceptibilityInflammation Mediatorsmedicine.symptomlcsh:Immunologic diseases. AllergyCardiac function curvemedicine.medical_specialtyIronImmunology03 medical and health sciencesHepcidinsInternal medicinemedicineHumansAgedCreatininebusiness.industryImmunitymedicine.diseaseFerritinOxidative Stress030104 developmental biologychemistryinflammationHeart failurebiology.proteintoll-like receptorlcsh:RC581-607businessBiomarkers030215 immunology
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Neuroprotective properties of mildronate, a mitochondria-targeted small molecule.

2010

Mildronate, a representative of the aza-butyrobetaine class of drugs with proven cardioprotective efficacy, was recently found to prevent dysfunction of complex I in rat liver mitochondria. The present study demonstrates that mildronate also acts as a neuroprotective agent. In a mouse model of azidothymidine (anti-HIV drug) neurotoxicity, mildronate reduced the azidothymidine-induced alterations in mouse brain tissue: it normalized the increase in caspase-3, cellular apoptosis susceptibility protein (CAS) and iNOS expression assessed by quantitative and semi-quantitative analysis. Mildronate also normalized the changes in cytochrome c oxidase (COX) expression, reduced the expression of glia…

MaleCell signalingAnti-HIV AgentsNitric Oxide Synthase Type IIMice Inbred StrainsMitochondrionPharmacologyNeuroprotectionElectron Transport Complex IVMiceCellular Apoptosis Susceptibility ProteinGlial Fibrillary Acidic ProteinmedicineAnimalsLymphocytesNeuroinflammationGlial fibrillary acidic proteinbiologyCaspase 3General NeuroscienceNeurodegenerationNeurotoxicityBrainmedicine.diseaseDisease Models AnimalNeuroprotective AgentsBiochemistrybiology.proteinNeurotoxicity SyndromesZidovudineCellular apoptosis susceptibility proteinMethylhydrazinesNeuroscience letters
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Effects of K(ATP) channel modulators on acetylcholine release from guinea-pig isolated atria and small intestine.

2002

The effects of K(ATP) channel blockers (glibenclamide, HMR 1883, HMR 1372) and openers (cromakalim, pinacidil, diazoxide) on the electrically-evoked (5 Hz) release of [(3)H]acetylcholine were studied in isolated guinea-pig atria and myenteric plexus-longitudinal muscle preparations which had been preincubated with [(3)H]choline. Atria: Cromakalim (0.3 microM and 1 microM), pinacidil (10 microM) and diazoxide (30 microM) significantly reduced the stimulation-evoked release of [(3)H]acetylcholine. The inhibition produced by cromakalim and pinacidil was prevented by 1 microM of either HMR 1883, HMR 1372 or glibenclamide. The blockers alone significantly increased the release at concentrations …

MaleCromakalimPotassium ChannelsGuinea PigsNeuromuscular JunctionMyenteric PlexusPharmacologyIn Vitro Techniqueschemistry.chemical_compoundGlyburideIntestine SmallmedicineDiazoxidePotassium Channel BlockersAnimalsChannel blockerHeart AtriaPharmacologySulfonamidesPinacidilDiazoxideThioureaPotassium channel blockerMuscle SmoothGeneral Medicinemusculoskeletal systemAtrial FunctionMyocardial ContractionHMR 1883Potassium channelAcetylcholinechemistryAnesthesiaPinacidilcardiovascular systemFemaleCromakalimAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Probability of major depression diagnostic classification based on the SCID, CIDI and MINI diagnostic interviews controlling for Hospital Anxiety and…

2020

Objective Two previous individual participant data meta-analyses (IPDMAs) found that different diagnostic interviews classify different proportions of people as having major depression overall or by symptom levels. We compared the odds of major depression classification across diagnostic interviews among studies that administered the Depression subscale of the Hospital Anxiety and Depression Scale (HADS-D). Methods Data accrued for an IPDMA on HADS-D diagnostic accuracy were analysed. We fit binomial generalized linear mixed models to compare odds of major depression classification for the Structured Clinical Interview for DSM (SCID), Composite International Diagnostic Interview (CIDI), and…

MaleDepressive disordersSCHEDULESACCURACYSocio-culturaleHospital Anxiety and Depression ScaleOdds03 medical and health sciences0302 clinical medicineSDG 3 - Good Health and Well-beingIndividual participant data meta-analysisMedicineHumansMajor depression030212 general & internal medicineVALIDITYDepression (differential diagnoses)Mini-international neuropsychiatric interviewProbabilityPsychiatric Status Rating ScalesDepressive Disorder MajorDepressive disorders Diagnostic interviews Hospital Anxiety and Depression Scale Individual participant data meta-analysis Major depressionbusiness.industryIndividual participant dataOdds ratioCIDIAn individual participant data meta-analysis of 73 primary studies.- Journal of psychosomatic research cilt.129 ss.109892 2020 [Wu Y. Levis B. Sun Y. Krishnan A. He C. Riehm K. Rice D. Azar M. Yan X. Neupane D. et al. -Probability of major depression diagnostic classification based on the SCID CIDI and MINI diagnostic interviews controlling for Hospital Anxiety and Depression Scale - Depression subscale scores]3. Good healthPsychiatry and Mental healthClinical PsychologyHospital Anxiety and Depression ScaleMeta-analysisDiagnostic interviews/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemalebusiness030217 neurology & neurosurgeryClinical psychology
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N-Valproyl-L-Phenylalanine as new potential antiepileptic drug: Synthesis, characterization and in vitro studies on stability, toxicity and anticonvu…

2013

Valproic acid (VPA) is considered first-line drug in treatment of generalized idiopathic seizures such as absence, generalized tonic-clonic and myoclonic seizures. Among major antiepileptic drugs, VPA is also considered effective in childhood epilepsies and infantile spasms. Due to its broad activity, VPA acts as a mood stabilizer in bipolar disorder and it is useful in migraine prophylaxis. Despite its long-standing usage, severe reactions to VPA, such as liver toxicity and teratogenicity, are reported. To circumvent side effects due to structural characteristics of VPA, we synthesized in good yield a new VPA-aminoacid conjugate, the N-valproyl-L-Phenylalanine, and characterized by FT-IR, …

MaleDrugCell Membrane PermeabilityAminoacidic derivative Astrocytes toxicity CNS-Targeting Enzymatic Stability Hippocampal epilepsy Valproic acid.Cell Survivalmedicine.drug_classPhenylalaninemedicine.medical_treatmentmedia_common.quotation_subjectPrimary Cell CulturePhenylalaninePharmacologySettore BIO/09 - FisiologiaHippocampusTissue Culture TechniquesDrug StabilityDrug DiscoverymedicineAnimalsRats WistarEvoked Potentialsmedia_commonValproic AcidChemistryHydrolysisValproic AcidBiological TransportMood stabilizerMicrotomyHydrogen-Ion ConcentrationIn vitroRatsAnticonvulsantSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoAstrocytesToxicityAnticonvulsantslipids (amino acids peptides and proteins)Conjugatemedicine.drug
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''Effect of stroke on arginase expression and localization in the rat brain''

2013

Quirie, Aurore | Demougeot, C. Eline | Bertrand, Nathalie | Mossiat, Claude | Garnier, Philippe | Marie, Christine | Prigent-Tessier, Anne; International audience; ''Because arginase and nitric oxide (NO) synthases (NOS) compete to degrade l-arginine, arginase plays a crucial role in the modulation of NO production. Moreover, the arginase 1 isoform is a marker of M2 phenotype macrophages that play a key role in tissue remodeling and resolution of inflammation. While NO has been extensively investigated in ischemic stroke, the effect of stroke on the arginase pathway is unknown. The present study focuses on arginase expression/activity and localization before and after (1, 8, 15 and 30days) …

MaleGene Expressionchemistry.chemical_compound0302 clinical medicineNeurotrophic factorsMACROPHAGESIN-VIVONeuronsAXONAL REGENERATION0303 health sciencesGlial fibrillary acidic proteinGeneral NeuroscienceBrainGLIAL RESPONSESCerebral InfarctionStrokeNitric oxide synthaseArginasemedicine.anatomical_structureBiochemistry[ SCCO.NEUR ] Cognitive science/NeuroscienceARGININE METABOLISMmedicine.symptom2'-DIPYRIDYLmedicine.medical_specialtyCentral nervous systemIRON CHELATOR 2InflammationBiologyFOCAL ISCHEMIANitric oxideLesion03 medical and health sciencesInternal medicineGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarNITRIC-OXIDE SYNTHASE030304 developmental biologyArginaseCEREBRAL-ISCHEMIABrain-Derived Neurotrophic Factor[SCCO.NEUR]Cognitive science/NeuroscienceCENTRAL-NERVOUS-SYSTEM''NITRIC-OXIDE SYNTHASERatsEndocrinologychemistryAstrocytesbiology.proteinMACROPHAGES''030217 neurology & neurosurgery
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