Search results for "CIDI"

showing 10 items of 778 documents

Global Mortality From Firearms, 1990-2016

2018

IMPORTANCE: Understanding global variation in firearm mortality rates could guide prevention policies and interventions. OBJECTIVE: To estimate mortality due to firearm injury deaths from 1990 to 2016 in 195 countries and territories. DESIGN, SETTING, AND PARTICIPANTS: This study used deidentified aggregated data including 13 812 location-years of vital registration data to generate estimates of levels and rates of death by age-sex-year-location. The proportion of suicides in which a firearm was the lethal means was combined with an estimate of per capita gun ownership in a revised proxy measure used to evaluate the relationship between availability or access to firearms and firearm injury …

MaleGlobal Health01 natural sciences0302 clinical medicineFirearm injuryArmas de FuegoHomicideGlobal healthMedicine030212 general & internal medicineYoung adultChildAged 80 and overMortality rateArmes de foc11 Medical And Health SciencesGeneral MedicineMiddle Aged16. Peace & justiceFirearm suicideSuicideGun ownershipChild PreschoolViolenciaFemaleHomicideAdultFirearmsAdolescentUncertainty intervalViolenceYoung Adult03 medical and health sciencesAge DistributionGeneral & Internal MedicineMortalitatHumansVital registrationSuicidioSex Distribution0101 mathematicsMortalityAgedbusiness.industry010102 general mathematicsInfant NewbornInfantHeridas por Arma de FuegoHomicidioMortalidadWounds GunshotHuman medicinebusinessDemographyJAMA - Journal of the American Medical Association
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Carotenoid trade-off between parasitic resistance and sexual display: an experimental study in the blackbird (Turdus merula).

2008

Many parasites depress the expression of the carotenoid-based colour displays of their hosts, and it has been hypothesized that animals face a trade-off in carotenoid allocation between immune functions and ‘degree of ornamentation’. While numerous correlative studies suggest that parasite infection decreases the intensity of carotenoid-based colour displays, the existence of this trade-off has never been demonstrated experimentally in a host–parasite model. In this study, we used the blackbird ( Turdus merula ) and Isospora (an intestinal parasite) to assess whether this trade-off does indeed exist. Blackbirds were supplemented with carotenoids while simultaneously being exposed to parasi…

MaleMESH : Host-Parasite InteractionsMESH : Analysis of VarianceTrade-offmedicine.disease_causeSongbirds[ SDV.BBM.BC ] Life Sciences [q-bio]/Biochemistry Molecular Biology/Biomolecules [q-bio.BM][ SDV.EE.IEO ] Life Sciences [q-bio]/Ecology environment/SymbiosisParasite hostingbill colourCarotenoidGeneral Environmental Sciencetrade-offchemistry.chemical_classificationPigmentationMESH : PigmentationBeakcarotenoidsfood and beveragesMESH : IsosporaGeneral MedicineIsosporaBeakGeneral Agricultural and Biological SciencesResearch Article[ SDV.MP.PAR ] Life Sciences [q-bio]/Microbiology and Parasitology/ParasitologyMESH : MaleZoologyIntestinal parasiteBiologyParasitic infectionGeneral Biochemistry Genetics and Molecular BiologyHost-Parasite InteractionsCoccidiaBotanymedicineAnimalsBody Weights and MeasuresMESH : Dietary SupplementsMESH : SongbirdsMESH : CarotenoidsAnalysis of VarianceIsosporaGeneral Immunology and Microbiologyorganic chemicalscoccidiaMESH : Body Weights and Measuresbiology.organism_classificationMESH : Beakchemistryexperimental infectionDietary SupplementsMESH : Animals
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Temperature modulates the response of the thermophilous sea urchin Arbacia lixula early life stages to CO2-driven acidification

2014

The increasing abundances of the thermophilous black sea urchin Arbacia lixula in the Mediterranean Sea are attributed to the Western Mediterranean warming. However, few data are available on the potential impact of this warming on A. lixula in combination with other global stressors such as ocean acidification. The aim of this study is to investigate the interactive effects of increased temperature and of decreased pH on fertilization and early development of A. lixula. This was tested using a fully crossed design with four temperatures (20, 24, 26 and 27 °C) and two pH levels (pHNBS 8.2 and 7.9). Temperature and pH had no significant effect on fertilization and larval survival (2d) for te…

MaleMediterranean climateSettore BIO/07 - EcologiaEmbryo NonmammalianEffects of global warming on oceansEmbryonic DevelopmentAquatic ScienceOceanographyHuman fertilizationMediterranean seabiology.animalAnimalsSeawaterPluteusSea urchinArbacia lixulaArbaciabiologyEcologyTemperatureOcean acidificationGeneral MedicineCarbon DioxideHydrogen-Ion Concentrationbiology.organism_classificationPollutionClimate change Ocean warming Ocean acidification Calcification Sea urchinFertilizationFemale
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MyD88 is dispensable for resistance toParacoccidioides brasiliensisin a murine model of blood-borne disseminated infection

2008

We have studied the role of MyD88, an adaptor protein of Toll-like receptors (TLRs), in murine defenses against Paracoccidioides brasiliensis in a model of blood-borne disseminated infection. Wild-type (WT) and MyD88-deficient mice infected intravenously with P. brasiliensis yeast cells showed an equivalent fungal burden, as well as similar levels of proinflammatory IL-1beta, IL-6, IL-12p70, tumor necrosis factor (TNF)-alpha and MIP-2, T-helper type 1 (Th1) (IFN-gamma) and Th2 cytokines (IL-4) in tissue homogenates. In vitro production of TNF-alpha, IFN-gamma and IL-12p70, by antigen-stimulated splenocytes from infected animals, was also similar in both types of mice; this production of Th1…

MaleMicrobiology (medical)medicine.medical_treatmentImmunologyNerve Tissue ProteinsMicrobiologyParacoccidioidesMicrobiologyProinflammatory cytokineMicePeritoneal cavitymedicineAnimalsHumansImmunology and AllergyLectins C-TypeMice KnockoutParacoccidioides brasiliensisbiologyMembrane ProteinsParacoccidioidesGeneral MedicineTh1 Cellsbiology.organism_classificationToll-Like Receptor 2Mice Inbred C57BLToll-Like Receptor 4Disease Models AnimalTLR2Infectious Diseasesmedicine.anatomical_structureCytokineMyeloid Differentiation Factor 88Macrophages PeritonealTLR4CytokinesTumor necrosis factor alphaParacoccidioidomycosisFungemiaFEMS Immunology & Medical Microbiology
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Neuroprotective Properties of Mildronate, a Small Molecule, in a Rat Model of Parkinson’s Disease

2010

Previously, we have found that mildronate [3-(2,2,2-trimethylhydrazinium) propionate dihydrate], a small molecule with charged nitrogen and oxygen atoms, protects mitochondrial metabolism that is altered by inhibitors of complex I and has neuroprotective effects in an azidothymidine-neurotoxicity mouse model. In the present study, we investigated the effects of mildronate in a rat model of Parkinson’s disease (PD) that was generated via a unilateral intrastriatal injection of the neurotoxin 6-hydroxydopamine (6‑OHDA). We assessed the expression of cell biomarkers that are involved in signaling cascades and provide neural and glial integration: the neuronal marker TH (tyrosine hydroxylase); …

MaleNitric Oxide Synthase Type IIlcsh:ChemistryUbiquitinNeurotoxinlcsh:QH301-705.5Receptor Notch3SpectroscopyNeuronsReceptors NotchbiologyGlial fibrillary acidic proteinMicrofilament ProteinsGeneral MedicineComputer Science ApplicationsCell biologySubstantia NigraNitric oxide synthaseNeuroprotective Agentsmedicine.anatomical_structureBiochemistryNeurogliaNeurogliaMethylhydrazinesneuroimmunological biomarkersTyrosine 3-Monooxygenasesmall moleculeSubstantia nigraParkinson’s disease; 6-OHDA model; neuroimmunological biomarkers; mildronate; small moleculeNeuroprotectionArticleCatalysisInorganic ChemistryGlial Fibrillary Acidic ProteinmedicineAnimalsParkinson Disease SecondaryRats WistarPhysical and Theoretical ChemistryOxidopamineMolecular BiologyTyrosine hydroxylase6-OHDA modelCalcium-Binding ProteinsmildronateOrganic ChemistryCorpus StriatumRatslcsh:Biology (General)lcsh:QD1-999nervous systemParkinson’s diseasebiology.proteinBiomarkersInternational Journal of Molecular Sciences
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BLBP-expression in astrocytes during experimental demyelination and in human multiple sclerosis lesions

2011

Several lines of evidence indicate that remyelination represents one of the most effective mechanisms to achieve axonal protection. For reasons that are not yet understood, this process is often incomplete or fails in multiple sclerosis (MS). Activated astrocytes appear to be able to boost or inhibit endogenous repair processes. A better understanding of remyelination in MS and possible reasons for its failure is needed. Using the well-established toxic demyelination cuprizone model, we created lesions with either robust or impaired endogenous remyelination capacity. Lesions were analyzed for mRNA expression levels by Affymetrix GeneChip® arrays. One finding was the predominance of immune a…

MalePathologyPlatelet-derived growth factormedicine.medical_treatmentCell CountBehavioral Neurosciencechemistry.chemical_compoundMice0302 clinical medicineFluorescent Antibody Technique IndirectOligonucleotide Array Sequence AnalysisPlatelet-Derived Growth Factor0303 health sciencesGlial fibrillary acidic proteinbiologyExperimental autoimmune encephalomyelitisAstrocytomaMiddle AgedImmunohistochemistrymedicine.anatomical_structureFemaleFibroblast Growth Factor 2Fatty Acid-Binding Protein 7Adultmedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisImmunologyBlotting WesternNerve Tissue ProteinsFatty Acid-Binding ProteinsReal-Time Polymerase Chain ReactionTransfection03 medical and health sciencesCuprizoneCell Line TumorGlial Fibrillary Acidic ProteinmedicineAnimalsHumansRNA MessengerRemyelination030304 developmental biologyAgedEndocrine and Autonomic SystemsMultiple sclerosisGrowth factorTumor Suppressor Proteinsmedicine.diseaseOligodendrocyteMice Inbred C57BLchemistryAstrocytesbiology.proteinOsteopontinCarrier Proteins030217 neurology & neurosurgeryDemyelinating Diseases
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Upregulation of antibody response to heat shock proteins and tissue antigens in an ocular ischemia model.

2011

PURPOSE. The aim of this study was to characterize the serum antibody reactivities occurring after ocular ischemia reperfusion. The time course of serum antibody responses was examined. METHODS. Wistar rats were exposed to transient ocular ischemia by elevating intraocular pressure to 130 mm Hg for 60 minutes. Axonal damage was evaluated on optic-nerve sections 2 and 4 weeks later. Blood samples collected before and several times after ischemia were used for antibody detection via customized protein microarrays. Different tissue antigens, including heat shock proteins (HSPs) and crystallins, were selected based on previous identification of antibody reactivities in studies on ischemic event…

MalePathologymedicine.medical_specialtyIschemiaHSP27 Heat-Shock ProteinsProtein Array AnalysisVimentinBiologyAutoantigensDownregulation and upregulationAntigenRetinal DiseasesHeat shock proteinGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarEye ProteinsIntraocular PressureAutoantibodiesGlial fibrillary acidic proteinRetinal VesselsSpectrinMyelin Basic ProteinOptic Nervemedicine.diseaseAxonsRatsUp-RegulationMyelin-Associated GlycoproteinShock (circulatory)Immunoglobulin GReperfusion Injurybiology.proteinMyelin-Oligodendrocyte Glycoproteinmedicine.symptomAntibodyMyelin ProteinsInvestigative ophthalmologyvisual science
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Pleomorphic pineocytoma with extensive neuronal differentiation: report of two cases

1994

Two pineal parenchymal tumors are presented, arising in a 54-year-old man and a 72-year-old woman; respectively. They showed isomorphic, cellular areas of small cells, often with characteristic pineocytomatous rosettes, and of medium-sized cells, as well as less cellular regions with highly pleomorphic, often ganglioid large cells. Immunohistochemistry disclosed extensive neuronal differentiation. There was intense positivity for neurofilament protein and microtubule-associated protein 2 in the pleomorphic areas and more variable expression in the isomorphic regions. Diffuse synaptophysin positivity was seen, accentuated along the borders of pleomorphic cells and in the rosettes, as well as…

MalePathologymedicine.medical_specialtyNeurofilamentEnolasePineal GlandPathology and Forensic MedicineCellular and Molecular NeurosciencemedicineCentral neurocytomaHumansAgedNeuronsPineoblastomaGlial fibrillary acidic proteinbiologyBrain NeoplasmsPineocytomaMiddle Agedmedicine.diseaseImmunohistochemistrybiology.proteinSynaptophysinImmunohistochemistryFemaleNeurology (clinical)PinealomaActa Neuropathologica
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Esthesioneuroblastoma: Ultrastructural, immunohistological and biochemical investigation of one case

1984

A case of esthesioneuroblastoma, the pathological diagnosis of which almost always causes great difficulties, was investigated ultrastructurally, biochemically, and immunohistologically, using antibodies against the five known types of intermediate filaments [keratin, vimentin, desmin, glial fibrillary acidic protein (GFAP) and neurofilaments]. The tumour cells did not react with antibodies against any of the five intermediate filament proteins. Ultrastructural investigations showed dense cored secretory granules in the cytoplasm and cell processes. Thus, immunohistology offers by "exclusion" a differential diagnosis to avoid often misdiagnosed tumours (undifferentiated carcinomas, embryona…

MalePathologymedicine.medical_specialtyNeurofilamentVimentinmacromolecular substancesCytoplasmic GranulesImmunofluorescenceDiagnosis DifferentialVanilmandelic Acid03 medical and health sciences0302 clinical medicineEsthesioneuroblastomaKeratinmedicineHumansNeuroectodermal Tumors Primitive Peripheral030223 otorhinolaryngologyIntermediate filamentchemistry.chemical_classificationbiologymedicine.diagnostic_testGlial fibrillary acidic proteinHomovanillic AcidGeneral MedicineMiddle Agedmedicine.disease3. Good healthOtorhinolaryngologychemistry030220 oncology & carcinogenesisbiology.proteinDesminParanasal Sinus NeoplasmsArchives of Oto-Rhino-Laryngology
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Fate of autologous dermal stem cells transplanted into the spinal cord after traumatic injury (TSCI)

2003

Rat dermis is a source of cells capable of growing in vitro and, in appropriate conditions, forming floating spheres constituted by nestin-positive cells. We have clonally grown these spheres up to the 15th generation. These spheres can be dissociated into cells that differentiate in vitro under appropriate conditions, these cells are labeled by antibodies to immature neuron markers such as nestin and beta-tubulin III and, later, to mature neuron markers such as microtubule-associated protein 2 and neurofilaments. However, most cells are positive to the astroglial marker glia fibrillary acidic protein (GFAP). When sphere-derived cells are transplanted into the spinal cord after traumatic in…

MalePathologymedicine.medical_specialtyTime Factorstiming of transplantationNeurofilamentCellular differentiationBlotting Westernstem cell migrationPolymerase Chain ReactionRats Sprague-DawleyCell MovementGlial Fibrillary Acidic ProteinmedicineAnimalsstem cell differentiationSpinal Cord InjuriesNeuronsrecovery from disabilityGlial fibrillary acidic proteinbiologystem cell migration; stem cell differentiation; timing of transplantation; recovery from disabilityStem CellsGeneral NeuroscienceCell DifferentiationDermisRecovery of FunctionNestinRatsTransplantationmedicine.anatomical_structureImmunologySettore BIO/14 - Farmacologiabiology.proteinSettore MED/26 - NeurologiaNeuronAntibodyStem cellStem Cell TransplantationNeuroscience
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