Search results for "CLASSIFICATION"

showing 10 items of 29475 documents

Regenerative Medicine: Does Erythropoietin have a Role?

2009

Regenerative Medicine, a recent new medical domain, aims to develop new therapies through the stimulation of natural regenerative processes also in human beings. In this field, Erythropoietin (EPO) represents a significant subject of research. Several studies allow the assertion that EPO, in different concentrations, has protective effects mainly on the central nervous system, cardiovascular system and renal tissue. This action is carried out through one of few regenerative activities of human beings: angiogenesis. This mechanism, which involves endothelial stem cells and VEGF (Vascular Endothelial Growth Factor), has been experimentally demonstrated with Recombinant human erythropoietin (r…

medicine.medical_specialtyAngiogenesisNeovascularization PhysiologicRegenerative MedicineBioinformaticsModels BiologicalAngiogenesis; Erythropoietin; Regenerative medicineRegenerative medicineNeovascularizationchemistry.chemical_compoundModelsNeoplasmshemic and lymphatic diseasesInternal medicineDrug DiscoverymedicineAnimalsHumansRegenerationPhysiologicErythropoietinZebrafishNeovascularizationPathologicPharmacologyNeovascularization Pathologicbiologybusiness.industryMechanism (biology)FishesAnimals Erythropoietin Fishes Humans Models; Biological Neoplasms Neovascularization; Pathologic Neovascularization; Physiologic Regeneration Regenerative MedicineBiologicalbiology.organism_classificationVascular endothelial growth factorEndothelial stem cellEndocrinologychemistryErythropoietinmedicine.symptombusinessmedicine.drugCurrent Pharmaceutical Design
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Non-wild-type cryptococcosis in a child with multivisceral organ transplant who owned bird pets.

2020

Cryptococcosis is the third most common invasive fungal infection in solid-organ transplant recipients, although it is rarely reported in transplanted children. It typically appears as a late-onset infection with central nervous system or pulmonary involvement. We present a case of cryptococcosis caused by a non-wild strain in a 10-year-old child who owned two pet birds, and review the existent literature.

medicine.medical_specialtyAntifungal AgentsCentral nervous systemDrug resistance030230 surgeryOrgan transplantationBirds03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansChildCryptococcus neoformansTransplantationNon wild typebiologybusiness.industryCryptococcosisOrgan TransplantationPetsbiology.organism_classificationmedicine.diseaseInfectious Diseasesmedicine.anatomical_structureImmunologyCryptococcosisCryptococcus neoformans030211 gastroenterology & hepatologySolid organ transplantationbusinessTransplant infectious disease : an official journal of the Transplantation SocietyREFERENCES
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Fungal Necrotizing Scleritis After Intravitreal Injection Therapy

2020

PURPOSE To report a case of infectious necrotizing scleritis secondary to Aspergillus terreus after intravitreal injection therapy. METHODS This is a case report with literature review. RESULTS A 98-year-old woman receiving intravitreal aflibercept injections for neovascular age-related macular degeneration in the left eye presented with severe pain, redness, and purulent discharge at the injection site. She was initially treated with topical fortified antibiotics, and clinical improvement was achieved, although microbial cultures showed negative results. Two months later, she presented with severe ocular pain and was diagnosed with anterior necrotizing scleritis. Scleral scrapings were col…

medicine.medical_specialtyAntifungal Agentsmedicine.drug_classRecombinant Fusion ProteinsAntibioticsAngiogenesis InhibitorsmedicineAspergillosisHumansAspergillus terreusNecrotizing scleritisAfliberceptAged 80 and overVoriconazolebiologybusiness.industryInjection therapyMacular degenerationmedicine.diseasebiology.organism_classificationSurgeryOphthalmologyAspergillusReceptors Vascular Endothelial Growth FactorAcute DiseaseIntravitreal InjectionsWet Macular DegenerationEtiologyFemalebusinessEye Infections FungalScleraScleritismedicine.drugCornea
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Diabetes, oxidative stress and therapeutic strategies.

2014

Abstract Background Diabetes has emerged as a major threat to health worldwide. Scope of Review The exact mechanisms underlying the disease are unknown; however, there is growing evidence that excess generation of reactive oxygen species (ROS), largely due to hyperglycemia, causes oxidative stress in a variety of tissues. Oxidative stress results from either an increase in free radical production, or a decrease in endogenous antioxidant defenses, or both. ROS and reactive nitrogen species (RNS) are products of cellular metabolism and are well recognized for their dual role as both deleterious and beneficial species. In type 2 diabetic patients, oxidative stress is closely associated with ch…

medicine.medical_specialtyAntioxidantEndogenous Factorsmedicine.medical_treatmentBiophysicsInflammationEndogeny030204 cardiovascular system & hematologyBiologyPharmacologymedicine.disease_causeBiochemistryAntioxidants03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineDiabetes mellitusmedicineDiabetes MellitusHumansMolecular BiologyReactive nitrogen speciesComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesmedicine.disease3. Good health[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemOxidative StressEndocrinologychemistrymedicine.symptomOxidoreductasesReactive Oxygen SpeciesOxidative stressBiochimica et biophysica acta
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The Activation Pattern of the Antioxidant Enzymes in the Right Ventricle of Rat in Response to Pressure Overload is of Heart Failure Type

2003

In the left ventricle subjected to pressure overload activity, the antioxidant enzymes increased at the hyperfunctional stage. During the transition to heart failure, these enzymes are down-regulated, oxidative stress increases, and apoptosis progresses. Maladaptative activation of the antioxidant enzymes at an early stage may contribute to the intrinsic vulnerability of right ventricle to pressure overload. The authors studied changes in expression and activity of the enzymes manganese and copper-zinc superoxide dismutases, glutathione peroxidase, and catalase in the right ventricle of rat following induction of pulmonary hypertension by injection of monocrotaline. Increase in the manganes…

medicine.medical_specialtyAntioxidantHeart Ventriclesmedicine.medical_treatmentmedicine.disease_causeAntioxidantsSuperoxide dismutaseInternal medicinePressuremedicineAnimalsRats WistarHeart Failurechemistry.chemical_classificationPressure overloadGlutathione PeroxidaseBase SequenceHypertrophy Right VentricularbiologySequence Analysis RNASuperoxide Dismutasebusiness.industryGlutathione peroxidaseCatalasemedicine.diseasePulmonary hypertensionRatsOxidative Stressmedicine.anatomical_structureEndocrinologychemistryVentricleHeart failureModels Animalbiology.proteinCardiologyReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessOxidative stressHeart Disease
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Janus-faced role of endothelial NO synthase in vascular disease: uncoupling of oxygen reduction from NO synthesis and its pharmacological reversal

2006

Endothelial NO synthase (eNOS) is the predominant enzyme responsible for vascular NO synthesis. A functional eNOS transfers electrons from NADPH to its heme center, where L-arginine is oxidized to L-citrulline and NO. Common conditions predisposing to atherosclerosis, such as hypertension, hypercholesterolemia, diabetes mellitus and smoking, are associated with enhanced production of reactive oxygen species (ROS) and reduced amounts of bioactive NO in the vessel wall. NADPH oxidases represent major sources of ROS in cardiovascular pathophysiology. NADPH oxidase-derived superoxide avidly interacts with eNOS-derived NO to form peroxynitrite (ONOO(-)), which oxidizes the essential NOS cofactor…

medicine.medical_specialtyAntioxidantNitric Oxide Synthase Type IIImedicine.medical_treatmentClinical BiochemistryNitric Oxidemedicine.disease_causeBiochemistrychemistry.chemical_compoundEnosInternal medicinemedicineAnimalsHumansVascular DiseasesEnzyme InhibitorsMolecular BiologyHemeJanus Kinaseschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxidebiology.organism_classificationOxygenEndocrinologychemistrybiology.proteinPeroxynitriteOxidative stressBiological Chemistry
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Pentaerithrityl tetranitrate improves angiotensin II induced vascular dysfunction via induction of heme oxygenase-1

2010

The organic nitrate pentaerythritol tetranitrate is devoid of nitrate tolerance, which has been attributed to the induction of the antioxidant enzyme heme oxygenase (HO)-1. With the present study, we tested whether chronic treatment with pentaerythritol tetranitrate can improve angiotensin II–induced vascular oxidative stress and dysfunction. In contrast to isosorbide-5 mononitrate (75 mg/kg per day for 7 days), treatment with pentaerythritol tetranitrate (15 mg/kg per day for 7 days) improved the impaired endothelial and smooth muscle function and normalized vascular and cardiac reactive oxygen species production (mitochondria, NADPH oxidase activity, and uncoupled endothelial NO synthase)…

medicine.medical_specialtyAntioxidantNitric Oxide Synthase Type IIImedicine.medical_treatmentVasodilator AgentsBlotting WesternFluorescent Antibody TechniquePentaerythritol tetranitratemedicine.disease_causePentaerythritolArticlechemistry.chemical_compoundInternal medicineRats Inbred SHRInternal MedicinemedicineAnimalsPentaerythritol TetranitrateEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesAnalysis of VarianceAngiotensin IImedicine.diseaseAngiotensin IIMitochondriaRatsHeme oxygenaseOxidative StressEndocrinologychemistryHeminEndothelium VascularReactive Oxygen SpeciesOxidative stressHeme Oxygenase-1
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Prevention of apoptosis by deferoxamine during 4 hours of cold cardioplegia and reperfusion: in vitro study of isolated working rat heart model.

2002

INTRODUCTION: Heart transplantation is often accompanied by multiple functional alterations, especially in reperfusion period. These are probably related to the reactive oxygen species (ROS) formation catalyzed by transition metals such as iron and copper, and thus the preservation time of the donor hearts is limited. Metabolic protection of the heart grafts is a permanent objective of numerous experiments. Recently, an iron chelator deferoxamine (DFX) was proposed as antioxidant agent for storage solutions in heart grafts. Oxidative stress is also known to mediate the apoptotic cell death in different tissues during ischemia-reperfusion. METHODS: The aim of this study was to evaluate a pos…

medicine.medical_specialtyAntioxidantmedicine.medical_treatment030204 cardiovascular system & hematologyPharmacologymedicine.disease_causePathology and Forensic Medicine03 medical and health sciences0302 clinical medicinePhysiology (medical)Internal medicineHeart ratemedicine030304 developmental biologychemistry.chemical_classificationHeart transplantation0303 health sciencesReactive oxygen speciesbusiness.industry3. Good healthDeferoxaminemedicine.anatomical_structurechemistryVentricleApoptosisCardiologybusinessOxidative stressmedicine.drugPathophysiology : the official journal of the International Society for Pathophysiology
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Effect of a Diet Supplemented with alpha-Tocopherol and beta-Carotene on ATP and Antioxidant Levels after Hepatic Ischemia-Reperfusion.

2008

Ischemia-reperfusion injury associated with liver transplantation remains a serious complication in clinical practice. In the present study the effect of intake of alpha-tocopherol or beta-carotene to limit liver injury by oxidative stress in ischemia and reperfusion was explored. Wistar rats were fed with diets enriched with alpha-tocopherol (20 mg/day) or beta-carotene (3 mg/day) for 21 days. After 21 days, their livers were subjected to 15 and 30 min of ischemia and afterwards were reperfused for 60 min. The recovery of levels of ATP during reperfusion was better in the group of rats whose diets were supplemented with alpha-tocopherol or beta-carotene than in the group control. The suppl…

medicine.medical_specialtyAntioxidantmedicine.medical_treatmentClinical BiochemistryIschemiaMedicine (miscellaneous)medicine.disease_causeSuperoxide dismutasechemistry.chemical_compoundInternal medicineβ-carotenemedicineGSHoxidative stresschemistry.chemical_classificationLiver injuryNutrition and Dieteticsα-tocopherolbiologybusiness.industryGlutathione peroxidaseGlutathionemedicine.diseaseischemia/reperfusionEndocrinologychemistryBiochemistrybiology.proteinOriginal ArticlebusinessReperfusion injuryOxidative stressJournal of clinical biochemistry and nutrition
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Redox modulation of mitochondriogenesis in exercise. Does antioxidant supplementation blunt the benefits of exercise training?

2015

Physical exercise increases the cellular production of reactive oxygen species (ROS) in muscle, liver, and other organs. This is unlikely due to increased mitochondrial production but rather to extramitochondrial sources such as NADPH oxidase or xanthine oxidase. We have reported a xanthine oxidase-mediated increase in ROS production in many experimental models from isolated cells to humans. Originally, ROS were considered as detrimental and thus as a likely cause of cell damage associated with exhaustion. In the past decade, evidence showing that ROS act as signals has been gathered and thus the idea that antioxidant supplementation in exercise is always recommendable has proved incorrect.…

medicine.medical_specialtyAntioxidantmedicine.medical_treatmentPhysical exerciseBiochemistryAntioxidantsSuperoxide dismutasechemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineAnimalsHumansMuscle SkeletalXanthine oxidaseExercisechemistry.chemical_classificationReactive oxygen speciesOrganelle BiogenesisNADPH oxidasebiologyMuscle adaptationGlutathione peroxidaseAdaptation PhysiologicalMitochondria MuscleOxidative StressEndocrinologychemistryDietary Supplementsbiology.proteinOxidation-ReductionFree Radical Biology and Medicine
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