Search results for "CORTICOSTERONE"

showing 10 items of 72 documents

Affinity and efficacy of racemic, (+)-, and (−)-methacholine in muscarinic inhibition of [3H]-noradrenaline release

1985

The right postganglionic sympathetic nerves of rat isolated perfused hearts (previously loaded with [3H]-noradrenaline) were stimulated electrically with 10 trains of 10 pulses at 10 Hz. The inhibition by methacholine of stimulation-evoked [3H]-noradrenaline overflow into the perfusate (determined in the presence of corticosterone, desipramine, phentolamine, and propranolol) was taken as a measure for activation of presynaptic muscarinic receptors. The evoked [3H]-noradrenaline overflow was inhibited by (+)-, racemic, and (-)-methacholine in a reversible and concentration-dependent manner. The concentration causing 50% inhibition (IC50) was 0.1, 0.26, and 65 microM, respectively, resulting …

MaleReceptor complexSympathetic Nervous SystemIntrinsic activityPhenoxybenzamineStereochemistryPhysostigminePropranololIn Vitro TechniquesPropylbenzilylcholine MustardNorepinephrineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsDrug InteractionsReceptorMethacholine ChlorideNeuronsPharmacologyPhenoxybenzamineChemistryHeartRats Inbred StrainsStereoisomerismReceptors MuscarinicElectric StimulationRatsDissociation constantSynapsesMethacholineCorticosteroneResearch Articlemedicine.drugBritish Journal of Pharmacology
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Effect of recurrent yohimbine on immediate and post-hoc behaviors, stress hormones, and energy homeostatic parameters

2013

Evidence from experimental models has suggested that acute activation of brain stress and anxiety pathways impacts subsequent behaviors that are mediated or modulated by limbic circuitry. There have been limited investigations of prior or chronic activation of these pathways on subsequent limbic-mediated behaviors. In this study, we tested whether recurrent administration of the anxiogenic compound yohimbine (YOH) could have post-injection effects on brain activation, stress hormones, and performance in sucrose self-administration and startle response paradigms. Rats received six injections across two weeks of either 2mg/kg YOH or saline. Behavioral evaluation confirmed the continued effica…

MaleReflex Startlemedicine.medical_specialtyStartle responseHippocampusExperimental and Cognitive PsychologyAdrenocorticotropic hormoneMotor ActivityAmygdalaArticleEatingBehavioral Neurosciencechemistry.chemical_compoundAdrenocorticotropic HormoneDietary SucroseCorticosteroneInternal medicinemedicineAnimalsPsychotropic Drugsmedicine.diagnostic_testBody WeightBrainYohimbineFeeding BehaviorImmunohistochemistryRatsYohimbineVentral tegmental areamedicine.anatomical_structureEndocrinologyAnxiogenicchemistryCorticosteronePsychologyProto-Oncogene Proteins c-fosStress Psychologicalmedicine.drugPhysiology & Behavior
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Novel sequential stress model for functional dyspepsia: Efficacy of the herbal preparation STW5

2015

Abstract Background Many screening procedures for agents with potential usefulness in functional dyspepsia (FD) rely on animals exposed to stress early in life (neonatal maternal separation, NMS) or in adulthood (restraint stress, RS). Purpose Since many clinical cases of FD have been associated with stress in early life followed by stress in adulthood, a sequential model simulating the clinical situation is described. To explore the validity of the model, the efficacy of STW5, a multicomponent herbal preparation of proven usefulness in FD, was tested. Study design/methods A sequential stress model established where rats are exposed to NMS after birth followed later by RS in adulthood. Stre…

MaleRestraint PhysicalDrugFunctional dyspepsiaPathologymedicine.medical_specialtyCorticotropin-Releasing Hormonemedia_common.quotation_subjectPharmaceutical SciencePharmacologySTW5chemistry.chemical_compoundCorticosteroneDrug DiscoverymedicineAnimalsNeonatal maternal separationDyspepsiaRats WistarScreening proceduresmedia_commonPharmacologyRestraint stressPlant Extractsbusiness.industryMaternal DeprivationGastric accommodationPlasma levelsGhrelinRatsDisease Models AnimalchemistryComplementary and alternative medicineMolecular MedicineFemaleGhrelinAnalysis of varianceRestraint stressCorticosteroneGastrointestinal MotilitybusinessStress PsychologicalHormonePhytomedicine
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c-fos expression, behavioural, endocrine and autonomic responses to acute social stress in male rats after chronic restraint: modulation by serotonin

2000

The effects in male rats of serotonin depletion (using the neurotoxin 5,7-dihydroxytryptamine) on the cross-sensitization of an acute social stress (defeat by a larger resident male) by previous repeated restraint stress (10 days, 60 min per day) was studied. Previous restraint increased freezing responses during social defeat in sham-operated rats, but this was not observed in those with depleted serotonin (83% or more in different regions of the brain). In contrast, neither heart rate (tachycardia) nor core temperature responses (hyperthermia) were accentuated in previously restrained rats (i.e. neither showed heterotypical sensitization), and neither adapted to repeated restraint (there …

MaleRestraint PhysicalSerotoninmedicine.medical_specialty57-DihydroxytryptamineAutonomic Nervous SystemAmygdalaBody TemperatureRats Sprague-DawleySocial defeatSerotonin AgentsDorsal raphe nucleusHeart RateInternal medicinemedicineAnimalsSocial BehaviorBrain ChemistrySocial stressBehavior AnimalGeneral NeuroscienceCentral nucleus of the amygdalaNeurosecretory SystemsRatsEndocrinologymedicine.anatomical_structureHypothalamusLocus coeruleusSerotoninCorticosteronePsychologyProto-Oncogene Proteins c-fosStress PsychologicalNeuroscience
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Impaired hippocampal neuroligin-2 function by chronic stress or synthetic peptide treatment is linked to social deficits and increased aggression.

2014

Neuroligins (NLGNs) are cell adhesion molecules that are important for proper synaptic formation and functioning, and are critical regulators of the balance between neural excitation/inhibition (E/I). Mutations in NLGNs have been linked to psychiatric disorders in humans involving social dysfunction and are related to similar abnormalities in animal models. Chronic stress increases the likelihood for affective disorders and has been shown to induce changes in neural structure and function in different brain regions, with the hippocampus being highly vulnerable to stress. Previous studies have shown evidence of chronic stress-induced changes in the neural E/I balance in the hippocampus. Ther…

MaleRestraint PhysicalhippocampusmoodCell Adhesion Molecules NeuronalNeurexinstress disordersHippocampusPoison controlNeuroliginNerve Tissue ProteinsReceptors Cell Surfacebehavioral scienceHippocampal formationneuropharmacologyHippocampussocial behaviorRats Sprague-DawleystressmedicineNeuritesAnimalsChronic stressRats WistarSocial BehaviorCells CulturedPharmacologyNeuronsAggressionaggressionneuropeptideschronic restraint stressOrgan SizeanxietyRatsAggressionsociabilityPsychiatry and Mental healthChronic DiseaseOriginal Articleneuroliginmedicine.symptomPsychologyCorticosteronePeptidesNeuroscienceStress PsychologicalSocial behaviorNeuropsychopharmacology : official publication of the American College of Neuropsychopharmacology
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Chronic restraint stress and chronic corticosterone treatment modulate differentially the expression of molecules related to structural plasticity in…

2004

Stress and stress-related hormones induce structural changes in neurons of the adult CNS. Neurons in the hippocampus, the amygdala and the prefrontal cortex undergo neurite remodeling after chronic stress. In the hippocampus some of these effects can be mimicked with chronic administration of adrenal steroids. These changes in neuronal structure may be mediated by certain molecules related to plastic events such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). The expression of PSA-NCAM persists in the adult hippocampus and it is up-regulated after chronic stress. The piriform cortex also displays considerable levels of PSA-NCAM during adulthood and indirect evide…

MaleRestraint Physicalmedicine.medical_specialtyDoublecortin ProteinHippocampusNeural Cell Adhesion Molecule L1AmygdalaRats Sprague-Dawleychemistry.chemical_compoundCorticosteroneStress PhysiologicalInternal medicinePiriform cortexmedicineAnimalsChronic stressOlfactory memoryPrefrontal cortexCerebral CortexNeuronal PlasticitybiologyGeneral NeuroscienceDoublecortinRatsmedicine.anatomical_structureEndocrinologynervous systemchemistryGene Expression RegulationChronic Diseasebiology.proteinSialic AcidsCorticosteroneNeuroscienceNeuroscience
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Chronic social defeat stress causes retinal vascular dysfunction

2021

Abstract Purpose The roles of vascular dysfunction and chronic stress have been extensively discussed in the pathophysiology of glaucoma. Our aim was to test whether chronic stress causes retinal vascular dysfunction and therewith induces retinal ganglion cells (RGCs) loss. Methods Twelve mice underwent chronic social defeat (CSD) stress, while 12 mice received control treatment only. Intraocular pressure (IOP) was measured with a rebound tonometer. Blood plasma corticosterone concentration and adrenal gland weight were used to assess stress levels. Brn-3a staining in retinas and PPD staining in optic nerve cross sections were conducted to assess the survival of RGCs and axons respectively.…

MaleRetinal Ganglion CellsIntraocular pressuremedicine.medical_specialtyCell SurvivalRetinal ArteryVideo RecordingVideo microscopyRetinal ganglionSocial DefeatMiceTonometry OcularCellular and Molecular Neurosciencechemistry.chemical_compoundRetinal DiseasesCorticosteroneInternal medicineAnimalsMedicineChronic stressIntraocular PressureTranscription Factor Brn-3ARetinaDisorder of Sex Development 46XYAdrenal Hyperplasia CongenitalEndothelin-1business.industryOptic NerveRetinalActinsSensory SystemsMice Inbred C57BLDisease Models AnimalOphthalmologyEndocrinologymedicine.anatomical_structurechemistryChronic DiseaseOptic nerveOcular Hypertensionsense organsCorticosteronebusinessStress PsychologicalExperimental Eye Research
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Central serotonin depletion modulates the behavioural, endocrine and physiological responses to repeated social stress and subsequent c-fos expressio…

1999

Abstract Intraspecific confrontation has been used to study effect of depleting central serotonin on the adaptation of male rats to repeated social stress (social defeat). Four groups of adult male rats were used (serotonin depletion/sham: stressed; serotonin depletion/sham: non-stressed). Central serotonin was reduced (by 59–97%) by a single infusion of the neurotoxin 5,7-dihydroxtryptamine (150 μg) into the cerebral ventricles; levels of dopamine and noradrenaline were unaltered (rats received appropriate uptake blockers prior to neurotoxic infusions). Sham-operated animals received solute only. Rats were then either exposed daily for 10 days to a second larger aggressive male in the latt…

MaleSerotoninmedicine.medical_specialty57-DihydroxytryptamineHypothalamusMotor ActivityAmygdalac-FosRats Sprague-DawleySocial defeatchemistry.chemical_compoundSerotonin AgentsHeart RateStress PhysiologicalCorticosteroneDopamineInternal medicineAdaptation PsychologicalmedicineAnimalsNeurotransmitterSocial stressbiologyGeneral NeuroscienceHydroxyindoleacetic AcidAmygdalaRatsAggressionEndocrinologymedicine.anatomical_structureSocial Dominancechemistrybiology.proteinFemaleSerotoninCorticosteronePsychologyProto-Oncogene Proteins c-fosBody Temperature RegulationBrain Stemmedicine.drugNeuroscience
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Adolescent pre-exposure to ethanol or MDMA prolongs the conditioned rewarding effects of MDMA

2011

Adolescents often take ethanol (EtOH) in combination with MDMA (3,4-methylenedioxymethylamphetamine). In the present work we studied the effect of repeated intermittent adolescent pre-exposure to both drugs on the behavioral and neurochemical effects of MDMA in mice. Sixteen days after pre-treatment, the rewarding and reinstating effects of MDMA in the conditioned place preference (CPP) paradigm were evaluated, along with the levels of biogenic amines, basal motor activity and corticosterone response to different challenges. Pre-exposure to EtOH, MDMA or EtOH+MDMA did not affect the CPP induced by 10mg/kg of MDMA. However, adolescent exposure to EtOH or MDMA increased the duration of the co…

MaleSerotoninmedicine.medical_specialtyDopamineN-Methyl-34-methylenedioxyamphetaminePoison controlExperimental and Cognitive PsychologyStriatumMotor ActivityChoice BehaviorHippocampusDrug Administration ScheduleExtinction PsychologicalMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalRewardCorticosteroneInternal medicineConditioning Psychologicalmental disordersAnimals Outbred StrainsmedicineAnimalsDrug InteractionsCerebral CortexEthanolIllicit DrugsMDMAExtinction (psychology)Hydroxyindoleacetic AcidCorpus StriatumConditioned place preferenceMonoamine neurotransmitterEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidCorticosteronePsychologypsychological phenomena and processesmedicine.drugPhysiology & Behavior
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The antidepressant-like effect of tDCS in mice: A behavioral and neurobiological characterization

2017

Abtract Background Transcranial direct current stimulation (tDCS) is a non-invasive method increasingly popular for the treatment of several brain disorders, such as major depression. Despite great enthusiasm and promising results, some studies report discrepant findings and no consensus exists for the clinical use of tDCS. Objective The present study aims to (i) determine the most effective stimulation parameters to optimize antidepressant-like effect of tDCS in the forced-swim test in mice and (ii) identify brain regions recruited by tDCS and possibly involved in its behavioral effect using Fos immunohistochemistry. Results We reported that tDCS induced long-lasting antidepressant-like ef…

MaleStimulation parametersMouse model of depressionmedicine.medical_treatmentBiophysicsPrefrontal CortexAntidepressantStimulationNucleus accumbensTranscranial Direct Current StimulationHippocampuslcsh:RC321-571Mice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineCorticosteronemedicineAnimalsPrefrontal cortexlcsh:Neurosciences. Biological psychiatry. NeuropsychiatrySwimmingDepressive Disorder MajorTranscranial direct-current stimulationLong-lasting effectsGeneral NeuroscienceForced-swim test030227 psychiatryMice Inbred C57BLVentral tegmental areamedicine.anatomical_structurechemistryAntidepressantFemaleNeurology (clinical)PsychologyNeuroscience030217 neurology & neurosurgeryBehavioural despair testBrain Stimulation
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