Search results for "CYTOKINE"

showing 10 items of 1787 documents

Is cell salvage safe in liver resection? A pilot study

2007

Abstract Study Objective To investigate the quality of cell salvaged (CS) blood in patients undergoing hemihepatectomy (study group) and compare it with CS-blood from aortic surgery (control group). Design Observational study. Setting Operating room in a university hospital. Measurements 6 patients undergoing hemihepatectomy or aortobifemoral bypass with intraoperative blood loss of more than 800 mL. Samples were drawn from the central venous catheter, from the reservoir of a CS recovery system, and from the processed blood in each patient to determine interleukin (IL)-6, IL-8, IL-10, tumor necrosis factor (TNF), complement C3a, and the terminal complement complex C5b-9. Microbiological ana…

medicine.medical_specialtyBlood transfusionmedicine.medical_treatmentCellPilot ProjectsComplement Membrane Attack ComplexResectionBlood Transfusion AutologousmedicineHepatectomyHumansInterleukin-6business.industryInterleukin-8InterleukinBacterial InfectionsInterleukin-10Complement systemSurgeryAnesthesiology and Pain Medicinemedicine.anatomical_structureCytokineComplement C3aErythrocyte TransfusionbusinessAnaerobic exerciseCentral venous catheterJournal of Clinical Anesthesia
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An Insight into the Role of Postmortem Immunohistochemistry in the Comprehension of the Inflammatory Pathophysiology of COVID-19 Disease and Vaccine-…

2021

On 11 March 2020, the World Health Organization (WHO) declared a pandemic due to the spread of COVID-19 from Wuhan, China, causing high mortality rates all over the world. The related disease, which mainly affects the lungs, is responsible for the onset of Diffuse Alveolar Damage (DAD) and a hypercoagulability state, frequently leading to Severe Acute Respiratory Syndrome (SARS) and multiorgan failure, particularly in old and severe-critically ill patients. In order to find effective therapeutic strategies, many efforts have been made aiming to shed light on the pathophysiology of COVID-19 disease. Moreover, following the late advent of vaccination campaigns, the need for the comprehension …

medicine.medical_specialtyCOVID-19 VaccinesQH301-705.5AutopsyReviewDiseaseCatalysisInorganic ChemistryvaccinePandemicHumansMedicineEndotheliumBiology (General)Physical and Theoretical ChemistryIntensive care medicineDiffuse alveolar damageAdverse effectQD1-999Molecular BiologypathophysiologySpectroscopypostmortemChAdOx1 nCov-19SARS-CoV-2business.industryMechanism (biology)Organic ChemistryCOVID-19ThrombosisGeneral MedicinePathophysiologyComputer Science ApplicationsVaccinationChemistryimmunohistochemistryCytokinesAutopsybusinessInternational Journal of Molecular Sciences
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A conceptually new treatment approach for relapsed glioblastoma: Coordinated undermining of survival paths with nine repurposed drugs (CUSP9) by the …

2013

Kast, Richard E. et al.

medicine.medical_specialtyCaptoprilTel avivMorpholinesArtesunateReviewGluconatesAngiotensinQuality of lifeAuranofinSertralineAntineoplastic Combined Chemotherapy ProtocolsDisulfiramTemozolomidemedicineRecurrent diseaseHumansIn patientNelfinavirTemozolomideBrain Neoplasmsbusiness.industrySuccinatesNeurokininCytotoxic chemotherapyUniversity hospitalmedicine.diseaseArtemisininsSurgeryDacarbazineKetoconazoleOncologyangiotensin aprepitant artesunate auranofin captopril cytokines disulfiram glioblastoma ketoconazole nelfinavir neurokinin sertraline temozolomideFamily medicineCytokinesNeoplasm Recurrence LocalGlioblastomabusinessAprepitantGlioblastomamedicine.drugOncotarget
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A new hepatocyte stimulating factor: cardiotrophin-1 (CT-1)

1995

Abstract Recently, a novel cytokine, cardiotrophin-1 (CT-1), was cloned and found to induce cardiac myocyte hypertrophy in vitro. Amino acid sequence similarity showed CT-1 to be a member of the IL-6/LIF/CNTF/OSM/IL-11 cytokine family. Since all known members of the IL-6 cytokine family induce an hepatic acute phase protein (APP) gene expression, we investigated the ability of CT-1 to induce a liver acute phase response. Upon stimulation of rat hepatoma cells, CT-1 and LIF induced the strongest rat fibrinogen mRNA expression, OSM and IL-6 induced a less pronounced response. When human hepatoma cells and primary rat hepatocytes were stimulated with CT-1, the expression of human haptoglobin a…

medicine.medical_specialtyCarcinoma HepatocellularCardiotrophin 1medicine.medical_treatmentBiophysicsGene ExpressionCiliary neurotrophic factorBiochemistryCardiotrophin 1Structural BiologyInternal medicineGene expressionGeneticsmedicineTumor Cells CulturedAnimalsHumansHepatocyteInterleukin 6Molecular BiologybiologyInterleukin-6Acute-phase proteinCell BiologyMolecular biologyMacroglobulinRatsmedicine.anatomical_structureEndocrinologyCytokineHepatocytebiology.proteinCytokinesInterleukin-6-cytokine familyAcute-Phase ProteinsAcute-phase responseFEBS Letters
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Effects of Norepinephrine and Cardiotrophin-1 on Phospholipase D Activity and Incorporation of Myristic Acid Into Phosphatidylcholine in Rat Heart

2004

The present study is part of a project on phospholipase D (PLD) in cardiac hypertrophy and analyzed effects on PLD activity of two growth stimuli, norepinephrine (NE) and cardiotrophin-1 (CT-1), in incubated rat heart. Phosphatidylcholine (PC) was labeled by 3H-myristic acid. PLD produced 3H-phosphatidylethanol (3H-PEth) from 3H-PC in the presence of ethanol and maintained a basal formation of 3H-PEth. Short-term and long-term exposure to NE for 2 or 13 h, respectively, enhanced the formation of 3H-PEth, which was blocked by prazosin. Long-term pretreatment with NE or CT-1 increased the incorporation of 3H-myristic acid into PC, which was blocked by atenolol. When the 3H-PEth formation was …

medicine.medical_specialtyCardiotrophin 1Heart VentriclesMyristic acidStimulationIn Vitro TechniquesMyristic AcidRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundReceptors Adrenergic alpha-1Internal medicinePhosphatidylcholineReceptors Adrenergic betaPhospholipase DmedicinePrazosinAnimalsPhospholipase D activityPharmacologyChemistryPhospholipase DMyocardiumlcsh:RM1-950AtenololRatsEnzyme Activationenzymes and coenzymes (carbohydrates)lcsh:Therapeutics. PharmacologyEndocrinologyPhosphatidylcholinesCytokinesMolecular Medicinelipids (amino acids peptides and proteins)Adrenergic alpha-Agonistsmedicine.drugJournal of Pharmacological Sciences
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Dihydrocucurbitacin B, isolated from Cayaponia tayuya, reduces damage in adjuvant-induced arthritis

2005

23,24-Dihydrocucurbitacin B, from the anti-rheumatic plant Cayaponia tayuya, was tested on arthritis induced by adjuvant to corroborate the anti-inflammatory properties of this plant. Arthritis was induced in Lewis rats; the resulting arthritic rats were then treated with dihydrocucurbitacin B (1 mg/kg orally, daily, 1 week). The effect of dihydrocucurbitacin B on the synthesis, release, and activity of pro-inflammatory enzymes (elastase, cyclooxygenase-2, and nitric oxide synthase-2) as well as its effect on different mediators (tumor necrosis factor-alpha and interleukin-1beta) were determined. Dihydrocucurbitacin B modified the evolution of the clinical symptoms, reducing the swelling an…

medicine.medical_specialtyCell Survivalmedicine.medical_treatmentAnti-Inflammatory AgentsAdministration OralNitric Oxide Synthase Type IIPainArthritisPlant RootsDinoprostoneCell LineNitric oxidechemistry.chemical_compoundSuperoxidesInternal medicinemedicineAnimalsLymphocytesNitritesPharmacologyDose-Response Relationship DrugPancreatic ElastasebiologyPlant Extractsbusiness.industryMacrophagesElastasemedicine.diseasebiology.organism_classificationArthritis ExperimentalTriterpenesCayaponia tayuyaRatsEnzyme ActivationNitric oxide synthaseCucurbitaceaeEndocrinologyCytokinechemistryCyclooxygenase 2Rats Inbred LewAntirheumatic AgentsToxicitybiology.proteinCytokinesFemaleTumor necrosis factor alphabusinessPhytotherapyEuropean Journal of Pharmacology
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The Dual Role of the GABAA Receptor in Peripheral Inflammation and Neuroinflammation: A Study in Hyperammonemic Rats

2021

Cognitive and motor impairment in minimal hepatic encephalopathy (MHE) are mediated by neuroinflammation, which is induced by hyperammonemia and peripheral inflammation. GABAergic neurotransmission in the cerebellum is altered in rats with chronic hyperammonemia. The mechanisms by which hyperammonemia induces neuroinflammation remain unknown. We hypothesized that GABAA receptors can modulate cerebellar neuroinflammation. The GABAA antagonist bicuculline was administrated daily (i.p.) for four weeks in control and hyperammonemic rats. Its effects on peripheral inflammation and on neuroinflammation as well as glutamate and GABA neurotransmission in the cerebellum were assessed. In hyperammone…

medicine.medical_specialtyCerebellumastrocyte activationcerebellumQH301-705.5hepatic encephalopathyNeurotransmissionCatalysisInorganic ChemistryGABA and glutamate transportersInternal medicineMedicinePhysical and Theoretical ChemistryBiology (General)Molecular BiologyQD1-999SpectroscopyNeuroinflammationMicrogliabusiness.industryGABAA receptorOrganic ChemistryGlutamate receptormicroglia phenotypeGeneral MedicineBicucullinecytokinesComputer Science ApplicationsChemistryEndocrinologymedicine.anatomical_structurenervous systemGABAergicbicucullinebusinessmedicine.drugInternational Journal of Molecular Sciences
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Dual specificity phosphatase 1 knockout mice show enhanced susceptibility to anaphylaxis but are sensitive to glucocorticoids.

2007

Dual specificity phosphatase DUSP1 (otherwise known as mitogen-activated phosphatase 1 or MKP-1) dephosphorylates MAPKs, particularly p38, and negatively regulates innate immunity. Recent studies have shown that the DUSP1 gene is transcriptionally up-regulated by glucocorticoids (GCs) and that the antiinflammatory action of GCs is impaired in DUSP1-/- mice. Here we show that GC-mediated dephosphorylation of ERK-1 and ERK-2 activated by IgE receptor cross-linking is unimpaired in bone marrow-derived mast cells (BMMCs) of DUSP1-/- mice. Dephosphorylation of phospho-p38 MAPK is impaired but only at early times of GC treatment. Proinflammatory cytokine and chemokine gene expression (CCL2, IL-6,…

medicine.medical_specialtyChemokinePhosphataseImmunoglobulin Ep38 Mitogen-Activated Protein KinasesProinflammatory cytokineDephosphorylationMiceEndocrinologyInternal medicineSepsisDual-specificity phosphatasemedicineAnimalsGenetic Predisposition to DiseaseMolecular BiologyAnaphylaxisGlucocorticoidsMice KnockoutMitogen-Activated Protein Kinase 1Mice Inbred C3HMitogen-Activated Protein Kinase 3biologyInterleukin-6Tumor Necrosis Factor-alphaDegranulationDual Specificity Phosphatase 1General MedicineMice Inbred C57BLEndocrinologyGene Expression RegulationMice Inbred DBAbiology.proteinCytokinesTumor necrosis factor alphaMolecular endocrinology (Baltimore, Md.)
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Oral administration of taurolidine ameliorates chronic DSS colitis in mice.

2007

Taurolidine (TRD) has antimicrobial and anti-inflammatory properties. However, the anti-inflammatory effects of TRD in inflammatory bowel diseases (IBD) have not been investigated. Here, we have analyzed the toxicity of TRD after oral long-term application in mice and examined the impact of oral TRD in a dextran sulfate sodium (DSS) model of experimental colitis. Female C57/BL6 mice received TRD in various concentrations (0.1% to 0.4%) for 60 days. Toxicity was evaluated by use of a disease activity index (DAI) and histological examination of major metabolic organs. Furthermore, the impact of 0.2% TRD on a chronic DSS colitis was examined by daily DAI, histological crypt damage score (CDS),…

medicine.medical_specialtyColonTaurineAdministration OralGastroenterologyInflammatory bowel diseasechemistry.chemical_compoundMiceOral administrationInternal medicinemedicineMesenteric lymph nodesAnimalsColitisThiadiazinesbusiness.industryDextran SulfateInterleukinTaurolidinemedicine.diseaseColitisInflammatory Bowel DiseasesMice Inbred C57BLmedicine.anatomical_structurechemistryCyclooxygenase 2Bacterial TranslocationImmunologyToxicityCytokinesSurgeryTumor necrosis factor alphaFemaleLymph NodesbusinessJournal of investigative surgery : the official journal of the Academy of Surgical Research
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The Role of Endothelium in COVID-19

2021

The 2019 novel coronavirus, known as severe acute respiratory syndrome-coronavirus 2 (SARS-CoV-2) or coronavirus disease 2019 (COVID-19), is causing a global pandemic. The virus primarily affects the upper and lower respiratory tracts and raises the risk of a variety of non-pulmonary consequences, the most severe and possibly fatal of which are cardiovascular problems. Data show that almost one-third of the patients with a moderate or severe form of COVID-19 had preexisting cardiovascular comorbidities such as diabetes mellitus, obesity, hypertension, heart failure, or coronary artery disease. SARS-CoV2 causes hyper inflammation, hypoxia, apoptosis, and a renin–angiotensin system imbalance …

medicine.medical_specialtyComplications Endothelium Pathogenesis COVID-19 Cytokine Release Syndrome Endothelial Cells Heart Failure Humans Renal Insufficiency Renin-Angiotensin System SARS-CoV-2 ThrombosisEndotheliumendotheliumcomplicationsQH301-705.5InflammationReviewCatalysisRenin-Angiotensin SystemInorganic ChemistryCoronary artery diseasePathogenesisInternal medicineDiabetes mellitusHumansMedicineRenal InsufficiencyBiology (General)Physical and Theoretical ChemistryEndothelial dysfunctionQD1-999Molecular BiologySpectroscopyHeart Failurebusiness.industrySARS-CoV-2pathogenesisOrganic ChemistryEndothelial CellsCOVID-19ThrombosisGeneral MedicineHypoxia (medical)medicine.diseaseComputer Science ApplicationsChemistrymedicine.anatomical_structureHeart failureCardiologymedicine.symptomCytokine Release SyndromebusinessInternational Journal of Molecular Sciences
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