Search results for "CYTOKINE"

showing 10 items of 1787 documents

Evaluation of cytokine polymorphisms (TNFalpha, IFNgamma and IL-10) in Down patients with coeliac disease.

2005

Abstract Background In Down syndrome there is an increased prevalence of coeliac disease, but the reasons for this association are yet unknown. Aims To evaluate a possible correlation between TNFα, IFNγ and IL-10 genotype polymorphisms with the susceptibility to coeliac disease in Down syndrome patients. Methods Single nucleotide polymorphisms of TNFα (−308G → A promoter region), IFNγ (+874T → A promoter region) and IL-10 (−1082G → A promoter region) have been studied in 10 Down patients with coeliac disease, in 40 Down patients without coeliac disease and in 220 healthy controls. Clinical features were also studied in coeliac disease–Down syndrome patients. Results The 10 coeliac disease–D…

medicine.medical_specialtyDown syndromeAdolescentSingle-nucleotide polymorphismGastroenterologyPolymorphism Single NucleotideCoeliac diseaseSerologyInterferon-gammaInternal medicineGenotypeBiopsymedicineHumansGenetic Predisposition to DiseaseChildPolymorphism GeneticHepatologymedicine.diagnostic_testbusiness.industryTumor Necrosis Factor-alphaGastroenterologynutritional and metabolic diseasesInfantmedicine.diseasesyndromedigestive system diseasesSurgeryInterleukin-10Interleukin 10Celiac DiseaseChild PreschoolCytokinesDownDown SyndromebusinessTrisomyDigestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
researchProduct

Effects of exercise on inflammation markers in type 2 diabetic subjects

2011

Endothelial dysfunction and plasma markers of inflammation are significantly increased in type 2 diabetics. Several proinflammatory cytokines, acute-phase proteins, and cell adhesion molecules, such as C-reactive protein (CRP), interleukines (IL), and tumor necrosis factor alpha (TNF-α), seem to play a role in the low-grade systemic inflammation observed in these subjects. Lifestyle changes are necessary to prevent atherosclerosis and cardiovascular events. Physical exercise is known to reduce markers of inflammation by decreasing adipocytokine production and cytokine release from skeletal muscles, endothelial cells, and immune system and also improving antioxidant status. In type 2 diabeti…

medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentInflammationPhysical exerciseSystemic inflammationProinflammatory cytokineEndocrinologyAdipokinesInternal medicinediabetesexerciseinflammation markersInternal MedicinemedicineHumansAerobic exerciseExerciseInflammationAdiponectinbusiness.industryGeneral MedicineExercise TherapyCytokineEndocrinologyDiabetes Mellitus Type 2CytokinesResistinmedicine.symptombusinessBiomarkersActa Diabetologica
researchProduct

Effects of alcohol-free beer on lipid profile and parameters of oxidative stress and inflammation in elderly women

2009

We assessed the influence of alcohol-free beer on factors implicated in atherosclerosis, such as lipid profile, oxidative stress parameters, and proinflammatory cytokines, in postmenopausal women, a population particularly at risk for atherosclerotic disease.The study was carried out in 29 nuns, 58 to 73 y old, who live in a convent with a disciplined, regular, and homogeneous lifestyle. The nuns maintained their habits and diet routine, but their meals were supplemented with 500 mL/d of alcohol-free beer (0.0%) divided into two doses over a 45-d period. Lipid profile, inflammatory markers such as C-reactive protein, interleukins 1 and 6, and tumor necrosis factor-alpha, and parameters of o…

medicine.medical_specialtyEndocrinology Diabetes and Metabolismmedicine.medical_treatmentPopulationInflammationmedicine.disease_causeThiobarbituric Acid Reactive SubstancesProinflammatory cytokineBeveragesLipid peroxidationchemistry.chemical_compoundInternal medicineHumansMedicineeducationInflammationeducation.field_of_studyNutrition and DieteticsEthanolmedicine.diagnostic_testTumor Necrosis Factor-alphabusiness.industryCholesterolInterleukinsVitamin EBeerMiddle AgedLipidsPostmenopauseOxidative StressC-Reactive ProteinEndocrinologychemistryCardiovascular DiseasesFemaleLipid PeroxidationInflammation Mediatorsmedicine.symptombusinessLipid profileOxidation-ReductionBiomarkersOxidative stressNutrition
researchProduct

Regulation of endotoxin-induced IL-6 production in liver sinusoidal endothelial cells and Kupffer cells by IL-10

1997

SUMMARY Sinusoidal endothelial cells and Kupffer cells are the first cell populations in the liver that come into contact with gut-derived endotoxin in portal blood. Although endotoxin concentrations as high as 1 ng/ml are physiologically present in portal blood, no local inflammation is seen. We show that the proinflammatory cytokine IL-6, which is central to the development of inflammatory reactions in the liver, is produced by sinusoidal endothelial cells and Kupffer cells in response to low concentrations of endotoxin (100 pg/ml to 1 ng/ml). The anti-inflammatory cytokine IL-10 down-regulated endotoxin-induced IL-6 release in endothelial and Kupffer cells. Importantly, Kupffer cells sec…

medicine.medical_specialtyEndotheliumKupffer Cellsmedicine.medical_treatmentImmunologyInflammationBiologyProinflammatory cytokineMiceInternal medicinemedicineAnimalsImmunology and AllergyInterleukin 4Mice Inbred BALB CInterleukin-6MicrocirculationKupffer cellOriginal ArticlesInterleukin-10EndotoxinsEndothelial stem cellInterleukin 10medicine.anatomical_structureCytokineEndocrinologyLiverImmunologyEndothelium Vascularmedicine.symptomClinical and Experimental Immunology
researchProduct

Pathophysiology of polymorphonuclear leukocyte in arterial hypertension

2009

This review shows how polymorphonuclear leukocytes (PMNs) play a pivotal role in the development of the organ injury that is associated with arterial hypertension. Elevated white blood cell count and higher levels of PMNs activation are risk factors for arterial hypertension and cardiovascular disease. Spontaneously activated PMNs release proinflammatory factors and reactive oxygen species, which have negative effects on vascular tone and on their adhesion to the endothelium. The oxidative stress in hypertensive PMNs is revealed by increased NADPH-oxidase production and lipid peroxidation and by decreased cytosolic and mitochondrial superoxide dismutase concentrations. The overexpression of…

medicine.medical_specialtyEndotheliumNeutrophilsPhysiologyPolymorphonuclear leukocyte Hypertension oxidative stress adhesion moleculesmedicine.disease_causeProinflammatory cytokineSuperoxide dismutaseLipid peroxidationLeukocyte Countchemistry.chemical_compoundPhysiology (medical)Internal medicinemedicineHumansLeukocyte Rollingchemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxide Dismutasebusiness.industryCell adhesion moleculeNADPH OxidasesHematologymedicine.anatomical_structureEndocrinologychemistryCD18 AntigensHypertensionImmunologybiology.proteinLipid PeroxidationCardiology and Cardiovascular MedicinebusinessOxidative stressClinical Hemorheology and Microcirculation
researchProduct

Association between Proinflammatory Markers, Leukocyte–Endothelium Interactions, and Carotid Intima–Media Thickness in Type 2 Diabetes: Role of Glyce…

2020

Glycated hemoglobin monitorization could be a tool for maintaining type 2 diabetes (T2D) under control and delaying the appearance of cardiovascular events. This cross-sectional study was designed to assess the role of glycemic control in modulating early-stage markers of cardiovascular complications. One hundred and eight healthy controls and 161 type 2 diabetic patients were recruited and distributed according to their glycemic control, setting the threshold at 6.5% (good control). Biochemical and anthropometrical parameters were registered during the initial visit, and peripheral blood was extracted to obtain polymorphonuclear cells and analyze inflammatory markers, adhesion molecules, l…

medicine.medical_specialtyEndotheliumPopulationlcsh:MedicineInflammationType 2 diabetes030204 cardiovascular system & hematologyGastroenterologyArticleProinflammatory cytokine03 medical and health scienceschemistry.chemical_compound0302 clinical medicineendothelial functionInternal medicinemedicineeducationcarotid intima–media thickness030304 developmental biologyGlycemic0303 health scienceseducation.field_of_studybusiness.industrylcsh:RGeneral Medicinemedicine.diseasemedicine.anatomical_structurechemistryIntima-media thicknessinflammationGlycated hemoglobintype 2 diabetesmedicine.symptombusinessglycated hemoglobinJournal of Clinical Medicine
researchProduct

Eosinophils, pruritus and psoriasis: effects of treatment with etretinate or cyclosporin-A

1998

Abstract Background The antipsoriatic drugs cyclosporin A (CyA) and etretinate have been found to influence proinflammatory eosinophilic leukocytes and pruritus. Aim We compared the number of blood eosinophils, concentration of serum eosinophil cationic protein (ECP), and pruritus in patients with psoriasis treated with either CyA or etretinate. Study design Patients with psoriasis vulgaris were randomly assigned to treatment for 10 weeks with either CyA ( n =21) or etretinate ( n =10). The psoriasis area-and-severity index (PASI-score) and pruritus (according to a 0–3 scale) served as clinical parameters, the blood esosinophil counts (Coulter Counter) and the serum ECP (RIA, Pharmacia) as …

medicine.medical_specialtyEosinophil cationic proteinbusiness.industryKeratolyticEtretinateDermatologymedicine.diseaseGastroenterologyBlood proteinsProinflammatory cytokineInfectious DiseasesPsoriasisInternal medicineCyclosporin aEosinophilicImmunologymedicinebusinessmedicine.drugJournal of the European Academy of Dermatology and Venereology
researchProduct

Role of exercise-induced hepatokines in metabolic disorders.

2019

International audience; The health-promoting effects of physical activity to prevent and treat metabolic disorders are numerous. However, the underlying molecular mechanisms are not yet completely deciphered. In recent years, studies have referred to the liver as an endocrine organ, since it releases specific proteins called hepatokines. Some of these hepatokines are involved in whole body metabolic homeostasis and are theorized to participate in the development of metabolic disease. In this regard, the present review describes the role of Fibroblast Growth Factor 21, Fetuin-A, Angiopoietin-like protein 4, and Follistatin in metabolic disease and their production in response to acute exerci…

medicine.medical_specialtyFGF21PhysiologyEndocrinology Diabetes and MetabolismPhysical activity030209 endocrinology & metabolism03 medical and health sciences0302 clinical medicineMetabolic DiseasesRegular exerciseNon-alcoholic Fatty Liver DiseasePhysiology (medical)Internal medicineEndocrine systemMedicineAnimalsHumansObesityMetabolic diseaseBeneficial effectsExercise030304 developmental biology0303 health sciencesbiologybusiness.industry[SDV.MHEP.EM]Life Sciences [q-bio]/Human health and pathology/Endocrinology and metabolism3. Good healthEndocrinologyDiabetes Mellitus Type 2Liverbiology.proteinCytokinesInsulin ResistanceWhole bodybusinessFollistatinAmerican journal of physiology. Endocrinology and metabolism
researchProduct

Potential involvement of fas and its ligand in the pathogenesis of Hashimoto's thyroiditis

1997

The mechanisms responsible for thyrocyte destruction in Hashimoto's thyroiditis (HT) are poorly understood. Thyrocytes from HT glands, but not from nonautoimmune thyroids, expressed Fas. Interleukin-1β (IL-1β), abundantly produced in HT glands, induced Fas expression in normal thyrocytes, and cross-linking of Fas resulted in massive thyrocyte apoptosis. The ligand for Fas (FasL) was shown to be constitutively expressed both in normal and HT thyrocytes and was able to kill Fas-sensitive targets. Exposure to IL-1β induced thyrocyte apoptosis, which was prevented by antibodies that block Fas, suggesting that IL-1β-induced Fas expression serves as a limiting factor for thyrocyte destruction. Th…

medicine.medical_specialtyFas Ligand Proteinmedicine.medical_treatmentThyroid GlandApoptosisPolymerase Chain ReactionThyroiditisFas ligandPathogenesisImmunoenzyme TechniquesInternal medicinemedicineTumor Cells CulturedHumansRNA Messengerfas ReceptorCells CulturedNucleic Acid Synthesis InhibitorsProtein Synthesis InhibitorsMultidisciplinaryMembrane GlycoproteinsChemistryThyroidThyroiditis AutoimmuneInterleukinAntibodies Monoclonalmedicine.diseaseFas receptorRecombinant ProteinsCytokinemedicine.anatomical_structureEndocrinologyApoptosisCytokinesInterleukin-1
researchProduct

Effects of Mancozeb and Metribuzin on in vitro proliferative responses and oxidative stress of human and rat spleen lymphocytes stimulated by mitogens

2011

Pesticides have been shown to possess marked immunotropic activity. The aim of this work was to study the in vitro effects of different concentrations (1-100 µM) of Mancozeb (fungicide) and Metribuzin (herbicide), on the proliferative responses of human and rat spleen lymphocytes stimulated by concanavalin A (ConA, mitogen), the Th1- (IL-2, INFγ) and Th2- (IL-4) cytokine secretion and on the intracellular oxidative status. The results showed that Mancozeb significantly reduced ConA lymphocyte proliferation in a dose-dependent manner in both humans and rats. It also decreased IL-2, INFγ and IL-4 secretion with a a shift away to Th1 phenotype. Metribuzin at low concentrations (1-10 µM) result…

medicine.medical_specialtyHealth Toxicology and Mutagenesismedicine.medical_treatmentLymphocyte proliferationMetribuzinmedicine.disease_causechemistry.chemical_compoundMetribuzinHuman lymphocytesInternal medicinemedicineMancozebRat splenocytesbiologyGeneral MedicineGlutathioneCytokineEndocrinologychemistryConcanavalin AOxidative stressbiology.proteinCytokinesCytokine secretionAgronomy and Crop ScienceOxidative stressMancozeb
researchProduct