Search results for "CYTOKINE"

showing 10 items of 1787 documents

Peripheral Blood and Cerebrospinal Fluid Cytokine Levels in Guillain Barré Syndrome: A Systematic Review and Meta-Analysis

2019

Background: Guillain Barre Syndrome (GBS) is an autoimmune disorder caused by the immune-mediated damage of the peripheral nervous system. Increasing evidence suggests that inflammatory cytokines are important mediators for the onset and progression of GBS. A number of clinical studies have demonstrated elevated levels of T helper-1 (Th1-), Th2-, and Th17-related cytokines in patients with GBS; however, the results were inconsistent across studies. Methods: We performed a systematic review and a meta-analysis of studies comparing the levels of inflammatory cytokines in the cerebrospinal fluid and peripheral blood between patients with GBS and healthy individuals, using Comprehensive Meta-An…

0301 basic medicinemedicine.medical_specialtymedicine.medical_treatmentInflammationGastroenterologycerebrospinal fluidlcsh:RC321-571Proinflammatory cytokine03 medical and health sciences0302 clinical medicineCerebrospinal fluidInternal medicinecytokineMedicinelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryGuillain-Barre syndromebusiness.industryGeneral Neuroscienceperipheral bloodmedicine.diseaseConfidence intervalmeta-analysisGuillain Barré Syndrome030104 developmental biologyCytokineinflammationMeta-analysisTumor necrosis factor alphaSystematic Reviewmedicine.symptombusiness030217 neurology & neurosurgeryNeuroscienceFrontiers in Neuroscience
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Role of Redox Signaling and Inflammation in Skeletal Muscle Adaptations to Training

2016

The inflammatory response to exercise-induced muscle damage has been extensively described. Exercise has important modulatory effects on immune function. These effects are mediated by diverse factors including pro-inflammatory cytokines, classical stress hormones, and hemodynamic effects leading to cell redistribution. As has been reported regarding oxidative stress, inflammation can have both detrimental and beneficial effects in skeletal muscle. In this review we will address the role of inflammation on protein metabolism in skeletal muscle. Specifically, we will review studies showing that treatment with cyclooxygenase-inhibiting drugs modulate the protein synthesis response to one bout …

0301 basic medicinemedicine.medical_specialtyprotein synthesisPhysiologyClinical BiochemistryInflammationReviewBiologyBioinformaticsmedicine.disease_causeBiochemistryProinflammatory cytokineMuscle hypertrophyprostaglandins03 medical and health sciences0302 clinical medicineImmune systemInternal medicinemedicineoxidative stressMolecular Biologychemistry.chemical_classificationReactive oxygen specieslcsh:RM1-950Skeletal muscle030229 sport sciencesCell Biology030104 developmental biologyEndocrinologymedicine.anatomical_structurelcsh:Therapeutics. Pharmacologychemistryanti-inflammatoriesmedicine.symptomhypertrophyOxidative stressHormoneAntioxidants
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New insights about the putative role of myokines in the context of cardiac rehabilitation and secondary cardiovascular prevention.

2017

Exercise training prevents the onset and the development of many chronic diseases, acting as an effective tool both for primary and for secondary prevention. Various mechanisms that may be the effectors of these beneficial effects have been proposed during the past decades: some of these are well recognized, others less. Muscular myokines, released during and after muscular contraction, have been proposed as key mediators of the systemic effects of the exercise. Nevertheless the availability of an impressive amount of evidence regarding the systemic effects of muscle-derived factors, few studies have examined key issues: (I) if skeletal muscle cells themselves are the main source of cytokin…

0301 basic medicinemedicine.medical_specialtyregular exercisemedicine.medical_treatmentInflammationBioinformaticsCachexiaCoronary artery disease03 medical and health sciencesMyokineMyokineReview Article on Myokines Exercise and Cardiovascular DiseasemedicineRehabilitationMyokines; cardiac rehabilitation; cardiovascular disease (CVD); cerebrovascular disease; regular exercisecardiovascular disease (CVD)business.industrySkeletal muscleGeneral Medicinemedicine.diseasecerebrovascular diseasecardiac rehabilitation030104 developmental biologyCytokinemedicine.anatomical_structureSarcopeniaPhysical therapymedicine.symptombusiness
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Blockade of Pannexin-1 Channels and Purinergic P2X7 Receptors Shows Protective Effects Against Cytokines-Induced Colitis of Human Colonic Mucosa

2018

Introduction: The pannexin-1 (Panx1) channels are found in many cell types, and ATP released from these channels can act on nearby cells activating purinergic P2X7 receptors (P2X7R) which lead to inflammation. Although Panx1 and P2X7R are implicated in the process of inflammation and cell death, few studies have looked at the role they play in inflammatory bowel disease in human. Hence, the aim of the present study was to investigate the function of Panx1 and P2X7R in an ex vivo colitis model developed from human colonic mucosal explants.Materials and Methods: Healthy human colonic mucosal strips (4 × 10 mm) were incubated in carbogenated culture medium at 37°C for 16 h. Proinflammatory cyt…

0301 basic medicinemedicine.medical_specialtytissue explantsCryptInflammationInflammatory bowel diseasecolonic inflammationProinflammatory cytokine03 medical and health sciencesInternal medicinemedicinePharmacology (medical)ColitisOriginal ResearchPharmacologyTight junctionChemistrylcsh:RM1-950Purinergic receptorpannexin-1medicine.diseaselcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologyP2X7 receptorhuman colitisTumor necrosis factor alphamedicine.symptomFrontiers in Pharmacology
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Systemic Oxidative Stress and Visceral Adipose Tissue Mediators of NLRP3 Inflammasome and Autophagy Are Reduced in Obese Type 2 Diabetic Patients Tre…

2020

Obesity is a low-grade inflammatory condition affecting a range of individuals, from metabolically healthy obese (MHO) subjects to type 2 diabetes (T2D) patients. Metformin has been shown to display anti-inflammatory properties, though the underlying molecular mechanisms are unclear. To study whether the effects of metformin are mediated by changes in the inflammasome complex and autophagy in visceral adipose tissue (VAT) of obese patients, a biopsy of VAT was obtained from a total of 68 obese patients undergoing gastric bypass surgery. The patients were clustered into two groups: MHO patients and T2D patients treated with metformin. Patients treated with metformin showed decreased levels o…

0301 basic medicinemedicine.medical_specialtyvisceral adipose tissue (VAT)obesityautophagyendocrine system diseasesPhysiologyinflammatory cytokinesClinical BiochemistryATG5Adipose tissue030209 endocrinology & metabolismLeukocyte homeostasisType 2 diabetesBiochemistryArticleProinflammatory cytokine03 medical and health sciences0302 clinical medicineInternal medicinemedicineoxidative stressMolecular Biologytype 2 diabetes (T2D)business.industrylcsh:RM1-950nutritional and metabolic diseasesInflammasomeCell Biologymedicine.diseaseMetforminlcsh:Therapeutics. Pharmacology030104 developmental biologyEndocrinologybusinessmetforminInflammasome complexmedicine.drugAntioxidants (Basel, Switzerland)
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Conditioned Medium from Human Amnion-Derived Mesenchymal Stromal/Stem Cells Attenuating the Effects of Cold Ischemia-Reperfusion Injury in an In Vitr…

2021

The clinical results of lung transplantation (LTx) are still less favorable than other solid organ transplants in both the early and long term. The fragility of the lungs limits the procurement rate and can favor the occurrence of ischemia-reperfusion injury (IRI). Ex vivo lung perfusion (EVLP) with Steen SolutionTM (SS) aims to address problems, and the implementation of EVLP to alleviate the activation of IRI-mediated processes has been achieved using mesenchymal stromal/stem cell (MSC)-based treatments. In this study, we investigated the paracrine effects of human amnion-derived MSCs (hAMSCs) in an in vitro model of lung IRI that includes cold ischemia and normothermic EVLP. We found tha…

0301 basic medicinemedicine.medical_treatmentApoptosislcsh:Chemistry0302 clinical medicinelcsh:QH301-705.5Cells CulturedSpectroscopyamnion-derived mesenchymal stem cellsCell CycleCold IschemiaNF-kappa BCell DifferentiationGeneral MedicineComputer Science Applicationsconditioned mediummedicine.anatomical_structureReperfusion Injury030220 oncology & carcinogenesisCytokinesStem cellStromal cellCell Survivalex vivo lung perfusionArticleCatalysisInorganic Chemistry03 medical and health sciencesParacrine signallingDownregulation and upregulationmedicineHumansLung transplantationAmnionlung ischemia-reperfusion injuryPhysical and Theoretical ChemistryMolecular BiologyLungbusiness.industryOrganic ChemistryMesenchymal stem cellMesenchymal Stem Cellsmedicine.disease030104 developmental biologyGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999A549 CellsAlveolar Epithelial CellsCulture Media ConditionedCancer researchbusinessReperfusion injuryInternational Journal of Molecular Sciences
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Filifactor alocis and Tumor Necrosis Factor-Alpha Stimulate Synthesis of Visfatin by Human Macrophages

2021

There is little known about the effect of the periodontopathogen Filifactor alocis on macrophages as key cells of the innate immune defense in the periodontium. Therefore, the aim of the present study was to investigate the effect of F. alocis and additionally of the pro-inflammatory cytokine tumor necrosis factor-alpha (TNF&alpha

0301 basic medicinemedicine.medical_treatmentCD14tumor necrosis factor<i>Filifactor alocis</i>610 Medicine & healthmacrophageArticleCatalysisProinflammatory cytokineInorganic Chemistrylcsh:Chemistry03 medical and health sciences0302 clinical medicinestomatognathic systemvisfatinmedicineMacrophagePhysical and Theoretical Chemistry610 Medicine &amp; healthMolecular Biologyperiodontitislcsh:QH301-705.5SpectroscopyPeriodontitisInnate immune systembusiness.industryOrganic Chemistry030206 dentistryGeneral Medicinemedicine.diseaseComputer Science ApplicationsFilifactor alocisTLR2030104 developmental biologyCytokinelcsh:Biology (General)lcsh:QD1-999ImmunologyTumor necrosis factor alphaMMP1businessCOX2International Journal of Molecular Sciences
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PRR signaling during in vitro macrophage differentiation from progenitors modulates their subsequent response to inflammatory stimuli.

2017

Toll-like receptor (TLR) agonists drive hematopoietic stem and progenitor cells (HSPCs) to differentiate along the myeloid lineage in vitro and also in vivo following infection. In this study, we used an in vitro model of HSPC differentiation to investigate the functional consequences (cytokine production) that exposing HSPCs to various pathogen-associated molecular patterns (PAMPs) and Candida albicans cells have on the subsequently derived macrophages. Mouse HSPCs (Lin- cells) were cultured with GM-CSF to induce macrophage differentiation in the presence or absence of the following pattern recognition receptor (PRR) agonists: Pam3CSK4 (TLR2 ligand), LPS (TLR4 ligand), depleted zymosan (wh…

0301 basic medicinemedicine.medical_treatmentClinical BiochemistryImmunologyProinflammatory cytokineMajor Histocompatibility Complex03 medical and health scienceschemistry.chemical_compoundMicemedicineEscherichia coliImmunology and AllergyAnimalsAntigens LyProgenitor cellCells CulturedChemistryMacrophagesZymosanPattern recognition receptorCell DifferentiationFlow CytometryCell biologyMice Inbred C57BLHaematopoiesisTLR2030104 developmental biologyCytokineReceptors Pattern RecognitionTLR4CytokinesFemaleSignal TransductionEuropean cytokine network
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Immunomodulatory Therapy of Inflammatory Liver Disease Using Selectin-Binding Glycopolymers

2017

Immunotherapies have the potential to significantly advance treatment of inflammatory disease and cancer, which are in large part driven by immune cells. Selectins control the first step in immune cell adhesion and extravasation, thereby guiding leukocyte trafficking to tissue lesions. We analyzed four different highly specific selectin-binding glycopolymers, based on linear poly(2-hydroxypropyl)-methacrylamide (PHPMA) polymers. These glycopolymers contain either the tetrasaccharide sialyl-LewisX (SLeX) or the individual carbohydrates fucose, galactose, and sialic acids mimicking the complex SLeX binding motive. The glycopolymers strongly bind to primary human macrophages, without activatin…

0301 basic medicinemedicine.medical_treatmentGeneral Physics and Astronomy02 engineering and technologyFucoseImmunomodulationMice03 medical and health scienceschemistry.chemical_compoundImmune systemPolysaccharidesmedicineAnimalsHumansGeneral Materials ScienceCell adhesionCells CulturedInflammationBinding SitesbiologyChemistryLiver DiseasesGeneral EngineeringImmunotherapy021001 nanoscience & nanotechnologyDynamic Light ScatteringExtravasationIn vitro3. Good healthMice Inbred C57BLDisease Models Animal030104 developmental biologyBiochemistryConcanavalin ASelectinsbiology.proteinCancer researchCytokines0210 nano-technologySelectinACS Nano
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Tissue microenvironment dictates the fate and tumor-suppressive function of type 3 ILCs

2017

Nussbaum et al. found that tumor suppression through innate lymphoid cells (ILCs) cannot be predicted solely based on the ILC phenotype and lineage but that their immune properties are shaped both by their ontogeny and by the tissue microenvironment they reside in.

0301 basic medicinemedicine.medical_treatmentImmunology314610 Medicine & healthBiology10263 Institute of Experimental ImmunologyArticle31103 medical and health sciencesMiceRAR-related orphan receptor gammaCell Line TumormedicineImmunology and AllergyAnimalsLymphocytesskin and connective tissue diseasesTranscription factorResearch ArticlesMice Knockout2403 ImmunologyInnate lymphoid cellNeoplasms ExperimentalNuclear Receptor Subfamily 1 Group F Member 3PhenotypeCell biologybody regionsKiller Cells NaturalMice Inbred C57BL030104 developmental biologyCytokineCellular MicroenvironmentCell cultureTumor progressionInterleukin 122723 Immunology and AllergyCytokines570 Life sciences; biologyTranscription Factors
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