Search results for "Calcium Channel Blockers"

showing 10 items of 102 documents

Cannabinoid receptor 1 modulates the autophagic flux independent of mTOR- and BECLIN1-complex

2013

Cannabinoid Receptor 1 (CB1) has been initially described as the receptor for Delta-9-Tetrahydrocannabinol in the central nervous system (CNS), mediating retrograde synaptic signaling of the endocannabinoid system. Beside its expression in various CNS regions, CB1 is ubiquituous in peripheral tissues, where it mediates, among other activities, the cell's energy homeostasis. We sought to examine the role of CB1 in the context of the evolutionarily conserved autophagic machinery, a main constituent of the regulation of the intracellular energy status. Manipulating CB1 by siRNA knockdown in mammalian cells caused an elevated autophagic flux, while the expression of autophagy-related genes rema…

Cannabinoid receptorMorpholinesGreen Fluorescent ProteinsDown-RegulationmTORC1NaphthalenesBiochemistryMiceCellular and Molecular NeurosciencePiperidinesReceptor Cannabinoid CB1RimonabantAutophagymedicineAnimalsHumansEnzyme InhibitorsCannabinoid Receptor AntagonistsCells CulturedPI3K/AKT/mTOR pathwayAdenine NucleotidesChemistryTOR Serine-Threonine KinasesAutophagyMembrane ProteinsCalcium Channel BlockersEmbryo MammalianEndocannabinoid systemBenzoxazinesCell biologyMice Inbred C57BLnervous systemAstrocytesPyrazolesBeclin-1lipids (amino acids peptides and proteins)MacrolidesSynaptic signalingRimonabantApoptosis Regulatory ProteinsFlux (metabolism)medicine.drugJournal of Neurochemistry
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WIN 55,212-2, agonist of cannabinoid receptors, prevents amyloid β1-42 effects on astrocytes in primary culture

2015

Alzheimer's disease (AD), a neurodegenerative illness involving synaptic dysfunction with extracellular accumulation of Aβ1-42 toxic peptide, glial activation, inflammatory response and oxidative stress, can lead to neuronal death. Endogenous cannabinoid system is implicated in physiological and physiopathological events in central nervous system (CNS), and changes in this system are related to many human diseases, including AD. However, studies on the effects of cannabinoids on astrocytes functions are scarce. In primary cultured astrocytes we studied cellular viability using MTT assay. Inflammatory and oxidative stress mediators were determined by ELISA and Western-blot techniques both in…

Cannabinoid receptormedicine.medical_treatmentInterleukin-1betaNitric Oxide Synthase Type IIlcsh:Medicinemedicine.disease_causeReceptors CannabinoidWIN 55212-2Receptorlcsh:ScienceCerebral CortexMultidisciplinaryCalcium Channel BlockersSistema nerviós Malaltiesmedicine.symptomSignal transductionResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyCell SurvivalMorpholinesPrimary Cell CultureInflammationNaphthalenesBiologyNeurologiaFetusInternal medicinemedicineAnimalsViability assayCannabinoid Receptor AgonistsAmyloid beta-PeptidesSuperoxide DismutaseTumor Necrosis Factor-alphalcsh:RTranscription Factor RelAPeptide FragmentsBenzoxazinesRatsPPAR gammaOxidative StressEndocrinologyGene Expression RegulationCyclooxygenase 2Astrocyteslcsh:QFisiologia humanaCannabinoidOxidative stress
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AISF position paper on liver disease and pregnancy.

2016

Abstract The relationship between liver disease and pregnancy is of great clinical impact. Severe liver disease in pregnancy is rare; however, pregnancy-related liver disease is the most frequent cause of liver dysfunction during pregnancy and represents a severe threat to foetal and maternal survival. A rapid differential diagnosis between liver disease related or unrelated to pregnancy is required in women who present with liver dysfunction during pregnancy. This report summarizes the recommendation of an expert panel established by the Italian Association for the Study of the Liver (AISF) on the management of liver disease during pregnancy. The article provides an overview of liver disea…

Cholagogues and CholereticsViral HepatitisBudd-Chiari SyndromeChronic liver diseaseAdrenal Cortex HormoneGastroenterologyHyperemesis gravidarumLiver disease0302 clinical medicinePre-EclampsiaAdrenal Cortex HormonesCholelithiasisMED/12 - GASTROENTEROLOGIAPregnancyHyperemesis GravidarumEclampsiaCholelithiasiThiaminePregnancy Complications InfectiousCholagogues and CholereticSocieties Medical030219 obstetrics & reproductive medicineFatty liverUrsodeoxycholic AcidGastroenterologyCalcium Channel BlockersLiver diseases; Pregnancy; Gastroenterology; HepatologyPregnancy ComplicationAntihypertensive AgentItalyVitamin B ComplexBudd–Chiari syndromeLiver diseases; Pregnancy030211 gastroenterology & hepatologyFemaleCalcium Channel BlockerLiver diseaseHumanViral Hepatitis Vaccinesmedicine.medical_specialtyHELLP SyndromeHepatitis Viral HumanHELLP syndromeCholestasis Intrahepatic03 medical and health sciencesMagnesium SulfateInternal medicinemedicineHumansIntensive care medicineAntihypertensive AgentsLiver diseasesPregnancyEclampsiaHepatologybusiness.industrymedicine.diseasePregnancy ComplicationsFatty LiverPregnancy Liver disease Viral HepatitisPregnancy Complications InfectiouFluid TherapybusinessViral Hepatitis Vaccine
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Extracellular site of action of phenylalkylamines on L-type calcium current in rat ventricular myocytes.

1995

The effects of the phenylalkylamines verapamil, gallopamil, and devapamil on L-type calcium currents (ICa) were studied in ventricular myocytes from rat hearts using the whole-cell patch-clamp technique. In particular, the question was addressed, whether the pharmacological binding sites for these drugs were located at the inner and/or at the outer surface of the cell membrane. Therefore, tertiary verapamil, gallopamil, and devapamil and their corresponding quaternary derivatives were applied either from the outside or the inside of the cell membrane. Extracellular application of verapamil, gallopamil and devapamil (each at 3 microM) reduced ICa to 16.1 +/- 8.6%, 11 +/- 8.9%, and 9.3 +/- 6%…

DevapamilGallopamilPatch-Clamp TechniquesHeart Ventricleschemistry.chemical_elementPharmacologyCalciumRats Sprague-Dawleychemistry.chemical_compoundmedicineExtracellularAnimalsPatch clampGallopamilPharmacologyBinding SitesChemistryCalcium channelCell MembraneGeneral MedicineCalcium Channel BlockersRatsVerapamilcardiovascular systemVerapamilCalcium ChannelsIntracellularmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Biowaiver Monographs for Immediate-Release Solid Oral Dosage Forms: Nifedipine

2015

Literature data relevant to the biopharmaceutical properties of the active pharmaceutical ingredient (API) nifedipine are reviewed to evaluate whether a waiver of in vivo bioequivalence (BE) testing of immediate-release (IR) dosage forms formulated as tablets and soft gelatin capsules is warranted. Nifedipine's solubility and permeability, its therapeutic use and index, pharmacokinetics, food drug interactions, and any reported BE/bioavailability problems were all taken into consideration. Solubility and BA data indicate conclusively that nifedipine is a class II substance of biopharmaceutics classification system (BCS) and that the formulation of drug product plays a key role on the dissol…

DrugNifedipineChemistry Pharmaceuticalmedia_common.quotation_subjectPharmaceutical ScienceCapsulesBioequivalencePharmacologyDosage formExcipientsFood-Drug InteractionsNifedipinePharmacokineticsmedicineAnimalsHumansmedia_commonActive ingredientChemistryCalcium Channel BlockersBiopharmaceutics Classification SystemBioavailabilityIntestinal AbsorptionSolubilityTabletsmedicine.drugJournal of Pharmaceutical Sciences
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Cytosolic Ca2+ and Phosphoinositide Hydrolysis Linked to Constitutively Active α1d-Adrenoceptors in Vascular Smooth Muscle

2003

In the present study, we analyzed changes in intracellular Ca2+ levels and inositol phosphate accumulation related to a population of alpha 1d-adrenoceptors in rat aorta resembling constitutively active receptors. Following intracellular Ca2+ store depletion by noradrenaline in Ca2+-free medium and removal of the agonist, restoration of extracellular Ca2+ induced four signals: a biphasic (transient and sustained) increase in [Ca2+]i, inositol phosphate accumulation, and a contractile response in the aorta. The transient increase in Ca2+, the inositol phosphate accumulation, and the contractile response were not observed in aortae incubated with prazosin or BMY 7378 [8-[2-[4-(2-methoxyphenyl…

Guanethidinemedicine.medical_specialtyVascular smooth muscleInositol PhosphatesPopulationchemistry.chemical_elementCalciumBiologyPhosphatidylinositolsMuscle Smooth VascularAdrenergic AgentsReceptors Adrenergic alpha-1Internal medicinemedicinePrazosinExtracellularAnimalsRats WistarInositol phosphateeducationAortaPharmacologyCalcium metabolismchemistry.chemical_classificationeducation.field_of_studyHydrolysisCalcium Channel BlockersRatsEndocrinologychemistryBiophysicsMolecular MedicineCalciumIntracellularSignal Transductionmedicine.drugJournal of Pharmacology and Experimental Therapeutics
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Possible Ca 2+ -dependent mechanism of apical outer hair cell modulation within the cochlea of the guinea pig

1998

Calcium ions were precipitated with potassium antimonate after injection of the inorganic calcium channel blocker MnCl2 or the inorganic potassium channel blockers BaCl2 or CsCl into the perilymph of the scala vestibuli of the guinea pig. The spatial distribution of the formed histochemical reaction products within the organ of Corti was studied by energy-filtering transmission-electron microscopy. Compared with untreated control ears, the number of the formed precipitates drastically increased at the extracellular side of the lamina reticularis after application of the various inorganic channel blockers. The apical side of the outer hair cells and the intervening Deiter cells were covered …

HistologyTectorial membraneBarium CompoundsGuinea PigsCesiumchemistry.chemical_elementPerilymphCalciumPathology and Forensic MedicineChloridesmedicineAnimalsChemical PrecipitationChannel blockerCochleaLamina reticularisCell BiologyAnatomyCalcium Channel BlockersPerilymphHair Cells Auditory OuterMicroscopy Electronmedicine.anatomical_structurechemistryOrgan of CortiBiophysicsCalciumHair cellElectron Probe MicroanalysisCell and Tissue Research
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Effects of Anandamide and Noxious Heat on Intracellular Calcium Concentration in Nociceptive DRG Neurons of Rats

2007

As an endogenous agonist at the cannabinoid receptor CB1 and the capsaicin-receptor TRPV1, anandamide may exert both anti- and pronociceptive actions. Therefore we studied the effects of anandamide and other activators of both receptors on changes in free cytosolic calcium ([Ca2+]i) in acutely dissociated small dorsal root ganglion neurons (diameter: ≤30 μm). Anandamide (10 μM) increased [Ca2+]iin 76% of the neurons. The EC50was 7.41 μM, the Hill slope was 2.15 ± 0.43 (mean ± SE). This increase was blocked by the competitive TRPV1-antagonist capsazepine (10 μM) and in Ca2+-free extracellular solution. Neither exclusion of voltage-gated sodium channels nor additional blockade of voltage-gate…

Hot TemperatureCannabinoid receptorPolyunsaturated AlkamidesPhysiologyTRPV1TRPV Cation ChannelsArachidonic AcidsPharmacologyCalcium in biologyRats Sprague-Dawleychemistry.chemical_compoundGanglia SpinalPhysical StimulationAnimalsDrug InteractionsDronabinolEgtazic AcidChelating AgentsNeuronsCalcium metabolismAnalysis of VarianceDose-Response Relationship DrugChemistryGeneral NeuroscienceExtracellular FluidAnandamideCalcium Channel BlockersEndocannabinoid systemRatsNociceptionCalciumCapsaicinEndogenous agonistEndocannabinoidsJournal of Neurophysiology
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Intracellular Na+ concentration influences short-term plasticity of glutamate transporter-mediated currents in neocortical astrocytes.

2012

Fast synaptic transmission requires a rapid clearance of the released neurotransmitter from the extracellular space. Glial glutamate transporters (excitatory amino acid transporters, EAATs) strongly contribute to glutamate removal. In this work, we investigated the paired-pulse plasticity of synaptically activated, glutamate transporter-mediated currents (STCs) in cortical layer 2/3 astrocytes. STCs were elicited by local electrical stimulation in layer 4 in the presence of ionotropic glutamate (AMPA and NMDA), GABAA, and GABAB receptor antagonists. In experiments with low [Na+]i (5 mM) intrapipette solution, STCs elicited by paired-pulse stimulation demonstrated paired-pulse facilitation (…

Intracellular FluidPatch-Clamp TechniquesAmino Acid Transport System X-AGBiophysicsNipecotic AcidsAction PotentialsNeocortexAMPA receptorTetrodotoxinBiologyGABAB receptorAnisolesIn Vitro TechniquesSynaptic TransmissionGABA AntagonistsCellular and Molecular NeuroscienceMiceCadmium ChlorideEthers CyclicOximesmedicineGABA transporterAnimalsgamma-Aminobutyric AcidBenzofuransAspartic AcidNeuronal PlasticityGABAA receptorRhodaminesSodiumGlutamate receptorCalcium Channel BlockersElectric StimulationMice Inbred C57BLmedicine.anatomical_structureNeurologyAnimals NewbornAstrocytesExcitatory postsynaptic potentialBiophysicsbiology.proteinNMDA receptorNeuroscienceExcitatory Amino Acid AntagonistsAstrocyteSodium Channel BlockersGlia
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Memory-enhancing and brain protein expression-stimulating effects of novel calcium antagonist in Alzheimer’s disease transgenic female mice

2016

The prevalence of Alzheimer's disease (AD) is higher in females than in males, and causes more severe cognitive, memory and behavioral impairments. Previously, in male transgenic (Tg) APPSweDI mice, we reported that the novel lipophilic 1,4-dihydropyridine (DHP) derivative AP-12 crossed the blood-brain barrier, blocked neuronal and vascular calcium channels, changed brain protein expression and improved behavior. In this study, we used female Tg APPSweDI mice to assess the effects of AP-12 on behavior, and brain protein expression, with a particular focus on those of the GABAergic system. The results showed that in female Tg mice, similar to male Tg mice, AP-12 improved spatial learning/mem…

Male0301 basic medicineCingulate cortexDihydropyridinesmedicine.medical_specialtyElevated plus mazeVesicular Inhibitory Amino Acid Transport ProteinsHippocampusMice TransgenicWater mazeBiologyHippocampal formationGyrus CinguliHippocampusArticleAmyloid beta-Protein PrecursorMice03 medical and health sciences0302 clinical medicineAlzheimer DiseaseMemoryInternal medicineNeuroplasticitymedicineAnimalsGABAergic NeuronsMaze LearningPharmacologyAmyloid beta-PeptidesNeuronal PlasticityGlutamate DecarboxylaseCalcium Channel BlockersUp-RegulationDisease Models Animal030104 developmental biologyEndocrinologyAnti-Anxiety AgentsBlood-Brain BarrierSynaptic plasticityGABAergicCalciumFemale030217 neurology & neurosurgeryPharmacological Research
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