Search results for "Calcium channels"

showing 10 items of 67 documents

Synaptopodin regulates denervation-induced homeostatic synaptic plasticity

2013

Synaptopodin (SP) is a marker and essential component of the spine apparatus (SA), an enigmatic cellular organelle composed of stacked smooth endoplasmic reticulum that has been linked to synaptic plasticity. However, SP/SA-mediated synaptic plasticity remains incompletely understood. To study the role of SP/SA in homeostatic synaptic plasticity we here used denervation-induced synaptic scaling of mouse dentate granule cells as a model system. This form of plasticity is of considerable interest in the context of neurological diseases that are associated with the loss of neurons and subsequent denervation of connected brain regions. In entorhino-hippocampal slice cultures prepared from SP-de…

Patch-Clamp TechniquesDendritic SpinesGreen Fluorescent ProteinsNonsynaptic plasticityMice TransgenicTetrodotoxinBiologyIn Vitro TechniquesHippocampusReceptors N-Methyl-D-AspartateMiceHomeostatic plasticitySynaptic augmentationMetaplasticityAnimalsEntorhinal CortexHomeostasisPromoter Regions GeneticMultidisciplinarySynaptic scalingNeuronal PlasticityMicrofilament ProteinsRyanodine Receptor Calcium Release ChannelBiological SciencesDenervationSpine apparatusMice Inbred C57BLSynaptic fatigueSynaptic plasticityDentate GyrusSynapsesCalcium ChannelsNeuroscience
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Kinetics and state-dependent effects of verapamil on cardiac L-type calcium channels.

1996

The voltage dependence and the kinetics of block by verapamil of L-type calcium current (ICa) were investigated in ventricular myocytes from rat hearts using the whole-cell patch-clamp technique. ICa was elicited repetitively in response to depolarizing voltage pulses from -80 mV to 0 mV at different pulse intervals and durations. Verapamil reduced the magnitude of ICa in a frequency-dependent manner without tonic component. The time course of ICa remained unchanged suggesting that not open but inactivated channels were affected by the drug. The interaction of verapamil with inactivated channels was investigated by the application of twin pulses. In the presence of verapamil, the duration o…

PharmacologyVoltage-dependent calcium channelChemistryKineticsTime constantAnalytical chemistryDepolarizationHeartGeneral MedicineIn Vitro TechniquesCalcium Channel BlockersRatsKineticsDrug concentrationNuclear magnetic resonanceVerapamilState dependentBariummedicineVerapamilAnimalsL-type calcium channelCalcium Channelsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Bax inhibitor-1 is likely a pH-sensitive calcium leak channel, not a H+/Ca2+ exchanger.

2014

The endoplasmic reticulum (ER) plays a key role in the synthesis, folding, and sorting of proteins, and disturbances of this delicate system can cause cell death. The ER also serves as the major intracellular calcium (Ca(2+)) store, and release of Ca(2+) from this store controls diverse cellular functions. At the interface of both these functions of the ER is Bax inhibitor-1 (BI-1), an evolutionarily conserved multifunctional protein that mediates Ca(2+) efflux from the ER and protects against ER stress. Several mechanisms have been proposed to explain how BI-1 might mediate Ca(2+) efflux from the ER. Chang et al. present structural evidence that a bacterial homolog of BI-1, BsYetJ, is a pH…

Programmed cell deathProtein familyProteolipidschemistry.chemical_elementCalciumBiologyEndoplasmic ReticulumBiochemistryModels BiologicalCalcium in biologySpecies SpecificityHumansMolecular BiologyBAX inhibitor 1Endoplasmic reticulumCell MembraneMembrane ProteinsCell BiologyHydrogen-Ion ConcentrationCell biologychemistryUnfolded protein responseCalciumEffluxCalcium ChannelsApoptosis Regulatory ProteinsBacillus subtilisScience signaling
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Stable expression in HEK-293 cells of the rat alpha3/beta4 subtype of neuronal nicotinic acetylcholine receptor.

1996

The alpha3/beta4 subtype of neuronal nicotinic acetylcholine receptor (nAChR) was stably expressed in human embryonic kidney (HEK) 293 cells that co-expressed a voltage-gated Ca2+ channel. alpha3/beta4-nAChR-expressing clones were identified using the fura-2 Ca2+ imaging technique, and were further characterised by single-cell and whole-cell patch-clamp studies. Acetylcholine (ACh) induced fast activating currents which showed desensitisation and inward rectification. The conductance of the ACh-activated channel was 29 pS. The order of potency of the nicotinic agonists tested was cytisine approximately = nicotine > acetylcholine. The EC50 value for ACh was 145 microM; the Hill coefficient w…

Stable expressionPatch-Clamp Techniquesα3/β4 nAChRBiophysicsNicotinic AntagonistsPharmacologyReceptors NicotinicTransfectionBiochemistryCell LineGanglionic nAChRCa2+ imagingGanglion type nicotinic receptorStructural BiologyMuscarinic acetylcholine receptorGeneticsmedicineAnimalsHumansNicotinic AgonistsNicotinic AntagonistHEK cellMolecular BiologyNeuronsurogenital systemChemistryMuscarinic acetylcholine receptor M3Cell BiologyAcetylcholineRecombinant ProteinsRatsNicotinic acetylcholine receptorNicotinic agonistCalciumCalcium ChannelsAlpha-4 beta-2 nicotinic receptorAcetylcholinemedicine.drugFEBS letters
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Astrocytic potassium and calcium channels as integrators of the inflammatory and ischemic CNS microenvironment.

2021

Abstract Astrocytes are key regulators of their surroundings by receiving and integrating stimuli from their local microenvironment, thereby regulating glial and neuronal homeostasis. Cumulating evidence supports a plethora of heterogenic astrocyte subpopulations that differ morphologically and in their expression patterns of receptors, transporters and ion channels, as well as in their functional specialisation. Astrocytic heterogeneity is especially relevant under pathological conditions. In experimental autoimmune encephalomyelitis (EAE), a mouse model of multiple sclerosis (MS), morphologically distinct astrocytic subtypes were identified and could be linked to transcriptome changes dur…

Voltage-gated ion channelVoltage-dependent calcium channelClinical BiochemistryExperimental autoimmune encephalomyelitisBiologymedicine.diseaseBiochemistryPotassium channelTransient receptor potential channelMicemedicine.anatomical_structureAstrocytesmedicinePotassiumAnimalsCalcium ChannelsMolecular BiologyNeuroscienceIon channelNeuroinflammationAstrocyteBiological chemistryReferences
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Diagnostic and therapeutic aspects of hemiplegic migraine

2020

Hemiplegic migraine (HM) is a clinically and genetically heterogeneous condition with attacks of headache and motor weakness which may be associated with impaired consciousness, cerebellar ataxia and intellectual disability. Motor symptoms usually last <72 hours and are associated with visual or sensory manifestations, speech impairment or brainstem aura. HM can occur as a sporadic HM or familiar HM with an autosomal dominant mode of inheritance. Mutations in CACNA1A, ATP1A2 and SCN1A encoding proteins involved in ion transport are implicated. The pathophysiology of HM is close to the process of typical migraine with aura, but appearing with a lower threshold and more severity. We review…

Weaknessmedicine.medical_specialtyMigraine with AuraElectroencephalographyMotor symptomsDiagnosis Differentialclinical neurology; EEG; headache; ion transportion transport03 medical and health sciences0302 clinical medicinePhysical medicine and rehabilitationIntellectual disabilitymedicineHumansclinical neurology1506EEGMigraine030304 developmental biology0303 health sciencesCerebellar ataxiamedicine.diagnostic_testbusiness.industryDisease ManagementEEG; clinical neurology; headache; ion transportmedicine.diseasePedigreeClinical neurologyImpaired consciousnessPsychiatry and Mental healthMutationHemiplegic migraineSettore MED/26 - NeurologiaSurgeryCalcium ChannelsNeurology (clinical)medicine.symptombusinessheadache030217 neurology & neurosurgeryJournal of Neurology, Neurosurgery & Psychiatry
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(±)- BIGI-3h: Pentatarget-Directed Ligand combining Cholinesterase, Monoamine Oxidase, and Glycogen Synthase Kinase 3β Inhibition with Calcium Channe…

2021

Multitarget-directed ligands (MTDLs) are considered a promising therapeutic strategy to address the multifactorial nature of Alzheimer's disease (AD). Novel MTDLs have been designed as inhibitors of human acetylcholinesterases/butyrylcholinesterases, monoamine oxidase A/B, and glycogen synthase kinase 3β and as calcium channel antagonists via the Biginelli multicomponent reaction. Among these MTDLs, (±)-BIGI-3h was identified as a promising new hit compound showing in vitro balanced activities toward the aforementioned recognized AD targets. Additional in vitro studies demonstrated antioxidant effects and brain penetration, along with the ability to inhibit the aggregation of both τ protein…

cholinesterasePhysiologyMonoamine oxidaseCognitive NeuroscienceLigandPharmacologyLigandsCalcium ChannelBiochemistry03 medical and health sciences0302 clinical medicineAlzheimer DiseaseIn vivoGSK-3HumansCholinesterasesCholinesterase InhibitorBiginelli reactionAlzheimer's disease; Biginelli reaction; calcium channel; cholinesterases; GSK 3β; MAO; Calcium Channel Blockers; Calcium Channels; Cholinesterase Inhibitors; Glycogen Synthase Kinase 3 beta; Humans; Ligands; Monoamine Oxidase; Alzheimer DiseaseMonoamine OxidaseGSK3B030304 developmental biologyCholinesterase0303 health sciencesGlycogen Synthase Kinase 3 betaVoltage-dependent calcium channelbiologyChemistryCalcium channelCell BiologyGeneral MedicineAlzheimer's diseaseCalcium Channel BlockersCalcium channel GSK 3β MAOMAObiology.proteinCalcium ChannelsCholinesterase InhibitorsGSK 3βMonoamine oxidase ACalcium Channel BlockerAlzheimer’s disease030217 neurology & neurosurgeryHuman
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Resonance properties of GABAergic interneurons in immature GAD67-GFP mouse neocortex.

2014

Subthreshold resonance is a characteristic membrane property of different neuronal classes, is critically involved in the generation of network oscillations, and tunes the integration of synaptic inputs to particular frequency ranges. In order to investigate whether neocortical GABAergic interneurons show resonant behavior already during early postnatal development, we performed whole-cell patch-clamp recordings from visually identified interneurons in supragranular layers of parietal regions in coronal neocortical slices from postnatal day (P) P6-P13 GAD67-GFP knock-in mice. Subthreshold resonance was analyzed by injection of sinusoidal current with varying frequency. About 50% of the inve…

genetic structuresSubthreshold membrane potential oscillationsGlutamate decarboxylaseGreen Fluorescent ProteinsNeocortexSodium ChannelsMembrane PotentialsCalcium Channels T-TypeMiceInterneuronsParietal LobemedicineAnimalsGene Knock-In TechniquesGABAergic NeuronsMolecular BiologyMembrane potentialNeocortexSubthreshold conductionChemistryGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyGeneral NeuroscienceResonanceMembrane hyperpolarizationmedicine.anatomical_structurenervous systemGABAergicNeurology (clinical)NeuroscienceDevelopmental BiologyBrain research
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Stromal Interaction Molecule 1 (STIM1) Is Involved in the Regulation of Mitochondrial Shape and Bioenergetics and Plays a Role in Oxidative Stress

2012

Calcium ions are involved in a plethora of cellular functions including cell death and mitochondrial energy metabolism. Store-operated Ca(2+) entry over the plasma membrane is activated by depletion of intracellular Ca(2+) stores and is mediated by the sensor STIM1 and the channel ORAI1. We compared cell death susceptibility to oxidative stress in STIM1 knock-out and ORAI1 knockdown mouse embryonic fibroblasts and in knock-out cells with reconstituted wild type and dominant active STIM1. We show that STIM1 and ORAI1 deficiency renders cells more susceptible to oxidative stress, which can be rescued by STIM1 and ORAI1 overexpression. STIM1 knock-out mitochondria are tubular, have a higher Ca…

inorganic chemicalsProgrammed cell deathORAI1 ProteinEukaryotic Initiation Factor-2Active Transport Cell NucleusApoptosisMitochondrionBiologymedicine.disease_causeBiochemistryMiceeIF-2 KinasemedicineAnimalsStromal Interaction Molecule 1PhosphorylationMolecular BiologyTranscription factorCells CulturedMice KnockoutEIF-2 kinaseMembrane GlycoproteinsEndoplasmic reticulumMolecular Bases of DiseaseSTIM1Cell BiologyFibroblastsEmbryo MammalianMitochondriaCell biologyOxidative Stressbiology.proteinCalciumCalcium ChannelsEnergy MetabolismIntracellularOxidative stressJournal of Biological Chemistry
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Alteration of loosely bound calcium in the guinea pig organ of Corti after treatment with diltiazem as calcium channel blocker

1997

After oral administration of the organic calcium channel blocker diltiazem to guinea pigs for 7 days, calcium ions were precipitated with potassium antimonate in the cochleae. The spatial distribution of the precipitates was studied by energy-filtering transmission electron microscopy and the amount of the ultrastructural reaction products formed was determined semiquantitatively by an image processing system. Compared with untreated control ears, the number of the formed precipitates was reduced drastically in the inner hair cells after diltiazem treatment. In addition, electron microscopic analysis revealed that the number of calcium precipitates attached at the basolateral membrane of th…

medicine.drug_classGuinea PigsAdministration Oralchemistry.chemical_elementCalcium channel blockerCalciumGuinea pigDiltiazemmedicineAnimalsDiltiazemOrgan of CortiLamina reticularisVoltage-dependent calcium channelbusiness.industryGeneral MedicineAnatomyCalcium Channel BlockersMicroscopy Electronmedicine.anatomical_structureOtorhinolaryngologychemistryOrgan of CortiBiophysicsUltrastructureCalciumCalcium Channelsbusinessmedicine.drugEuropean Archives of Oto-Rhino-Laryngology
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