Search results for "Cancer"

showing 10 items of 11546 documents

Digital control circuitry for the p53 dynamics in cancer cell and apoptosis

2010

Abstract Experimental work and theoretical models deduce a “digital” response of the p53 transcription factor when genomic integrity is damaged. The mutual influence of p53 and its antagonist, the Mdm2 oncogene, is closed in a feedback. This paper proposes an aerospace-based architecture for translating the p53/Mdm2/DNA damage network into a digital circuitry in which the optimal control theory is applied for obtaining the requested dynamic evolutions of some considered cell species for repairing a DNA damage. The purpose of this paper is to demonstrate the usefulness of such digital circuitry design to detect and predict the cell species dynamics for shedding light on their inner and mutua…

p53General Immunology and MicrobiologyMechanism (biology)DNA damageQH301-705.5General NeuroscienceapoptosisWiring diagramCell fate determinationBiologycellular circuitryBioinformaticsOptimal controlGeneral Biochemistry Genetics and Molecular Biologyprotein networks signallingfeedback controlCancer cellDigital controlBiology (General)General Agricultural and Biological SciencesBiological systemTranscription factorOpen Life Sciences
researchProduct

Mutations in p53 Gene Exons in a Sample from the South of Spain in Oral Cancer

2021

[Background+ Cancer is a genetic disease caused by mutations in DNA and epigenetic alterations that control gene expression. The majority of epidermoid carcinomas develop within the fields of epithelial genetic alterations. The mechanisms underlying tumorigenesis of epidermoid carcinoma are as yet unknown; therefore, precise identification of the risk factors is needed.

p53Geneticseducation.field_of_studyOral Medicine and PathologyResearchOral cancerPopulationExonCancerBiologymedicine.diseasemedicine.disease_causeChromosome 17 (human)ExonEpidermoid carcinomaDysplasiamedicineeducationCarcinogenesisGeneral DentistryGeneMutationsUNESCO:CIENCIAS MÉDICAS
researchProduct

THE HDAC INHIBITOR ITF2357 (GIVINOSTAT) AS A KEY PLAYER IN EPIGENETIC TARGETING OF MELANOMA AND COLON CANCER CELLS

2023

Histone deacetylase inhibitors (HDACIs) are epigenetic compounds that have been recently considered for their promising anti-tumor activity. The aim of this PhD thesis was to elucidate and characterize the anti-tumor effect of the HDAC inhibitor ITF2357 (Givinostat) in melanoma and colon cancer cells that are characterized by oncogenic BRAF mutations. Interestingly, data reported in this thesis demonstrate that ITF2357 exerts a remarkable anti-tumor effect in melanoma cells by inducing a switch from a pro-survival autophagy to caspase-dependent apoptosis. The thesis provides the first evidences that ITF2357 is able to target oncogenic BRAF and oncogenic p53. The ITF2357 decreasing effect on…

p53HDAC inhibitorSettore BIO/10 - BiochimicaAutophagyEpigeneticApoptosisMelanomaColon cancerBRAF
researchProduct

Targeting HSP90 with the small molecule inhibitor AUY922 (luminespib) as a treatment strategy against hepatocellular carcinoma

2018

Hepatocellular carcinoma (HCC) is a highly malignant tumor that responds very poorly to existing therapies, most probably due to its extraordinary inter- and intra-tumor molecular heterogeneity. The modest therapeutic response to molecular targeted agents underlines the need for new therapeutic approaches for HCC. In our study, we took advantage of well-characterized human HCC cell lines, differing in transcriptomic subtypes, DNA mutation and amplification alterations, reflecting the heterogeneity of primary HCCs, to provide a preclinical evaluation of the specific heat shock protein 90 (HSP90) inhibitor AUY922 (luminespib). Indeed, HSP90 is highly expressed in different tumor types, but it…

p53MaleCancer ResearchCellTranscriptome0302 clinical medicineHCCbeta CateninAged 80 and overLuminespibAUY922Liver NeoplasmsHep G2 CellsSorafenibMiddle AgedUp-RegulationGene Expression Regulation Neoplasticmedicine.anatomical_structureOncologyCaspases030220 oncology & carcinogenesisHepatocellular carcinomaFemaleNUPR1medicine.drugAdultSorafenibCarcinoma Hepatocellularβ-CateninMice NudeAntineoplastic AgentsSmall Molecule Libraries03 medical and health sciencesDownregulation and upregulationIn vivoCell Line TumormedicineHSP90AnimalsHumansHSP90 Heat-Shock ProteinsAgedCell growthbusiness.industryMcl-1IsoxazolesResorcinolsHCCSmedicine.diseasedigestive system diseasesMutationCancer researchTranscriptomebusinessInternational Journal of Cancer
researchProduct

Plausible Role of Estrogens in Pathogenesis, Progression and Therapy of Lung Cancer

2021

Malignant neoplasms are among the most common diseases and are responsible for the majority of deaths in the developed world. In contrast to men, available data show a clear upward trend in the incidence of lung cancer in women, making it almost as prevalent as breast cancer. Women might be more susceptible to the carcinogenic effect of tobacco smoke than men. Furthermore, available data indicate a much more frequent mutation of the tumor suppressor gene-p53 in non-small cell lung cancer (NSCLC) female patients compared to males. Another important factor, however, might lie in the female sex hormones, whose mitogenic or carcinogenic effect is well known. Epidemiologic data show a correlatio…

p53MaleLung NeoplasmsHealth Toxicology and Mutagenesismedicine.medical_treatmentlcsh:MedicineEstrogen receptorReviewNSCLCsex hormonessex hormone03 medical and health sciences0302 clinical medicineBreast cancerA549Carcinoma Non-Small-Cell LungmedicineCarcinomaestrogenNeoplasmEstrogen Receptor betaHumansLung cancerCarcinogennon-small cell lung cancer030304 developmental biology0303 health sciencesLungbusiness.industrylcsh:RPublic Health Environmental and Occupational Health17β-estradiolEstrogen Receptor alphaHormone replacement therapy (menopause)Estrogensmedicine.diseaselung adenocarcinomarespiratory tract diseaseslung cancermedicine.anatomical_structure17- estradiolReceptors Estrogen030220 oncology & carcinogenesisCancer researchFemalebusinessestrogen receptorInternational Journal of Environmental Research and Public Health
researchProduct

Molecular mechanisms of MYCN-dependent apoptosis and the MDM2-p53 pathway: an Achille’s heel to be exploited for the therapy of MYCN amplified neurob…

2012

The p53 oncosuppressor is very seldom mutated in neuroblastoma, but several mecha- nisms cooperate to its functional inactivation in this tumor. Increased MDM2 levels, due to genetic amplification or constitutive inhibition of p14ARF, significantly contribute to this event highlighting p53 reactivation as an attractive perspective for neuroblastoma treat- ment. In addition to its role in tumorigenesis, MYCN sensitizes untransformed and cancer cells to apoptosis. This is associated to a fine modulation of the MDM2-p53 pathway Indeed MYCN induces p53 and MDM2 transcription, and, by evoking a DNA damage response (DDR), it stabilizes p53 and its proapoptotic kinase Homeodomain Interacting Prote…

p53Programmed cell deathCancer ResearchHMGA1HIPK2Biologymedicine.disease_causelcsh:RC254-28203 medical and health sciencesNeuroblastoma0302 clinical medicineMDM2NeuroblastomaMYCNmedicineProtein kinase Aneoplasms030304 developmental biology0303 health sciencesKinaseHMGA1amedicine.diseaselcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensHMGA13. Good healthOncology030220 oncology & carcinogenesisCancer cellPerspective ArticleMDM2-antagonistsbiology.proteinCancer researchMdm2CarcinogenesisMDM2-antagonistFrontiers in Oncology
researchProduct

Oral paracoccidioidomycosis:a retrospective study of 95 cases from a single center and literature review

2023

The ecoepidemiological panorama of paracoccidioidomycosis (PCM) is dynamic and still ongoing in Brazil. In particular, data about the oral lesions of PCM are barely explored. The aim of this study was to report the clinicopathological features of individuals diagnosed with oral PCM lesions at an oral and maxillofacial pathology service in Rio de Janeiro, Brazil, in the light of a literature review. A retrospective study was conducted on oral biopsies obtained from 1958 to 2021. Additionally, electronic searches were conducted in PubMed, Embase, Scopus, Web of Science, Latin American and Caribbean Center on Health Sciences Information, and Brazilian Library of Dentistry to gather information…

paediatrichead and neck neoplasmsOtorhinolaryngologyadolescentSurgeryepidemiologybiopsyoral cancerGeneral DentistryUNESCO:CIENCIAS MÉDICAS
researchProduct

Reliability of Frail and Barthel Tests for Detecting Frailty in Palliative Oncological Patients in a Home Hospitalization Unit: A Comparative Study.

2022

Cancer is a condition that can increase the risk of frailty. In addition, palliative oncological patients in home hospitalization can find their activities of daily living affected. The main objective was to measure the degree of frailty in the oncological population in home hospitalization comparing Barthel and Frail-VIG Indexes. This is a descriptive cross-sectional study. A sample of oncological patients in home hospitalization (n = 50) that included 27 men and 23 women were recruited, and disability due to frailty was measured using the VIG frailty index and the Barthel scale for Activities of Daily Living (ADLs). Spearman’s correlation coefficients were categorized as weak (rs &l…

palliative carePaliative careFrailtyHospitalització domiciliàriafrailty; palliative care; cancer; home care; frail-VIG indexPaleontologyCáncerCuidados en casaCuidados paliativosHome careGeneral Biochemistry Genetics and Molecular BiologyOncologíaSpace and Planetary ScienceFrail-VIG indexTractament pal·liatiuÍndice VIG de vulnerabilidadEnfermeríaCàncer Pacientshuman activitiesVulnerabilidadEcology Evolution Behavior and SystematicsCancerLife (Basel, Switzerland)
researchProduct

Elpas trūkums onkoloģiskiem pacientiem. Biežums un problēmas risinājums

2018

Elpas trūkums ir viens no visbiežākajiem un mokošākajiem vēža pacientu simptomiem. Katram pacientam šo simptomu etioloģija, patoģenēze un smaguma pakāpe atšķiras, tas traucē efektīvai ārstēšanai un ir nepieciešama individualizēta pieeja. Šis pētījums izvērtē elpas trūkuma sastopamību un kā tā tiek menedžēta starp termināliem vēža slimniekiem, kā arī simptomu dinamiku un individuālās pacientu atšķirības. Tā kā elpas trūkums ir subjektīvs simptoms un vāji korelē ar objektīviem veselības parametriem, šādi mērījumi šajā pētījumā netiks iekļauti, liekot uzsvaru uz dažādām ārstēšanas metodēm gan farmakoloģiskām, gan nefarmakoloģiskām. Lai to paveiktu, tika izveidota anketa, kas tika izsniegta 46 …

palliative carebreathlessnesscancerdyspnoeadyspneaMedicīna
researchProduct

Genetic determined downregulation of both type 1 and type 2 cytokine pathways might be protective against pancreatic cancer

2008

pancreatic cancergenetic polymorphismCytokine
researchProduct