Search results for "Cardiology"

showing 10 items of 6064 documents

High Intestinal Cholesterol Absorption Is Associated With Cardiovascular Disease and Risk Alleles in ABCG8 and ABO

2013

Objectives This study sought to determine whether high intestinal cholesterol absorption represents a cardiovascular risk factor and to link ABCG8 and ABO variants to cardiovascular disease (CVD). Background Plant sterol–enriched functional foods are widely used for cholesterol lowering. Their regular intake yields a 2-fold increase in circulating plant sterol levels that equally represent markers of cholesterol absorption. Variants in ABCG8 and ABO have been associated with circulating plant sterol levels and CVD, thereby suggesting atherogenic effects of plant sterols or of cholesterol uptake. Methods The cholestanol-to-cholesterol ratio (CR) was used as an estimate of cholesterol absorpt…

medicine.medical_specialtySingle-nucleotide polymorphism030204 cardiovascular system & hematologyHigh cholesterolIntestinal absorption03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEzetimibeInternal medicineABO blood group systemmedicineRisk factor030304 developmental biology2. Zero hunger0303 health sciencesCholesterolbusiness.industrymedicine.diseaseEndocrinologychemistryIntestinal cholesterol absorptionlipids (amino acids peptides and proteins)businessCardiology and Cardiovascular Medicinemedicine.drugJournal of the American College of Cardiology
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Uncoupling Protein 2 as genetic risk factor for systemic lupus erythematosus: association with malondialdehyde levels and intima media thickness

2020

BACKGROUND Increased oxidative stress potentially leads to accelerated atherosclerosis and, consequently, cardiovascular diseases, the main cause of death in systemic lupus erythematous (SLE). To gain insight into these mechanisms, we studied the association of uncoupling protein (UCP) 2 genetic variants, gene involved in the mitochondrial production of reactive oxygen species, and oxidative stress with SLE and the presence of atherosclerosis. METHODS Genetic analysis of the UCP2 -866G/A and UCP2 Ins/Del polymorphisms was performed in 45 SLE patients and 36 healthy controls by RFLP-PCR. Oxidation status was determined by measuring malondialdehyde (MDA) levels. Presence of subclinical athero…

medicine.medical_specialtySingle-nucleotide polymorphism030204 cardiovascular system & hematologymedicine.disease_causeCarotid Intima-Media Thickness03 medical and health scienceschemistry.chemical_compound0302 clinical medicineGeneticRisk FactorsMalondialdehydeInternal medicineGenotypemedicineHumansUncoupling proteinUncoupling Protein 2030212 general & internal medicineAlleleskin and connective tissue diseaseschemistry.chemical_classificationReactive oxygen speciesbusiness.industryMalondialdehydeLupus erythematosus systemicEndocrinologychemistryIntima-media thicknessCardiology and Cardiovascular MedicinebusinessCardiovascular diseases.Oxidative stressMinerva Cardioangiologica
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Obstructive sleep apnea and blood pressure in young hypertensives: does it matter?

2020

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medicine.medical_specialtySleep Apnea Obstructivebusiness.industryBlood PressureSettore MED/10 - Malattie Dell'Apparato Respiratoriomedicine.diseaseObstructive sleep apneaBlood pressureInternal medicineHypertensionEmergency MedicineInternal MedicineCardiologyMedicineHumansBlood Pressure Humans Hypertension Sleep Apnea Obstructivebusiness
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Impact of Nonstationarities on Short Heart Rate Variability Recordings During Obstructive Sleep Apnea

2017

Obstructive sleep apnea (OSA) is a sleep disorder characterized by breathing pauses due to collapse of the upper airways. During OSA the autonomic modulation, as noninvasively assessed through heart period (HP) variability, is altered in a time-varying way even though time-varying properties of HP fluctuations are often disregarded by HP variability studies. We performed a time domain analysis computed over very short epochs corresponding to the sole OSA events explicitly accounting for HP variability nonstationarities. Length-matched epochs were extracted during OSA and quiet sleep (SLEEP) in 13 subjects suffering from OSA (11 males, age 55±11, apnea-hypopnea index 44±19). Mean HP, varianc…

medicine.medical_specialtySleep disorderbusiness.industryComputer Science (all)030204 cardiovascular system & hematologymedicine.diseaseSleep in non-human animalsAutonomic controlnervous system diseasesrespiratory tract diseasesObstructive sleep apnea03 medical and health sciencesQuiet sleep0302 clinical medicinestomatognathic systemInternal medicineSettore ING-INF/06 - Bioingegneria Elettronica E InformaticaBreathingmedicineCardiologyHeart rate variabilityAutonomic modulationCardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgeryComputing in Cardiology Conference (CinC)
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Obstructive sleep apnoea syndrome: What the anesthesiologist should know

2016

Obstructive sleep apnoea syndrome (OSAS) is a rather common sleep disorder and constitutes a risk or an aggravating factor for various underlying diseases. OSAS is characterised by repeated upper airway collapse during sleep causing fragmented sleep, hypoxemia and hypercapnia. It may also cause considerable changes in intrathoracic pressure and an increase in sympathetic nervous activity, which represent the basis of associated pathologies such as arterial hypertension, ischaemic heart disease, diabetes mellitus, stroke and sudden death [1]. Moreover, there is a well-established association between OSAS and postoperative complications [2, 3]. Nevertheless, a significant proportion of patien…

medicine.medical_specialtySleep disorderbusiness.industrymedicine.medical_treatmentSleep apneaSleep apneaPerioperativemedicine.diseaseSudden deathrespiratory tract diseasesHypoxemiaInternal medicinemedicineCardiologyContinuous positive airway pressuremedicine.symptomComplicationbusinessStroke
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Growth and differentiation factor 11 (GDF11): Functions in the regulation of erythropoiesis and cardiac regeneration

2015

International audience; Members of the TGF-β superfamily transduce their signals through type I and II receptor serine/threonine kinases. The binding of activins to activin type IIA (ActRIIA) or type IIB (ActRIIB) receptors induces the recruitment and phosphorylation of an activin type I receptor (ALK4 and/or ALK7), which then phosphorylates the Smad2 and Smad3 intracellular signaling proteins. The regulation of members of the TGF-β family is known to be complex, because many proteins able to bind the ligands and inhibit their activities have been identified. Growth and differentiation factor 11 (Gdf11) belongs to the TGF-β family. GDF11, like other members of the TGF-β superfamily, is prod…

medicine.medical_specialtySmad2 ProteinProtein Serine-Threonine Kinases030204 cardiovascular system & hematologyBiology03 medical and health sciences0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicineTGF beta signaling pathway[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineHumansRegeneration[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyPharmacology (medical)PhosphorylationCCL11Activin type 2 receptors030304 developmental biologyPharmacology0303 health sciencesR-SMADcardiac regenerationGrowth differentiation factorHeartActivins[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemCell biologyBMPR2Growth Differentiation FactorsEndocrinologyBone Morphogenetic ProteinsGDF11Smad2 ProteinSignal transductionActivin Receptors Type IerythropoiesisACVR2BSignal TransductionPharmacology & Therapeutics
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LDL size and subclasses in patients with abdominal aortic aneurysm

2009

Abstract Since the type of dyslipidemia in patients with abdominal aortic aneurysm (AAA) is still insufficiently defined, we measured plasma lipids and analyzed LDL size and subclasses by gradient gel electrophoresis in 30 male patients (69±6 years, BMI: 27±3) with newly diagnosed AAA and in 26 age- and BMI-matched male healthy controls. Patients with AAA had lower HDL-cholesterol ( p p =.0002) and smaller LDL size ( p p =.0210) in relation to controls. Multivariate analysis also showed that small LDL size was independently associated with the presence of AAA ( p =.0350). Increased levels of small, dense LDL may therefore represent a common feature in patients with AAA.

medicine.medical_specialtySmall dense ldl610 Medicine & healthmacromolecular substancesDense LDLSmallLDL sizeGastroenterology2705 Cardiology and Cardiovascular MedicineAortic aneurysmInternal medicinePlasma lipidsmedicineIn patientcardiovascular diseasesbusiness.industrymedicine.diseaseAbdominal aortic aneurysmEndocrinologymedicine.anatomical_structureMale patientcardiovascular systemAbdominal aortic aneurysmAbdomenlipids (amino acids peptides and proteins)10029 Clinic and Policlinic for Internal MedicineaterosclerosiCardiology and Cardiovascular MedicinebusinessDyslipidemia
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Lipid triad or atherogenic lipoprotein phenotype: a role in cardiovascular prevention?

2005

The term "lipid triad" or "atherogenic lipoprotein phenotype" has been introduced to describe a common form of dyslipidemia, characterized by three lipid abnormalities: increased plasma triglyceride levels, decreased HDL-cholesterol concentrations and the presence of small, dense LDL particles. It has been suggested that the clinical importance of the atherogenic lipoprotein phenotype probably exceeds that of LDL-cholesterol, because many more patients with coronary artery disease are found to have this trait than hypercholesterolaemia. There is a body of evidence that therapies effective against plasma HDL-cholesterol and triglycerides are associated with a strong reduction of cardiovascul…

medicine.medical_specialtySmall dense ldlBiologyCoronary artery diseaseCardiovascular preventionInternal medicineInternal MedicinemedicineHumansNational Cholesterol Education ProgramDyslipidemiasHypolipidemic AgentsLDL HDL triglycerides preventionBiochemistry (medical)LDL Particle SizeTriad (anatomy)Atherogenic lipoprotein phenotypemedicine.diseaseAtherosclerosisLipoproteins LDLEndocrinologymedicine.anatomical_structureCardiovascular Diseaseslipids (amino acids peptides and proteins)Cardiology and Cardiovascular MedicineDyslipidemiaJournal of atherosclerosis and thrombosis
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Tratamiento integral de la dislipidemia diabética: beneficios y nuevas alternativas terapéuticas

2010

Diabetic dyslipidemia, characterized by the lipid triad (elevated plasma triglycerides, low HDL cholesterol and predominance of small, dense LDL particles), is a significant contributor to the elevated cardiovascular risk of type 2 diabetic patients. Statin monotherapy has shown, in different prospective trials, significant reductions in cardiovascular events and mortality. However, the residual risk in these subjects remains elevated, probably due to the incomplete control of diabetic dyslipidemia. In this review we discuss the global therapeutic approach, underlying the need of combining statins with agents that more effective in reducing triglycerides and elevating HDL cholesterol, even …

medicine.medical_specialtySmall dense ldlStatinmedicine.diagnostic_testCholesterolbusiness.industrymedicine.drug_classnutritional and metabolic diseasesGeneral MedicineResidual riskClinical trialchemistry.chemical_compoundTherapeutic approachEndocrinologychemistryInternal medicineHypolipidemic AgentsmedicineCardiologylipids (amino acids peptides and proteins)Lipid profilebusinessMedicina Clínica
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Update on the role of the atherogenic lipoprotein phenotype in cardiovascular prevention

2009

Higher plasma triglyceride levels and decreased HDL-cholesterol concentrations are usually accompanied by the presence of small, dense LDL in the so-called lipid triad or ‘atherogenic lipoprotein phenotype’. This phenotype is highly atherogenic and its prevalence may suggest an even higher overall burden of atherosclerotic disease as compared with that associated with hypercholesterolemia. As stated by the National Cholesterol Education Program Adult Treatment Panel III, there is evidence suggesting each component of this lipid triad is individually atherogenic. However, the relative contribution of each component cannot be easily determined. Therefore, it has been suggested to consider th…

medicine.medical_specialtySmall dense ldlbusiness.industryAtherosclerotic diseaseAtherogenic lipoprotein phenotype dense LDL gradient gel electrophoresis HDL-cholesterol triglyceridesAtherogenic lipoprotein phenotypePhenotypeEndocrinologyCardiovascular preventionPlasma triglycerideInternal medicineMolecular MedicineMedicinelipids (amino acids peptides and proteins)Risk factorCardiology and Cardiovascular MedicinebusinessNational Cholesterol Education ProgramFuture Cardiology
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