Search results for "Cardiomyopathy"

showing 10 items of 242 documents

Cardiomyopathy due to PRDM16 mutation: First description of a fetal presentation, with possible modifier genes

2020

PRDM16 (positive regulatory domain 16) is localized in the critical region for cardiomyopathy in patients with deletions of chromosome 1p36, as defined by Gajecka et al., American Journal of Medical Genetics, 2010, 152A, 3074-3083, and encodes a zinc finger transcription factor. We present the first fetal case of left ventricular non-compaction (LVNC) with a PRDM16 variant. The third-trimester obstetric ultrasound revealed a hydropic fetus with hydramnios and expanded hypokinetic heart. After termination of pregnancy, foetopathology showed a eutrophic fetus with isolated cardiomegaly. Endocardial fibroelastosis was associated with non-compaction of the myocardium of the left ventricle. Exom…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyCardiomyopathyBiologyLabor PresentationGenetic HeterogeneityPregnancyExome SequencingGeneticsmedicineHumansMissense mutationGenetic Predisposition to DiseaseGenetics (clinical)Exome sequencingGeneticsFetusGenes ModifierGenetic heterogeneityInfant NewbornEndocardial fibroelastosisMiddle AgedFetal Presentationmedicine.diseasePedigreeDNA-Binding ProteinsMutationMedical geneticsFemaleCardiomyopathiesTranscription FactorsAmerican Journal of Medical Genetics Part C: Seminars in Medical Genetics
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Quantitative criteria for the diagnosis of the congenital absence of pericardium by cardiac magnetic resonance

2015

Congenital absence of the left ventricular pericardium (LCAP) is a rare and poorly known cardiac malformation. Cardiac Magnetic Resonance (CMR) is generally used for the diagnosis of LCAP because of its high soft tissue contrast, multiplanarity and cine capability, but the diagnosis is usually made by only qualitative criteria. The aim of the present study was to establish quantitative criteria for the accurate diagnosis of LCAP on CMR.We enrolled nine consecutive patients affected by LCAP (mean age 26±8years, 7 males), 13 healthy controls, 13 patients with dilated cardiomyopathy (DCM), 12 patients with hypertrophic cardiomyopathy (HCM) and 13 patients with right ventricular overload (RVO).…

AdultHeart Defects CongenitalMalemedicine.medical_specialtyMagnetic Resonance SpectroscopyCardiac magnetic resonanceLeft congenital absence of the pericardium030204 cardiovascular system & hematologyVolume change030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansPericardiumRadiology Nuclear Medicine and imagingcardiovascular diseasesbusiness.industryHealthy subjectsHypertrophic cardiomyopathyReproducibility of ResultsDilated cardiomyopathyMean ageGeneral MedicineSteady-state free precession imagingmedicine.diseasemedicine.anatomical_structurecardiovascular systemCardiologyFemaleRadiologybusinessCardiac magnetic resonancePericardiumHumanEuropean Journal of Radiology
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Circulating miR-323-3p is a biomarker for cardiomyopathy and an indicator of phenotypic variability in Friedreich’s ataxia patients

2017

AbstractMicroRNAs (miRNAs) are noncoding RNAs that contribute to gene expression modulation by regulating important cellular pathways. In this study, we used small RNA sequencing to identify a series of circulating miRNAs in blood samples taken from Friedreich’s ataxia patients. We were thus able to develop a miRNA biomarker signature to differentiate Friedreich’s ataxia (FRDA) patients from healthy people. Most research on FDRA has focused on understanding the role of frataxin in the mitochondria, and a whole molecular view of pathological pathways underlying FRDA therefore remains to be elucidated. We found seven differentially expressed miRNAs, and we propose that these miRNAs represent …

AdultMale0301 basic medicineSmall RNAAtaxiaSciencePopulationCardiomyopathyBioinformaticsArticleYoung Adult03 medical and health sciencesmicroRNAmedicineHumanseducationCells CulturedAgedCell ProliferationGeneticseducation.field_of_studyMultidisciplinarybiologyQRHigh-Throughput Nucleotide SequencingMiddle AgedPrognosismedicine.diseasePhenotypeMicroRNAs030104 developmental biologyBiological Variation PopulationFriedreich AtaxiaCase-Control StudiesFrataxinbiology.proteinBiomarker (medicine)MedicineFemalemedicine.symptomCardiomyopathiesBiomarkersFollow-Up StudiesScientific Reports
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Vulnerability of the right ventricle to cathodal, anodal, and bipolar stimulation at double diastolic threshold strength

1984

The repetitive ventricular response (RVR) to three stimulation techniques (bipolar, cathodal and anodal) was investigated in 35 patients. 26 patients suffered from coronary heart disease and 9 patients from dilative cardiomyopathy. The stimulation study was performed at a ventricular driving rate of 120/min with one and two premature ventricular extrastimuli. We used rectangular impulses of 1.8 ms duration at duable diastolic threshold strength. RVR was scored as follows: 0: no RVR, 1: one nonstimulated RVR, 2: two nonstimulated RVR, 3: three nonstimulated RVR, 4: four to ten nonstimulated RVR, 5: more than ten nonstimulated RVR lasting less than 2 minutes, 6: sustained ventricular tachycar…

AdultMaleCardiac Catheterizationmedicine.medical_specialtyPhysiologyHeart VentriclesDiastoleCoronary DiseaseStimulationDilative cardiomyopathyElectrocardiographyPhysiology (medical)Internal medicinemedicineHumansAgedStimulation techniquebusiness.industryEffective refractory periodMiddle Agedmedicine.diseaseElectric StimulationBipolar stimulationmedicine.anatomical_structureVentricleAnesthesiaVentricular fibrillationCardiologyFemaleCardiomyopathiesCardiology and Cardiovascular MedicinebusinessBasic Research in Cardiology
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Outcomes after acute myocardial infarction in HIV-infected patients: analysis of data from a French nationwide hospital medical information database.

2013

Background— We aimed to assess in-hospital case fatality and 1-year prognosis in HIV-infected patients with acute myocardial infarction. Methods and Results— From the PMSI (Program de Medicalisation des Systèmes d’informatique) database, data from 277 303 consecutive acute myocardial infarction patients hospitalized from January 1, 2005, to December 31, 2009, were analyzed. Surviving patients were followed up for 1 year after discharge. HIV-infected patients were compared with uninfected patients. Among the cohort, HIV-infected patients (n=608) accounted for 0.22%. All-cause hospital and 1-year mortality rates were lower in the HIV-infected group than in uninfected patients (3.1% versus 8.…

AdultMaleDatabases FactualStatistics as TopicMyocardial InfarctionHIV Infectionscomputer.software_genreCohort StudiesPhysiology (medical)Case fatality ratemedicineHumansMyocardial infarctionMedical Informatics ApplicationsAgedRetrospective StudiesAged 80 and overIschemic cardiomyopathyDatabasebusiness.industryMortality rateRetrospective cohort studyMiddle Agedmedicine.diseaseHospitalizationTreatment OutcomeCohortFemaleMyocardial infarction diagnosisFranceCardiology and Cardiovascular MedicinebusinesscomputerCohort studyCirculation
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Multislice multiecho T2* cardiovascular magnetic resonance for detection of the heterogeneous distribution of myocardial iron overload.

2006

Purpose To assess the tissue iron concentration of the left ventricle (LV) using a multislice, multiecho T2* MR technique and a segmental analysis. Materials and Methods T2* multiecho MRI was performed in 53 thalassemia major patients. Three short-axis views of the LV were obtained and analyzed with custom-written software. The myocardium was automatically segmented into 12 segments. The T2* value on each segment as well as the global T2* value were calculated. Cine dynamic images were also obtained to evaluate biventricular function parameters by quantitative analysis. Results For the T2* global value, the coefficient of variation (CoV) for intra-/interobserver and interstudy reproducibili…

AdultMaleLiver Iron ConcentrationthalassemiaAdolescentHeart VentriclesIronCoefficient of variationCardiomyopathyMagnetic Resonance Imaging CineImage Processing Computer-AssistedHumansMedicineDistribution (pharmacology)magnetic resonance imagingRadiology Nuclear Medicine and imagingMultisliceiron overloadChildObserver VariationReproducibilitymedicine.diagnostic_testbusiness.industryMyocardiumbeta-ThalassemiaReproducibility of Resultsrelaxation time (T2*)Magnetic resonance imagingMiddle Agedmedicine.diseasemedicine.anatomical_structureLiverVentricleFerritinsFemalebusinessNuclear medicinecardiomyopathy
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A series of West European patients with severe cardiac and skeletal myopathy associated with a de novo R406W mutation in desmin.

2003

Desminopathy is a familial or sporadic cardiac and skeletal muscular dystrophy associated with mutations in desmin. We have previously characterized a de novo desmin R406W mutation in a patient of European origin with early onset muscle weakness in the lower extremities and atrioventricular conduction block requiring a permanent pacemaker. The disease relentlessly progressed resulting in severe incapacity within 5 years after onset. We have now identified three other patients with early onset rapidly progressive cardiac and skeletal myopathy caused by this same desmin R406W mutation. The mutation was present in each studied patient, but not in their parents or other unaffected family member…

AdultMaleModels Molecularmedicine.medical_specialtyPathologyNeurologyHeart diseaseAdolescentAmino Acid MotifsCardiomyopathymacromolecular substancesDiseaseBiologyProtein Structure SecondaryDesmin03 medical and health sciences0302 clinical medicineMuscular DiseasesmedicineHumansMuscular dystrophyMyopathyMuscle SkeletalConserved Sequence030304 developmental biology0303 health sciencesMuscle WeaknessBase SequenceMyocardiumMuscle weaknessAnatomymedicine.diseasePedigreeEuropeHeart BlockNeurologyAmino Acid SubstitutionMutationDisease ProgressionDesminFemaleNeurology (clinical)medicine.symptomCardiomyopathies030217 neurology & neurosurgeryJournal of neurology
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Hereditary motor sensory neuropathy type II with neurofilament accumulation: new finding or new disorder?

1985

Peroneal muscular atrophy is now known to be heterogeneous and to be due to various underlying genetic mechanisms. Exploring this heterogeneity further, we report on a German kinship with the clinical, genetic, and nerve conduction features of hereditary motor and sensory neuropathy type II (HMSN type II) but whose sural nerves on biopsy were found to show infrequent axonal swellings with neurofilament accumulations not previously described. The dominant inheritance and absence of kinky hair set this disorder apart from giant axonal neuropathy. There was no history of toxic exposure to industrial chemicals. We conclude that the disorder either is a new type of HMSN or is HMSN type II with p…

AdultMaleNeurofilamentAdolescentBiopsyCardiomyopathyNeural ConductionCardiovascular System03 medical and health sciences0302 clinical medicineSural NerveEvoked Potentials SomatosensoryBiopsymedicineHumansAxonHereditary Sensory and Autonomic NeuropathiesChildCytoskeleton030304 developmental biologyGiant axonal neuropathyAged0303 health sciencesmedicine.diagnostic_testbusiness.industryElectromyographyPeroneal muscular atrophyMiddle Agedmedicine.diseaseAxonsPedigreeMicroscopy ElectronMuscular Atrophymedicine.anatomical_structureNeurologyEvoked Potentials VisualFemaleNeurology (clinical)AbnormalityHereditary motor and sensory neuropathybusinessNeuroscience030217 neurology & neurosurgeryAnnals of neurology
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GAA trinucleotide repeat expansion in variant Friedreich's ataxia families.

1997

Phenotypic variants in Friedreich's ataxia include late onset, preservation of the lower limbs tendon reflexes, and slow progression. We describe clinical and electrophysiological features from three families with Friedreichlike phenotypes. Friedreich's ataxia diagnosis was confirmed by finding two allelic expansions of the GAA trinucleotide repeat at the X25 gene. In family 1 both patients had a late-onset phenotype with preservation of knee and ankle jerks, lack of cardiomyopathy, and preserved H reflex. One of them did not have electrophysiologic evidence of sensory axonal neuropathy. Patients from family 2 showed variability in the age of onset, and 2 out of 3 affected children had hype…

AdultMaleReflex Stretchcongenital hereditary and neonatal diseases and abnormalitiesPathologymedicine.medical_specialtySensory axonal neuropathyAtaxiaPhysiologyGenetic LinkageAction PotentialsLate onsetBiologyH-ReflexCellular and Molecular NeuroscienceDegenerative diseaseTrinucleotide RepeatsPhysiology (medical)medicineHumansNeurons AfferentChildAllelesLegGenetic VariationDNACardiomyopathy Hypertrophicmedicine.diseasePedigreePeripheral neuropathyFriedreich AtaxiaReflexDisease ProgressionFemaleNeurology (clinical)medicine.symptomAge of onsetTrinucleotide repeat expansionMusclenerve
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Prospective risk stratification of sudden cardiac death in Marfan's syndrome.

2013

Marfan syndrome (MFS) is a variable, autosomal-dominant disorder of the connective tissue. In MFS serious ventricular arrhythmias and sudden cardiac death (SCD) can occur. The aim of this prospective study was to reveal underlying risk factors and to prospectively investigate the association between MFS and SCD in a long-term follow-up.77 patients with MFS were included. At baseline serum N-terminal pro-brain natriuretic peptide (NT-proBNP), transthoracic echocardiogram, 12-lead resting ECG, signal-averaged ECG (SAECG) and a 24-h Holter ECG with time- and frequency domain analyses were performed. The primary composite endpoint was defined as SCD, ventricular tachycardia (VT), ventricular fi…

AdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyCardiomyopathyVentricular tachycardiaRisk AssessmentSudden cardiac deathMarfan SyndromeYoung AdultInternal medicineClinical endpointMedicineHumanscardiovascular diseasesProspective StudiesUltrasonographybusiness.industryHazard ratioMiddle Agedmedicine.diseaseSignal-averaged electrocardiogramDeath Sudden CardiacVentricular fibrillationCardiologyFemaleTransthoracic echocardiogramCardiology and Cardiovascular MedicinebusinessFollow-Up StudiesInternational journal of cardiology
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