Search results for "Cardioprotection"

showing 5 items of 35 documents

Antineoplastic Drug-Induced Cardiotoxicity: A Redox Perspective

2018

Antineoplastic drugs can be associated with several side effects, including cardiovascular toxicity (CTX). Biochemical studies have identified multiple mechanisms of CTX. Chemoterapeutic agents can alter redox homeostasis by increasing the production of reactive oxygen species (ROS) and reactive nitrogen species RNS. Cellular sources of ROS/RNS are cardiomyocytes, endothelial cells, stromal and inflammatory cells in the heart. Mitochondria, peroxisomes and other subcellular components are central hubs that control redox homeostasis. Mitochondria are central targets for antineoplastic drug-induced CTX. Understanding the mechanisms of CTX is fundamental for effective cardioprotection, without…

Stromal cellPhysiologymedicine.medical_treatmentTyrosine kinase inhibitorChemotherapy; HER-2 inhibitors; Oxidative/nitrosative stress; Tyrosine kinase inhibitors; Vascular endothelial growth factorReviewOxidative phosphorylation030204 cardiovascular system & hematologyMitochondrionPharmacologyChemotherapy; HER-2 inhibitors; Oxidative/nitrosative stress; Tyrosine kinase inhibitors; Vascular endothelial growth factor; Physiology; Physiology (medical)chemotherapyHER-2 inhibitorlcsh:Physiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicinePhysiology (medical)tyrosine kinase inhibitorsMedicinechemotherapy HER-2 inhibitors oxidative/nitrosative stress vascular endothelial growth factor tyrosine kinase inhibitorsReactive nitrogen specieschemistry.chemical_classificationCardioprotectionReactive oxygen speciesChemotherapyCardiotoxicitylcsh:QP1-981vascular endothelial growth factorbusiness.industryOxidative/nitrosative strechemistry030220 oncology & carcinogenesisbusinessHER-2 inhibitorsoxidative/nitrosative stress
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Intermittent hypobaric hypoxia applicability in myocardial infarction prevention and recovery.

2012

Abstract Intermittent hypobaric hypoxia (IHH) has been the focus of important research in cardioprotection, and it has been associated with several mechanisms. Intermittent hypobaric hypoxia inhibits prolyl hydroxylases (PHD) activity, increasing the stabilization of hypoxia-inducible factor-1 (HIF-1) and activating crucial adaptative genes. It has been hence suggested that IHH might be a simple intervention, which may offer a thoughtful benefits to patients with acute myocardial infarction and no complications. Nevertheless, several doubts exist as to whether IHH is a really safe technique, with little to no complications in post-myocardial infarction patients. Intermittent hypobaric hypox…

endotheliumInfarctionCoronary artery diseasehypobaric chamberrecoverypreventionmedicineHumansMyocardial infarctionhypoxia-inducible factorCardioprotectionAir Pressuremyocardial infarction; prevention; recoverybusiness.industryAltitudeCell Biologymedicine.diseaseThrombosisbody regionsmyocardial infarctionPoint of ViewsHeart failureHypobaric chamberAnesthesiacardioprotectionIschemic Preconditioning MyocardialMolecular MedicineIschemic preconditioningerythropoietinbusiness
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Myocardial Protection by Retrograde Cardioplegic Perfusion in the Presence of Acute Coronary Artery Obstruction: An Experimental Study

1992

To investigate retrograde delivery of cardioplegic solutions as a means of enhancing myocardial protection in the presence of coronary artery occlusion, a two-part experimental model was devised. In part 1 (in vitro) the possibility of retroperfusing the entire myocardium during acute occlusion of the left anterior descending artery (LAD) was assessed. In part 2 (in vivo) acute LAD occlusion was performed in dogs, and during 2 hours of aortic cross-clamping crystalline cardioplegic solution was infused at 20-minute intervals. In group I the infusion was antegrade, via the aortic root, and in group II it was retrograde, via the coronary sinus. Thereafter the LAD snare was released and the do…

medicine.medical_specialtyGroup iiContrast MediaBlood PressureCoronary DiseaseCoronary AngiographyPotassium ChlorideDogsBody WaterHypothermia InducedInternal medicineOcclusionmedicineCarnivoraAnimalscardiovascular diseasesCardioplegic SolutionsAortaCoronary sinusCardioprotectionCardiopulmonary Bypassbiologybusiness.industryMyocardiumFissipediaHeartbiology.organism_classificationCoronary VesselsPerfusionmedicine.anatomical_structureAnesthesiaHeart Arrest Inducedcardiovascular systemCardiologyCardiology and Cardiovascular MedicinebusinessPerfusionArteryScandinavian Journal of Thoracic and Cardiovascular Surgery
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Impaired border zone formation and adverse remodeling after reperfused myocardial infarction in cannabinoid CB2 receptor deficient mice.

2014

Abstract Aims Reperfusion of myocardial infarction is associated with inflammatory reaction and subsequent myocardial remodeling with a rapid scar formation in mice. The cannabinoid receptor CB2 has been associated with cardioprotection and regulation of macrophage function. We investigated its role in remodeling of reperfused infarction. Main methods One hour LAD-occlusion was followed by reperfusion over 6 h and 1, 3 and 7 days in wild-type C57/BL6J (WT) and CB2 receptor-deficient (Cnr2 −/− ) mice (n = 8/group). Hearts were processed for functional, morphological and mRNA/protein analysis, and tissue concentration of endocannabinoids was determined using liquid chromatography-multiple rea…

medicine.medical_specialtyIschemiaMyocardial InfarctionInfarctionMyocardial Reperfusion InjuryGeneral Biochemistry Genetics and Molecular BiologyReceptor Cannabinoid CB2MiceInternal medicinemedicineCannabinoid receptor type 2AnimalsMyocytes CardiacMyocardial infarctionGeneral Pharmacology Toxicology and PharmaceuticsCardioprotectionInflammationMice KnockoutbiologyChemistryMyocardiumTenascin CHemodynamicsGranulation tissueGeneral Medicinemedicine.diseaseEndocannabinoid systemMice Inbred C57BLEndocrinologymedicine.anatomical_structureCardiologybiology.proteinGranulation TissueCytokinesLife sciences
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Erythropoietin and the heart: physiological effects and the therapeutic perspective.

2014

Erythropoietin (Epo) has been thought to act exclusively on erythroid progenitor cells. The identification of Epo receptor (EpoR) in non-haematopoietic cells and tissues including neurons, astrocytes, microglia, immune cells, cancer cell lines, endothelial cells, bone marrow stromal cells, as well as cells of myocardium, reproductive system, gastrointestinal tract, kidney, pancreas and skeletal muscle indicates that Epo has pleiotropic actions. Epo shows signals through protein kinases, anti-apoptotic proteins and transcription factors. In light of interest of administering recombinant human erythropoietin (rhEpo) and its analogues for limiting infarct size and left ventricular (LV) remodel…

medicine.medical_specialtyStromal cellCardiotonic AgentsAngiogenesisNeovascularization PhysiologicInflammationerythroid progenitor cellshemic and lymphatic diseasesInternal medicineEpo receptorReceptors ErythropoietinMedicineHumansErythropoietinCardioprotectionMicrogliabusiness.industryHeartErythropoietin receptorErythropoietin; Epo receptor; erythroid progenitor cellsEndocrinologymedicine.anatomical_structureErythropoietinCancer researchAirway RemodelingBone marrowmedicine.symptomCardiology and Cardiovascular Medicinebusinessmedicine.drugInternational journal of cardiology
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