Search results for "Cell Communication"

showing 10 items of 153 documents

Impaired Vitamin D Signaling in Endothelial Cell Leads to an Enhanced Leukocyte-Endothelium Interplay: Implications for Atherosclerosis Development

2015

Endothelial cell activation leading to leukocyte recruitment and adhesion plays an essential role in the initiation and progression of atherosclerosis. Vitamin D has cardioprotective actions, while its deficiency is a risk factor for the progression of cardiovascular damage. Our aim was to assess the role of basal levels of vitamin D receptor (VDR) on the early leukocyte recruitment and related endothelial cell-adhesion-molecule expression, as essential prerequisites for the onset of atherosclerosis. Knockdown of VDR in endothelial cells (shVDR) led to endothelial cell activation, characterized by upregulation of VCAM-1, ICAM-1 and IL-6, decreased peripheral blood mononuclear cell (PBMC) ro…

Malemedicine.medical_specialtyEndotheliumMedicinalcsh:MedicineDown-RegulationVascular Cell Adhesion Molecule-1InflammationCell CommunicationBiologyCalcitriol receptorCell LineEndothelial activationMiceEndothelial cellNF-KappaB Inhibitor alphaInternal medicineCell AdhesionmedicineAnimalsHumansMacrophageEndotheliumVitamin Dlcsh:ScienceCell adhesionMultidisciplinaryInterleukin-6lcsh:RNF-kappa BEndothelial CellsAtherosclerosisIntercellular Adhesion Molecule-1Plaque AtheroscleroticUp-RegulationMice Inbred C57BLEndothelial stem cellIκBαmedicine.anatomical_structureEndocrinologyLeukocytes MononuclearReceptors Calcitriollcsh:QFemaleI-kappa B Proteinslipids (amino acids peptides and proteins)medicine.symptomSignal TransductionResearch ArticlePLOS ONE
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Regulation of cell-to-cell communication in non-tumorigenic and malignant human prostate epithelial cells.

2002

BACKGROUND Gap-junction-mediated intercellular communication (GJIC) is required for normal development and tissue homeostasis. However, the role of GJIC in human prostate carcinogenesis and progression remains ill-defined. METHODS The ability of hormones, anti-hormones, and the anti-hypertensive drug, forskolin, to restore GJIC in non-tumorigenic (RWPE-1 and PWR-1E) and malignant (RWPE-2, LNCaP, DU-145) human prostate epithelial cell lines, was examined by Scrape-Loading/Dye Transfer (SL/DT) and Fluorescence Recovery After Photobleaching (FRAP) methods using an Ultima laser cytometer. RESULTS Results from both assays show that PWR-1E, RWPE-2, LNCaP, and DU-145 cells have weak or absent GJIC…

Malemedicine.medical_specialtyEstroneUrologyCell CommunicationBiologyurologic and male genital diseasesmedicine.disease_causeConnexinschemistry.chemical_compoundProstate cancerCell–cell interactionInternal medicineLNCaPmedicineTumor Cells CulturedHumansTissue homeostasisForskolinColforsinGap JunctionsProstatic NeoplasmsEpithelial Cellsmedicine.diseaseEndocrinologyCell Transformation NeoplasticOncologychemistryCell cultureCancer researchCarcinogenesisImmortalised cell lineThe Prostate
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Gastric antisecretory drugs induce leukocyte-endothelial cell interactions through gastrin release and activation of CCK-2 receptors.

2007

Antisecretory drugs are effective antiulcer agents, but its chronic use generates hypergastrinemia and accelerates the development of atrophic gastritis in Helicobacter pylori-positive patients. We have recently shown that gastrin exerts a proinflammatory effect in rats through CCK-2 receptor activation that contributes to the inflammation induced by H. pylori. The present study was designed to examine whether gastrin hypersecretion in response to treatment with antisecretory drugs induces an inflammatory response that could promote mucosal atrophy. The effects of omeprazole or famotidine on leukocyte/endothelial cell interactions in vivo were analyzed in rat mesenteric venules using intrav…

Malemedicine.medical_specialtyProglumidemedicine.drug_classInflammationCD18Cell CommunicationProinflammatory cytokineRats Sprague-DawleyInternal medicineGastrinsmedicineLeukocytesAnimalsOmeprazoleGastrinPharmacologyInflammationMicroscopy Videobusiness.industryEndothelial CellsProton Pump InhibitorsReceptor antagonistAnti-Ulcer AgentsFlow CytometryImmunohistochemistryReceptor Cholecystokinin BRatsFamotidineChemotaxis LeukocyteEndocrinologyGastric MucosaMolecular Medicinemedicine.symptombusinessmedicine.drugThe Journal of pharmacology and experimental therapeutics
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Expression of Cx36 in mammalian neurons

2000

Cx36 is the first mammalian member of a novel subgroup of the connexin family, characterized by a long cytoplasmic loop, a peculiar gene structure and a preferential expression in cell types of neural origin. In the present review we summarize the evidence in favour of its predominant expression in neuronal cells in the mammalian central nervous system, such as results from experiments with specific neurotoxins and co-localization of Cx36 mRNA and a neuronal marker. We also report a detailed description of Cx36 mRNA distribution in the rat and human central nervous system by in situ hybridization and, for each brain region, we correlate the novel findings with previous morphological or func…

MammalsMessenger RNAGeneral NeuroscienceCentral nervous systemGap JunctionsGene ExpressionConnexinCell CommunicationMolecular neuroscienceIn situ hybridizationBiologyCell junctionConnexinsmedicine.anatomical_structureSynapsesGene expressionmedicineAnimalsHumanssense organsNeurology (clinical)NeuronEye ProteinsNeuroscienceBrain Research Reviews
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Autophagy Induces Expression of IL-6 in Human Periodontal Ligament Fibroblasts Under Mechanical Load and Overload and Effects Osteoclastogenesis in v…

2021

Frontiers in physiology 12, 716441 (2021). doi:10.3389/fphys.2021.716441 special issue: "Alveolar Bone: a Pivotal Role in Periodontal Disease Pathobiology and Treatment, Volume I / Fani Anagnostou, Beatriz Castaneda, Frédéric Lézot and Petros Papagerakis"

Mechanical overloadautophagymechanical loadPhysiologymedicine.medical_treatmentADAM10Matrix metalloproteinaseOsteoprotegerinhuman periodontal ligament fibroblastsPhysiology (medical)medicineQP1-981cell-cell communicationOriginal ResearchIL-6ADAM17biologyChemistryAutophagyADAM10OsteoblastCell biologymechanical overloadCytokinemedicine.anatomical_structureRANKLbiology.proteinFrontiers in Physiology
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Electrical Coupling in Ensembles of Nonexcitable Cells: Modeling the Spatial Map of Single Cell Potentials

2015

We analyze the coupling of model nonexcitable (non-neural) cells assuming that the cell membrane potential is the basic individual property. We obtain this potential on the basis of the inward and outward rectifying voltage-gated channels characteristic of cell membranes. We concentrate on the electrical coupling of a cell ensemble rather than on the biochemical and mechanical characteristics of the individual cells, obtain the map of single cell potentials using simple assumptions, and suggest procedures to collectively modify this spatial map. The response of the cell ensemble to an external perturbation and the consequences of cell isolation, heterogeneity, and ensemble size are also ana…

Membrane potentialChemistryCellNanotechnologyCell CommunicationHydrogen-Ion ConcentrationModels BiologicalIon ChannelsMembrane PotentialsQuantitative Biology::Cell BehaviorSurfaces Coatings and FilmsCoupling (electronics)medicine.anatomical_structureMembraneMaterials ChemistrymedicineSpatial mapsPhysical and Theoretical ChemistryExtracellular SpaceLipid bilayerBiological systemElectromagnetic PhenomenaIon channelBiophysical chemistryThe Journal of Physical Chemistry B
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Inter- and intracellular signaling in amyotrophic lateral sclerosis: role of p38 mitogen-activated protein kinase.

2006

The pathogenetic processes underlying the selective motor neuron degeneration in amyotrophic lateral sclerosis (ALS) are complex and still not completely understood even in the cases of inherited disease caused by mutations in the Cu/Zn superoxide dismutase-dependent (SOD1) gene. Recent evidence supports the view that ALS is not a cell-autonomous disease and that glial-neuron cross-talk, throughout cytokines and other toxic factors like the nitric oxide and superoxide, is a crucial determinant for the induction of motor neuron death. This cell-cell interaction may determine the progression of the disease through processes that are likely independent of the initial trigger and that may conve…

Motor NeuronsCell signalingp38 mitogen-activated protein kinasesSOD1Amyotrophic Lateral SclerosisNeurotoxicityCell CommunicationReceptor Cross-TalkMotor neuronBiologymedicine.diseasep38 Mitogen-Activated Protein Kinasesmedicine.anatomical_structurenervous systemNeurologyMitogen-activated protein kinasemedicinebiology.proteinAnimalsHumansNeurology (clinical)Amyotrophic lateral sclerosisNeuroscienceNeurogliaNeuroinflammation
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Myeloid dendritic cell: From sentinel of immunity to key player of peripheral tolerance?

2009

Myeloid dendritic cells (DC) are "sentinels" of immunity, ideally positioned throughout the body gateways and equipped with unique properties to transport antigens from the periphery to lymphoid tissues. They are professional antigen-presenting cells transmitting incoming infectious signals to T cells, the key players of adaptive immunity. For induction of effective antigen-specific T-cell immunity, crosstalk of DC and naive T cells is mandatory. However, besides this essential immunostimulatory function of DC, consolidated findings from the DC research field in the last 10 years have shown that DC have an additional important function. They act as pivotal players in the peripheral toleranc…

MyeloidImmunologyCell CommunicationBiologyImmune toleranceMiceImmune systemAntigenImmunityT-Lymphocyte SubsetsmedicineImmune ToleranceImmunology and AllergyAnimalsHumansMyeloid CellsImmunologic SurveillancePeripheral toleranceGeneral MedicineDendritic CellsAcquired immune systemCrosstalk (biology)medicine.anatomical_structureImmunity ActiveImmunologyCytokinesHuman immunology
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Friend retrovirus infection of myeloid dendritic cells impairs maturation, prolongs contact to naïve T cells, and favors expansion of regulatory T ce…

2007

AbstractRetroviruses have developed immunmodulatory mechanisms to avoid being attacked by the immune system. The mechanisms of this retrovirus-associated immune suppression are far from clarified. Dendritic cells (DCs) have been attributed a decisive role in these pathogenic processes. We have used the Friend retrovirus (FV) mouse model in order to acquire further knowledge about the role of infection of DCs in virus-induced immunosuppression. About 20% of the myeloid DCs that were generated from the bone marrow of FV-infected mice carried FV proteins. The infection was productive, and infected DCs transmitted the virus in cell culture and in vivo. FV infection of DCs led to a defect in DC …

MyeloidImmunologyPopulationMedizinBone Marrow CellsMice Transgenicchemical and pharmacologic phenomenaCell CommunicationBiologyLymphocyte ActivationT-Lymphocytes RegulatoryBiochemistryMiceImmune systemAntigenImmune TolerancemedicineAnimalsCytotoxic T cellMyeloid CellseducationCell ProliferationAntigen PresentationMice Inbred BALB Ceducation.field_of_studyFollicular dendritic cellsModels ImmunologicalFOXP3hemic and immune systemsDendritic CellsCell BiologyHematologyFriend murine leukemia virusCell biologymedicine.anatomical_structureImmunologyBone marrowRetroviridae InfectionsBlood
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NG2-expressing cells in the nervous system: role of the proteoglycan in migration and glial–neuron interaction

2005

The NG2 glycoprotein is a type I membrane protein expressed in the developing and adult central nervous system (CNS) by subpopulations of glia including oligodendroglial precursor cells (OPCs), and in the developing CNS additionally by pericytes. In the mouse CNS, expression of NG2 protein is already observed at embryonic day 13 and peaks between postnatal days 8 and 12. NG2+ cells persist in grey and white matter in adult mouse brain: cells in the developing and adult brain show clear differences in migration, cell-cycle length and lineage restriction. Several groups have provided evidence that subpopulations of NG2+ cells can generate neurons in vivo. Neuronal stimulation in the developin…

Nervous systemHistologyPDZ domainNeurexinAMPA receptorReviewCell CommunicationBiologyCell MovementmedicineCell AdhesionAnimalsHumansAntigensMolecular BiologyEcology Evolution Behavior and SystematicsNeuronsNG2 proteoglycanStem CellsGlutamate receptorBrainCell BiologyCell biologyOligodendrogliamedicine.anatomical_structurenervous systemImmunologyProteoglycansNeuronAnatomyNeurogliaDevelopmental BiologyAstrocyte
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