Search results for "Cell Cycle"

showing 10 items of 804 documents

p53 as the main traffic controller of the cell signaling network

2010

Among different pathological conditions that affect human beings, cancer has received a great deal of attention primarily because it leads to significant morbidity and mortality. This is essentially due to increasing world-wide incidence of this disease and the inability to discover the cause and molecular mechanisms by which normal human cells acquire the characteristics that define cancer cells. Since the discovery of p53 over a quarter of a century ago, it is now recognized that virtually all cell fate pathways of live cells and the decision to die are under the control of p53. Such extensive involvement indicates that p53 protein is acting as a major traffic controller in the cell signa…

Cell signalingSettore MED/06 - Oncologia MedicaApoptosisDiseaseCell fate determinationBiologyNeoplasmsmedicineApoptosis; Cellular Senescence; Gene Expression Regulation Neoplastic; Humans; Mutation; Neoplasms; Polymorphism Genetic; Signal Transduction; Tumor Suppressor Protein p53HumansCellular SenescencePolymorphism GeneticCancerApoptosiCell cyclemedicine.diseaseCell biologyGene Expression Regulation NeoplasticThe Hallmarks of CancerApoptosisCancer cellMutationNeoplasmTumor Suppressor Protein p53HumanSignal Transduction
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Fusion of bone-marrow-derived cells with Purkinje neurons, cardiomyocytes and hepatocytes

2003

Recent studies have suggested that bone marrow cells possess a broad differentiation potential, being able to form new liver cells, cardiomyocytes and neurons1,2. Several groups have attributed this apparent plasticity to ‘transdifferentiation’3,4,5. Others, however, have suggested that cell fusion could explain these results6,7,8,9. Using a simple method based on Cre/lox recombination to detect cell fusion events, we demonstrate that bone-marrow-derived cells (BMDCs) fuse spontaneously with neural progenitors in vitro. Furthermore, bone marrow transplantation demonstrates that BMDCs fuse in vivo with hepatocytes in liver, Purkinje neurons in the brain and cardiac muscle in the heart, resul…

Cell typeCell signalingBone Marrow CellsBiologyBioinformaticsGiant CellsModels BiologicalCell FusionMicePurkinje CellsmedicineAnimalsMyocyteMyocytes CardiacProgenitor cellBone Marrow TransplantationMultidisciplinaryCell fusionStem CellsTransdifferentiationCell DifferentiationCell cycleCell biologyMice Inbred C57BLmedicine.anatomical_structureHepatocytesBone marrow
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Terminally differentiated postmitotic tumor cells in a rat rhabdomyosarcoma cell line.

1988

A permanent rat rhabdomyosarcoma cell line (BA-HAN-1C) has been established, the phenotype of which is characterized by the coexistence of undifferentiated mononuclear cells and differentiated multinuclear myotube-like giant cells. The failure of attempts to separate these two cell types by repeated recloning procedures indicates their close histogenetic relationship and suggests that differentiation in this tumor proceeds in a similar manner to that in normal striated muscle where postmitotic myotubes arise from mononuclear myoblasts by fusion. The morphologically undifferentiated mononuclear tumor cells were shown to be actively proliferating and to incorporate thymidine methyl-3H(3H-TdR)…

Cell typePathologymedicine.medical_specialtyCellular differentiationCell DifferentiationNeoplasms ExperimentalBiologyCell cyclePeripheral blood mononuclear cellPathology and Forensic MedicineCell biologyRatsGiant cellCell cultureRhabdomyosarcomamedicineMitotic IndexTumor Cells CulturedAnimalsClonogenic assayFloxuridineMitosisCell DivisionVirchows Archiv. B, Cell pathology including molecular pathology
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Differential expression levels of Sox9 in early neocortical radial glial cells regulate the decision between stem cell maintenance and differentiation

2021

ABSTRACTRadial glial progenitor cells (RGCs) in the dorsal forebrain directly or indirectly produce excitatory projection neurons and macroglia of the neocortex. Recent evidence shows that the pool of RGCs is more heterogeneous than originally thought and that progenitor subpopulations can generate particular neuronal cell types. Using single cell RNA sequencing, we have studied gene expression patterns of two subtypes of RGCs that differ in their neurogenic behavior. One progenitor type rapidly produces postmitotic neurons, whereas the second progenitor remains relatively quiescence before generating neurons. We have identified candidate genes that are differentially expressed between thes…

Cell typeTranscription GeneticNeurogenesisEpendymoglial CellsGenetic VectorsNeocortexNerve Tissue ProteinsBiologyMiceradial glia cellsprogenitors diversityGenes ReporterPregnancyGene expressionmedicineAnimalscortical developmentProgenitors diversityCell Self RenewalProgenitor cellPromoter Regions GeneticTranscription factorResearch ArticlesInjections IntraventricularProgenitorNeuronsNeocortexCortical developmentGeneral NeuroscienceCell CycleGene Expression Regulation DevelopmentalSOX9 Transcription FactorEmbryonic stem cellCell biologyMice Inbred C57BLCorticogenesisElectroporationmedicine.anatomical_structureCerebral cortexForebrainFemalesense organsSingle-Cell AnalysisStem cellNeuroscienceNeurogliaRadial glia cellsCellular/MolecularSox9
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The effect of cadmium on brain cells in culture

2009

Cadmium is a long-living heavy metal, abundantly present in the environment, which accumulates in the body. In this study, we investigated the effects of cadmium on the expression of molecular chaperones, and of certain cell-specific proteins, in a variety of brain cell types in culture, namely primary cultures of rat cortical neurons and astrocytes, a brain capillary endothelial cell line (RB4E.B cells), and pheochromocytoma cells (PC12), induced or not to differentiate by NGF treatment. The metal induces a dose-dependent increase of Hsp70 in all cell types. Responses to the metal are cell-specific in the case of Hsc70 and Hsp90: i) in astrocytes, as well as in PC12 cells, cadmium has no s…

Cell typecadmium brain cells molecular chaperones PIPPinCell SurvivalCellBlotting Westernchemistry.chemical_elementNerve Tissue ProteinsBiologyPC12 CellsSettore BIO/10 - BiochimicaNerve Growth FactorGeneticsmedicineAnimalsCytoskeletonCell ShapeCells CulturedFluorescent DyesCerebral CortexNeuronsCadmiumBrainEndothelial CellsRNA-Binding ProteinsCell DifferentiationGeneral MedicineCell cycleMolecular biologyHsp70Cell biologyRatsEndothelial stem cellmedicine.anatomical_structurechemistryApoptosisAstrocytesCadmiumMolecular Chaperones
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A nuclear glutathione cycle within the cell cycle

2010

The complex antioxidant network of plant and animal cells has the thiol tripeptide GSH at its centre to buffer ROS (reactive oxygen species) and facilitate cellular redox signalling which controls growth, development and defence. GSH is found in nearly every compartment of the cell, including the nucleus. Transport between the different intracellular compartments is pivotal to the regulation of cell proliferation. GSH co-localizes with nuclear DNA at the early stages of proliferation in plant and animal cells. Moreover, GSH recruitment and sequestration in the nucleus during the G1- and S-phases of the cell cycle has a profound impact on cellular redox homoeostasis and on gene expression. F…

CellBiologyBiochemistrychemistry.chemical_compoundGene expressionmedicineAnimalsHumansNuclear proteinMolecular BiologyCell ProliferationCell NucleusCell growthCell CycleCell BiologyGlutathioneCell cycleGlutathioneCell CompartmentationCell biologymedicine.anatomical_structureBiochemistrychemistryOxidation-ReductionNucleusIntracellularBiochemical Journal
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Powerful tumor cell growth-inhibiting activity of a synthetic derivative of atractyligenin: Involvement of PI3K/Akt pathway and thioredoxin system

2014

The semi-synthetic ent-kaurane 15-ketoatractyligenin methyl ester (SC2017) has been previously reported to possess high antiproliferative activity against several solid tumor-derived cell lines. Our study was aimed at investigating SC2017 tumor growth-inhibiting activity and the underlying mechanisms in Jurkat cells (T-cell leukemia) and xenograft tumor models. METHODS: Cell viability was evaluated by MTT assay. Cell cycle progression, reactive oxygen species (ROS) elevation and apoptotic hallmarks were monitored by flow cytometry. Inhibition of thioredoxin reductase (TrxR) by biochemical assays. Levels and/or activation status of signaling proteins were assessed by western blotting. Xenogr…

CellBiophysicsAntineoplastic AgentsApoptosisAtractylosideBiologyCell cycleBiochemistryJurkat cellsMicePhosphatidylinositol 3-KinasesThioredoxinsTumor Cells CulturedmedicineAnimalsHumansMTT assayViability assaySettore BIO/15 - Biologia FarmaceuticaMolecular BiologyProtein kinase BPI3K/AKT/mTOR pathwayCell ProliferationPI3K/AktHCT 116 xenograftCytochromes cApoptosiThioredoxin systemSettore CHIM/06 - Chimica OrganicaCell cycleXenograft Model Antitumor AssaysCell biologymedicine.anatomical_structureCaspasesCancer researchThioredoxinDiterpenes KauraneProto-Oncogene Proteins c-aktEnt-kaurane
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Dihydrocucurbitacin B Inhibits Delayed Type Hypersensitivity Reactions by Suppressing Lymphocyte Proliferation

2007

We have studied the effects of dihydrocucurbitacin B, a triterpene isolated from Cayaponia tayuya roots, on different models of delayed type hypersensitivity (DTH) in mice, as well as on T-lymphocyte proliferation and the mediators involved. In experiments with mice, dihydrocucurbitacin B inhibited the inflammatory reactions induced by oxazolone, dinitrofluorobenzene, and sheep red blood cells, reducing both the edema and cell infiltration. Moreover, the analysis of inflamed tissues showed that dihydrocucurbitacin B reduced the presence of the most relevant cytokines implicated in these processes, including interleukin-1 beta, interleukin-4, and tumor necrosis factor-alpha. Dihydrocucurbita…

CellLymphocyte proliferationLymphocyte ActivationResting Phase Cell CycleOxazoloneMicechemistry.chemical_compoundCyclinsmedicineAnimalsHypersensitivity DelayedCyclinInflammationPharmacologyNFATC Transcription FactorsbiologyNFATCell cyclebiology.organism_classificationMolecular biologyTriterpenesCayaponia tayuyaDisease Models Animalmedicine.anatomical_structurechemistryImmunologyCytokinesMolecular MedicineTumor necrosis factor alphaJournal of Pharmacology and Experimental Therapeutics
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Cell-Free Coelomic Fluid Extracts of the Sea Urchin Arbacia lixula Impair Mitochondrial Potential and Cell Cycle Distribution and Stimulate Reactive …

2020

Triple-negative breast cancer (TNBC) is a highly malignant tumor histotype which lacks effective targeted therapies, thereby being considered as the most aggressive form of breast carcinoma. To identify novel compounds which could counteract TNBC cell growth, we explored the in vitro effects of crude extracts and &lt

CellSettore BIO/05 - ZoologiaOcean Engineering03 medical and health scienceslcsh:Oceanography0302 clinical medicinebreast cancerlcsh:VM1-989biology.animalmedicineViability assaylcsh:GC1-1581Settore BIO/06 - Anatomia Comparata E CitologiaArbacia lixulaSea urchin030304 developmental biologyWater Science and TechnologyCivil and Structural Engineeringchemistry.chemical_classification0303 health sciencesReactive oxygen speciesbiologylcsh:Naval architecture. Shipbuilding. Marine engineeringCell cyclebiology.organism_classificationechinodermmedicine.anatomical_structurechemistryApoptosis030220 oncology & carcinogenesisCancer researchcytotoxicityBreast carcinomaJournal of Marine Science and Engineering
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Role of nuclear glutathione as a key regulator of cell proliferation.

2009

Glutathione (GSH) is essential for survival of eukaryotic but not in prokaryotic cells. Its functions in nucleated cells are far from being known. In fact GSH plays an important role in cell proliferation. The purpose of the present review is to summarize the relationship between glutathione and the important events that take place in the nucleus during the cell cycle. Most GSH co-localizes with nuclear DNA when cells are proliferating. However, when cells were confluent no differences between nucleus and cytoplasm could be seen. A number of relevant nuclear proteins are strictly dependent on nuclear redox status. For instance, we found that telomerase is regulated by shifts in glutathione …

CellsClinical BiochemistryBiochemistryEpigenesis Geneticchemistry.chemical_compoundAnimalsHumansEpigeneticsNuclear proteinCell Cycle ProteinMolecular BiologyTelomeraseCell ProliferationbiologyCell growthGeneral MedicineGlutathioneCell cycleGlutathioneCell biologyOxidative StressHistoneBiochemistrychemistryCytoplasmbiology.proteinMolecular MedicineMolecular aspects of medicine
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