Search results for "Cell Death"

showing 10 items of 824 documents

Serotherapy with thymoglobulin and alemtuzumab differentially influences frequency and function of natural killer cells after allogeneic stem cell tr…

2007

Although thymoglobulin and alemtuzumab are frequently used in hematopoietic stem cell transplantation (HSCT), little is known of their effects on NK cells, which mediate important functions in post-transplantation immunology. In the present study, we determined NK cell death in vitro using propidium iodide and Annexin V. The NK cell activity in 34 patients at day +30 after allogeneic HSCT was assessed using the CD107a assay. Alemtuzumab and thymoglobulin were similarly very potent in inducing NK cell death in vitro. Even in low concentrations (1 microg/ml) the antibodies induced apoptosis and necrosis in a relevant percentage of NK cells (30%). However, the number of tumor reactive (CD107a+…

AdultAdolescentAntibodies Neoplasmmedicine.medical_treatmentApoptosisHematopoietic stem cell transplantationAntibodies Monoclonal HumanizedLymphocyte DepletionNatural killer cellCell Line TumormedicineHumansTransplantation HomologousProgenitor cellAlemtuzumabAgedAntilymphocyte SerumTransplantationCell DeathThymoglobulinbusiness.industryHematopoietic Stem Cell TransplantationAntibodies MonoclonalHematologyMiddle Agedmedicine.diseaseKiller Cells NaturalTransplantationmedicine.anatomical_structureGraft-versus-host diseaseImmunologyAlemtuzumabStem cellbusinessImmunosuppressive Agentsmedicine.drugBone Marrow Transplantation
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Low bcl-2 expression and increased spontaneous apoptosis in T-lymphocytes from newly-diagnosed IDDM patients.

1995

The bcl-2 gene product has been shown to regulate apoptotic cell death, and its dysregulation has been shown to induce several abnormalities in the immune system. No data exist regarding bcl-2 expression in autoimmune diseases, such as human insulin-dependent diabetes mellitus (IDDM). We investigated bcl-2 protein expression by testing T lymphocytes from 15 newly-diagnosed (3 weeks) IDDM patients in comparison to 10 age-matched control subjects. The expression of bcl-2 on CD3+ lymphocyte subsets was investigated after membrane permeabilization by two- or three-colour immunofluorescence. When the percentage and mean fluorescence intensity (MFI) of bcl-2+/CD3+ cells from normal individuals an…

AdultBlood GlucoseMaleProgrammed cell deathmedicine.medical_specialtyAdolescentCD3 ComplexEndocrinology Diabetes and MetabolismLymphocyteCD3T-LymphocytesGene ExpressionApoptosisBiologychemistry.chemical_compoundImmune systemAntigens CDReference ValuesRisk FactorsT-Lymphocyte SubsetsInternal medicineProto-Oncogene ProteinsInternal MedicinemedicineHumansFamilyPropidium iodideAutoantibodiesAutoimmune diseaseGlycated HemoglobinT lymphocytemedicine.diseaseFlow Cytometrymedicine.anatomical_structureEndocrinologyDiabetes Mellitus Type 1chemistryProto-Oncogene Proteins c-bcl-2ApoptosisCase-Control Studiesbiology.proteinFemaleDiabetologia
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CD4+ CCR5+ and CD4+ CCR3+ lymphocyte subset and monocyte apoptosis in patients with acute visceral leishmaniasis

2004

The potential involvement of apoptosis in the pathogenesis of visceral leishmaniasis (VL) was examined by studying spontaneous and Leishmania antigen (LAg)-induced apoptosis using cryopreserved peripheral blood mononuclear cells (PBMC) of Sicilian patients with VL. Results indicate that monocytes and T lymphocytes from acute VL patients show a significantly higher level of apoptosis compared with that observed in healed subjects. The percentage of apoptotic cells was higher in monocytes than in T lymphocytes. T cells involved in programmed cell death (PCD) were mainly of the CD4(+) phenotype. In particular, the T helper 1-type (Th1) subset, as evaluated by chemokine receptor-5 (CCR5) expres…

AdultCD4-Positive T-LymphocytesProgrammed cell deathChemokineReceptors CCR5Receptors CCR3ImmunologyAntigens ProtozoanApoptosisCD8-Positive T-LymphocytesBiologyPeripheral blood mononuclear cellMonocytesParacrine signallingAntigenmedicineHumansImmunology and Allergyfas ReceptorAutocrine signallingCells CulturedMonocyteOriginal ArticlesTh1 CellsLymphocyte Subsetsmedicine.anatomical_structureApoptosisAcute DiseaseImmunologyLeukocytes Mononuclearbiology.proteinLeishmaniasis VisceralReceptors ChemokineImmunology
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Impact of a preceding radiotherapy on the outcome of immune checkpoint inhibition in metastatic melanoma: a multicenter retrospective cohort study of…

2020

BackgroundImmune checkpoint inhibition (ICI) is an essential treatment option in melanoma. Its outcome may be improved by a preceding radiation of metastases. This study aimed to investigate the impact of a preceding radiotherapy on the clinical outcome of ICI treatment.MethodsThis multicenter retrospective cohort study included patients who received anti-cytotoxic T-lymphocyte-associated protein 4 (CTLA-4) or anti-programmed cell death protein 1 (PD-1) ICI with or without preceding radiotherapy for unresectable metastatic melanoma. ICI therapy outcome was measured as best overall response (BOR), progression-free (PFS) and overall survival (OS). Response and survival analyses were adjusted …

AdultMale0301 basic medicineOncologyCancer Researchmedicine.medical_specialtySkin NeoplasmsMetastatic melanoma2435medicine.medical_treatmentProgrammed Cell Death 1 ReceptorImmunologyMedizin03 medical and health sciences0302 clinical medicineInternal medicinemedicineHumansImmunology and AllergyCTLA-4 Antigen1506Immune Checkpoint InhibitorsMelanomaradiotherapyRC254-282Survival analysisRetrospective StudiesClinical/Translational Cancer ImmunotherapyPharmacologybusiness.industryMelanomaConfoundingNeoplasms. Tumors. Oncology. Including cancer and carcinogensRetrospective cohort studyChemoradiotherapyMiddle Agedmedicine.diseaseProgression-Free SurvivalImmune checkpointRadiation therapy030104 developmental biologyOncology030220 oncology & carcinogenesisRelative riskMolecular MedicineFemalebusinessJournal for ImmunoTherapy of Cancer
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Cigarette smoke causes caspase-independent apoptosis of bronchial epithelial cells from asthmatic donors

2015

BackgroundEpidemiologic studies have demonstrated important links between air pollution and asthma. Amongst these pollutants, environmental cigarette smoke is a risk factor both for asthma pathogenesis and exacerbation. As the barrier to the inhaled environment, the bronchial epithelium is a key structure that is exposed to cigarette smoke.ObjectivesSince primary bronchial epithelial cells (PBECs) from asthmatic donors are more susceptible to oxidant-induced apoptosis, we hypothesized that they would be susceptible to cigarette smoke-induced cell death.MethodsPBECs from normal and asthmatic donors were exposed to cigarette smoke extract (CSE); cell survival and apoptosis were assessed by fl…

AdultMaleProgrammed cell deathDNA damageScienceCaspase 3ApoptosisBronchiBiologyTobacco smokeAntioxidantschemistry.chemical_compoundYoung Adultparasitic diseasesHumansAgricultural and Biological Sciences (all); Biochemistry Genetics and Molecular Biology (all); Medicine (all)MultidisciplinaryBiochemistry Genetics and Molecular Biology (all)Caspase-Independent ApoptosisCaspase 3Medicine (all)QSmokingREpithelial CellsGlutathioneMiddle AgedAscorbic acid3. Good healthchemistryAgricultural and Biological Sciences (all)13. Climate actionApoptosisImmunologyMedicineFemaleResearch Article
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Apoptosis resistance in epithelial tumors is mediated by tumor-cell-derived interleukin-4

2008

We investigated the mechanisms involved in the resistance to cell death observed in epithelial cancers. Here, we identify that primary epithelial cancer cells from colon, breast and lung carcinomas express high levels of the antiapoptotic proteins PED, cFLIP, Bcl-xL and Bcl-2. These cancer cells produced interleukin-4 (IL-4), which amplified the expression levels of these antiapoptotic proteins and prevented cell death induced upon exposure to TRAIL or other drug agents. IL-4 blockade resulted in a significant decrease in the growth rate of epithelial cancer cells and sensitized them, both in vitro and in vivo, to apoptosis induction by TRAIL and chemotherapy via downregulation of the antia…

AdultMaleProgrammed cell deathLung NeoplasmsTime Factorsapoptosis interleukin-4 cancer stem cells cancer chemiotherapy cytokinesCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinAntineoplastic AgentsApoptosisBreast NeoplasmsBiologyTNF-Related Apoptosis-Inducing LigandTumor Cells CulturedmedicineHumansAutocrine signallingMolecular BiologyInterleukin 4AgedCell ProliferationSettore MED/04 - Patologia GeneraleCell DeathDose-Response Relationship DrugCell growthCarcinomaIntracellular Signaling Peptides and ProteinsAntibodies MonoclonalInterleukin-4 Receptor alpha SubunitCorrectionCancerCell BiologyMiddle AgedPhosphoproteinsmedicine.diseaseUp-RegulationCell biologyAutocrine CommunicationProto-Oncogene Proteins c-bcl-2Drug Resistance NeoplasmApoptosisColonic NeoplasmsCancer cellFemaleInterleukin-4Interleukin-4 Cancer stem cellsSignal transductionApoptosis Regulatory ProteinsSignal Transduction
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Leukocytes in neuronal ceroid-lipofuscinoses: function and apoptosis

1997

The neuronal ceroid-lipofuscinoses (NCL) are a group of progressive encephalopathies with a fatal course that are mostly of autosomal recessive inheritance. The pathophysiological mechanisms causing the diseases are not known. The characteristic histomorphological feature of the NCL is an abnormal lysosomal accumulation of lipopigments in neural and extraneural cells, including peripheral blood leukocytes. We studied the function of peripheral venous blood immunocompetent cells in ten patients with NCL and in age- and sex-matched controls to determine how, if at all, the accumulation of intracytoplasmic storage material influences the functional capacity of affected tissue. Our results did …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyAdolescentmedicine.medical_treatmentImmunoglobulinsApoptosisImmunoglobulin EImmunophenotypingPathogenesisDevelopmental NeuroscienceNeuronal Ceroid-LipofuscinosesSuperoxidesLeukocytesmedicineHumansChildRespiratory BurstbiologyInterleukin-6Interleukin-8General Medicinemedicine.diseasePathophysiologyCytokineApoptosisChild PreschoolPediatrics Perinatology and Child HealthImmunologybiology.proteinFemaleNeuronal ceroid lipofuscinosisNeurology (clinical)AntibodyBiomarkersCell DivisionInterleukin-1Brain and Development
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Apoptosis-related Proteins in Skeletal Muscle Fibers of Spinal Muscular Atrophy

1997

There is evidence that apoptosis in spinal muscular atrophies (SMA) is not restricted to motor neurons but also affects muscle fibers. Studying the expression of several apoptosis-associated proteins we found constant expression of bax in muscle fibers, which promoted cell death. The expression of bax correlated with defective innervation of muscle fibers was also indicated by upregulation of N-CAM. While in early-onset SMA atrophic as well as normo- and hypertrophic muscle fibers displayed expression of bax, muscle fibers in late-onset SMA and peripheral neuropathies showed bax-expression only in atrophic fibers. Other investigated apoptosis-associated factors comprised interleukin-1 beta …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyMuscle Fibers Skeletalbcl-X ProteinMuscle ProteinsApoptosisBiologyMicrofilamentPathology and Forensic MedicineMuscular Atrophy SpinalCellular and Molecular NeuroscienceReference ValuesProto-Oncogene ProteinsmedicineHumansMyocyteMuscle SkeletalActinAgedbcl-2-Associated X ProteinCaspase 1InfantPeripheral Nervous System DiseasesGeneral MedicineSpinal muscular atrophyMiddle AgedSMA*Spinal muscular atrophiesmedicine.diseaseCell biologyCysteine EndopeptidasesProto-Oncogene Proteins c-bcl-2NeurologyFemaleNeural cell adhesion moleculeNeurology (clinical)Journal of Neuropathology and Experimental Neurology
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NF-κB protects Behçet's disease T cells against CD95-induced apoptosis up-regulating antiapoptotic proteins

2005

Objective To determine whether prolongation of the inflammatory reaction in patients with Behcet's disease (BD) is related to apoptosis resistance and is associated with the up-regulation of antiapoptotic factors. Methods The percentage of cell death was evaluated by flow cytometry in peripheral blood mononuclear cells from 35 patients with BD and 30 healthy volunteers. The expression levels of antiapoptotic factors and NF-κB regulatory proteins were measured using Western blotting and immunohistochemical analyses. To down-regulate NF-κB nuclear translocation, BD T lymphocytes were exposed in vitro to thalidomide and subjected to transfection with NF-κB small interfering RNA. Results Althou…

AdultMaleSmall interfering RNAProgrammed cell deathT-LymphocytesT cellImmunologyCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinApoptosisCaspase 3TransfectionCaspase 8RheumatologyHumansImmunology and AllergyMedicinePharmacology (medical)fas ReceptorRNA Small InterferingCells CulturedDose-Response Relationship Drugbusiness.industryBehcet SyndromeIntracellular Signaling Peptides and ProteinsNF-kappa BTransfectionFlow CytometryFas receptorThalidomideUp-Regulationmedicine.anatomical_structureGene Expression RegulationProto-Oncogene Proteins c-bcl-2ApoptosisImmunologyLeukocytes MononuclearCancer researchFemalebusinessArthritis & Rheumatism
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PD-1, PD-L1 and PD-L2 Gene Expression on T-Cells and Natural Killer Cells Declines in Conjunction with a Reduction in PD-1 Protein during the Intensi…

2015

Background The PD-1 axis is a cell intrinsic immunoregulatory pathway that mediates T cell exhaustion in chronic infection particularly in some viral infections. We hypothesized that PD-1, PD-L1 and PD-L2 would be highly expressed in untreated tuberculosis patients compared to controls due to their chronic infection and would decrease with successful TB treatment. Materials and Methods Untreated tuberculosis patients (n = 26) were recruited at diagnosis and followed up during treatment. Household contacts (n = 24) were recruited to establish baseline differences. Blood gene expression ex vivo was investigated using qRT-PCR. Flow cytometry was performed to establish protein expression patter…

AdultMaleTuberculosisT cellProgrammed Cell Death 1 ReceptorAntitubercular Agentslcsh:MedicineDown-RegulationB7-H1 AntigenImmunophenotypingMycobacterium tuberculosisYoung AdultImmunophenotypingT-Lymphocyte SubsetsPD-L1medicineCytotoxic T cellHumansTuberculosisLymphocyte Countlcsh:ScienceAgedMultidisciplinarybiologylcsh:RMycobacterium tuberculosisMiddle Agedbiology.organism_classificationmedicine.diseaseProgrammed Cell Death 1 Ligand 2 ProteinGranzyme BKiller Cells Naturalmedicine.anatomical_structureGene Expression RegulationImmunologybiology.proteinlcsh:QFemaleCD8Research Article
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