Search results for "Cell Division"

showing 10 items of 457 documents

CORRELATED BIOCHEMICAL AND ULTRASTRUCTURAL CHANGES IN NITROGEN-STARVED EUGLENA GRACILIS1

1996

Growth of Euglena gracilis Z Pringsheim under photoheterotrophic conditions in a nitrogen-deprived medium resulted in progressive loss of chloroplastic material until total bleaching of the cells occurred. Biochemical analysis and ultrastructural observation of the first stages of the starvation process demonstrated an early lag phase (from 0 to 9 h) in which cells increased in size, followed by a period of cell division, apparently supported by the mobilization of some chloroplastic proteins such as the photosynthetic CO2-fixing enzyme ribulose-1,5-bisphosphate carboxylase/oxygenase. The degradation of the enzyme started after 9 h of starvation and was preceded by a transient concentration…

OxygenaseEuglena gracilisbiologyCell divisionved/biologyved/biology.organism_classification_rank.speciesPlant ScienceAquatic Sciencebiology.organism_classificationPhotosynthesisEuglenaPyrenoidChloroplastchemistry.chemical_compoundBiochemistrychemistryParamylonJournal of Phycology
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MCC1019, a selective inhibitor of the Polo-box domain of Polo-like kinase 1 as novel, potent anticancer candidate

2019

Polo-like kinase (PLK1) has been identified as a potential target for cancer treatment. Although a number of small molecules have been investigated as PLK1 inhibitors, many of which showed limited selectivity. PLK1 harbors a regulatory domain, the Polo box domain (PBD), which has a key regulatory function for kinase activity and substrate recognition. We report on 3-bromomethyl-benzofuran-2-carboxylic acid ethyl ester (designated: MCC1019) as selective PLK1 inhibitor targeting PLK1 PBD. Cytotoxicity and fluorescence polarization-based screening were applied to a library of 1162 drug-like compounds to identify potential inhibitors of PLK1 PBD. The activity of compound MC1019 against the PLK1…

PBD Polo box domainMTD maximal tolerance doseCDC25 cell division cycle 25HIF-1α hypoxia-inducible factor 1 αMST microscale thermophoresisIC50 50% inhibition concentrationMFP M phase promoting factorPARP-1 poly(ADP-ribose) polymerase-10302 clinical medicineFOXO forkhead box ONec-1 necrostatin 1CDC2 cell division cycle protein 2 homologGeneral Pharmacology Toxicology and PharmaceuticsMitotic catastropheCDK cyclin-dependent kinase0303 health sciencesChemistryPolo-like kinaseMono-targeted therapyCell cycleBUBR1 budding uninhibited by benzimidazole-related 1Polo box domain030220 oncology & carcinogenesisPLK1 Polo-like kinaseNecroptosisSpindle damagePLK1IHC immunohistochemistryOriginal articleNecroptosisCell cyclePLK1APC/C anaphase-promoting complex/cyclosomePLK3ABC avidin-biotin complexPI propidium iodide03 medical and health sciencesFBS fetal bovine serumPDB Protein Data BankKd the dissociation constantKinase activity030304 developmental biologyAkt/PKB signaling pathwayCell growthlcsh:RM1-950LC3 light chain 3lcsh:Therapeutics. PharmacologyCancer researchDAPKs death-associated protein kinase3-MA 3-methyladenineDAPI 4′6-diamidino-2-phenylindoleSAC spindle assembly checkpointActa Pharmaceutica Sinica B
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“Super p53” Mice Display Retinal Astroglial Changes

2013

Tumour-suppressor genes, such as the p53 gene, produce proteins that inhibit cell division under adverse conditions, as in the case of DNA damage, radiation, hypoxia, or oxidative stress (OS). The p53 gene can arrest proliferation and trigger death by apoptosis subsequent to several factors. In astrocytes, p53 promotes cell-cycle arrest and is involved in oxidative stress-mediated astrocyte cell death. Increasingly, astrocytic p53 is proving fundamental in orchestrating neurodegenerative disease pathogenesis. In terms of ocular disease, p53 may play a role in hypoxia due to ischaemia and may be involved in the retinal response to oxidative stress (OS). We studied the influence of the p53 ge…

PathologyAnatomy and PhysiologyCell divisionMouselcsh:MedicineFluorescent Antibody Techniquemedicine.disease_causechemistry.chemical_compoundMiceMolecular Cell Biologylcsh:ScienceMultidisciplinaryGlial fibrillary acidic proteinAnimal ModelsCell biologymedicine.anatomical_structureMedicineOftalmologíaDNA modificationAstrocyteResearch ArticleSignal TransductionProgrammed cell deathmedicine.medical_specialtyCell PhysiologyHistologyOcular AnatomyNeurocienciasMice TransgenicBiologyRetinaModel OrganismsOcular SystemGlial Fibrillary Acidic ProteinmedicineGeneticsAnimalsBiologyRetinaStaining and Labelinglcsh:RRetinalAnatomía ocularMice Inbred C57BLGenética médicaOphthalmologychemistryApoptosisAstrocytesbiology.proteinlcsh:QGene expressionGene FunctionTumor Suppressor Protein p53Animal GeneticsOxidative stress
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Expression of T-cadherin in tumor cells influences invasive potential of human hepatocellular carcinoma

2006

Overexpression of T-cadherin (T-cad) transcripts occurs in approximately 50% of human hepatocellular carcinomas (HCCs). To elucidate T-cad functions in HCC, we examined T-cad protein expression in normal and tumoral human livers and hepatoma cell lines and investigated its influence on invasive potential of HCC using RNA interference silencing of T-cad expression in Mahlavu cells. Whereas T-cad expression was restricted to endothelial cells (EC) from large blood vessels in normal livers, it was up-regulated in sinusoidal EC from 8/15 invasive HCCs. Importantly, in three of them (38%) T-cad was detected in tumor cells within regions in which E-cadherin expression was absent. Among six hepato…

Pathologymedicine.medical_specialtyCarcinoma HepatocellularTranscription GeneticLiver cytologyCell Culture TechniquesMotilityBiologyTransfectionBiochemistryRNA interferenceCell MovementCell Line TumorGeneticsmedicineGene silencingAnimalsHumansNeoplasm Invasivenesscardiovascular diseasesRNA Small InterferingMolecular BiologyDNA PrimersWound Healingprimary tumors cadherin switch cell invasion hepatoma cell lines RNA interferenceLiver NeoplasmsEndothelial CellsTransfectionHCCSFibroblastsCadherinsdigestive system diseasesT-cadherinLiverCell cultureCancer researchHepatocytesRabbitsCell DivisionBiotechnology
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New cell lines of gastric and pancreatic cancer: distinct morphology, growth characteristics, expression of epithelial and immunoregulatory antigens.

1995

Two new cell lines from stomach cancers and one from a pancreatic carcinoma are presented. MZ-GC-1 was established from a hepatic metastasis of a well differentiated gastric adenocarcinoma. MZ-GC-2 was derived from ascites induced by a poorly differentiated gastric adenocarcinoma. MZ-PC-1 originated from the pleural effusion of a moderately well differentiated pancreatic ductal adenocarcinoma. MZ-GC-1 cells were adherent and partially polarized, connected tightly via desmosomes. In contrast MZ-GC-2 cells consisted of slightly adherent or floating subpopulations and displayed no desmosomes. MZ-PC-1 cells were adherent and showed polarized growth, connected by apical junctional complexes. Cel…

Pathologymedicine.medical_specialtyCell divisionCellular differentiationCellBiologyAdenocarcinomaEpitheliumPathology and Forensic MedicineCytokeratinNude mouseStomach NeoplasmsPancreatic cancermedicineBiomarkers TumorHumansNeoplasm MetastasisMolecular BiologyCell Line TransformedLiver NeoplasmsCell DifferentiationCell BiologyGeneral Medicinebiology.organism_classificationmedicine.diseasePancreatic NeoplasmsMicroscopy Electronmedicine.anatomical_structureCell cultureAntigens SurfaceCancer researchMicroscopy Electron ScanningPancreasCell DivisionVirchows Archiv : an international journal of pathology
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Adhesion, growth and cytoskeletal characteristics of 8701-BC breast carcinoma cells cultured in the presence of type V collagen

1990

Type V collagen is one of the minor components of the extracellular matrix (ECM) whose content is increased in cases of ductal infiltrating carcinomas of the breast. In order to clarify its biological role, we have investigated the effect of this molecule, both as substrate and as soluble factor, on the behaviour of a breast carcinoma cell line (8701-BC) grown in vitro. Cell-collagen adhesion was monitored for 24 h from plating in the absence or presence of serum. The influence of type V collagen on cell growth was followed during 9 days of culture, and the actin-vinculin arrangement was studied by simultaneous fluorescent immuno-staining. The results indicate that type V collagen is not a …

Pathologymedicine.medical_specialtyCell growthBreast NeoplasmsAdhesionBiologyMolecular biologyIn vitroExtracellular matrixCytoskeletal ProteinsCarcinoma Intraductal NoninfiltratingOncologyCell cultureCell AdhesionTumor Cells CulturedmedicineHumansNeoplastic cellCollagenCytoskeletonBreast carcinomaCell DivisionEuropean Journal of Cancer and Clinical Oncology
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Proliferating macrophages, dendritic cells, natural killer cells, T and B lymphocytes in the middle ear and Eustachian tube mucosa during experimenta…

2001

SummaryAlthough many studies focus on the increase of immunocompetent cells within the middle ear mucosa during acute otitis media it is poorly understood how this increase is mediated. The differentiation between two possible causes, i.e. immigration and local proliferation, would help to better understand the pathophysiology of this disease. Therefore, the number of proliferating macrophages, dendritic cells, natural killer cells and T and B lymphocytes was studied during acute otitis media in the rat middle ear mucosa (ME mucosa) and Eustachian tube mucosa (ET mucosa) by labelling proliferating leucocytes with the DNA precursor bromodeoxyuridine (BrdU). By removing the middle ear and Eus…

Pathologymedicine.medical_specialtyEustachian tubeT-LymphocytesImmunologyEar MiddleBiologyOtitis Media SuppurativeNatural killer cellchemistry.chemical_compoundmedicineAnimalsImmunology and AllergyMacrophageAntigen-presenting cellB-LymphocytesMucous MembraneEustachian TubeMacrophagesDNADendritic CellsDendritic cellT lymphocyteEar DiseaseRatsKiller Cells Naturalmedicine.anatomical_structureBromodeoxyuridinechemistryRats Inbred LewAcute DiseaseImmunologyMiddle earFemaleCell DivisionBromodeoxyuridineClinical and Experimental Immunology
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Usefulness of immunohistochemical staining for p53 in the prognosis of breast carcinomas: correlations with established prognosis parameters and with…

1995

Mutations of the p53 gene often result in the overexpression of p53 protein. Previous studies have suggested that the function of p53 and its mutant protein forms may be linked with the disease course of patients with a breast carcinoma. In the present study, we tested 462 primary breast carcinomas for the presence of p53 antigen using immunohistochemical methods employing antibodies against the clone, DO-1. These tumors were also immunohistochemically stained using the monoclonal antibody, MIB-1, in order to demonstrate the presence of Ki67. Comparison of the presence of p53 with other prognostic parameters revealed highly significant negative correlations with estrogen- and progesterone-r…

Pathologymedicine.medical_specialtyMammary glandBreast NeoplasmsDisease-Free SurvivalAntigenmedicineCarcinomaBiomarkers TumorHumansProliferation MarkerCell NucleusEpitheliomabiologybusiness.industryObstetrics and GynecologyAntibodies Monoclonalmedicine.diseasePrognosisImmunohistochemistrySurvival Analysismedicine.anatomical_structureOncologyMutationCancer researchbiology.proteinImmunohistochemistryRegression AnalysisFemaleAntibodyTumor Suppressor Protein p53Breast carcinomabusinessCell DivisionGynecologic oncology
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Effects of nano-scaled particles on endothelial cell function in vitro: studies on viability, proliferation and inflammation.

2004

Recent studies give support for a connection between the presence of inorganic particles (of microm and nm size) in different organs and tissues and the development of inflammatory foci, called granulomas. As the potential source of particles (e.g. porcelain dental bridges) and the location of particle detection were topographically far apart, a distribution via the blood stream appears highly probable. Thus, endothelial cells, which line the inner surface of blood vessels, would come into direct contact with these particles, making particle-endothelial interactions potentially pathogenically relevant. The objective of this study was to evaluate the effects that five different nano-scaled p…

Pathologymedicine.medical_specialtyMaterials scienceEffectsCell divisionCell Survivalnano-scaledproliferationCellBiomedical EngineeringBiophysicsBiocompatible MaterialsBioengineeringInflammationBiomaterialsNickelIn vivoMaterials TestingendothelialmedicineHumansInterleukin 8Particle SizePolyvinyl ChlorideCells CulturedTitaniumparticlesfunctionNanotubesForeign-Body ReactionviabilityInterleukin-8Endothelial Cellsin vitroCobaltcellSilicon DioxideEndothelial stem cellKi-67 Antigenmedicine.anatomical_structureinflammationBiophysicsParticle sizemedicine.symptomEffects; nano-scaled; particles; endothelial; cell; function; in vitro; viability; proliferation; inflammationCell DivisionBlood vessel
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Assessment of proliferative activity in breast cancer: MIB-1 immunohistochemistry versus mitotic figure count.

1999

Abstract The proliferative activity is one of the most important single prognostic parameters in breast cancer diagnosis and the time-honored measure of proliferative activity, the mitotic figure count, is an integral component of most combined prognostic scores. The detection of the cell cycle-specific antigens Ki-67, and the development of anti-Ki-67 antibodies, including the paraffin-reactive antibody MIB-1, have established immunohistochemical detection of cell cycle-specific antigens as a measure of proliferative activity in breast cancer diagnosis. The current study was performed to correlate mitotic figure counts with the proliferative activity as assessed by MIB-1 immunohistochemist…

Pathologymedicine.medical_specialtyMitotic indexMammary glandBreast NeoplasmsPathology and Forensic MedicineBreast cancermedicineCarcinomaMitotic IndexHumansHigh-power fieldObserver VariationbiologyCarcinoma Ductal BreastNuclear ProteinsAntigens Nuclearmedicine.diseaseImmunohistochemistrymedicine.anatomical_structureKi-67 AntigenKi-67biology.proteinMitotic FigureImmunohistochemistryCell DivisionHuman pathology
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