Search results for "Cell Lineage"

showing 10 items of 126 documents

Development, Differentiation, and Diversity of Innate Lymphoid Cells

2014

Recent years have witnessed the discovery of an unprecedented complexity in innate lymphocyte lineages, now collectively referred to as innate lymphoid cells (ILCs). ILCs are preferentially located at barrier surfaces and are important for protection against pathogens and for the maintenance of organ homeostasis. Inappropriate activation of ILCs has been linked to the pathogenesis of inflammatory and autoimmune disorders. Recent evidence suggests that ILCs can be grouped into two separate lineages, cytotoxic ILCs represented by conventional natural killer (cNK) cells and cytokine-producing helper-like ILCs (i.e., ILC1s, ILC2s, ILC3s). We will focus here on current work in humans and mice th…

Transcription GeneticLymphocyteCellular differentiationImmunologyBiologyArticleTight Junctions03 medical and health sciencesMice0302 clinical medicinemedicineTranscriptional regulationCytotoxic T cellImmunology and AllergyAnimalsHumansCell Lineageskin and connective tissue diseases030304 developmental biologyRegulation of gene expression0303 health sciencesStem CellsInnate lymphoid cellCell DifferentiationT-Lymphocytes Helper-InducerImmunity InnateKiller Cells Naturalbody regionsMulticellular organismmedicine.anatomical_structureInfectious DiseasesGene Expression RegulationImmunologyCytokinesStem cell030215 immunologySignal TransductionImmunity
researchProduct

Human Fetal Aorta Contains Vascular Progenitor Cells Capable of Inducing Vasculogenesis, Angiogenesis, and Myogenesis in Vitro and in a Murine Model …

2007

Vasculogenesis, the formation of blood vessels in embryonic or fetal tissue mediated by immature vascular cells (ie, angioblasts), is poorly understood. We report the identification of a population of vascular progenitor cells (hVPCs) in the human fetal aorta composed of undifferentiated mesenchymal cells that coexpress endothelial and myogenic markers. Under culture conditions that promoted cell differentiation, hVPCs gave rise to a mixed population of mature endothelial and mural cells when progenitor cells were stimulated with vascular endothelial growth factor-A or platelet-derived growth factor-betabeta. hVPCs grew as nonadherent cells and, when embedded in a three-dimensional collagen…

Vascular Endothelial Growth Factor AAngiogenesisBecaplerminNeovascularization PhysiologicAntigens CD34BiologyMuscle DevelopmentMural cellPathology and Forensic MedicineAngiopoietin-2MiceFetusVasculogenesisAntigens CDIschemiaAnimalsHumansCell LineageAC133 AntigenProgenitor cellAortaCells CulturedGlycoproteinsPlatelet-Derived Growth FactorStem CellsProto-Oncogene Proteins c-sisVascular Endothelial Growth Factor Receptor-2Cell biologyEndothelial stem cellVascular endothelial growth factor BVascular endothelial growth factor AVascular endothelial growth factor CImmunologyBlood VesselsPeptidesBiomarkersRegular ArticlesThe American Journal of Pathology
researchProduct

Heart infarct in NOD-SCID mice: therapeutic vasculogenesis by transplantation of human CD34+ cells and low dose CD34+KDR+ cells

2004

Hematopoietic (Hem) and endothelial (End) lineages derive from a common progenitor cell, the hemangioblast: specifically, the human cord blood (CB) CD34+KDR+ cell fraction comprises primitive Hem and End cells, as well as hemangioblasts. In humans, the potential therapeutic role of Hem and End progenitors in ischemic heart disease is subject to intense investigation. Particularly, the contribution of these cells to angiogenesis and cardiomyogenesis in myocardial ischemia is not well established. In our studies, we induced myocardial infarct (MI) in the immunocompromised NOD-SCID mouse model, and monitored the effects of myocardial transplantation of human CB CD34+ cells on cardiac function.…

Vascular Endothelial Growth Factor AneoangiogenesisTime FactorsAngiogenesisCell TransplantationHeart VentriclesCD34Myocardial InfarctionAntigens CD34ApoptosisMice SCIDBiologySCIDPeripheral blood mononuclear cellBiochemistryCulture Media Serum-FreeSerum-FreeCell FusionMiceVasculogenesisMice Inbred NODparasitic diseasesGeneticsAnimalsHumansVentricular Functionendothelial precursorsCell LineageProgenitor cellAntigensMolecular Biologyneoangiogenesis endothelial precursors hematopoietic stem cellsHemodynamicsFetal BloodVascular Endothelial Growth Factor Receptor-2Coculture Techniqueshematopoietic stem cellsCulture MediaTransplantationAutocrine CommunicationCord bloodImmunologycardiovascular systemCancer researchHemangioblastInbred NODCD34neoangiogenesis; endothelial precursors; hematopoietic stem cells; Animals; Antigens CD34; Apoptosis; Autocrine Communication; Cell Fusion; Cell Lineage; Coculture Techniques; Culture Media Serum-Free; Fetal Blood; Heart Ventricles; Hemodynamics; Humans; Mice; Mice Inbred NOD; Mice SCID; Myocardial Infarction; Time Factors; Vascular Endothelial Growth Factor A; Vascular Endothelial Growth Factor Receptor-2; Ventricular Function; Cell Transplantation; Biotechnology; Biochemistry; Molecular Biology; GeneticsBiotechnology
researchProduct

Electrophysiological and morphological properties of Cajal–Retzius cells with different ontogenetic origins

2010

International audience; The different origins of Cajal-Retzius cells (CRc) as well as their diverse molecular profile suggest that this cell type may represent different neuronal subpopulations. In order to investigate whether CRc from different origins show distinct functional or morphological characteristics we used transgenic Dbx1(cre);ROSA26(YFP) mice in which two subpopulations of CRc, originating from the septum and ventral pallium (VP) at the pallial-subpallial border (PSB), were permanently labeled by yellow fluorescent protein (YFP) expression. Electrophysiological properties of YFP(+) and YFP(-) CRc were investigated by whole-cell patch-clamp recordings, while a thorough somatoden…

Yellow fluorescent proteinCell typePatch-Clamp TechniquesNeurogenesisAction PotentialsGlutamic AcidMice Transgenicmacromolecular substancesReceptors N-Methyl-D-AspartateMembrane PotentialsMice03 medical and health scienceschemistry.chemical_compoundOrgan Culture Techniques0302 clinical medicineBiocytinAnimalsCell LineagePatch clampCell Shapegamma-Aminobutyric AcidImage Cytometry030304 developmental biologyCerebral CortexNeurons0303 health sciencesbiologyStem CellsGeneral NeurosciencefungiCell DifferentiationDendritesHyperpolarization (biology)digestive system diseasesCell biologyLuminescent ProteinsElectrophysiologynervous systemchemistrybiology.proteinGABAergic[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]DBX1Nerve NetExcitatory Amino Acid Antagonists030217 neurology & neurosurgeryNeuroscience
researchProduct

Pharmacogenomic identification of small molecules for lineage specific manipulation of subventricular zone germinal activity

2017

Strategies for promoting neural regeneration are hindered by the difficulty of manipulating desired neural fates in the brain without complex genetic methods. The subventricular zone (SVZ) is the largest germinal zone of the forebrain and is responsible for the lifelong generation of interneuron subtypes and oligodendrocytes. Here, we have performed a bioinformatics analysis of the transcriptome of dorsal and lateral SVZ in early postnatal mice, including neural stem cells (NSCs) and their immediate progenies, which generate distinct neural lineages. We identified multiple signaling pathways that trigger distinct downstream transcriptional networks to regulate the diversity of neural cells …

animal diseasesGene Identification and AnalysisGenetic NetworksAPC-PAIDMiceNeural Stem CellsCell SignalingLateral VentriclesDatabases GeneticGene Regulatory NetworksBiology (General)WNT Signaling CascadeNotch SignalingOrganic CompoundsBB/M029379/1GenomicsSignaling CascadesOligodendrogliaChemistryBBSRCPhysical Sciences[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Network AnalysisNeurovetenskaperSignal TransductionResearch ArticleBiotechnologyComputer and Information SciencesSignal InhibitionQH301-705.5NeurogenesisResearch and Analysis MethodsSmall Molecule LibrariesGenetics/dk/atira/pure/core/subjects/biomedicalsciencesAnimalsAdultsCell LineageComputer Simulation[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Molecular Biology TechniquesMolecular BiologyOrganic ChemistryGene MappingChemical CompoundsNeurosciencesBiology and Life SciencesRCUKBiomedical SciencesCell BiologyNerve RegenerationSignaling NetworksGene Expression Regulationnervous systemSmall MoleculesAge GroupsPeople and PlacesPopulation GroupingsTranscriptome
researchProduct

Context-dependent Pax-5 repression of a PU.1/NF-κB regulated reporter gene in B lineage cells

2001

Enhancers located in the 3' end of the locus in part regulate immunoglobulin heavy chain (IgH) gene expression. One of these enhancers, HS 1,2, is developmentally regulated by DNA binding proteins like NF-kappaB, Pax-5 and the protein complex NF-alphaP in B lineage cells. Here we report that NF-alphaP is the ets protein PU.1. A glutathione-S-transferase (GST)-pulldown assay demonstrated that PU.1 can physically interact with NF-kappaB in solution. Experiments in COS cells showed that PU.1 and NF-kappaB (p50/c-Rel) can activate transcription of an enhancer linked reporter gene. The paired domain protein Pax-5 has previously been shown to repress enhancer-dependent transcription. Additional c…

animal structuresLymphomaTranscription GeneticEnhancer RNAsBiologyDNA-binding proteinMiceSOX4Genes ReporterTranscription (biology)CricetinaeProto-Oncogene ProteinsGene expressionGeneticsAnimalsCell LineageBinding siteEnhancerCells CulturedB-LymphocytesReporter geneNF-kappa BPAX5 Transcription FactorNuclear ProteinsGeneral MedicineMolecular biologyGlobinsDNA-Binding ProteinsEnhancer Elements GeneticGene Expression RegulationCOS Cellsembryonic structuresTrans-ActivatorsTranscription FactorsGene
researchProduct

The pattern of neuroblast formation, mitotic domains and proneural gene expression during early brain development in Drosophila.

2003

In the Drosophila embryo, studies on CNS development have so far mainly focused on the relatively simply structured ventral nerve cord. In the trunk, proneural genes become expressed in small cell clusters at specific positions of the ventral neuroectoderm. A lateral inhibition process mediated by the neurogenic genes ensures that only one cell within each proneural cluster delaminates as a neural stem cell (neuroblast). Thus, a fixed number of neuroblasts is formed, according to a stereotypical spatiotemporal and segmentally repeated pattern, each subsequently generating a specific cell lineage. Owing to higher complexity and hidden segmental organisation, the mechanisms underlying the dev…

animal structuresMitosisProneural genesBiologyNeuroblastLateral inhibitionEctodermMorphogenesisAnimalsCell LineageNeurons AfferentMolecular BiologyIn Situ HybridizationGeneticsNeuronsNeuroectodermGenes HomeoboxBrainGene Expression Regulation DevelopmentalNeural stem cellDrosophila melanogasterVentral nerve cordembryonic structuresScuteNeuroscienceGanglion mother cellNeurogliaBiomarkersDevelopmental BiologyDevelopment (Cambridge, England)
researchProduct

Instruction of haematopoietic lineage choices, evolution of transcriptional landscapes and cancer stem cell hierarchies derived from an AML1-ETO mous…

2013

The t(8;21) chromosomal translocation activates aberrant expression of the AML1-ETO (AE) fusion protein and is commonly associated with core binding factor acute myeloid leukaemia (CBF AML). Combining a conditional mouse model that closely resembles the slow evolution and the mosaic AE expression pattern of human t(8;21) CBF AML with global transcriptome sequencing, we find that disease progression was characterized by two principal pathogenic mechanisms. Initially, AE expression modified the lineage potential of haematopoietic stem cells (HSCs), resulting in the selective expansion of the myeloid compartment at the expense of normal erythro- and lymphopoiesis. This lineage skewing was foll…

cancer stem cellsCancer stem cells; Core binding factor acute myeloid leukaemia; Preclinical mouse model; Therapy target validation; Whole transcriptome sequencingMyeloidtherapy target validationOncogene Proteins FusionCloseupsBiologyGranulocyte-Macrophage Progenitor CellsTranslocation Geneticwhole transcriptome sequencingImmunophenotypingMiceGranulocyte-Macrophage Progenitor CellsCancer stem cellhemic and lymphatic diseasesmedicineAML1-ETOAnimalsCell Lineageacute myeloid leukaemiaLymphopoiesisProgenitor cellt(8;21)Research Articlespreclinical mouse modelGeneticsRegulation of gene expressionAntibiotics AntineoplasticSequence Analysis RNAcore binding factor acute myeloid leukaemiainducible mouse-modelHematopoietic Stem CellsMice Inbred C57BLDisease Models AnimalLeukemia Myeloid AcuteHaematopoiesisPhenotypemedicine.anatomical_structureGene Expression RegulationDoxorubicinCancer researchNeoplastic Stem CellsMolecular MedicineStem cell
researchProduct

Genetic and Molecular Characterization of The Human Osteosarcoma 3AB-OS Cancer Stem Cell Line: A Possible Model For Studying Osteosarcoma Origin and …

2013

Finding new treatments targeting cancer stem cells (CSCs) within a tumor seems to be critical to halt cancer and improve patient survival. Osteosarcoma is an aggressive tumor affecting adolescents, for which there is no second-line chemotherapy. Uncovering new molecular mechanisms underlying the development of osteosarcoma and origin of CSCs is crucial to identify new possible therapeutic strategies. Here, we aimed to characterize genetically and molecularly the human osteosarcoma 3AB-OS CSC line, previously selected from MG63 cells and which proved to have both in vitro and in vivo features of CSCs. Classic cytogenetic studies demonstrated that 3AB-OS cells have hypertriploid karyotype wit…

cancer stem cellsPhysiologyClinical Biochemistrymedicine.disease_causePolymerase Chain ReactionOsteosarcoma cancer stem cellSettore BIO/10 - BiochimicaChromosomes HumanGene Regulatory NetworksCopy-number variationOligonucleotide Array Sequence AnalysisGeneticsComparative Genomic HybridizationOsteosarcomabiologychromosomal aberrationGene Expression Regulation NeoplasticPhenotypemiRNAsNeoplastic Stem CellsOsteosarcomaMitosisBone NeoplasmsHMGA2Cancer stem cellCell Line TumormicroRNABiomarkers Tumorgene expression profilingmedicineHumansOsteosarcoma cancer stem cells; karyotype; chromosomal aberrations; gene expression profiling; miRNAsCell LineageGenetic Predisposition to DiseaseRNA MessengerCell NucleusChromosome AberrationsPloidiesModels GeneticComputational BiologyCancerCell Biologymedicine.diseasekaryotypeMicroRNAsKaryotypingbiology.proteinCancer researchCarcinogenesisComparative genomic hybridization
researchProduct

Topography of somatostatin gene expression relative to molecular progenitor domains during ontogeny of the mouse hypothalamus

2010

The hypothalamus comprises alar, basal, and floor plate developmental compartments. Recent molecular data support a rostrocaudal subdivision into rostral (terminal) and caudal (peduncular) halves. In this context, the distribution of neuronal populations expressing somatostatin (Sst) mRNA was analyzed in the developing mouse hypothalamus, comparing with the expression pattern of the genes Orthopedia (Otp), Distal-less 5 (Dlx5), Sonic Hedgehog (Shh), and Nk2 homeobox 1 (Nkx2.1). At embryonic day 10.5 (E10.5), Sst mRNA was first detectable in the anterobasal nucleus, a Nkx2.1-, Shh-, and Otp-positive basal domain. By E13.5, nascent Sst expression was also related to two additional Otp-positiv…

endocrine systemBasal plate (neural tube)forebrain[SDV]Life Sciences [q-bio]OtpNeuroscience (miscellaneous)Shhlcsh:RC321-571lcsh:QM1-69503 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineArcuate nucleusmedicine[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologySonic hedgehoghypothalamuslcsh:Neurosciences. Biological psychiatry. Neuropsychiatry[SDV.BDD]Life Sciences [q-bio]/Development BiologyOriginal Research030304 developmental biologyFloor plate0303 health sciencesAlar platebiologyDlk5forebrain;hypothalamus;Sst;Otp;Dlk5;Nkx2.1;Shh;in situ hybridization;CONTAINING NEURON SYSTEM;SONIC-HEDGEHOG;FOREBRAIN DEVELOPMENT;VENTRAL FOREBRAIN;DEVELOPMENTAL EXPRESSION;BRAIN-DEVELOPMENT;RAT HYPOTHALAMUS;GROWTH-HORMONE;CELL LINEAGES;DIENCEPHALONlcsh:Human anatomyCiencias naturales y ciencias de la saludSstNkx2.1medicine.anatomical_structureHypothalamusForebrainembryonic structuresNeuranatomybiology.protein[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]in situ hybridizationAnatomyNucleusNeuroscience030217 neurology & neurosurgery
researchProduct