Search results for "Cell Proliferation"

showing 10 items of 1056 documents

Histamine and spontaneously released mast cell granules affect the cell growth of human hepatocellular carcinoma cells

2007

The role of mast cells in tumor growth is still controversial. In this study we analyzed the effects of both histamine and pre-formed mediators spontaneously released by mast cells on the growth of two human hepatocellular carcinoma cell lines, HA22T/VGH and HuH-6, with different characteristics of differentiation, biological behavior and genetic defects. We showed that total mast cell releasate, exocytosed granules (granule remnants) and histamine reduced cell viability and proliferation in HuH-6 cells. In contrast, in HA22T/VGH cells granule remnants and histamine induced a weak but significant increase in cell growth. We showed that both cell lines expressed histamine receptors H(1) and …

medicine.medical_specialtyCarcinoma HepatocellularCell SurvivalSurvivinClinical BiochemistryHistamine AntagonistsApoptosisHistamine H1 receptorBiologyRanitidineBiochemistryExocytosisInhibitor of Apoptosis ProteinsHistamine receptorchemistry.chemical_compoundInternal medicineCell Line TumormedicineAnimalsHumansHistamine H4 receptorMast CellsEnterochromaffin-like cellRats WistarMolecular BiologyCells Culturedbeta CateninCell ProliferationCell growthCaspase 3Liver NeoplasmsMast cellMolecular biologyNeoplasm ProteinsRatsEnzyme ActivationEndocrinologymedicine.anatomical_structurechemistryCell cultureCyclooxygenase 2Molecular MedicineReceptors HistamineFemaleTerfenadinePoly(ADP-ribose) PolymerasesMicrotubule-Associated ProteinsHistamineHistamine
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Developmental and tumoral vascularization is regulated by G protein-coupled receptor kinase 2

2012

Tumor vessel dysfunction is a pivotal event in cancer progression. Using an in vivo neovascularization model, we identified G protein–coupled receptor kinase 2 (GRK2) as a key angiogenesis regulator. An impaired angiogenic response involving immature vessels was observed in mice hemizygous for Grk2 or in animals with endothelium-specific Grk2 silencing. ECs isolated from these animals displayed intrinsic alterations in migration, TGF-β signaling, and formation of tubular networks. Remarkably, an altered pattern of vessel growth and maturation was detected in postnatal retinas from endothelium-specific Grk2 knockout animals. Mouse embryos with systemic or endothelium-selective Grk2 ablation …

medicine.medical_specialtyG-Protein-Coupled Receptor Kinase 2Angiogenic SwitchAngiogenesisMedicinaActivin Receptors Type IIMelanoma ExperimentalReceptor Transforming Growth Factor-beta Type INeovascularization PhysiologicProtein Serine-Threonine KinasesBiologyMural cellGrk2Transforming Growth Factor beta1NeovascularizationMiceDownregulation and upregulationCell MovementPregnancyInternal medicinemedicineAnimalsHumansCell ProliferationHemizygoteMice KnockoutG protein-coupled receptor kinaseTumorNeovascularization PathologicEndothelial CellsRetinal VesselsG proteinGeneral MedicineCell biologyEndocrinologymedicine.anatomical_structurecardiovascular systemFemalePericyteSignal transductionmedicine.symptomActivin Receptors Type IReceptors Transforming Growth Factor betaSignal TransductionResearch Article
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Diacylglycerol kinase α mediatses 17-β-estradiol-induced proliferation, motility, and anchorage-independent growth of Hec-1A endometrial cancer cell …

2011

Increased levels of endogenous and/or exogenous estrogens are one of the well known risk factors of endometrial cancer. Diacylglycerol kinases (DGKs) are a family of enzymes which phosphorylate diacylglycerol (DAG) to produce phosphatidic acid (PA), thus turning off and on DAG-mediated and PA-mediated signaling pathways, respectively. DGK α activity is stimulated by growth factors and oncogenes and is required for chemotactic, proliferative, and angiogenic signaling in vitro. Herein, using either specific siRNAs or the pharmacological inhibitor R59949, we demonstrate that DGK α activity is required for 17-β-estradiol (E2)-induced proliferation, motility, and anchorage-independent growth of …

medicine.medical_specialtyGPR30medicine.drug_classCell SurvivalDiacylglycerol kinaseMotilityEstrogen receptorEnzyme AssayEndometrial carcinomaBiologyQuinazolinoneReceptors G-Protein-CoupledPiperidinePiperidinesCell MovementInternal medicineCell Line TumormedicineCell AdhesionHumansEndometrial NeoplasmEnzyme AssaysQuinazolinonesDiacylglycerol kinaseCell ProliferationEstradiolCell growthKinaseCell BiologyDiacylglycerol kinase; Endometrial carcinoma; Estrogen; GPR30; Cell BiologyEstrogenEndometrial NeoplasmsCell biologyEnzyme ActivationLipoprotein LipaseEndocrinologyReceptors EstrogenEstrogenGene Knockdown TechniquesGene Knockdown TechniqueFemaleRNA InterferenceSignal transductionGPERHuman
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Phosphodiesterase-4 inhibition improves corticosteroid insensitivity in pulmonary endothelial cells under oxidative stress.

2012

Several clinical studies have shown that smoking in asthmatics and chronic obstructive pulmonary disease patients is closely associated with corticosteroid refractoriness. In this work, we have analyzed glucocorticoid insensitivity in human pulmonary artery endothelial cells (HPAECs) under cigarette smoke extract (CSE) exposure as well as the possible additive effects of the combination therapy with a phosphodiesterase (PDE)-4 inhibitor. Interleukin (IL)-8 was measured in cell supernatants by ELISA. Histone deacetylase (HDAC), histone acetylase (HAT), and intracellular cAMP levels were measured by colorimetric assays and enzyme immunoassay, respectively. PDE4 isotypes and glucocorticoid rec…

medicine.medical_specialtyImmunologyApoptosisDexamethasoneHistone DeacetylasesGlucocorticoid receptorReceptors GlucocorticoidAdrenal Cortex HormonesInternal medicinemedicineCyclic AMPImmunology and AllergyHumansReceptorLungDexamethasoneRolipramCell ProliferationHistone AcetyltransferasesChemistryTumor Necrosis Factor-alphaInterleukin-8InterleukinPhosphodiesteraseEndothelial CellsAparato respiratorioCyclic Nucleotide Phosphodiesterases Type 4Enzyme ActivationOxidative StressEndocrinologyHistone deacetylasePhosphodiesterase 4 InhibitorsPulmonesReactive Oxygen SpeciesRolipramGlucocorticoidmedicine.drugAllergy
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Gastrin: an acid-releasing, proliferative and immunomodulatory peptide?

2010

Gastrin release is affected by gastric inflammatory conditions. Antral G cells respond to inflammatory mediators by increasing gastrin secretion. Accumulating experimental evidence suggests that gastrin exerts immunomodulatory and proinflammatory effects. Gastrin could be a contributing factor to these pathologies, which may constitute a new justification for pharmacological blockade of gastrin action.

medicine.medical_specialtyInflammationdigestive systemProinflammatory cytokineGastric AcidImmunomodulationInternal medicineDrug DiscoveryGastrinsmedicineAnimalsHumansSecretionGastrin-Secreting CellsProtein PrecursorsAntrumGastrinCell ProliferationPharmacologyChemistrydigestive oral and skin physiologyGeneral MedicineBlockadeImmunomodulatory peptideEndocrinologyGastrinomaGastritisReceptors CholecystokininG cellmedicine.symptomhormones hormone substitutes and hormone antagonistsSignal TransductionMini reviews in medicinal chemistry
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The CB1 cannabinoid receptor mediates excitotoxicity-induced neural progenitor proliferation and neurogenesis.

2007

Endocannabinoids are lipid signaling mediators that exert an important neuromodulatory role and confer neuroprotection in several types of brain injury. Excitotoxicity and stroke can induce neural progenitor (NP) proliferation and differentiation as an attempt of neuroregeneration after damage. Here we investigated the mechanism of hippocampal progenitor cell engagement upon excitotoxicity induced by kainic acid administration and the putative involvement of the CB1 cannabinoid receptor in this process. Adult NPs express kainate receptors that mediate proliferation and neurosphere generation in vitro via CB1 cannabinoid receptors. Similarly, in vivo studies showed that excitotoxicity-induce…

medicine.medical_specialtyKainic acidCannabinoid receptorNeurotoxinsExcitotoxicityKainate receptorBiologymedicine.disease_causeBiochemistryNeuroprotectionHippocampuschemistry.chemical_compoundMiceReceptor Cannabinoid CB1Epidermal growth factorInternal medicinemedicineAnimalsMolecular BiologyCell ProliferationMice KnockoutNeuronsKainic AcidStem CellsNeurogenesisCell BiologyEndocannabinoid systemCell biologyNerve RegenerationEndocrinologynervous systemchemistrylipids (amino acids peptides and proteins)Fibroblast Growth Factor 2The Journal of biological chemistry
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An O2-sensitive glomus cell-stem cell synapse induces carotid body growth in chronic hypoxia.

2013

Summary Neural stem cells (NSCs) exist in germinal centers of the adult brain and in the carotid body (CB), an oxygen-sensing organ that grows under chronic hypoxemia. How stem cell lineage differentiation into mature glomus cells is coupled with changes in physiological demand is poorly understood. Here, we show that hypoxia does not affect CB NSC proliferation directly. Rather, mature glomus cells expressing endothelin-1, the O 2 -sensing elements in the CB that secrete neurotransmitters in response to hypoxia, establish abundant synaptic-like contacts with stem cells, which express endothelin receptors, and instruct their growth. Inhibition of glomus cell transmitter release or their sel…

medicine.medical_specialtyMice TransgenicBiologyGeneral Biochemistry Genetics and Molecular BiologyProlyl HydroxylasesMiceGlomus cellNeural Stem CellsInternal medicinemedicineAnimalsRats WistarCell ProliferationCarotid BodyCell growthBiochemistry Genetics and Molecular Biology(all)Germinal centerCell DifferentiationHypoxia (medical)Respiratory CenterNeural stem cellRatsOxygenEndocrinologymedicine.anatomical_structureCarotid bodyStem cellmedicine.symptomEndothelin receptorCell
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Tetralogy of Fallot as a Model to Study Cardiac Progenitor Cell Migration and Differentiation During Heart Development

2009

Tetralogy of Fallot (ToF) has long been considered a congenital disorder that occurs due to environmental alterations during gestation. Recently, several mutated genes have been discovered that are thought to be responsible for the malformations observed in ToF. These genetic mutations, which are microdeletions, are sporadic and are frequently also present in trisomy 21 patients. The ToF malformations can be lethal, but for the last 50 years, surgical repairs that place an artificial patch to repair the four features of ToF have improved the survival of patients with ToF. However, 0.5% to 6% of patients who survive after surgical repair of ToF die of sudden cardiac death caused by ventricul…

medicine.medical_specialtyOrganogenesisBiologyVentricular tachycardiaSudden cardiac deathHomeobox protein Nkx-2.5Cell MovementInternal medicinemedicineAnimalsHumansCell LineageCell ProliferationTetralogy of FallotHomeodomain ProteinsSurgical repairHeart developmentMyocardiumStem CellsGene Expression Regulation DevelopmentalCell DifferentiationToF Cardiac Stem Cellsmedicine.diseaseGATA4 Transcription Factormedicine.anatomical_structureVentricleMutationHomeobox Protein Nkx-2.5Tetralogy of FallotCardiologyCardiology and Cardiovascular MedicineTranscription FactorsCongenital disorderTrends in Cardiovascular Medicine
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Current perspectives on parathyroid hormone (PTH) and PTH-related protein (PTHrP) as bone anabolic therapies.

2013

Osteoporosis is characterized by low bone mineral density and/or poor bone microarchitecture leading to an increased risk of fractures. The skeletal alterations in osteoporosis are a consequence of a relative deficit of bone formation compared to bone resorption. Osteoporosis therapies have mostly relied on antiresorptive drugs. An alternative therapeutic approach for osteoporosis is currently available, based on the intermittent administration of parathyroid hormone (PTH). Bone anabolism caused by PTH therapy is mainly accounted for by the ability of PTH to increase osteoblastogenesis and osteoblast survival. PTH and PTH-related protein (PTHrP)-an abundant local factor in bone- interact wi…

medicine.medical_specialtyOsteoporosisParathyroid hormoneBone healingBiochemistryBone resorptionBone remodelingOsteogenesisInternal medicinemedicineAnimalsHumansBone regenerationCell ProliferationReceptor Parathyroid Hormone Type 1PharmacologyBone mineralOsteoblastsBone Density Conservation Agentsbusiness.industryParathyroid Hormone-Related ProteinOsteoblastCell Differentiationmedicine.diseaseProtein Structure Tertiarymedicine.anatomical_structureEndocrinologyGene Expression RegulationParathyroid HormoneOsteoporosisFemalebusinesshormones hormone substitutes and hormone antagonistsSignal TransductionBiochemical pharmacology
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Resistance of the internal mammary artery to restenosis: a histomorphologic study of various porcine arteries.

2007

<i>Background/Aims:</i> Restenosis after percutaneous transluminal angioplasty (PTA) of the internal mammary artery (IMA) grafts is much less pronounced than in other arteries and venous grafts. The aim of the study was to test whether various arteries respond differently to dilatation. <i>Methods:</i> PTA of the IMA, carotid, renal and circumflex coronary (RCx) arteries was performed in 9 pigs (balloon to artery ratio of 1:1.5). After 8 weeks, angiography was repeated and vessels prepared for histological analysis. Immunohistochemical staining was done to examine proliferative activity (Ki67) and to identify the vasa vasorum of the adventitia (F VIII-RA). <i>R…

medicine.medical_specialtyPercutaneousTime FactorsPhysiologySwinemedicine.medical_treatmenteducationRenal Artery ObstructionCoronary AngiographyRenal Artery ObstructionCoronary RestenosisRenal ArteryRestenosisInternal medicinemedicine.arteryAngioplastyotorhinolaryngologic diseasesmedicineMitotic IndexAnimalsCarotid StenosisRenal arteryAngioplasty Balloon CoronaryMammary ArteriesCell Proliferationmedicine.diagnostic_testbusiness.industryAngiographyGraft Occlusion Vascularmedicine.diseaseTunica intimaCoronary VesselsImmunohistochemistrysurgical procedures operativemedicine.anatomical_structureCarotid ArteriesTreatment OutcomeAngiographyCirculatory systemModels AnimalCardiologyCardiology and Cardiovascular MedicinebusinessTunica IntimaTunica MediaAngioplasty BalloonJournal of vascular research
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