Search results for "Cell Signaling"

showing 10 items of 228 documents

Identification of the cell-wall derived xyloglucan as a new damage-associated molecular pattern (DAMP) eliciting plant immunity in Vitis vinifera and…

2018

Activation of the plant immune responses requires recognition of common pathogen-associated molecular pattern (PAMP) by their cognate pattern recognition receptors (PRR). Chitin, a major component of fungal cell walls, is a well-known PAMP that triggers defense responses in several mammal and plant species.In the first part of this study, we show that two chitooligosaccharides, chitin and chitosan, act as PAMPs in grapevine (Vitis vinifera) as they elicit immune signaling events, defense gene expression, and resistance against pathogens. These two PAMPs are active in grapevine suggesting that at least one perception system exists. Phylogenetic analysis clearly distinguished three V. vinifer…

ChitosanImmunité des plantes[SDV.SA] Life Sciences [q-bio]/Agricultural sciencesGrapevine (Vitis vinifera)Récepteurs (PRR)Arabidopsis thalianaVigne (Vitis vinifera)Signalisation cellulaire et moléculaireChitinePattern Recognition Receptors (PRR)Plant immunityCERK1LysM RK[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologyMolecular cell signaling
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SMART: Unique splitting-while-merging framework for gene clustering

2014

© 2014 Fa et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited. Successful clustering algorithms are highly dependent on parameter settings. The clustering performance degrades significantly unless parameters are properly set, and yet, it is difficult to set these parameters a priori. To address this issue, in this paper, we propose a unique splitting-while-merging clustering framework, named "splitting merging awareness tactics" (SMART), which does not require any a priori knowledge of either the number …

Clustering algorithmsMicroarrayslcsh:MedicineGene ExpressionBioinformaticscomputer.software_genreCell SignalingData MiningCluster Analysislcsh:ScienceFinite mixture modelOligonucleotide Array Sequence AnalysisPhysicsMultidisciplinarySMART frameworkConstrained clusteringCompetitive learning modelBioassays and Physiological AnalysisMultigene FamilyCanopy clustering algorithmEngineering and TechnologyData miningInformation TechnologyGenomic Signal ProcessingAlgorithmsResearch ArticleSignal TransductionComputer and Information SciencesFuzzy clusteringCorrelation clusteringResearch and Analysis MethodsClusteringMolecular GeneticsCURE data clustering algorithmGeneticsGene RegulationCluster analysista113Gene Expression Profilinglcsh:RBiology and Life SciencesComputational BiologyCell BiologyDetermining the number of clusters in a data setComputingMethodologies_PATTERNRECOGNITIONSplitting-merging awareness tactics (SMART)Signal ProcessingAffinity propagationlcsh:QGene expressionClustering frameworkcomputer
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Material-driven fibronectin assembly rescues matrix defects due to mutations in collagen IV in fibroblasts

2020

Basement membranes (BMs) are specialised extracellular matrices that provide structural support to tissues as well as influence cell behaviour and signalling. Mutations in COL4A1/COL4A2, a major BM component, cause a familial form of eye, kidney and cerebrovascular disease, including stroke, while common variants in these genes are a risk factor for intracerebral haemorrhage in the general population. These phenotypes are associated with matrix defects, due to mutant protein incorporation in the BM and/or its absence by endoplasmic reticulum (ER) retention. However, the effects of these mutations on matrix stiffness, the contribution of the matrix to the disease mechanism(s) and its effects…

Collagen Type IVCell signalingPopulationIntegrinBiophysicsBioengineering02 engineering and technologyMatrix (biology)medicine.disease_causeBasement MembraneArticleBiomaterialsExtracellular matrix03 medical and health sciences0302 clinical medicineLamininmedicineExtracellularHumanseducationCell adhesion030304 developmental biologyeducation.field_of_study0303 health sciencesMutationbiologyChemistryEndoplasmic reticulumFibroblasts021001 nanoscience & nanotechnologyPhenotypeExtracellular MatrixFibronectinsCell biologyFibronectinMechanics of MaterialsMutationCeramics and Compositesbiology.protein0210 nano-technology030217 neurology & neurosurgeryBiomaterials
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Loss of Neuroglobin Expression Alters Cdkn1a/Cdk6-Expression Resulting in Increased Proliferation of Neural Stem Cells

2018

Abstract: In the quest to unravel its functional significance, neuroglobin (Ngb), a brain-specific neuroprotective protein, has recently been proposed as an actor in neurodevelopment. As neural stem cells (NSCs) are fundamental during brain development, the present study aimed at investigating the role of Ngb in the growth and proliferation of NSCs by comparing an Ngb-floxed (Ngb(fl)-)NSC line, equivalent to the wild-type cellular situation, with an in-house created Ngb knockout (Ngb(KO)-)NSC line. Ngb(KO)-NSCs were characterized by an increased growth and proliferation capacity in vitro, supported by RNA sequencing and western blot results reporting the downregulation of Cdkn1a and the upr…

Cyclin-Dependent Kinase Inhibitor p21Male0301 basic medicineCell signalingDown-RegulationNeuroglobinNerve Tissue ProteinsBiologyNeuroprotectionTranscriptomeMice03 medical and health sciences0302 clinical medicineNeural Stem CellsDownregulation and upregulationAnimalsBiologyCells CulturedCell ProliferationCell CycleCyclin-Dependent Kinase 6Cell BiologyHematologyCell cycleNeural stem cellUp-RegulationCell biologyMice Inbred C57BL030104 developmental biologynervous systemNeuroglobinbiology.proteinFemaleHuman medicineCyclin-dependent kinase 6Tumor Suppressor Protein p53Proto-Oncogene Proteins c-akt030217 neurology & neurosurgerySignal TransductionDevelopmental BiologyStem Cells and Development
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Lrp4, a Novel Receptor for Dickkopf 1 and Sclerostin, Is Expressed by Osteoblasts and Regulates Bone Growth and Turnover In Vivo

2009

Lrp4 is a multifunctional member of the low density lipoprotein-receptor gene family and a modulator of extracellular cell signaling pathways in development. For example, Lrp4 binds Wise, a secreted Wnt modulator and BMP antagonist. Lrp4 shares structural elements within the extracellular ligand binding domain with Lrp5 and Lrp6, two established Wnt co-receptors with important roles in osteogenesis. Sclerostin is a potent osteocyte secreted inhibitor of bone formation that directly binds Lrp5 and Lrp6 and modulates both BMP and Wnt signaling. The anti-osteogenic effect of sclerostin is thought to be mediated mainly by inhibition of Wnt signaling through Lrp5/6 within osteoblasts. Dickkopf1 …

Genetic Markersmusculoskeletal diseasesmedicine.medical_specialtylcsh:MedicineBiologyBone morphogenetic proteinBone and BonesCell LineMicechemistry.chemical_compoundInternal medicineBiochemistry/Cell Signaling and Trafficking StructuresmedicineAnimalsHumanslcsh:ScienceLDL-Receptor Related ProteinsAdaptor Proteins Signal TransducingGlycoproteinsBone growthBone DevelopmentOsteoblastsMultidisciplinarylcsh:RWnt signaling pathwayLRP6Rheumatology/Bone and Mineral MetabolismLRP5OsteoblastPhenotypemedicine.anatomical_structureEndocrinologyGene Expression RegulationReceptors LDLGenetics and Genomics/Disease ModelschemistryOsteocyteBone Morphogenetic ProteinsIntercellular Signaling Peptides and ProteinsSclerostinlcsh:QSignal TransductionResearch ArticlePLoS ONE
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Aβ and tau toxicities in Alzheimer’s are linked via oxidative stress-induced p38 activation: Protective role of vitamin E

2014

AbstractOxidative stress is a hallmark of Alzheimer’s disease (AD). We propose that rather than causing damage because of the action of free radicals, oxidative stress deranges signaling pathways leading to tau hyperphosphorylation, a hallmark of the disease. Indeed, incubation of neurons in culture with 5 µM beta-amyloid peptide (Aβ) causes an activation of p38 MAPK (p38) that leads to tau hyperphosphorylation. Inhibition of p38 prevents Aβ-induced tau phosphorylation. Aβ-induced effects are prevented when neurons are co-incubated with trolox (the water-soluble analog of vitamin E).We have confirmed these results in vivo, in APP/PS1 double transgenic mice of AD. We have found that APP/PS1 …

Genetically modified mouseMalemedicine.medical_specialtyCell signalingAntioxidantP-p38p38 mitogen-activated protein kinasesmedicine.medical_treatmentClinical BiochemistryMice Transgenictau ProteinsBiologyBeta-amyloidmedicine.disease_causeProtective AgentsBiochemistryHippocampusp38 Mitogen-Activated Protein KinasesArticlechemistry.chemical_compoundMiceAlzheimer DiseaseInternal medicinemental disordersmedicineVitamin EAnimalsPhosphorylationlcsh:QH301-705.5Cells CulturedNeuronslcsh:R5-920Amyloid beta-PeptidesVitamin EOrganic Chemistrymedicine.diseaseRatsDisease Models AnimalOxidative StressEndocrinologylcsh:Biology (General)chemistryTroloxAlzheimer's diseaseAntioxidantlcsh:Medicine (General)Oxidative stressRedox Biology
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The Free Radical Theory of Aging Revisited: The Cell Signaling Disruption Theory of Aging

2013

AbstractSignificance: The free radical theory of aging has provided a theoretical framework for an enormous amount of work leading to significant advances in our understanding of aging. Up to the turn of the century, the theory received abundant support from observations coming from fields as far apart as comparative physiology or molecular biology. Recent Advances: Work from many laboratories supports the theory, for instance showing that overexpression of antioxidant enzymes results in increases in life-span. But other labs have shown that in some cases, there is an increased oxidative stress and increased longevity. The discovery that free radicals can not only cause molecular damage to …

GerontologyAgingCell signalingForum Review ArticleAging (J. Viña Ed.)Free RadicalsPhysiologymedia_common.quotation_subjectRadicalClinical BiochemistryMitochondria LiverBiologymedicine.disease_causeModels BiologicalBiochemistryAntioxidantsmedicineAnimalsHumansMolecular BiologyGeneral Environmental ScienceFree-radical theory of agingmedia_commonchemistry.chemical_classificationReactive oxygen speciesLongevityCell BiologyOxidative StressPhysiological AdaptationschemistryGeneral Earth and Planetary SciencesOxidation-ReductionNeuroscienceOxidative stressSignal Transduction
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Light-induced Changes in the Dimerization Interface of Bacteriophytochromes

2015

Phytochromes are dimeric photoreceptor proteins that sense red light levels in plants, fungi, and bacteria. The proteins are structurally divided into a light-sensing photosensory module consisting of PAS, GAF, and PHY domains and a signaling output module, which in bacteriophytochromes typically is a histidine kinase (HK) domain. Existing structural data suggest that two dimerization interfaces exist between the GAF and HK domains, but their functional roles remain unclear. Using mutational, biochemical, and computational analyses of the Deinococcus radiodurans phytochrome, we demonstrate that two dimerization interfaces between sister GAF and HK domains stabilize the dimer with approximat…

Histidine KinaseLightProtein ConformationMutantCrystallography X-RayBiochemistryProtein structureBacterial Proteinsx-ray scatteringcell signalingDeinococcusMolecular BiologybiologyPhytochromeHistidine kinaseMutagenesista1182Photoreceptor proteinDeinococcus radioduransCell Biologybiology.organism_classificationphotoreceptormolecular dynamicsProtein Structure TertiaryBiochemistryhigh performance liquid chromatography (HPLC)BiophysicsDeinococcusPhytochromeDimerizationProtein KinasesmutagenesisMolecular BiophysicsJournal of Biological Chemistry
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Molecular basis of filamin a-filGAP interaction and its impairment in congenital disorders associated with filamin a mutations

2008

Background Mutations in filamin A (FLNa), an essential cytoskeletal protein with multiple binding partners, cause developmental anomalies in humans. Methodology/Principal Findings We determined the structure of the 23rd Ig repeat of FLNa (IgFLNa23) that interacts with FilGAP, a Rac-specific GTPase-activating protein and regulator of cell polarity and movement, and the effect of the three disease-related mutations on this interaction. A combination of NMR structural analysis and in silico modeling revealed the structural interface details between the C and D β-strands of the IgFLNa23 and the C-terminal 32 residues of FilGAP. Mutagenesis of the predicted key interface residues confirmed the b…

ImmunoprecipitationFilaminsMolecular Sequence Dataeducationlcsh:MedicineComputational Biology/Protein Structure PredictionBiologyFilaminCell Biology/Cell SignalingCongenital AbnormalitiesBiochemistry/Protein Folding03 medical and health sciences0302 clinical medicineProtein structureContractile ProteinsCell Biology/CytoskeletonFLNAHumansFLNBFLNCAmino Acid Sequencelcsh:Science030304 developmental biologyGenetics0303 health sciencesMultidisciplinaryBinding SitesMolecular StructureSequence Homology Amino AcidPoint mutationlcsh:RGTPase-Activating ProteinsMicrofilament Proteins3. Good healthBiochemistry/BioinformaticsMutationProtein foldinglcsh:Q118 Biological sciences030217 neurology & neurosurgeryResearch Article
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New agents and approaches for targeting the RAS/RAF/MEK/ERK and PI3K/AKT/mTOR cell survival pathways.

2012

The Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR cascades are often activated by genetic alterations in upstream signaling molecules such as receptor tyrosine kinases (RTK). Targeting these pathways is often complex and can result in pathway activation depending on the presence of upstream mutations (e.g., Raf inhibitors induce Raf activation in cells with wild type (WT) RAF in the presence of mutant, activated RAS) and rapamycin can induce Akt activation. Targeting with inhibitors directed at two constituents of the same pathway or two different signaling pathways may be a more effective approach. This review will first evaluate potential uses of Raf, MEK, PI3K, Akt and mTOR inhibitors that have…

MAPK/ERK pathway0303 health sciencesCell signalingbiologyChemistryAKTApoptosisGrowth factorRafOncogens: Signaling pathway3. Good healthMalignant transformation03 medical and health sciences0302 clinical medicineApoptosis030220 oncology & carcinogenesisCancer researchbiology.proteinEpidermal growth factor receptorSignal transductionpi3kProtein kinase BRaPI3K/AKT/mTOR pathway030304 developmental biology
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