Search results for "Cellular Neuroscience"

showing 10 items of 51 documents

Correction: Recovery from Toxic-Induced Demyelination Does Not Require the NG2 Proteoglycan.

2018

[This corrects the article DOI: 10.1371/journal.pone.0163841.].

ImmunologyGene ExpressionMouse ModelsCell MigrationResearch and Analysis MethodsPathology and Laboratory MedicineCorpus CallosumDirected Cell MigrationModel OrganismsNerve FibersSigns and SymptomsAnimal CellsDiagnostic MedicineMedicine and Health SciencesGeneticsImmune ResponseNeuronsInflammationChemotaxisBiology and Life SciencesBrainCell DifferentiationAnimal ModelsCell BiologyAxonsCell MotilityCellular NeuroscienceCellular TypesAnatomyResearch ArticleDevelopmental BiologyNeurosciencePloS one
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Optogenetically blocking sharp wave ripple events in sleep does not interfere with the formation of stable spatial representation in the CA1 area of …

2016

During hippocampal sharp wave/ripple (SWR) events, previously occurring, sensory inputdriven neuronal firing patterns are replayed. Such replay is thought to be important for plasticity-related processes and consolidation of memory traces. It has previously been shown that the electrical stimulation-induced disruption of SWR events interferes with learning in rodents in different experimental paradigms. On the other hand, the cognitive map theory posits that the plastic changes of the firing of hippocampal place cells constitute the electrophysiological counterpart of the spatial learning, observable at the behavioral level. Therefore, we tested whether intact SWR events occurring during th…

LightPhysiologylcsh:MedicineHippocampusTetrodesMiceAnimal Cells571 PhysiologyMedicine and Health Scienceslcsh:ScienceNeuronsLight PulsesBrain MappingNeuronal PlasticityPyramidal CellsPhysicsElectromagnetic RadiationBrainLaboratory EquipmentSignal Filteringsharp wave ripple eventesBioassays and Physiological AnalysisOptical EquipmentVacuum ApparatusPhysical SciencesEngineering and TechnologyFemaleCellular TypesAnatomyResearch ArticleGanglion CellsArchaeal ProteinsSpatial LearningEquipmentResearch and Analysis Methodsuni (lepotila)AnimalshippokampusCA1 Region HippocampalLaserslcsh:RCorrectionBiology and Life SciencesNeurophysiological AnalysisCell BiologyBrain WavesMice Inbred C57BLOptogeneticsCellular NeuroscienceSignal ProcessingExploratory Behavior570 Life sciences; biologylcsh:QPhysiological ProcessesSleepNeuroscience
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Neuronal activity triggers uptake of hematopoietic extracellular vesicles in vivo

2019

Communication with the hematopoietic system is a vital component of regulating brain function in health and disease. Traditionally, the major routes considered for this neuroimmune communication are by individual molecules such as cytokines carried by blood, by neural transmission, or, in more severe pathologies, by the entry of peripheral immune cells into the brain. In addition, functional mRNA from peripheral blood can be directly transferred to neurons via extracellular vesicles (EVs), but the parameters that determine their uptake are unknown. Using varied animal models that stimulate neuronal activity by peripheral inflammation, optogenetics, and selective proteasome inhibition of dop…

LipopolysaccharidesMaleGene ExpressionStimulationHippocampusBiochemistryStereotaxic Techniques0302 clinical medicineShort ReportsAnimal CellsMedicine and Health SciencesPremovement neuronal activityBiology (General)Routes of AdministrationNeurons0303 health sciencesBrain MappingKainic AcidBrainAnimal ModelsPeripheralCell biologyHaematopoiesisBioassays and Physiological AnalysisExperimental Organism SystemsHippocampus ; Yellow flourescent protein ; Intravenous injections ; Marker genes ; Gene expression ; Neurons ; Microglial cells ; OptogeneticsFemaleCellular TypesSignal TransductionProteasome Endopeptidase ComplexQH301-705.5Yellow Fluorescent ProteinMice TransgenicGlial CellsMouse ModelsStimulus (physiology)BiologyResearch and Analysis Methods03 medical and health sciencesExtracellular VesiclesImmune systemModel OrganismsIn vivoIntravenous InjectionsGeneticsAnimalsddc:610Molecular Biology TechniquesMolecular BiologyMicroglial Cells030304 developmental biologyInflammationPharmacologyMessenger RNABlood CellsUbiquitinDopaminergic NeuronsBiology and Life SciencesProteinsMarker GenesCell BiologyNeurophysiological AnalysisOptogeneticsLuminescent ProteinsCellular NeuroscienceAnimal Studies030217 neurology & neurosurgeryNeuroscience
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New neurons use Slit-Robo signaling to migrate through the glial meshwork and approach a lesion for functional regeneration

2018

Appropriate positioning of new neurons in the brain promotes post-stroke functional recovery.

Male0301 basic medicineanimal structuresNeurogenesisBiologyLesionMice03 medical and health sciences0302 clinical medicineDevelopmental NeuroscienceNeuroblastCell MovementNeuroblast migrationmental disordersmedicineAnimalsRegenerationcardiovascular diseasesReceptors ImmunologicProgenitor cellcdc42 GTP-Binding ProteinResearch Articlesreproductive and urinary physiologyMice KnockoutNeuronsMultidisciplinaryRegeneration (biology)fungiNeurogenesisSciAdv r-articlesBrainActin cytoskeletonSlit-RoboActin Cytoskeleton030104 developmental biologynervous systemCellular NeuroscienceAstrocytesembryonic structuresIntercellular Signaling Peptides and ProteinsProtein Multimerizationmedicine.symptomNeurogliaNeurosciencepsychological phenomena and processes030217 neurology & neurosurgeryResearch ArticleProtein BindingSignal TransductionScience Advances
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Lung injury does not aggravate mechanical ventilation-induced early cerebral inflammation or apoptosis in an animal model.

2018

INTRODUCTION:The acute respiratory distress syndrome is not only associated with a high mortality, but also goes along with cognitive impairment in survivors. The cause for this cognitive impairment is still not clear. One possible mechanism could be cerebral inflammation as result of a "lung-brain-crosstalk". Even mechanical ventilation itself can induce cerebral inflammation. We hypothesized, that an acute lung injury aggravates the cerebral inflammation induced by mechanical ventilation itself and leads to neuronal damage. METHODS:After approval of the institutional and state animal care committee 20 pigs were randomized to one of three groups: lung injury by central venous injection of …

MaleARDSCritical Care and Emergency MedicinePulmonologySwinePhysiologymedicine.medical_treatmentVentilator-Induced Lung InjuryInterleukin-1betalcsh:MedicineApoptosisPathology and Laboratory MedicineHippocampusPositive-Pressure RespirationRandom Allocation0302 clinical medicineAnimal CellsImmune PhysiologyMedicine and Health Scienceslcsh:ScienceImmune ResponseAcute Respiratory Distress SyndromeTidal volumeCerebral CortexNeuronsCognitive ImpairmentRespiratory Distress SyndromeInnate Immune SystemMultidisciplinarymedicine.diagnostic_testCognitive NeurologyBrainGeneral MedicineLung InjuryNeurologyAnesthesiaBreathingCytokinesTumor necrosis factor alphamedicine.symptomAnatomyCellular TypesGeneral Agricultural and Biological SciencesResearch ArticleHistologyCognitive NeuroscienceImmunology10208 Institute of NeuropathologyInflammation610 Medicine & healthGenetics and Molecular BiologyGlial Cells1100 General Agricultural and Biological SciencesLung injury03 medical and health sciencesSigns and SymptomsRespiratory Failure1300 General Biochemistry Genetics and Molecular BiologyDiagnostic MedicinemedicineAnimalsMicroglial CellsMechanical ventilationInflammation1000 Multidisciplinarybusiness.industryInterleukin-6Tumor Necrosis Factor-alphalcsh:RBiology and Life Sciences030208 emergency & critical care medicineCell BiologyMolecular Developmentmedicine.diseaseRespiration ArtificialBronchoalveolar lavage030228 respiratory systemImmune SystemCellular NeuroscienceGeneral Biochemistry570 Life sciences; biologyCognitive Sciencelcsh:QbusinessDevelopmental BiologyNeurosciencePloS one
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Therapeutic effects of hMAPC and hMSC transplantation after stroke in mice.

2012

Stroke represents an attractive target for stem cell therapy. Although different types of cells have been employed in animal models, a direct comparison between cell sources has not been performed. The aim of our study was to assess the effect of human multipotent adult progenitor cells (hMAPCs) and human mesenchymal stem cells (hMSCs) on endogenous neurogenesis, angiogenesis and inflammation following stroke. BALB/Ca-RAG 2(-/-) γC(-/-) mice subjected to FeCl(3) thrombosis mediated stroke were intracranially injected with 2 × 10(5) hMAPCs or hMSCs 2 days after stroke and followed for up to 28 days. We could not detect long-term engraftment of either cell population. However, in comparison w…

MaleAnatomy and PhysiologyMousemedicine.medical_treatmentCell therapyMiceCell MovementMolecular Cell BiologyNeurobiology of Disease and RegenerationMedicineMultidisciplinaryNeuronal MorphologyNeurogenesisQRBrainInfarction Middle Cerebral ArteryAnimal ModelsStem-cell therapyStrokeAdult Stem Cellsmedicine.anatomical_structureNeurologyMedicineResearch ArticleAdult stem cellCell PhysiologyHistologyCell SurvivalCerebrovascular DiseasesScienceTherapeutic effectsNeurophysiologyNeovascularization PhysiologicSubventricular zoneMesenchymal Stem Cell TransplantationNeurological SystemModel OrganismsAnimalsHumansProgenitor cellBiologyTransplantationbusiness.industryMultipotent Stem CellsMesenchymal stem cellMultipotent Stem CellCellular NeuroscienceImmunologyCancer researchMolecular NeurosciencebusinesshMAPC and hMSCNeurosciencePLoS ONE
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Glia talk back.

2014

This study shows that the activity of neurons can trigger shedding of a protein, NG2, from the surface of oligodendrocyte precursor cells; this protein in turn modulates synaptic transmission, revealing a two-way conversation between neurons and glia.

MaleCell signalingNeural NetworksQH301-705.5AMPA receptorBiologyNeurotransmissionResearch and Analysis MethodsMechanical Treatment of SpecimensGeneral Biochemistry Genetics and Molecular BiologyBehavioral NeuroscienceNeurotransmitter receptormedicineBiological neural networkPremovement neuronal activityAnimalsBiology (General)AntigensNeuronsGeneral Immunology and MicrobiologyGeneral NeuroscienceMembrane ProteinsBiology and Life SciencesLong-term potentiationADAM ProteinsOligodendrogliamedicine.anatomical_structureElectroporationnervous systemSpecimen DisruptionSpecimen Preparation and TreatmentCellular NeuroscienceImmunologyProteoglycansNeuronAmyloid Precursor Protein SecretasesMolecular NeuroscienceGeneral Agricultural and Biological SciencesNeuroscienceResearch ArticleNeurosciencePLoS biology
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Aspartoacylase-lacZ knockin mice: an engineered model of Canavan disease.

2011

Canavan Disease (CD) is a recessive leukodystrophy caused by loss of function mutations in the gene encoding aspartoacylase (ASPA), an oligodendrocyte-enriched enzyme that hydrolyses N-acetylaspartate (NAA) to acetate and aspartate. The neurological phenotypes of different rodent models of CD vary considerably. Here we report on a novel targeted aspa mouse mutant expressing the bacterial β-Galactosidase (lacZ) gene under the control of the aspa regulatory elements. X-Gal staining in known ASPA expression domains confirms the integrity of the modified locus in heterozygous aspa lacZ-knockin (aspa(lacZ/+)) mice. In addition, abundant ASPA expression was detected in Schwann cells. Homozygous (…

MaleCentral Nervous SystemCerebellumPathologyAnatomy and PhysiologyCanavan DiseaseMouseMutantlcsh:MedicineNeural HomeostasisBiochemistryMiceNeurobiology of Disease and Regenerationlcsh:ScienceSex CharacteristicsMultidisciplinaryNeuromodulationNeurochemistryGenomicsAnimal ModelsFunctional Genomicsmedicine.anatomical_structureLac OperonNeurologyHomeostatic MechanismsMedicineFemaleNeurochemicalsGenetic EngineeringResearch ArticleNervous System PhysiologyBiotechnologymedicine.medical_specialtyTransgeneCentral nervous systemNeurophysiologyMice TransgenicNeuroimagingBiologyNeurological SystemAmidohydrolasesWhite matterModel OrganismsGeneticsmedicineAnimalsBiologyNeuropeptidesLeukodystrophylcsh:RComputational Biologymedicine.diseaseMolecular biologyCanavan diseaseAspartoacylaseDisease Models AnimalMetabolismnervous systemSmall MoleculesCellular NeuroscienceMetabolic DisordersMutationGenetics of DiseaseNervous System Componentslcsh:QGene FunctionMolecular NeuroscienceAnimal GeneticsNeurosciencePLoS ONE
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Longitudinal changes in physical activity, sedentary behavior and body mass index in adolescence: Migrations towards different weight cluster

2016

This study examined longitudinal changes in physical activity, sedentary behavior and body mass index in adolescents, specifically their migrations towards a different weight cluster. A cohort of 755 adolescents participated in a three-year study. A clustering Self-Organized Maps Analysis was performed to visualize changes in subjects' characteristics between the first and second assessment, and how adolescents were grouped. Also a classification tree was used to identify the behavioral characteristics of the groups that changed their weight cluster. Results indicated that boys were more active and less sedentary than girls. Boys were especially keen to technological-based activities while …

MaleDecision AnalysisPhysiologyHealth Behaviorlcsh:MedicineOverweightAdolescentsBody Mass Index0302 clinical medicineAnimal CellsMedicine and Health SciencesPublic and Occupational HealthLongitudinal Studies030212 general & internal medicineYoung adultlcsh:ScienceChildNeuronsMultidisciplinarySedentary behaviorPhysiological ParametersResearch DesignCohortEngineering and TechnologyFemaleCellular Typesmedicine.symptomPsychologyManagement EngineeringResearch ArticleAdultmedicine.medical_specialtyAdolescentMotor ActivityResearch and Analysis MethodsDisease clusterYoung Adult03 medical and health sciencesThinnessmedicineHumansObesityExercise physiologyExerciseBehaviorlcsh:RBody WeightDecision TreesBiology and Life SciencesCell BiologyPhysical Activity030229 sport sciencesOverweightmedicine.diseaseObesitySpainAge GroupsCellular NeurosciencePeople and PlacesPhysical therapylcsh:QPopulation GroupingsSedentary BehaviorBody mass indexNeuroscienceDemographyPLOS ONE
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Association of ocular, cardiovascular, morphometric and lifestyle parameters with retinal nerve fibre layer thickness.

2017

BACKGROUND Glaucoma is a neurodegenerative disease, leading to thinning of the retinal nerve fibre layer (RNFL). The exact influence of ocular, cardiovascular, morphometric, lifestyle and cognitive factors on RNFL thickness (RNFLT) is unknown and was analysed in a subgroup of the Gutenberg Health Study (GHS). METHODS Global peripapillary RNFLT was measured in 3224 eyes of 1973 subjects (49% female) using spectral-domain optical coherence tomography (SD-OCT). The association of age, sex, ocular, cardiovascular, morphometric, lifestyle and cognitive factors on RNFLT was analysed using Pearson correlation coefficient and fitting a linear mixed model. RESULTS In the univariable analysis highest…

MaleHomocysteineEye Diseasesgenetic structuresGlaucomalcsh:MedicineBlood PressureOtologyVascular MedicineDiagnostic Radiologychemistry.chemical_compoundTinnitus0302 clinical medicineNerve FibersAnimal CellsMedicine and Health SciencesProspective StudiesProspective cohort studylcsh:Science610 Medicine & healthTomographyHearing DisordersAged 80 and overNeuronsMultidisciplinaryAlcohol ConsumptionRadiology and ImagingAxial lengthMiddle AgedAxial Length EyeLifestyle factorsNeurologyRegression AnalysisFemaleCellular TypesAnatomyTomography Optical CoherenceResearch ArticleAdultmedicine.medical_specialtyImaging TechniquesNerve fibre layer610 Medicine & healthHomocysteine levelsResearch and Analysis MethodsRetina03 medical and health sciencesSex FactorsOcular SystemDiagnostic MedicineOphthalmologymedicineHumansddc:610Life StyleAgedNutritionbusiness.industryMorphometrylcsh:RBiology and Life SciencesRetinalGlaucomaCell Biologymedicine.diseaseeye diseasesDietOphthalmologychemistryOtorhinolaryngologyCellular Neuroscience030221 ophthalmology & optometryEyeslcsh:Qsense organsbusinessHead030217 neurology & neurosurgeryNeuroscience
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