Search results for "Cellular"

showing 10 items of 6449 documents

Extracellular Vesicle Encapsulated MicroRNAs in Patients with Type 2 Diabetes Are Affected by Metformin Treatment

2019

Recently, microRNAs (miRNAs) in circulating extracellular vesicles (EVs), have emerged as a source of potential biomarkers for various pathophysiological conditions, including metabolic disorders such as diabetes. Type 2 diabetes mellitus (T2DM), is the most prevalent form of diabetes in the USA, with 30 million diagnosed patients. Identifying miRNA biomarkers that can be used to assess response to glucose lowering treatments would be useful. Using patient plasma samples from a subset of the Danish Metagenomics of the Human Intestinal Tract (MetaHIT) cohort, we characterized miRNAs from whole plasma, plasma-derived EVs, and EV-depleted plasma by small RNA-sequencing to identify T2DM associa…

endocrine system diseaseslcsh:Medicine030209 endocrinology & metabolismType 2 diabetesPharmacologyArticle03 medical and health sciences0302 clinical medicineDiabetes mellitusmicroRNAmedicine030304 developmental biology0303 health sciencesbusiness.industrylcsh:RType 2 Diabetes Mellitusnutritional and metabolic diseasesGeneral MedicineExtracellular vesiclemedicine.diseasePathophysiology3. Good healthMetforminmicroRNAsCohorttype 2 diabetesbusinessextracellular vesiclesmetforminmedicine.drugJournal of Clinical Medicine
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Sterigmatocystin-induced cytotoxicity via oxidative stress induction in human neuroblastoma cells.

2020

Abstract Sterigmatocystin (STE) is a mycotoxin produced by fungi of the genus Aspergillus. Considering that the effect of STE on neuronal system has not been well studied, the aim of the present study consists to investigate the cytotoxic effects of STE in human neuroblastoma (SH-SY5Y) cells. Moreover, the role of oxidative stress and intracellular defense systems was assessed by evaluating reactive oxygen species (ROS) generation, lipid peroxidation (LPO) and antioxidant no-enzymatic (GSH) levels and enzymatic (GPx, GST, CAT and SOD) activity. Our results revealed that STE decreased cell viability in a dose and time-dependent manner. Furthermore, after 24 h of exposure, STE induced an incr…

endocrine systemAntioxidantCell Survivalmedicine.medical_treatmentSterigmatocystinToxicologymedicine.disease_causeLipid peroxidation03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyCell Line TumormedicineHumansViability assay030304 developmental biologyGlutathione Transferasechemistry.chemical_classification0303 health sciencesReactive oxygen speciesGlutathione PeroxidaseSuperoxide Dismutase04 agricultural and veterinary sciencesGeneral MedicineGlutathioneMycotoxinsCatalase040401 food scienceMolecular biologyGlutathioneOxidative StresschemistryLipid PeroxidationReactive Oxygen SpeciesOxidative stressIntracellularFood ScienceSterigmatocystinFood and chemical toxicology : an international journal published for the British Industrial Biological Research Association
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Topography of somatostatin gene expression relative to molecular progenitor domains during ontogeny of the mouse hypothalamus

2010

The hypothalamus comprises alar, basal, and floor plate developmental compartments. Recent molecular data support a rostrocaudal subdivision into rostral (terminal) and caudal (peduncular) halves. In this context, the distribution of neuronal populations expressing somatostatin (Sst) mRNA was analyzed in the developing mouse hypothalamus, comparing with the expression pattern of the genes Orthopedia (Otp), Distal-less 5 (Dlx5), Sonic Hedgehog (Shh), and Nk2 homeobox 1 (Nkx2.1). At embryonic day 10.5 (E10.5), Sst mRNA was first detectable in the anterobasal nucleus, a Nkx2.1-, Shh-, and Otp-positive basal domain. By E13.5, nascent Sst expression was also related to two additional Otp-positiv…

endocrine systemBasal plate (neural tube)forebrain[SDV]Life Sciences [q-bio]OtpNeuroscience (miscellaneous)Shhlcsh:RC321-571lcsh:QM1-69503 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineArcuate nucleusmedicine[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologySonic hedgehoghypothalamuslcsh:Neurosciences. Biological psychiatry. Neuropsychiatry[SDV.BDD]Life Sciences [q-bio]/Development BiologyOriginal Research030304 developmental biologyFloor plate0303 health sciencesAlar platebiologyDlk5forebrain;hypothalamus;Sst;Otp;Dlk5;Nkx2.1;Shh;in situ hybridization;CONTAINING NEURON SYSTEM;SONIC-HEDGEHOG;FOREBRAIN DEVELOPMENT;VENTRAL FOREBRAIN;DEVELOPMENTAL EXPRESSION;BRAIN-DEVELOPMENT;RAT HYPOTHALAMUS;GROWTH-HORMONE;CELL LINEAGES;DIENCEPHALONlcsh:Human anatomyCiencias naturales y ciencias de la saludSstNkx2.1medicine.anatomical_structureHypothalamusForebrainembryonic structuresNeuranatomybiology.protein[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]in situ hybridizationAnatomyNucleusNeuroscience030217 neurology & neurosurgery
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Extracellular heat shock proteins in cancer: From early diagnosis to new therapeutic approach

2021

In cancer, human cells lose the ability to properly control the series of events that occur constantly during cell growth and division, including protein expression, stability, and dynamics. Heat shock proteins (Hsps) are key molecules in these events, constitutively expressed at high levels and could furthermore be induced by the response to cancer-induced stress. In tumor cells, Hsps have been shown to be implicated in the regulation of apoptosis, immune responses, angiogenesis and metastasis; in some cases, they can be overexpressed and dysregulated, representing important cancer hallmarks. In the past few years, it has been demonstrated that Hsps can be released by tumor cells through s…

endocrine systemCancer ResearchAngiogenesisApoptosischemical and pharmacologic phenomenaBiologyMetastasisImmune systemNeoplasmsHeat shock proteinBiomarkers TumorTumor MicroenvironmentmedicineExtracellularHumansHeat-Shock ProteinsEarly Detection of CancerTumor microenvironmentSettore BIO/16 - Anatomia UmanaCell growthCancerextracellular vesicles Heat shock proteins Liquid biopsyMolecular chaperones.Cancerhemic and immune systemsmedicine.diseaseCell biologybiological sciencesSeminars in Cancer Biology
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Annexin-1 downregulation in thyroid cancer correlates to the degree of tumour differentiation

2006

We investigated the expression of annexin-1 (ANXA1) in thyroid carcinoma cell lines and in thyroid cancers with a different degree of differentiation. The highest level of ANXA1 expression examined by Western blotting was detected in the papillary carcinoma cells (NPA) and in the follicular cells (WRO). On the other hand, the most undifferentiated thyroid carcinoma cells (ARO and FRO) presented the lowest level of ANXA1 expression. In surgical tissue specimens from 32 patients with thyroid cancers, we found high immunoreactivity for ANXA1 in papillary (PTC) and follicular (FTC) thyroid cancers while in undifferentiated thyroid cancers (UTC) the expression of the protein was barely detectabl…

endocrine systemCancer ResearchPathologymedicine.medical_specialtyannexin-1endocrine system diseasesCellular differentiationThyroid Glandmedicine.disease_causeThyroid carcinomaDownregulation and upregulationannexinopathieTumor Cells CulturedmedicineHumansThyroid Neoplasmsdifferentiation markerThyroid cancerThyroid NeoplasmAnnexin A1PharmacologyRegulation of gene expressionbusiness.industryThyroidCell Differentiationmedicine.diseaseapoptosithyroid carcinomaGene Expression Regulation Neoplasticmedicine.anatomical_structureOncologyCell cultureMolecular MedicineCarcinogenesisbusinessHumanCancer Biology & Therapy
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RASSF1A inhibits estrogen receptor alpha expression and estrogen-independent signalling: implications for breast cancer development

2012

The Ras association domain family 1 isoform A (RASSF1A) is a tumor suppressor whose inactivation is implicated in the development of many human cancers, including breast carcinomas. Little is known about the tumor-suppressive function of RASSF1A in breast tissue and whether its inactivation is mechanistically involved in the initiation and progression of breast tumors. Here, we show that RASSF1A inhibits breast cancer growth in vivo, and suppresses estrogen receptor (ERα) expression and function. Reconstitution of RASSF1A in MCF7 cells led to decreased ERα levels and reduced sensitivity to estrogen (E2). Concomitantly, we observed decreased expression of Id1 as well as the E2-responsive gen…

endocrine systemCancer Researchmedicine.medical_specialtyCell SurvivalGene ExpressionEstrogen receptorApoptosisBreast NeoplasmsCell Cycle ProteinsMice SCIDBiologyMiceBreast cancerDownregulation and upregulationMice Inbred NODInternal medicineGeneticsmedicineAnimalsHumansFulvestrantMolecular BiologyCellular SenescenceCell ProliferationRegulation of gene expressionEstradiolFulvestrantTumor Suppressor ProteinsEstrogen AntagonistsEstrogen Receptor alphaCancerEstrogensCell Cycle Checkpointsmedicine.diseaseGene Expression Regulation NeoplasticEndocrinologyProteolysisMCF-7 CellsCancer researchFemaleEctopic expressionEstrogen receptor alphaNeoplasm TransplantationSignal Transductionmedicine.drugOncogene
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Cytotoxic effects of individual and combined sterigmatocystin and nivalenol on liver hepatocellular carcinoma cells

2020

Abstract Since humans are exposed to different mycotoxins through daily intake, there is increasing concern about the adverse effects of the interactions between them. Cytotoxicity of sterigmatocystin (STE) and nivalenol (NIV) alone and in combination in human hepatocarcinoma (HepG2) cells was evaluated by MTT assay. Furthermore, ROS production and alteration of ΔΨm as mechanisms of action were assessed. Cells were treated with concentrations ranging from 0.15 to 5 μM for NIV and from 0.78 to 50 μM for STE individually and in binary combinations. The combination ratio between the mixture STE + NIV was 10:1. The IC50 values of NIV ranged from 0.96 to 0.66 μM, whereas no IC50 values were obta…

endocrine systemCarcinoma HepatocellularSterigmatocystinAntineoplastic AgentsPharmacologyToxicology03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyIc50 valuesmedicineHumansCytotoxic T cellMTT assayCytotoxicityMycotoxinAdverse effect030304 developmental biology0303 health sciencesMolecular StructureLiver NeoplasmsDrug SynergismHep G2 Cells04 agricultural and veterinary sciencesGeneral Medicinemedicine.disease040401 food sciencechemistryHepatocellular carcinomaTrichothecenesFood ScienceSterigmatocystinFood and Chemical Toxicology
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Development of an in vitro neuroblastoma 3D model and its application for sterigmatocystin-induced cytotoxicity testing

2021

Abstract Given the increasing importance of establishing better risk assessments for mycotoxins, novel in vitro tools for the evaluation of their toxicity are mandatory. In this study, an in vitro 3D spheroid model from SH-SY5Y cells, a human neuroblastoma cell line, was developed, optimized and characterized to test the cytotoxic effects caused by the mycotoxin sterigmatocystin (STE). STE induced a concentration- and time-dependent cell viability decrease in spheroids. Spheroids displayed cell disaggregation after STE exposure, increasing in a dose-dependent manner and over time. STE also induced apoptosis as confirmed by immunofluorescence staining and Western blot. Following the decrease…

endocrine systemCytotoxicitySterigmatocystinBlotting WesternCellFluorescent Antibody TechniqueToxicology3D cell culturechemistry.chemical_compoundNeuroblastomaCell MovementCell Line TumorSpheroids CellularMechanisms of actionToxicity TestsmedicineHumansCell Culture Techniques Three DimensionalViability assayCytotoxicityChemistryGeneral MedicineMycotoxins3D spheroidCytostasisCell biologymedicine.anatomical_structure3D spheroid; Cytotoxicity; Mechanisms of action; Neuroblastoma; SterigmatocystinCell cultureApoptosisComet AssayReactive Oxygen SpeciesFood ScienceSterigmatocystin
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Immunohistochemical localization of the pro-peptide processing enzymes PC1/PC3 and PC2 in the human anal canal.

1997

Abstract HORsch, D., R. Day, N. G. Seidah, E. Weihe and M. K.-H. SchAFer. Immunohistochemical localization of the pro-peptide processing enzymes PC1/PC3 and PC2 in the human anal canal. Peptides 18(5) 755–760, 1997.—The distribution of prohormone/pro-peptide convertases PC1/PC3 and PC2 was investigated in the human anal canal by immunohistochemistry. Both prohormone convertases exhibited region-specific distribution patterns and were observed in neural and neuroendocrine cells and in nonneuroendocrine cellular elements. PC1/PC3 immunoreactivity was present in enteric neurons, subsets of nerve fibers, and neuroendocrine cells, and also in epithelial cells like intestinal stem cells, and a su…

endocrine systemPathologymedicine.medical_specialtyPhysiologyProhormoneNeuropeptideRectumAnal CanalBiologyBiochemistryImmunoenzyme TechniquesCellular and Molecular NeuroscienceEndocrinologymedicineChromograninsAspartic Acid EndopeptidasesHumansSubtilisinsAnal Transitional ZoneNeuronsNeuropeptidesAnal canalNeurosecretory SystemsEpitheliumNeoplasm Proteinsmedicine.anatomical_structureProprotein Convertase 2Fluorescent Antibody Technique DirectChromogranin AProprotein ConvertasesStem cellImmunostainingmedicine.drugPeptides
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Tenascin gene expression in rat liver and in rat liver cells. In vivo and in vitro studies.

1991

Tenascin is a major glycoprotein constituent of the extracellular matrix with a strong affinity to fibronectin; its distribution is believed to be temporarily and spatially limited. Tenascin gene expression is increased during wound healing processes. As repair mechanisms in chronic liver diseases resemble wound healing we studied tenascin gene expression in rat liver and in isolated rat liver cells. In normal rat liver a tenascin specific antiserum stains sinusoidal cells with fiber-like prolongations, which at the same time are desmin-positive (ITO-cells). In the CCl4-acutely-damaged liver a strong tenascin staining is detected in cells located among the mononuclear cells of the inflammat…

endocrine systemPathologymedicine.medical_specialtyanimal structuresKupffer CellsCell Adhesion Molecules NeuronalTenascinConnective tissueFluorescent Antibody TechniqueGene ExpressionLiver Cirrhosis Experimentaldigestive systemDesminmedicineAnimalsEndotheliumCarbon TetrachlorideCells CulturedExtracellular Matrix ProteinsbiologyTenascin CMuscle SmoothRats Inbred StrainsTenascinFibroblastsmusculoskeletal systemMolecular biologyRatsFibronectinEndothelial stem cellmedicine.anatomical_structureLiverCell cultureembryonic structuresbiology.proteinHepatic stellate cellWound healingVirchows Archiv. B, Cell pathology including molecular pathology
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