Search results for "Cerebral Arteries"

showing 7 items of 67 documents

Longitudinal imaging and evaluation of SAH-associated cerebral large artery vasospasm in mice using micro-CT and angiography

2019

Longitudinal in vivo imaging studies characterizing subarachnoid hemorrhage (SAH)-induced large artery vasospasm (LAV) in mice are lacking. We developed a SAH-scoring system to assess SAH severity in mice using micro CT and longitudinally analysed LAV by intravenous digital subtraction angiography (i.v. DSA). Thirty female C57Bl/6J-mice (7 sham, 23 SAH) were implanted with central venous ports for repetitive contrast agent administration. SAH was induced by filament perforation. LAV was assessed up to 14 days after induction of SAH by i.v. DSA. SAH-score and neuroscore showed a highly significant positive correlation (rsp = 0.803, p < 0.001). SAH-score and survival showed a negative sig…

medicine.medical_specialtySubarachnoid hemorrhageBiopsy030204 cardiovascular system & hematologySeverity of Illness IndexMice03 medical and health sciences0302 clinical medicineCerebral vasospasmIn vivomedicineAnimalsVasospasm Intracranialcardiovascular diseasesMicro ctmedicine.diagnostic_testbusiness.industryLarge arteryVasospasmOriginal ArticlesX-Ray MicrotomographyCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseaseImmunohistochemistrynervous system diseasesCerebral AngiographyDisease Models AnimalNeurologyAngiographyFemaleNeurology (clinical)RadiologyCardiology and Cardiovascular MedicinebusinessBiomarkers030217 neurology & neurosurgeryPreclinical imagingJournal of Cerebral Blood Flow & Metabolism
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Changes in the adrenergic mechanisms of cerebral arteries after subarachnoid hemorrhage in goats.

1994

We have examined the effects of experimental subarachnoid hemorrhage (SAH), induced by delivering autologous blood into the subarachnoid space, on the adrenergic mechanisms of the goat cerebrovascular bed. To achieve this, the response to noradrenaline was recorded both in vivo, by measuring cerebral blood flow in unanesthetized animals, and in vitro, by recording isometric tension in isolated cerebral arteries. In addition, we checked the function of adrenergic innervation by measuring the tritium efflux evoked by electrical stimulation in cerebral arteries preloaded with [3H]-noradrenaline, and we examined this innervation by using both fluorescent and electron transmission microscopy. Al…

medicine.medical_specialtySubarachnoid hemorrhageCerebral arteriesAdrenergicNorepinephrine (medication)NorepinephrineCerebral vasospasmInternal medicinemedicineAnimalscardiovascular diseasesbusiness.industryVascular diseaseGoatsBrainCerebral ArteriesSubarachnoid Hemorrhagemedicine.diseasenervous system diseasesMicroscopy ElectronEndocrinologymedicine.anatomical_structureCerebral blood flowIschemic Attack TransientNerve DegenerationSurgeryFemaleVascular ResistanceNeurology (clinical)Subarachnoid spacebusinessAdrenergic FibersBlood Flow Velocitymedicine.drugNeurosurgery
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Modulatory action of acetylcholine on cerebrovascular sympathetic neurotransmission

1991

1. Acetylcholine (10 micrograms/min) diminished the electrically-induced cerebral blood flow reductions. Atropine (1-2 mg) partially blocked this inhibitory effect. 2. Exogenously administered noradrenaline (1-10 micrograms) and tyramine (50-500 micrograms) reduced cerebral blood flow but this effect was unchanged by acetylcholine infusion. 3. Acetylcholine inhibited the nonadrenergic component of the electrically-induced contraction at a concentration greater than or equal to 10(-6) M and potentiated the adrenergic component at a concentration greater than or equal to 10(5) M. Atropine 10(-7) M) inhibited both of these effects. In addition, acetylcholine (10(-4) M) enhanced the electricall…

medicine.medical_specialtySympathetic Nervous SystemContraction (grammar)Cerebral arteriesTyramineAdrenergicTetrodotoxinIn Vitro TechniquesSynaptic TransmissionMuscle Smooth VascularNorepinephrinechemistry.chemical_compoundIsometric ContractionInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPharmacologyChemistryGoatsMuscarinic acetylcholine receptor M3Cerebral ArteriesTyramineAcetylcholineElectric StimulationAtropineEndocrinologyCerebrovascular CirculationFemaleAcetylcholinemedicine.drugGeneral Pharmacology: The Vascular System
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Normal Doppler Reference Values of the Pericallosal Artery

2015

Purpose: To provide the normal reference values of the Doppler flow of the pericallosal artery in relation to gestational age from 18 to 41 weeks of gestation. Materials and Methods: The pericallosal artery (PCA) was studied in 466 normal pregnancies. The pulsed Doppler evaluation of the pericallosal artery was done in A3 and A4 segments, and records from PI, RI and Vmax were studied. Results: The resistance index of the pericallosal artery in A3 / A4 segments exhibits a plateau from 18 to 31 weeks of gestation. After 31 weeks, a marked decrease becomes apparent. The pulsatility index of the pericallosal artery in A3 / A4 segments shows a plateau until 36 weeks of gestation. During the fina…

medicine.medical_specialtyUltrasonography Doppler TranscranialHemodynamicsGestational AgeUltrasonography PrenatalCorpus Callosumsymbols.namesakeText miningPregnancyReference ValuesInternal medicineHumansMedicineRadiology Nuclear Medicine and imagingFetusPregnancybusiness.industryInfant NewbornGestational ageAnatomyCerebral ArteriesPericallosal Arterymedicine.diseaseCross-Sectional Studiesmedicine.anatomical_structurePulsatile FlowReference valuesCardiologyVascular resistancesymbolsGestationFemaleVascular ResistancebusinessNuclear medicineDoppler effectUltraschall in der Medizin - European Journal of Ultrasound
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Possible Adverse Effects of Hyperventilation on rCBF during the Acute Phase of Total Proximal Occlusion of a Main Cerebral Artery

1969

Recent studies seem to favor a beneficial effect of hyperventilation (HV) in cases of cerebrovascular occlusion [1, 4] and brain trauma [2, 5]. On the other hand, it seems that, at least under certain circumstances, HV may exert an adverse effect upon cerebral blood flow and metabolism [3]. Since this question is not settled yet, it seems worthwhile to report briefly the present case of reversible total occlusion of the middle cerebral artery closely followed by successive rCBF studies (133Xe-gamma-clearance method), in which, during the acute phase, HV caused a latent ischemia to become manifest.

medicine.medical_specialtybusiness.industryCerebral arteriesIschemiamedicine.diseaseCerebral blood flowAnesthesiaInternal medicinemedicine.arteryHyperventilationMiddle cerebral arterymedicineCardiologyAnterior cerebral arterymedicine.symptomAdverse effectbusinessProximal occlusion
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Basics of Image Interpretation in Interventional Neuroradiology

2011

The majority of patients with cerebral vascular diseases, demanding neuroradiological interventions are those, who present with potential life-threatening spontaneous intracranial/intracerebral haemorrhages due to cerebral aneurysms or/and arteriovenous malformations (AVMs) or with cerebral infarction due to intra-arterial thrombi in cerebral arteries. Neuroradiological procedures (interventional neuroradiology) (IN), always, performed in general anaesthesia can be divided into occluding and opening interventions.

medicine.medical_specialtymedicine.diagnostic_testCerebral infarctionbusiness.industryCerebral arteriesArteriovenous malformationDigital subtraction angiographymedicine.diseaseAneurysmcardiovascular systemmedicineGeneral anaesthesiacardiovascular diseasesRadiologybusinessInterventional neuroradiology
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Endothelin action on goat cerebral arteries.

1990

Abstract Cumulative application of endothelin-1 (human) markedly constricted goat isolated cerebral arteries in a concentration-dependent manner. Contractile responses were not affected by removal of endothelial cells. Removal of extracellular calcium or addition of the calcium channel blocker nicardipine (10−7 M) failed to abolish responses to endothelin. The results suggest that the endothelium-independent constriction of cerebral arteries produced by endothelin cannot be explained solely by voltage-dependent calcium channels. The contractile responses are likely to be mediated by stimulation of specific receptors for this peptide.

medicine.medical_specialtymedicine.drug_classCerebral arteriesNicardipinePharmaceutical Sciencechemistry.chemical_elementStimulationCalcium channel blockerBiologyCalciumIn Vitro TechniquesMuscle Smooth VascularNicardipineInternal medicinemedicineAnimalsPharmacologyVoltage-dependent calcium channelEndothelinsGoatsCerebral ArteriesEndocrinologymedicine.anatomical_structurechemistrycardiovascular systemCalciumFemaleEndothelium VascularEndothelin receptormedicine.drugBlood vesselMuscle ContractionThe Journal of pharmacy and pharmacology
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