Search results for "Cerebral cortex"

showing 10 items of 529 documents

Study of spindle-spike interactions: Features of basal ganglia control

1987

Summary Changes in cortical spindle distribution following penicillin (PCN) injections were studied in feline generalized PCN epilepsy. PCN activation caused no substantial changes in spindle duration, frequency and intraburst frequency, while significant reductions in the amplitude of the negative waves were noted. At the same time combinations of spindle waves and epileptic complexes were recorded with one or more spikes randomly occurring at the beginning, in the middle, or at the end of a spindle envelope. Low frequency stimulation of the caudate nucleus induced a certain degree of enhancement in cortical precruciate spike frequency while high frequency activation of the entopeduncular …

MalePhysiologyCaudate nucleusPenicillinsElectroencephalographyGlobus PallidusBasal GangliaEpilepsySpike frequencyBasal gangliamedicineAnimalsLow frequency stimulationCerebral CortexDecerebrate StateEpilepsymedicine.diagnostic_testChemistryElectroencephalographymedicine.diseaseElectric StimulationSpindle envelopeCatsFemaleNeurology (clinical)Caudate NucleusNeuroscienceEntopeduncular nucleusRevue d&'apos;Electroencéphalographie et de Neurophysiologie Clinique
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Quantitative mass spectrometry for human melanocortin peptides in vitro and in vivo suggests prominent roles for β-MSH and desacetyl α-MSH in energy …

2018

Objective The lack of pro-opiomelanocortin (POMC)-derived melanocortin peptides results in hypoadrenalism and severe obesity in both humans and rodents that is treatable with synthetic melanocortins. However, there are significant differences in POMC processing between humans and rodents, and little is known about the relative physiological importance of POMC products in the human brain. The aim of this study was to determine which POMC-derived peptides are present in the human brain, to establish their relative concentrations, and to test if their production is dynamically regulated. Methods We analysed both fresh post-mortem human hypothalamic tissue and hypothalamic neurons derived from …

MalePluripotent Stem CellsLeptinlcsh:Internal medicineendocrine systemhPSC human pluripotent stem cellsPro-Opiomelanocortin[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyHypothalamusMass SpectrometryTandem Mass Spectrometry[SDV.BBM.GTP]Life Sciences [q-bio]/Biochemistry Molecular Biology/Genomics [q-bio.GN]beta-MSHHomeostasisHumansHuman pluripotent stem cellObesitylcsh:RC31-1245MSHNeuronsintegumentary systemReceptors MelanocortinLC-MS/MS liquid chromatography tandem mass spectrometryNeuropeptidesdigestive oral and skin physiologyPOMCPVH the paraventricular nucleus of the hypothalamusCTX cerebral cortexMelanocortinsNeuropeptidealpha-MSHOriginal ArticleFemalehormones hormone substitutes and hormone antagonistsChromatography Liquid
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Resonance of cortico-cortical connections of the motor system with the observation of goal directed grasping movements

2010

Goal directed movements require the activation of parietal, premotor and primary motor areas. In monkeys, neurons of these areas become active also during the observation of movements performed by others, especially for coding the goal of the action (mirror system). Using bifocal transcranial magnetic stimulation (TMS) in healthy subjects, we tested whether the observation of goal directed reach to grasp actions may lead to specific changes in the short-latency connections linking key areas of the mirror system, such as the anterior intraparietal cortex (AIP) and the ventral premotor cortex (PMv), with the primary motor cortex (M1). We found that AIP-M1 and PMv-M1 cortico-cortical interacti…

MalePremotor cortexmedicine.medical_treatmentObservationParietal cortexGoalBehavioral NeuroscienceCortex (anatomy)Neural PathwaysEvoked PotentialsMirror neuronCerebral CortexConnectivityBrain MappingHand StrengthMIRROR NEURONSBody movementSkeletalTranscranial Magnetic Stimulationmedicine.anatomical_structureMotorMuscleFemaleSettore MED/26 - NeurologiaPrimary motor cortexPsychologyGoalsHumanAdultConnectivity; Observation; Parietal cortex; Premotor cortex; Transcranial magnetic stimulation;Cognitive NeuroscienceMovementGoals; Movement; Muscle Skeletal; Male; Young Adult; Electromyography; Female; Evoked Potentials Motor; Cerebral Cortex; Humans; Photic Stimulation; Psychomotor Performance; Observation; Hand Strength; Neural Pathways; Transcranial Magnetic Stimulation; Brain Mapping; AdultPosterior parietal cortexExperimental and Cognitive PsychologyConnectivity; Observation; Parietal cortex; Premotor cortex; Transcranial magnetic stimulationNOPremotor cortexNeural PathwayYoung AdultMotor systemmedicineHumansMuscle SkeletalSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaElectromyographyEvoked Potentials MotorTranscranial magnetic stimulationGoals; Young Adult; Humans; Electromyography; Observation; Movement; Muscle Skeletal; Cerebral Cortex; Photic Stimulation; Brain Mapping; Evoked Potentials Motor; Adult; Neural Pathways; Hand Strength; Psychomotor Performance; Transcranial Magnetic Stimulation; Female; MaleTMSNeurosciencePhotic StimulationPsychomotor Performance
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Intrinsically determined cell death of developing cortical interneurons.

2009

The cell death of inhibitory neurons, which originate far from the cortical areas to which they migrate during embryonic development, is determined autonomously rather than by competition for trophic signals from other cell types. It has long been known that apoptosis, a form of programmed cell death, eliminates young cells from developing tissues. In the field of neurobiology, it is widely believed that developmental neuronal-cell death results from cellular competition for environmentally derived survival signals that selects for an optimally sized and properly wired population of neurons. This study of developmental cell death in the mouse cortex in vivo, in vitro and after transplantati…

MaleProgrammed cell deathInterneurongenetic structuresCell SurvivalPopulationApoptosisCell CountNeocortexBiologyArticle03 medical and health sciencesMice0302 clinical medicineNeural Stem CellsInterneuronsmedicineAnimalseducationCellular Senescence030304 developmental biologybcl-2-Associated X Protein0303 health scienceseducation.field_of_studyMultidisciplinaryNeocortexMembrane GlycoproteinsCaspase 3musculoskeletal neural and ocular physiologyPyramidal CellsfungiProtein-Tyrosine KinasesCell biologyTransplantationMice Inbred C57BLmedicine.anatomical_structurenervous systemAnimals NewbornInhibitory Postsynaptic PotentialsCerebral cortexbiology.proteinFemaleCell aging030217 neurology & neurosurgeryNeurotrophinNature
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Tissue oxygenation in brain, muscle and fat in a rat model of sleep apnea: differential effect of obstructive apneas and intermittent hypoxia.

2011

Study Objectives: To test the hypotheses that the dynamic changes in brain oxygen partial pressure (PtO 2) in response to obstructive apneas or to intermittent hypoxia differ from those in other organs and that the changes in brain PtO 2 in response to obstructive apneas is a source of oxidative stress. Design: Prospective controlled animal study. Setting: University laboratory. Participants: 98 Sprague-Dawley rats. Interventions: Cerebral cortex, skeletal muscle, or visceral fat tissues were exposed in anesthetized animals subjected to either obstructive apneas or intermittent hypoxia (apneic and hypoxic events of 15 s each and 60 events/h) for 1 h. Measurements and Results: Arterial oxyge…

MaleRat modelSettore MED/10 - Malattie Dell'Apparato RespiratorioTissue Oxygenation in Brain Muscle and Fat in Rat Model of ApneaRats Sprague-DawleySleep Apnea SyndromesPhysiology (medical)medicineAnimalsHSP70 Heat-Shock ProteinsHypoxiaMuscle SkeletalCerebral CortexAnalysis of VarianceSleep Apnea Obstructivebusiness.industryVascular Endothelial Growth FactorsApneaSleep apneaIntermittent hypoxiaHypoxia (medical)medicine.diseaseLipid MetabolismGlutathioneTissue oxygenation obstructive apnea intermittent hypoxia animal model oxidative stressRatsOxygenDisease Models Animalmedicine.anatomical_structureTissue oxygenationCerebral cortexAnesthesiaObstructive ApneaNeurology (clinical)Lipid Peroxidationmedicine.symptombusiness
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Chronic restraint stress and chronic corticosterone treatment modulate differentially the expression of molecules related to structural plasticity in…

2004

Stress and stress-related hormones induce structural changes in neurons of the adult CNS. Neurons in the hippocampus, the amygdala and the prefrontal cortex undergo neurite remodeling after chronic stress. In the hippocampus some of these effects can be mimicked with chronic administration of adrenal steroids. These changes in neuronal structure may be mediated by certain molecules related to plastic events such as the polysialylated form of the neural cell adhesion molecule (PSA-NCAM). The expression of PSA-NCAM persists in the adult hippocampus and it is up-regulated after chronic stress. The piriform cortex also displays considerable levels of PSA-NCAM during adulthood and indirect evide…

MaleRestraint Physicalmedicine.medical_specialtyDoublecortin ProteinHippocampusNeural Cell Adhesion Molecule L1AmygdalaRats Sprague-Dawleychemistry.chemical_compoundCorticosteroneStress PhysiologicalInternal medicinePiriform cortexmedicineAnimalsChronic stressOlfactory memoryPrefrontal cortexCerebral CortexNeuronal PlasticitybiologyGeneral NeuroscienceDoublecortinRatsmedicine.anatomical_structureEndocrinologynervous systemchemistryGene Expression RegulationChronic Diseasebiology.proteinSialic AcidsCorticosteroneNeuroscienceNeuroscience
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Expression of α4-1 and α5 Nicotinic Cholinoceptor mRNA in the Aging Rat Cerebral Cortex

1997

Although important in neurodegeneration, systematic studies of nicotinic acetylcholine receptor expression in normal aging human brains are difficult to perform. We have studied the expression of nicotinic receptor alpha 4-1 and alpha 5 mRNA in the frontal and parietal isocortex of 3- (young adult), 24- (late middle aged), and 33-month-old (old) rats by nonisotopic in situ hybridization. In all groups transcripts were mainly present in layer II/III and V pyramidal neurons. The numerical densities of alpha 4-1 mRNA-containing neurons with respect to those of cresyl violet-stained neurons decreased with aging in the rat frontal and parietal cortex, while those of alpha 5 mRNA-containing neuro…

MaleSenescenceAgingmedicine.medical_specialtyPosterior parietal cortexReceptors NicotinicBiologyNicotineInternal medicinemedicineAnimalsRNA MessengerRats WistarIn Situ HybridizationAcetylcholine receptorCerebral CortexGeneral NeuroscienceNeurodegenerationmedicine.diseaseImmunohistochemistryRatsNicotinic acetylcholine receptorNicotinic agonistEndocrinologymedicine.anatomical_structureCerebral cortexNeurology (clinical)Geriatrics and GerontologyDevelopmental Biologymedicine.drugNeurobiology of Aging
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Acute behavioural and neurotoxic effects of MDMA plus cocaine in adolescent mice.

2008

The poly-drug pattern is the most common among those observed in MDMA users, with cocaine being a frequently associated drug. This study evaluates the acute effects of MDMA (5, 10 and 20 mg/kg), alone or in combination with cocaine (25 mg/kg), on motor activity, anxiety (elevated plus maze and social interaction test), memory and brain monoamines in adolescent mice, Both drugs, administered alone or concurrently, produced hyperactivity and a decrease in social contacts. However, an anxiolytic effect, studied by means of the elevated plus maze and expressed as an increase in the time spent on the open arms, was observed only in those animals treated with cocaine and MDMA. The passive avoidan…

MaleSerotoninElevated plus mazeMDMAmedicine.drug_classDopamineN-Methyl-34-methylenedioxyamphetamineStriatumPharmacologyAnxietyMotor ActivityToxicologyAnxiolyticHippocampusCellular and Molecular NeuroscienceMiceSerotonin AgentsDevelopmental NeuroscienceCocaineDopaminemental disordersmedicineAvoidance LearningAnimalsBiogenic MonoaminesInterpersonal RelationsBrain ChemistryCerebral CortexBehavior AnimalMDMACortex (botany)NeostriatumSocial behaviourAnxietyNeurotoxicity SyndromesSerotoninmedicine.symptomElevated plus mazePsychologypsychological phenomena and processesmedicine.drugNeurotoxicology and teratology
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Adolescent pre-exposure to ethanol or MDMA prolongs the conditioned rewarding effects of MDMA

2011

Adolescents often take ethanol (EtOH) in combination with MDMA (3,4-methylenedioxymethylamphetamine). In the present work we studied the effect of repeated intermittent adolescent pre-exposure to both drugs on the behavioral and neurochemical effects of MDMA in mice. Sixteen days after pre-treatment, the rewarding and reinstating effects of MDMA in the conditioned place preference (CPP) paradigm were evaluated, along with the levels of biogenic amines, basal motor activity and corticosterone response to different challenges. Pre-exposure to EtOH, MDMA or EtOH+MDMA did not affect the CPP induced by 10mg/kg of MDMA. However, adolescent exposure to EtOH or MDMA increased the duration of the co…

MaleSerotoninmedicine.medical_specialtyDopamineN-Methyl-34-methylenedioxyamphetaminePoison controlExperimental and Cognitive PsychologyStriatumMotor ActivityChoice BehaviorHippocampusDrug Administration ScheduleExtinction PsychologicalMiceBehavioral Neurosciencechemistry.chemical_compoundNeurochemicalRewardCorticosteroneInternal medicineConditioning Psychologicalmental disordersAnimals Outbred StrainsmedicineAnimalsDrug InteractionsCerebral CortexEthanolIllicit DrugsMDMAExtinction (psychology)Hydroxyindoleacetic AcidCorpus StriatumConditioned place preferenceMonoamine neurotransmitterEndocrinologychemistryAnesthesia34-Dihydroxyphenylacetic AcidCorticosteronePsychologypsychological phenomena and processesmedicine.drugPhysiology & Behavior
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TLR4 response mediates ethanol-induced neurodevelopment alterations in a model of fetal alcohol spectrum disorders

2017

Background Inflammation during brain development participates in the pathogenesis of early brain injury and cognitive dysfunctions. Prenatal ethanol exposure affects the developing brain and causes neural impairment, cognitive and behavioral effects, collectively known as fetal alcohol spectrum disorders (FASD). Our previous studies demonstrate that ethanol activates the innate immune response and TLR4 receptor and causes neuroinflammation, brain damage, and cognitive defects in the developmental brain stage of adolescents. We hypothesize that by activating the TLR4 response, maternal alcohol consumption during pregnancy triggers the release of cytokines and chemokines in both the maternal …

MaleSerum0301 basic medicineChemokineDevelopmental Disabilitiesmedicine.medical_treatmentlcsh:RC346-429MiceMyelin0302 clinical medicineNeuroinflammationPregnancyTLR4Maternal BehaviorFetal alcohol spectrum disordersMice KnockoutMicrogliabiologyGeneral NeuroscienceAge FactorsBrainCerebral cortexBehavior impairmentsmedicine.anatomical_structureCytokineNeurologyPrenatal Exposure Delayed EffectsCytokinesFemalemedicine.symptomMyelin ProteinsAmniotic fluidmedicine.medical_specialtyOffspringImmunologyNerve Tissue ProteinsBrain damage03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicineAvoidance LearningmedicineAnimalsMaze Learninglcsh:Neurology. Diseases of the nervous systemNeuroinflammationEthanolbusiness.industryResearchBody WeightCentral Nervous System DepressantsMice Inbred C57BLToll-Like Receptor 4Disease Models AnimalMicroscopy Electron030104 developmental biologyEndocrinologyAnimals NewbornPrenatal ethanol exposureImmunologybiology.proteinTLR4business030217 neurology & neurosurgeryJournal of Neuroinflammation
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