Search results for "Chemotaxis"

showing 10 items of 145 documents

Vitamin D Receptor Activation Reduces Angiotensin-II–Induced Dissecting Abdominal Aortic Aneurysm in Apolipoprotein E–Knockout Mice

2015

Objective— Abdominal aortic aneurysm (AAA) is a vascular disorder characterized by chronic inflammation of the aortic wall. Low concentrations of vitamin D 3 are associated with AAA development; however, the potential direct effect of vitamin D 3 on AAA remains unknown. This study evaluates the effect of oral treatment with the vitamin D 3 receptor (VDR) ligand, calcitriol, on dissecting AAA induced by angiotensin-II (Ang-II) infusion in apoE −/− mice. Approach and Results— Oral treatment with calcitriol reduced Ang-II–induced dissecting AAA formation in apoE −/− mice, which was unrelated to systolic blood pressure or plasma cholesterol concentrations. Immunohistochemistry and reverse-tran…

MaleVascular Endothelial Growth Factor A0301 basic medicineDissecting Abdominal Aortic Aneurysm030204 cardiovascular system & hematologyLigandsCalcitriol receptorchemistry.chemical_compound0302 clinical medicineAorta AbdominalCells CulturedMice KnockoutAngiotensin IIVascular endothelial growth factorChemotaxis LeukocyteVascular endothelial growth factor APhenotypeMatrix Metalloproteinase 9Vitamin D3 ReceptorMatrix Metalloproteinase 2RNA Interferencelipids (amino acids peptides and proteins)ChemokinesMitogen-Activated Protein KinasesCardiology and Cardiovascular MedicineSignal Transductionmedicine.drugmedicine.medical_specialtyCalcitriolBiologyTransfectionProinflammatory cytokine03 medical and health sciencesApolipoproteins ECalcitriolInternal medicineHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansGenetic Predisposition to DiseaseRetinoid X Receptor alphaMacrophagesAngiotensin IIMice Inbred C57BLAortic DissectionDisease Models Animal030104 developmental biologyEndocrinologychemistryReceptors CalcitriolAortic Aneurysm AbdominalArteriosclerosis, Thrombosis, and Vascular Biology
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CXCR2 blockade impairs angiotensin II-induced CC chemokine synthesis and mononuclear leukocyte infiltration.

2007

Objective—Angiotensin II (Ang-II) and mononuclear leukocytes are involved in atherosclerosis. This study reports the inhibition of Ang-II–induced mononuclear cell recruitment by CXCR2 antagonism and the mechanisms involved.Methods and Results—Ang-II (1 nmol/L, i.p. in rats) induced CXC and CC chemokines, followed by neutrophil and mononuclear cell recruitment. Administration of the CXCR2 antagonist, SB-517785-M, inhibited the infiltration of both neutrophils (98%) and mononuclear cells (60%). SB-517785-M had no effect on the increase in CXC chemokine levels but reduced MCP-1, RANTES, and MIP-1α release by 66%, 63%, and 80%, respectively. Intravital microscopy showed that pretreatment with S…

Malemedicine.medical_specialtyChemokineCXCR3Peripheral blood mononuclear cellLosartanReceptors Interleukin-8BRats Sprague-DawleyChemokine receptorInternal medicinemedicineCell AdhesionCCL17AnimalsHumansCXC chemokine receptorsSplanchnic CirculationChemokine CCL7Chemokine CCL4Chemokine CCL5Cells CulturedChemokine CCL2Chemokine CCL3InflammationbiologyAngiotensin IIMicrocirculationEndothelial CellsMacrophage Inflammatory ProteinsAtherosclerosisAngiotensin IIMonocyte Chemoattractant ProteinsRatsMononuclear cell infiltrationChemotaxis LeukocyteEndocrinologyNeutrophil Infiltrationbiology.proteinLeukocytes MononuclearCardiology and Cardiovascular MedicineAngiotensin II Type 1 Receptor BlockersArteriosclerosis, thrombosis, and vascular biology
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Granulocyte integrins before and after activation in acute ischaemic stroke.

2001

We examined in 19 subjects with acute ischaemic stroke (AIS) the PMN integrin pattern (CD11a, CD11b, CD11c, CD18), using indirect immunofluorescence and adopting a flow cytometer, at baseline and during activation, prolonged for 5 and 15 min, with 4-phorbol 12-myristate 13-acetate (PMA). At baseline, an increase in the expression of CD11c and CD18 and a decrease in the CD11b were evident in AIS subjects compared to normals. After activation, we found in normals a constant and significant increase of all PMN adhesive molecules, while in AIS subjects, we found an increase in CD11b and CD18, a decrease in CD11a and no variation in CD11c. While the basal upregulation of CD11c and CD18 may depen…

Malemedicine.medical_specialtyIntegrinsIntegrinCD11cIntegrin alphaXbeta2Macrophage-1 Antigenchemical and pharmacologic phenomenaCD18CD11aGranulocyteBrain IschemiaDownregulation and upregulationInternal medicinemedicineHumansFluorescent Antibody Technique IndirectAgedAged 80 and overintegumentary systembiologybusiness.industryhemic and immune systemsMiddle AgedPathophysiologyLymphocyte Function-Associated Antigen-1StrokeChemotaxis LeukocyteEndocrinologymedicine.anatomical_structureNeurologyIntegrin alpha MCD18 AntigensImmunologyAcute Diseasebiology.proteinCarcinogensTetradecanoylphorbol AcetateFemaleNeurology (clinical)businessGranulocytesJournal of the neurological sciences
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Muscarinic receptors, leukotriene B4 production and neutrophilic inflammation in COPD patients.

2005

Background:  Acetylcholine (ACh) plays an important role in smooth muscle contraction and in the development of airway narrowing; preliminary evidences led us to hypothesize that ACh might also play a role in the development of airways inflammation in chronic obstructive pulmonary disease (COPD). Methods:  We evaluated the concentrations of leukotriene B4 (LTB4) in induced sputum, and the expression of Ach M1, M2, and M3 receptors in sputum cells (SC) obtained from 16 patients with COPD, 11 smokers, and 14 control subjects. The SC were also treated with ACh and the production of LTB4 assessed in the presence or absence of a muscarinic antagonist (oxitropium). In blood monocytes, we evaluate…

Malemedicine.medical_specialtyLeukotriene B4NeutrophilsImmunologyProtozoan ProteinsLeukotriene B4chemistry.chemical_compoundPulmonary Disease Chronic ObstructiveInternal medicineMuscarinic acetylcholine receptorImmunology and AllergyMedicineHumansReceptorCells CulturedAgedFlavonoidsCOPDMitogen-Activated Protein Kinase 3business.industryCalcium-Binding ProteinsSputumMuscarinic acetylcholine receptor M3Muscarinic antagonistSmooth muscle contractionMiddle Agedmedicine.diseaseImmunohistochemistryReceptors MuscarinicAcetylcholinerespiratory tract diseasesChemotaxis LeukocyteEndocrinologychemistryLeukocytes MononuclearFemalebusinessAcetylcholinemedicine.drugAllergy
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Acute Ischemic Stroke

2000

Background and Purpose —Several reports have considered the role of systemic leukocytes in acute ischemic stroke (AIS). Initially, greater attention was focused on the leukocyte count and subsequently on their adhesiveness, aggregation, rheology, and activation. The aim of this study was the evaluation of certain polymorphonuclear leukocyte (PMN) parameters, reflecting their rheology and activation, in subjects with AIS. Methods —In a group of 19 subjects with AIS and in a control group of 18 subjects with asymptomatic vascular atherosclerotic disease, we evaluated the PMN membrane fluidity and cytosolic Ca 2+ concentration at baseline and after in vitro chemotactic activation with 4-phorb…

Malemedicine.medical_specialtyMembrane FluidityNeutrophilsNeutrophileIschemiaAsymptomaticNeutrophil ActivationBrain IschemiaBrain ischemiaCytosolInternal medicinemedicineMembrane fluidityHumansStrokeAgedAged 80 and overAdvanced and Specialized Nursingbusiness.industryChemotaxisChemotaxisMiddle Agedmedicine.diseasePathophysiologyStrokeEndocrinologyAcute DiseaseCalciumFemaleNeurology (clinical)medicine.symptomCardiology and Cardiovascular MedicinebusinessStroke
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Gastric antisecretory drugs induce leukocyte-endothelial cell interactions through gastrin release and activation of CCK-2 receptors.

2007

Antisecretory drugs are effective antiulcer agents, but its chronic use generates hypergastrinemia and accelerates the development of atrophic gastritis in Helicobacter pylori-positive patients. We have recently shown that gastrin exerts a proinflammatory effect in rats through CCK-2 receptor activation that contributes to the inflammation induced by H. pylori. The present study was designed to examine whether gastrin hypersecretion in response to treatment with antisecretory drugs induces an inflammatory response that could promote mucosal atrophy. The effects of omeprazole or famotidine on leukocyte/endothelial cell interactions in vivo were analyzed in rat mesenteric venules using intrav…

Malemedicine.medical_specialtyProglumidemedicine.drug_classInflammationCD18Cell CommunicationProinflammatory cytokineRats Sprague-DawleyInternal medicineGastrinsmedicineLeukocytesAnimalsOmeprazoleGastrinPharmacologyInflammationMicroscopy Videobusiness.industryEndothelial CellsProton Pump InhibitorsReceptor antagonistAnti-Ulcer AgentsFlow CytometryImmunohistochemistryReceptor Cholecystokinin BRatsFamotidineChemotaxis LeukocyteEndocrinologyGastric MucosaMolecular Medicinemedicine.symptombusinessmedicine.drugThe Journal of pharmacology and experimental therapeutics
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Leukocyte Rheology Before and After Chemotactic Activation in some Venous Diseases

1999

Abstract Objective: to evaluate leukocyte rheology, polymorphonuclear leukocyte (PMN) membrane fluidity and cytosolic Ca2+ concentration in subjects with post-phlebitic leg syndrome (PPS) and acute deep-venous leg thrombosis (DVT). Subjects: twenty-two subjects with leg PPS and 14 subjects with leg DVT. Methods: we evaluated the leukocyte filtration (unfractionated, mononuclear cells (MN) and PMN), the PMN membrane fluidity and the PMN cytosolic Ca2+ concentration. Subsequently, we evaluated the same PMN variables after in vitro chemotactic activation with 4-phorbol 12-myristate 13-acetate (PMA) and N -formyl-methionyl-leucyl-phenylalanine (fMLP). Results: at baseline we observed a signific…

Malemedicine.medical_specialtyTime FactorsMembrane FluidityNeutrophilsPostphlebitic SyndromeIn Vitro TechniquesNeutrophil activation.Peripheral blood mononuclear cellMonocytesNeutrophil ActivationCytosolWhite blood cell filtrationInternal medicinemedicineMembrane fluidityHumansMedicine(all)Venous ThrombosisPolymorphonuclear leukocytebusiness.industrySignificant differenceChemotaxishemic and immune systemsChronic venous insufficiencyMiddle Agedmedicine.diseaseThrombosisIn vitroChemotaxis LeukocyteCytosolEndocrinologyHemorheologyImmunologyDeep venous thrombosisCalciumFemaleSurgeryCardiology and Cardiovascular MedicinebusinessEuropean Journal of Vascular and Endovascular Surgery
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An asymmetric electrospun membrane for the controlled release of ciprofloxacin and FGF-2: Evaluation of antimicrobial and chemoattractant properties.

2021

Here, an asymmetric double-layer membrane has been designed and fabricated by electrospinning as a tool for a potential wound healing application. A hydrophobic layer has been produced by using a polyurethane-polycaprolactone (PU-PCL) copolymer and loaded with the antibacterial ciprofloxacin whereas an ion responsive hydrophilic layer has been produced by using an octyl derivative of gellan gum (GG-C8) and polyvinyl alcohol (PVA) and loaded with the growth factor FGF-2. This study investigated how the properties of this asymmetric membrane loaded with actives, were influenced by the ionotropic crosslinking of the hydrophilic layer. In particular, the treatment in DPBS and the crosslinking i…

Materials sciencePolyurethanesNanofibersBioengineeringmacromolecular substances02 engineering and technologyChemotaxis (FGF-2)Antimicrobial activity (CPX); Chemotaxis (FGF-2); Double layer electrospun membrane; Gellan gum alkyl-derivative; Polyurethanes010402 general chemistry01 natural sciencesPolyvinyl alcoholGellan gum alkyl-derivativeBiomaterialschemistry.chemical_compoundAnti-Infective AgentsCiprofloxacinCopolymerDouble layer electrospun membraneChemotactic Factorstechnology industry and agriculture021001 nanoscience & nanotechnologyAntimicrobialControlled releaseBandagesGellan gumElectrospinning0104 chemical sciencesAnti-Bacterial AgentsAntimicrobial activity (CPX)MembranechemistryMechanics of MaterialsSettore CHIM/09 - Farmaceutico Tecnologico ApplicativoDelayed-Action PreparationsBiophysicsFibroblast Growth Factor 20210 nano-technologyLayer (electronics)Materials scienceengineering. C, Materials for biological applications
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Demyelination patterns in a mathematical model of multiple sclerosis.

2016

In this paper we derive a reaction-diffusion-chemotaxis model for the dynamics of multiple sclerosis. We focus on the early inflammatory phase of the disease characterized by activated local microglia, with the recruitment of a systemically activated immune response, and by oligodendrocyte apoptosis. The model consists of three equations describing the evolution of macrophages, cytokine and apoptotic oligodendrocytes. The main driving mechanism is the chemotactic motion of macrophages in response to a chemical gradient provided by the cytokines. Our model generalizes the system proposed by Calvez and Khonsari (Math Comput Model 47(7–8):726–742, 2008) and Khonsari and Calvez (PLos ONE 2(1):e…

Multiple Sclerosismedicine.medical_treatmentInflammationApoptosisBiology01 natural sciencesModels BiologicalConcentric ring03 medical and health sciences0302 clinical medicineTuring instabilitymedicineHumansMultiple sclerosi0101 mathematicsSettore MAT/07 - Fisica MatematicaInflammationMicrogliaOligodendrocyte apoptosisPatternMultiple sclerosisTuring instabilityApplied MathematicsChemotaxismedicine.diseaseAgricultural and Biological Sciences (miscellaneous)Magnetic Resonance Imaging010101 applied mathematicsChemotaxis PDE modelCytokinemedicine.anatomical_structureModeling and SimulationImmunologymedicine.symptomNeuroscience030217 neurology & neurosurgeryDemyelinating DiseasesJournal of mathematical biology
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Phospholipase A2 and arachidonic acid: a common link in the generation of the eosinophil chemotactic factor (ECF) from human PMN by various stimuli.

1980

An eosinophil chemotactic factor (ECF) of low molecular weight can be generated and released from human polymorphonuclear neutrophils by the calcium ionophore, phagocytosis of zymosan particles, arachidonic acid, and phospholipase A2. Since the activation of cells by the ionophore and during the phagocytic event leads to phospholipid turnover, with the subsequent generation of arachidonic acid, it is reasonable that phospholipase A2 represents the common link for ECF production. The kinetics of ECF release by phospholipase A2 is similar to the pattern observed with the various stimuli. After a rapid rise in activity a decline occurred at later times of secretion, suggesting a mechanism of i…

NeutrophilsPhagocytosisChemotactic Factors EosinophilImmunologyPhospholipidArachidonic AcidsBiologyPhospholipases Achemistry.chemical_compoundLipoxygenasePhospholipase A2Phospholipase DHumansCalcimycinCells CulturedChemotactic FactorsZymosanZymosanChemotaxisGeneral MedicineEosinophilsChemotaxis LeukocytePhospholipases A2chemistryBiochemistryPhospholipasesType C Phospholipasesbiology.proteinArachidonic acidCell fractionationSubcellular FractionsScandinavian journal of immunology
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