Search results for "Cholecystokinin"

showing 10 items of 23 documents

A possible association between the CCK-AR gene and persistent auditory hallucinations in schizophrenia.

2004

AbstractRecent studies have suggested that DNA variations in the CCK-AR gene might predispose individuals to schizophrenia and particularly to auditory hallucinations (AH). The aim of this study is to assess the association between AH, using a specific scale for AH in schizophrenia (PSYRATS), and the CCK-AR polymorphism at 779 in a Spanish sample. A total of 105 DSM-IV schizophrenic patients with AH and 93 unrelated controls were studied. Twenty-two patients were considered as persistent auditory hallucinators, which showed similar clinical and demographic characteristic than patients with episodic AH, but with the exception of the PSYRATS values. The persistent AH group showed an excess of…

AdultMalemedicine.medical_specialtyPsychosisPeriodicityGenotypeHallucinationsSeverity of Illness IndexGenetic determinismLinkage Disequilibrium03 medical and health sciences0302 clinical medicineGene FrequencyInternal medicineSurveys and QuestionnairesSeverity of illnessmedicineHumansAlleleSex DistributionPsychiatryAllele frequencyGenetic associationDNA PrimersDemographyAuditory hallucinationPolymorphism Geneticmedicine.diseaseIntrons030227 psychiatryReceptor Cholecystokinin ADiagnostic and Statistical Manual of Mental DisordersPsychiatry and Mental healthEndocrinologySchizophreniaSchizophreniaFemalemedicine.symptomPsychology030217 neurology & neurosurgeryEuropean psychiatry : the journal of the Association of European Psychiatrists
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Direct identification of the agonist binding site in the human brain cholecystokinin-B receptor

1999

In investigating the agonist binding site of the human brain cholecystokininB receptor (CCKBR), we employed the direct protein chemical approach using a photoreactive tritiated analogue of sulfated cholecystokinin octapeptide, which contains the p-benzoylbenzoyl moiety at the N-terminus, followed by purification of the affinity-labeled receptor to homogeneity. This probe bound specifically, saturably, and with high affinity (KD = 1.2 nM) to the CCKBR and has full agonistic activity. As the starting material for receptor purification, we used stably transfected HEK 293 cells overexpressing functional CCKBR. Covalent labeling of the WGA-lectin-enriched receptor revealed a 70-80 kDa glycoprote…

ElectrophoresisModels MolecularAgonistProtein Conformationmedicine.drug_classPeptidePhotoaffinity LabelsTritiumBiochemistryMass SpectrometrySincalidemedicineHumansBinding siteReceptorCells Culturedchemistry.chemical_classificationBinding SitesEdman degradationHEK 293 cellsBrainMolecular biologyReceptor Cholecystokinin BchemistryBiochemistryCholecystokinin B receptorChromatography GelMutagenesis Site-DirectedReceptors CholecystokininCholecystokininGlycoproteinSequence Analysis
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Synergistic interaction between CCK and leptin to regulate food intake.

2000

Abstract Leptin administered (either intracerebroventricularly, icv, or intraperitoneally, ip) acts in synergy with CCK to suppress food intake and body weight in lean mice or rats. The potentiating effect induced by the co-injection of ip CCK and leptin to inhibit food consumption in mice is mediated by the CCK-A receptor and capsaicin sensitive afferents. In vitro, studies in rats showed that a subset of gastric vagal afferent fibers responded to leptin injected directly into the gastric artery only after a prior intra-arterial CCK injection. Moreover, the tonic activity of gastric-related neurons in the nucleus tractus solitarius (NTS) increased when leptin was delivered into the gastric…

Leptinmedicine.medical_specialtyPhysiologyClinical BiochemistryNeuropeptideBiochemistryCellular and Molecular Neurosciencechemistry.chemical_compoundEatingMiceEndocrinologyInternal medicinemedicineAnimalsHumansReceptorCholecystokininGastric emptyingLeptindigestive oral and skin physiologyArea postremaBody WeightDrug SynergismRatsEndocrinologynervous systemchemistryHypothalamusCapsaicinCholecystokininhormones hormone substitutes and hormone antagonistsRegulatory peptides
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Inactivation of Socs3 in the Hypothalamus Enhances the Hindbrain Response to Endogenous Satiety Signals via Oxytocin Signaling.

2012

Leptin is an adipocyte-derived hormone that controls energy balance by acting primarily in the CNS, but its action is lost in common forms of obesity due to central leptin resistance. One potential mechanism for such leptin resistance is an increased hypothalamic expression of Suppressor of cytokine signaling 3 (Socs3), a feedback inhibitor of the Jak-Stat pathway that prevents Stat3 activation. Ample studies have confirmed the important role of Socs3 in leptin resistance and obesity. However, the degree to which Socs3 participates in the regulation of energy homeostasis in nonobese conditions remains largely undetermined. In this study, using adult mice maintained under standard diet, we d…

Leptinmedicine.medical_specialty[SDV.OT]Life Sciences [q-bio]/Other [q-bio.OT][SDV]Life Sciences [q-bio][ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionHypothalamusHindbrainSuppressor of Cytokine Signaling ProteinsBiologyOxytocinDevazepideSatiety ResponseEnergy homeostasis03 medical and health sciencesEatingMice0302 clinical medicineHormone AntagonistsInternal medicinemedicineAnimals[SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]SOCS3[ SDV.OT ] Life Sciences [q-bio]/Other [q-bio.OT]ComputingMilieux_MISCELLANEOUS030304 developmental biology2. Zero hunger0303 health sciences[SDV.OT] Life Sciences [q-bio]/Other [q-bio.OT]General NeuroscienceLeptindigestive oral and skin physiologyArticlesRhombencephalonEndocrinologyOxytocinHypothalamusSuppressor of Cytokine Signaling 3 Protein[ SDV.NEU ] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]Receptors Cholecystokinin[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryHomeostasisHormonemedicine.drugSignal Transduction
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Novel GPR120 agonist TUG891 modulates fat taste perception and preference and activates tongue-brain-gut axis in mice

2020

GPR120 is implicated as a lipid receptor in the oro-sensory detection of dietary fatty acids. However, the effects of GPR120 activation on dietary fat intake or obesity are not clearly understood. We investigated to determine whether the binding of TUG891, a novel GPR120 agonist, to lingual GPR120 modulates fat preference in mice. We explored the effects of TUG891 on obesity-related hormones and conducted behavioral choice tests on mice to better understand the physiologic relevance of the action of TUG891. In cultured mouse and human taste bud cells (TBCs), TUG891 induced a rapid increase in Ca2+ by acting on GPR120. A long-chain dietary fatty acid, linoleic acid (LA), also recruited Ca2+ …

Male0301 basic medicineAgonistlinoleic acidmedicine.medical_specialtyTasteextracellular signal-regulated kinase 1/2obesitymedicine.drug_classLinoleic acidQD415-436030204 cardiovascular system & hematologyBiochemistryReceptors G-Protein-CoupledMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyTongueInternal medicinemedicineAnimalsHumansReceptorCells CulturedResearch ArticlesCholecystokininchemistry.chemical_classificationPhenylpropionatesBiphenyl CompoundsBrainTaste PerceptionFatty acidGPR120Cell BiologyTaste BudsGastrointestinal MicrobiomeMice Inbred C57BL030104 developmental biologyEndocrinologychemistryglucagon-like peptide-1Hormone
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Age and Protein Restriction Followed by Balanced Refeeding Affect Pancreatic Digestive Enzyme Outputs and Turnover Times in Rats

1991

Outputs and turnover times of trypsinogen 2, chymotrypsinogen 1, lipase and amylase were determined in pancreatic juice of growing male Wistar rats at various times during protein restriction (5% protein) followed by balanced refeeding (20% protein). In control rats fed a 20% protein diet, trypsinogen 2, chymotrypsinogen 1 and amylase outputs increased progressively with age, those of lipase remained constant and the turnover times of the four hydrolases were shortened. With time, protein restriction induced the most rapid decrease in trypsinogen 2 output, followed by that of amylase, then by those of trypsinogen 1 and lipase. Compared with controls, protein restriction enhanced specific ra…

MaleAgingmedicine.medical_specialtyTrypsinogenMedicine (miscellaneous)ChymotrypsinogenBiologydigestive systemCholecystokinin receptorchemistry.chemical_compoundInternal medicinemedicineAnimalsAmylaseLipasePancreasCholecystokininNutrition and DieteticsBody WeightRats Inbred StrainsLipaseRatsEndocrinologychemistryFoodAmylasesPancreatic juiceDigestive enzymeTrypsinogenbiology.proteinDietary ProteinsIsoelectric FocusingFood DeprivationThe Journal of Nutrition
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The dendritic spines of interneurons are dynamic structures influenced by PSA-NCAM expression.

2013

Excitatory neurons undergo dendritic spine remodeling in response to different stimuli. However, there is scarce information about this type of plasticity in interneurons. The polysialylated form of the neural cell adhesion molecule (PSA-NCAM) is a good candidate to mediate this plasticity as it participates in neuronal remodeling and is expressed by some mature cortical interneurons, which have reduced dendritic arborization, spine density, and synaptic input. To study the connectivity of the dendritic spines of interneurons and the influence of PSA-NCAM on their dynamics, we have analyzed these structures in a subpopulation of fluorescent spiny interneurons in the hippocampus of glutamic …

MaleDendritic spineTime FactorsInterneuronCognitive NeuroscienceDendritic SpinesGreen Fluorescent ProteinsHippocampusNeuraminidaseMice TransgenicNerve Tissue ProteinsNeural Cell Adhesion Molecule L1BiologyHippocampal formationIn Vitro TechniquesHippocampus03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineOrgan Culture TechniquesInterneuronsmedicineAnimals030304 developmental biology0303 health sciencesPolysialic acidGlutamate DecarboxylaseDendritic filopodiamedicine.anatomical_structurenervous systemAnimals NewbornGene Expression RegulationCalbindin 2Excitatory postsynaptic potentialSialic AcidsNeural cell adhesion moleculeCholecystokininSomatostatinNeuroscience030217 neurology & neurosurgeryVasoactive Intestinal PeptideCerebral cortex (New York, N.Y. : 1991)
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Photoperiod effects on bombesin- and cholecystokinin-like immunoreactivity in the suprachiasmatic nuclei of the Djungarian hamster (Phodopus sungorus)

1991

The immunocytochemical distribution of the putative satiety peptides bombesin (BBS) and cholecystokinin (CCK) were studied in the hypothalamic suprachiasmatic nuclei (SCN) of male and female Djungarian hamsters (Phodopus sungorus) held under either long (light/dark, LD 16:8 h) or short (LD 8:16) photoperiod. The animals were killed by perfusion with a fixative at the middle of the light period and the tissue was processed by routine immunohistochemical methods. Perikarya exhibiting BBS- or CCK-like immunoreactivity (LI) were found in the SCN of animals of all groups. Sex-related differences were not observed. In contrast, long-term exposure to short days decreased the number of neurons exhi…

MalePeriodicityendocrine systemmedicine.medical_specialtyTime FactorsLightNeuropeptideHamsterBiologychemistry.chemical_compoundCricetinaeInternal medicinemedicineAnimalsTissue DistributionCholecystokininSuprachiasmatic nucleusGeneral Neurosciencedigestive oral and skin physiologyBombesinbiology.organism_classificationImmunohistochemistryPhodopusEndocrinologymedicine.anatomical_structurechemistryHypothalamusBombesinFemaleSuprachiasmatic NucleusCholecystokininPeriventricular nucleushormones hormone substitutes and hormone antagonistsNeuroscience Letters
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Gastric antisecretory drugs induce leukocyte-endothelial cell interactions through gastrin release and activation of CCK-2 receptors.

2007

Antisecretory drugs are effective antiulcer agents, but its chronic use generates hypergastrinemia and accelerates the development of atrophic gastritis in Helicobacter pylori-positive patients. We have recently shown that gastrin exerts a proinflammatory effect in rats through CCK-2 receptor activation that contributes to the inflammation induced by H. pylori. The present study was designed to examine whether gastrin hypersecretion in response to treatment with antisecretory drugs induces an inflammatory response that could promote mucosal atrophy. The effects of omeprazole or famotidine on leukocyte/endothelial cell interactions in vivo were analyzed in rat mesenteric venules using intrav…

Malemedicine.medical_specialtyProglumidemedicine.drug_classInflammationCD18Cell CommunicationProinflammatory cytokineRats Sprague-DawleyInternal medicineGastrinsmedicineLeukocytesAnimalsOmeprazoleGastrinPharmacologyInflammationMicroscopy Videobusiness.industryEndothelial CellsProton Pump InhibitorsReceptor antagonistAnti-Ulcer AgentsFlow CytometryImmunohistochemistryReceptor Cholecystokinin BRatsFamotidineChemotaxis LeukocyteEndocrinologyGastric MucosaMolecular Medicinemedicine.symptombusinessmedicine.drugThe Journal of pharmacology and experimental therapeutics
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Cholecystokinin-8 sulfate modulates the anticonvulsant efficacy of vigabatrin in an experimental model of partial complex epilepsy in the rat.

2009

Summary Purpose:  We evaluated the possible additive effect induced by the administration of the anticonvulsant vigabatrin (VGB) and cholecystokinin-8 sulfate (CCK-8S) on an experimental model of partial complex seizures (maximal dentate gyrus activation, MDA). Moreover, the functional involvement of γ-aminobutyric acid (GABA) neurotransmission was tested by iontophoretically administering bicuculline (GABA receptor antagonist) in the dentate gyrus. Methods:  Urethane anesthetized rats were pretreated with VGB (50, 100 or 200 mg/kg, i.p.) or CCK-8S (8 nmol/kg, i.p.) alone or coadministered with VGB (50 mg/kg, i.p.). Dentate gyrus epileptic activity was obtained through the repetitive electr…

Malemedicine.medical_treatmentStimulationConvulsantsNeurotransmissionPharmacologyBicucullineRat Partial epilepsy Vigabatrin Cholecystokinin-8 sulfate ControlVigabatrinDrug Administration ScheduleSincalideVigabatrinEpilepsy Complex PartialmedicineReaction TimeAnimalsRats WistarEvoked PotentialsNootropic AgentsAnalysis of VarianceIontophoresisDose-Response Relationship DrugChemistryDentate gyrusDrug SynergismBicucullineGABA receptor antagonistElectric StimulationRatsDisease Models AnimalAnticonvulsantNeurologyAnesthesiaDentate GyrusAnticonvulsantsDrug Therapy CombinationNeurology (clinical)medicine.drugEpilepsia
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