Search results for "Cholesterol"

showing 10 items of 1211 documents

Association of C677T polymorphism in MTHFR gene, high homocysteine and low HDL cholesterol plasma values in heterozygous familial hypercholesterolemi…

2010

Aim: to investigate the association of C677T polymorphism in the methylene tetrahydrofolate reductase (MTHFR) gene, homocysteine plasma values (Hcy), and plasma HDL cholesterol in heterozy-gous familial hypercholesterolemia (hFH).Methods: One hundred and twenty-five hFH subjects were studied. Plasma lipid, lipoprotein, vitamin B12, folic acid and Hcy values were determined. C677T polymorphism in the MTHFR gene was detected by SSCP-PCR. Genetic diagnosis of FH was determined by a three-step protocol using SSCP-PCR, Southern blot, long PCR and automatic sequencing.Results: We found significant differences in plasma HDL-C (CC 1.39±0.34, CT 1.33±0.39 and TT 1.14±0.26 mmol/L, p=0.028) between th…

AdultMalemedicine.medical_specialtyHeterozygoteApolipoprotein BHomocysteineHypercholesterolemiaFamilial hypercholesterolemiaPolymerase Chain Reactionchemistry.chemical_compoundInternal medicineGenotypeInternal MedicinemedicineHumansVitamin B12HomocysteineMethylenetetrahydrofolate Reductase (NADPH2)Polymorphism Single-Stranded ConformationalApolipoproteins BGeneticsPolymorphism GeneticbiologyCholesterolbusiness.industryBiochemistry (medical)Cholesterol HDLMiddle Agedmedicine.diseaseEndocrinologychemistryReceptors LDLMethylenetetrahydrofolate reductaseMutationbiology.proteinFemaleCardiology and Cardiovascular MedicinebusinessLipoproteinJournal of atherosclerosis and thrombosis
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Familial HDL deficiency due to ABCA1 gene mutations with or without other genetic lipoprotein disorders

2004

Mutations in ABCA1 have been shown to be the cause of Tangier disease (TD) and some forms of familial hypoalphalipoproteinemia (HA), two genetic disorders characterized by low plasma HDL levels. Here we report six subjects with low HDL, carrying seven ABCA1 mutations, six of which are previously unreported. Two mutations (R557X and H160FsX173) were predicted to generate short truncated proteins; two mutations (E284K and Y482C) were located in the first extracellular loop and two (R1901S and Q2196H) in the C-terminal cytoplasmic domain of ABCA1. Two subjects found to be compound heterozygotes for ABCA1 mutations did not have overt clinical manifestations of TD. Three subjects, all with prema…

AdultMalemedicine.medical_specialtyHeterozygoteSettore MED/09 - Medicina InternaApolipoprotein BAdolescentPremature coronary artery diseaseTangier diseaseCoronary DiseaseBiologyGene mutationmedicine.disease_causeCompound heterozygosityTangier diseaseInternal medicineGenotypeABCA1 genemedicineHumansChildHypoalphalipoproteinemiaSelection BiasAgedApolipoproteins BGeneticsMutationFamilial defective Apo B (FDB)Apolipoprotein A-ICholesterol HDLnutritional and metabolic diseasesMiddle Agedmedicine.diseaseLipoprotein lipaseTangier disease; Familial HDL deficiency; ABCA1 gene; Familial defective Apo B (FDB); Lipoprotein lipase; Premature coronary artery diseaseEndocrinologyChild PreschoolMutationbiology.proteinlipids (amino acids peptides and proteins)Allelic heterogeneityATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineFamilial HDL deficiencyATP Binding Cassette Transporter 1
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Plasma PCSK9 is a late biomarker of severity in patients with severe trauma injury.

2013

PCSK9 (proprotein convertase subtilisin kexin type 9) is a secreted protease that modulates cholesterol homeostasis by decreasing low-density lipoprotein receptor expression. Low levels of plasma lipoproteins are related to severity of illness and survival in patients of intensive care units (ICU).The aim of the study was to investigate the regulation of plasma PCSK9 and its association with plasma lipid parameters and clinical markers of severity during critical illness.The plasma biobank from the previously published HYPOLYTE prospective study was used to measure PCSK9 concentrations by ELISA at days 0 and 8 in 111 patients admitted to surgical ICU for severe multiple trauma. Patients wer…

AdultMalemedicine.medical_specialtyHydrocortisoneEndocrinology Diabetes and MetabolismReceptor expressionCritical IllnessClinical BiochemistryContext (language use)PlaceboBiochemistryGastroenterologySeverity of Illness IndexPlacebosEndocrinologyIntensive careInternal medicineSeverity of illnessmedicineHumansProspective cohort studyInfusion PumpsHydrocortisoneTrauma Severity Indicesbusiness.industryPCSK9Biochemistry (medical)Serine EndopeptidasesCholesterol LDLPrognosisIntensive Care UnitsEndocrinologyImmunologyWounds and InjuriesFemaleProprotein ConvertasesProprotein Convertase 9businessBiomarkersmedicine.drugThe Journal of clinical endocrinology and metabolism
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The supracondylar approach to the jugular tubercle and hypoglossal canal.

1998

BACKGROUND Circumscribed lesions of the hypoglossal canal and of the jugular tubercle still remain a surgical challenge. So far, transpetrosal, transcondylar suboccipital, and extreme lateral approaches have been used to access this region. These surgical procedures bear a high risk for neurological deficits. Therefore, we introduce a new minimally invasive extradural approach to the hypoglossal canal that also allows access to the lateral aspects of the jugular tubercle. METHODS After a paramedian retromastoid skin incision, a basal suboccipital craniectomy lateral to the foramen magnum toward the jugular tubercle is performed. With this approach the occipital condyle and the lateral osseo…

AdultMalemedicine.medical_specialtyHypoglossal NerveDura materHypoglossal canalNeurosurgical ProceduresJugular tuberclemedicineCranial nerve diseaseBone CystsHumansCranial Nerve NeoplasmsForamen magnumGranulomabusiness.industrySkullAnatomyMiddle AgedOccipital condyleSurgerymedicine.anatomical_structureCholesterolOccipital Bonecardiovascular systemSurgeryNeurology (clinical)Dura Matermedicine.symptomBone DiseasesbusinessHypoglossal nerveJugular foramenNeurilemmomaPetrous BoneSurgical neurology
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Increased expression of markers of early atherosclerosis in patients with inflammatory bowel disease.

2016

Recent studies documented an increased cardiovascular risk in patients with inflammatory bowel disease (IBD). Our study aimed at investigating the prevalence of intima-media thickness (IMT) of the carotid arteries and the arterial stiffness indices as markers of early atherosclerosis in young IBD patients.We recruited 68 consecutive IBD patients, and 38 matched healthy controls less than 45years old (median age 31.6±8.1years). Clinical and demographic features, cardiovascular risk factors, history of cardiovascular events, concomitant therapies were registered on a dedicate database. Carotid IMT was evaluated by using high resolution B-mode ultrasonography. Arterial stiffness was assessed b…

AdultMalemedicine.medical_specialtyIBD030204 cardiovascular system & hematologyPulse Wave AnalysisInflammatory bowel diseaseCarotid Intima-Media ThicknessCarotid intima-media thickne03 medical and health sciencesYoung Adult0302 clinical medicineVascular StiffnessCrohn DiseaseRisk FactorsInternal medicineInternal MedicinemedicineHumansMesalaminePulse wave velocityTriglyceridesSubclinical infectionCrohn's diseasebiologybusiness.industryC-reactive proteinAnti-Inflammatory Agents Non-SteroidalCholesterol HDLCholesterol LDLmedicine.diseaseAtherosclerosisInflammatory Bowel DiseasesArterial stiffneBlood pressureCarotid ArteriesCholesterolIntima-media thicknessCase-Control StudiesImmunologyEarly atherosclerosiArterial stiffnessCardiologybiology.protein030211 gastroenterology & hepatologyColitis UlcerativeFemalebusinessBiomarkersEuropean journal of internal medicine
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In the Identification of Cardiovascular Risk With the SCORE Model, Could We Recommend Its Calculation Interchangeably With Total Cholesterol or Ather…

2011

The SCORE table indiscriminately recommends the use of total cholesterol (SCORE-TC) or atherogenic index (SCORE-AI) for calculating cardiovascular (CV) risk. We evaluated reliability and agreement between both methods and the clinical implications for the identification of high CV risk. Observational study (n = 8942) in a 40- to 65-year-old population. Spearman’s Rho correlation was 0.987 (P < .001), the agreement intraclass correlation coefficient was 0.671 (IC 95% 0.413–0.796; with Bland–Altman’s method, the average of the differences between models was 0.74. Kappa index was poor, 0.297 (P < .001) and positive specific agreement was 0.31. Discrepancies fitted individuals with high CV risk…

AdultMalemedicine.medical_specialtyIndex (economics)Intraclass correlationConcordancePopulationRisk AssessmentKappa indexCorrelationInternal medicineTotal cholesterolmedicineHumanseducationAgededucation.field_of_studyModels Statisticalbusiness.industryGeneral MedicineMiddle AgedAtherosclerosisLipidsCholesterolCardiovascular DiseasesSpainFemaleObservational studybusinessRevista Española de Cardiología (English Edition)
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Alterations in lipid, carbohydrate and iron metabolism in patients with non-alcoholic steatohepatitis (NASH) and metabolic syndrome

2010

NASH (non-alcoholic steatohepatitis) is considered the hepatic manifestation of the metabolic syndrome (MS). We aimed to analyze lipid, carbohydrate, and iron metabolism in NASH.37 patients with MS (17 M/20 F, 51+/-15 years), elevated transaminases; 25 patients had histologically proven NASH (NAS score≥5), 12 patients had toxic background (nonNASH). 37 age, sex, BMI-matched healthy controls. Lipid variables, LDL-subfractions, iron, ferritin, transferrin (T), transferrin saturation (TS), and hepcidin (H) were measured in patients/controls. Oral glucose tolerance tests were performed.NASH patients with steatosis gr. 2 and 3 (33% hepatic fat) had higher sd-LDL (mg/dl) concentrations than patie…

AdultMalemedicine.medical_specialtyIronInsulin resistanceHepcidinsNon-alcoholic Fatty Liver DiseaseInternal medicineInternal MedicinemedicineHumansInsulinAgedMetabolic Syndromechemistry.chemical_classificationGlucose tolerance testC-Peptidemedicine.diagnostic_testTransferrin saturationbusiness.industryTransferrinnutritional and metabolic diseasesLipid metabolismCholesterol LDLGlucose Tolerance TestMiddle AgedLipid Metabolismmedicine.diseasedigestive system diseasesFatty LiverEndocrinologychemistryTransferrinFerritinsMetabolomeCarbohydrate MetabolismFemaleMetabolic syndromeSteatosisSteatohepatitisbusinessAntimicrobial Cationic PeptidesEuropean Journal of Internal Medicine
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Oxidative stress and antioxidant enzyme values in lymphomonocytes after an oral unsaturated fat load test in familial hypercholesterolemic subjects

2012

Oxidative stress (OS) has been observed in conditions affecting the cardiovascular system. Familial hypercholesterolemia (FH) is associated with an increased risk of premature coronary heart disease. In the postprandial state, circulating lipids and lipoproteins can modulate OS status. Our aim was to study the response of lymphomonocyte OS status and reactive oxygen species by-products after an oral unsaturated fat load test (OFLT) in those with FH and to compare this response with that obtained in normolipidemic, normoglycemic subjects. We studied 12 patients with FH and 20 healthy controls. In both groups, lymphomonocyte, oxidized/reduced glutathione ratio, and malondialdehyde were determ…

AdultMalemedicine.medical_specialtyIsoprostaneAdolescentFamilial hypercholesterolemiamedicine.disease_causeAntioxidantsMonocytesHyperlipoproteinemia Type IIchemistry.chemical_compoundMalondialdehydePhysiology (medical)Internal medicinemedicineHumansLymphocytesAgedchemistry.chemical_classificationGlutathione Disulfidebusiness.industryGlutathione peroxidaseBiochemistry (medical)Unsaturated fatPublic Health Environmental and Occupational HealthArea under the curveGeneral MedicineMiddle AgedPostprandial PeriodMalondialdehydemedicine.diseaseGlutathioneFats UnsaturatedOxidative StressEndocrinologyPostprandialchemistryFemalebusinessOxidative stressTranslational Research
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Association of selected ABC gene family single nucleotide polymorphisms with postprandial lipoproteins: results from the population-based Hortega stu…

2009

The aim of the study was to determine the influence of twenty single nucleotide polymorphisms (SNPs) of the ABCA1, ABCG1, ABCG5 and ABCG8 genes on the plasmatic concentrations of total cholesterol (TC), HDL and LDL cholesterol (HDLc, LDLc) in the postprandial state with a representative Spanish Caucasian population (1473 individuals, 50.0% women, ages ranging 21-85 years). In men, subjects with the AA genotype of the ABCA1 rs2230806 (R219K) polymorphism were associated with increased plasma LDLc levels, while the ABCA1 haplotype, which included the rs2230806 A allele, was associated with higher TC and LDLc plasma concentrations. In women, significant relationships were found between rs18935…

AdultMalemedicine.medical_specialtyLipoproteinsBlood lipidsSingle-nucleotide polymorphismHyperlipidemiasBiologyPolymorphism Single Nucleotidechemistry.chemical_compoundHigh-density lipoproteinPolymorphism (computer science)Internal medicineGenotypemedicineHumansATP Binding Cassette Transporter Subfamily G Member 5AllelesATP Binding Cassette Transporter Subfamily G Member 1AgedGeneticsAged 80 and overCholesterolHaplotypeATP Binding Cassette Transporter Subfamily G Member 8Cholesterol HDLMiddle AgedAtherosclerosisPostprandial PeriodPostprandialEndocrinologyCholesterolchemistryHaplotypesSpainlipids (amino acids peptides and proteins)ATP-Binding Cassette TransportersFemaleCardiology and Cardiovascular MedicineATP Binding Cassette Transporter 1Atherosclerosis
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Resistance Training Induces Antiatherogenic Effects on Metabolomic Pathways

2019

INTRODUCTION Arising evidence suggests that resistance training has the potential to induce beneficial modulation of biomarker profile. To date, however, only immediate responses to resistance training have been investigated using high-throughput metabolomics whereas the effects of chronic resistance training on biomarker profile have not been studied in detail. METHODS A total of 86 recreationally active healthy men without previous systematic resistance training background were allocated into (i) a resistance training (RT) group (n = 68; age, 33 ± 7 yr; body mass index, 28 ± 3 kg·m) and (ii) a non-RT group (n = 18; age, 31 ± 4 yr; body mass index, 27 ± 3 kg·m). Blood samples were collecte…

AdultMalemedicine.medical_specialtyMagnetic Resonance SpectroscopyWeight LiftingApolipoprotein BLipoproteinsPhysical Therapy Sports Therapy and Rehabilitation030204 cardiovascular system & hematologyBody Mass Index03 medical and health scienceschemistry.chemical_compound0302 clinical medicineRisk FactorsInternal medicinemedicineMetabolomeHumansMetabolomicsOrthopedics and Sports MedicineLongitudinal StudiesMuscle StrengthAmino Acids2. Zero hungerbiologymedicine.diagnostic_testCholesterolbusiness.industryFatty AcidsResistance TrainingCholesterol LDL030229 sport sciencesAtherosclerosisLipidsDiet3. Good healthCholesterolEndocrinologychemistryBody CompositionLean body massbiology.proteinBiomarker (medicine)businessLipid profileBody mass indexBiomarkersLipoproteinMedicine &amp; Science in Sports &amp; Exercise
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