Search results for "Chromaffin cell"

showing 10 items of 27 documents

Evidence that neuronally released vasoactive intestinal polypeptide inhibits the release of serotonin from enterochromaffin cells of the guinea pig s…

1991

Abstract. Isolated small intestinal segments of the guinea pig were arterially perfused and the release of serotonin (5-hydroxytryptamine) and 5-hydroxyindoleacetic acid into the portal venous effluent was determined by HPLC with electrochemical detection. Test substances were intra-arterially applied. The muscarine receptor agonist oxotremorine (1 μmol/l inhibited the release of 5-hydroxytryptamine by about 50%. In the presence of the neurotoxin tetrodotoxin, oxotremorine enhanced the release of 5-hydroxytryptamine by 145%, indicating that the inhibitory effect of oxotremorine was mediated by the release of a neurotransmitter. Exogenous vasoactive intestinal polypeptide ( 1-100 pmol/l inhi…

MaleSerotoninmedicine.medical_specialtyEndocrinology Diabetes and MetabolismGuinea PigsVasoactive intestinal peptideTetrodotoxinBiologyAntibodiesGuinea pigchemistry.chemical_compoundEndocrinologyInternal medicineIntestine SmallEnterochromaffin CellsOxotremorinemedicineAnimalsNeurotransmitterChromatography High Pressure LiquidNeuronsMuscarineOxotremorineGeneral MedicineHydroxyindoleacetic AcidSmall intestineKineticsmedicine.anatomical_structureEndocrinologychemistryEnterochromaffin cellFemaleSerotoninVasoactive Intestinal Peptidemedicine.drugActa Endocrinologica
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Temperature-dependent effects of increased intraluminal pressure on serotonin release from the vascularly perfused guinea pig ileum

1987

Isolated segments of the guinea pig ileum were vascularly perfused and the release of endogenous serotonin into the portal effluent was measured. Peristalsis was induced by raising the intraluminal hydrostatic pressure by 500 Pa for 5 min. Serotonin release increased during peristalsis induced by fluid of 37 degrees C, but decreased when the temperature of the intraluminal fluid was between 13 degrees C and 22 degrees C. In the presence of naloxone (0.3 mumol/l) raising the intraluminal pressure with fluid of 37 degrees C caused an inhibition of the serotonin release which was blocked by scopolamine (0.1 mumol/l). Naloxone did not affect the inhibition of serotonin release during peristalsi…

MaleSerotoninmedicine.medical_specialtyGuinea PigsIndomethacinScopolamineHydrostatic pressureIleumIn Vitro TechniquesBiologyGuinea pigIndometacinIleumInternal medicinePressuremedicineAnimalsPeristalsisPharmacologyNaloxoneTemperatureGeneral MedicineHydroxyindoleacetic AcidSmall intestinePerfusionEndocrinologymedicine.anatomical_structureEnterochromaffin cellPeristalsisSerotoninmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Adrenergic modulation of the release of 5-hydroxytryptamine from the vascularly perfused ileum of the guinea-pig

1988

1. Isolated segments of the guinea-pig ileum were vascularly perfused and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent was determined by h.p.l.c. with electrochemical detection. Test substances were applied via the arterial perfusion medium. 2. Isoprenaline (0.1 microM) increased the outflow of 5-HT and 5-HIAA maximally by about 75% and this was antagonized by propranolol (0.1 microM). Forskolin (1-10 microM) increased the outflow of 5-HT by approximately 105% and that of 5-HIAA by approximately 55%. The phosphodiesterase inhibitor AH 21-132 (0.1-1 microM) increased the outflow of 5-HT and 5-HIAA by about 70%. Isoprenaline…

MaleSerotoninmedicine.medical_specialtyPhosphodiesterase InhibitorsAdrenergic beta-AntagonistsGuinea PigsPropranololClonidinechemistry.chemical_compoundIleumIsoprenalineInternal medicinemedicinePrazosinAnimalsNaphthyridinesPhosphodiesterase inhibitorAdrenergic alpha-AntagonistsPharmacologyForskolinColforsinIsoproterenolPhosphodiesteraseAdrenergic beta-AgonistsHydroxyindoleacetic AcidPerfusionEndocrinologychemistryTetrodotoxinEnterochromaffin cellAdrenergic alpha-AgonistsResearch Articlemedicine.drugBritish Journal of Pharmacology
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Cisplatin increases the release of 5-hydroxytryptamine (5-HT) from the isolated vascularly perfused small intestine of the guinea-pig: Involvement of…

1991

Isolated segments of the guinea-pig small intestine were vascularly perfused and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent determined by high pressure liquid chromatography with electrochemical detection. Release of acetylcholine from isolated superfused intestinal segments was determined as outflow of [3H]radioactivity from preparations preincubated with [3H]choline. Cisplatin (3 microM) increased the outflow of 5-HT and 5-HIAA by about 90%. At 30 and 100 microM cisplatin decreased the outflow of 5-HT and its metabolite by 40%-50%. The stimulatory effect of cisplatin was consistently observed only when the bicarbonate-…

MaleSerotoninmedicine.medical_specialtymedicine.drug_classMetaboliteGuinea PigsTetrodotoxinIn Vitro Techniqueschemistry.chemical_compoundInternal medicineIntestine SmallEnterochromaffin CellsmedicineAnimalsReceptor5-HT receptorPharmacologyCisplatinDose-Response Relationship DrugImidazolesGeneral MedicineHydroxyindoleacetic AcidReceptor antagonistOndansetronAcetylcholineSmall intestinePerfusionEndocrinologymedicine.anatomical_structurechemistryReceptors SerotoninFemaleHexamethoniumCisplatinAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Cholinergic modulation of the release of 5-hydroxytryptamine from the guinea pig ileum.

1987

Isolated segments of the guinea pig ileum were vascularly perfused and the release of 5-HT and its metabolite 5-HIAA into the portal venous effluent determined by HPLC with electrochemical detection. Test substances were applied via the arterial perfusion medium. Oxotremorine inhibited concentration-dependently the release of 5-HT and 5-HIAA (by 47% at 1 mumol/l). Scopolamine (0.1 mumol/l) did not affect the release of 5-HT and 5-HIAA, but antagonized the effect of oxotremorine. In the presence of TTX (1 mumol/l), oxotremorine (1 mumol/l) increased the release of 5-HT by 150% and that of 5-HIAA by 220%. This increase was completely blocked by scopolamine. Hexamethonium (100 mumol/l) and TTX…

Malemedicine.medical_specialtySerotoninMetaboliteGuinea PigsScopolamineHexamethonium CompoundsBiologyIn Vitro TechniquesReceptors NicotinicHexamethoniumGuinea pigchemistry.chemical_compoundIleumInternal medicineMuscarinic acetylcholine receptorOxotremorinemedicineEnterochromaffin CellsAnimalsReceptors CholinergicIntestinal MucosaPharmacologyMuscarineOxotremorineGeneral MedicineEndocrinologynervous systemchemistryEnterochromaffin cellHexamethoniumSerotoninmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Effect of vasoactive intestinal polypeptide on the release of serotonin from the in vitro vascularly perfused small intestine of guinea pig.

1989

Isolated segments of the guinea pig small intestine were vascularly perfused and the release of endogenous serotonin (5-HT) and its metabolite 5-hydroxyindoleacetic acid (5-HIAA) into the portal vein was measured. All test substances were intraarterially perfused. Vasoactive intestinal polypeptide (VIP, 1 pmol/l-100 nmol/l) inhibited the spontaneous release of 5-HT and 5-HIAA. The maximal inhibitory effect (about 60%) was seen at 100 pmol/l. The effect of VIP on the spontaneous release of 5-HT and 5-HIAA was not changed in the presence of 1 mumol/l tetrodotoxin (TTX). Raising intraluminal pressure by 500 Pa for 5 min increased the release of 5-HT and 5-HIAA by about 25%. Raising the intralu…

Malemedicine.medical_specialtySerotoninMetaboliteVasoactive intestinal peptideGuinea PigsTetrodotoxinBiologyIn Vitro TechniquesGuinea pigchemistry.chemical_compoundInternal medicineIntestine SmallmedicineAnimalsPharmacologyMuscle SmoothGeneral MedicineHydroxyindoleacetic AcidSmall intestineEndocrinologymedicine.anatomical_structurenervous systemGastrointestinal hormonechemistryEnterochromaffin cellTetrodotoxinSerotoninhormones hormone substitutes and hormone antagonistsMuscle ContractionVasoactive Intestinal PeptideNaunyn-Schmiedeberg's archives of pharmacology
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Identification of SNARE complex modulators that inhibit exocytosis from an alpha-helix-constrained combinatorial library.

2003

Synthetic peptides patterned after the proteins involved in vesicle fusion [the so-called SNARE (soluble N-ethylmaleimide-sensitive fusion protein attachment protein receptor) proteins] are potent inhibitors of SNARE complex assembly and neuronal exocytosis. It is noteworthy that the identification of peptide sequences not related to the SNARE proteins has not been accomplished yet; this is due, in part, to the structural constraints and the specificity of the protein interactions that govern the formation of the SNARE complex. Here we have addressed this question and used a combinatorial approach to identify peptides that modulate the assembly of the SNARE core complex and inhibit neuronal…

Models MolecularVesicle fusionMacromolecular SubstancesChromaffin CellsMolecular Sequence DataVesicular Transport ProteinsBiologyBiochemistryExocytosisExocytosisProtein Structure SecondaryPeptide LibraryAnimalsAmino Acid SequencePeptide libraryMolecular BiologyCells CulturedSNARE complex assemblyNeuronsSTX1AMembrane ProteinsMunc-18Cell BiologyFusion proteinCell biologyRatsCattleSNARE complexPeptidesSNARE ProteinsResearch ArticleThe Biochemical journal
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Concomitant Small Cell Neuroendocrine Carcinoma of Gallbladder and Breast Cancer

2014

The neuroendocrine carcinoma is defined as a high-grade malignant neuroendocrine neoplasm arising from enterochromaffin cells, usually disposed in the mucosa of gastric and respiratory tracts. The localization in the gallbladder is rare. Knowledge of these gallbladder tumors is limited and based on isolated case reports. We describe a case of an incidental finding of small cell neuroendocrine carcinoma of the gallbladder, observed after cholecystectomy for cholelithiasis, in a 55-year-old female, who already underwent quadrantectomy and sentinel lymph-node biopsy for breast cancer. The patient underwent radiotherapy for breast cancer and six cycles of chemotherapy with cisplatin and etoposi…

Pathologymedicine.medical_specialtybusiness.industryGallbladdermedicine.medical_treatmentlcsh:SurgeryCase Reportlcsh:RD1-811Settore MED/08 - Anatomia PatologicaMalignancymedicine.diseaseRadiation therapySettore MED/18 - Chirurgia Generalemedicine.anatomical_structureBreast cancerNeuroendocrine carcinomamedicineEnterochromaffin cellPharmacology (medical)CholecystectomyNeoplastic transformationbusinessQuadrantectomy
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Production of biologically active light chain of tetanus toxin inEscherichia coli

1993

AbstractThe activity of the light (L) chain of tetanus toxin, and of mutants constructed by site-directed mutagenesis, was studied by expression and purification of the proteins from E. coli. Wild-type recombinant L chain (pTet87) was active in the inhibition of exocytosis from cultured bovine adrenal chromaffin cells, although at a level 5–15% of that of L chain purified from tetanus toxin. L chain mutants which terminated at Leu-438 (pTet89), or which contained a Cys-to-Ser mutation at residue 439 (pTet88) were equally as active as the full-length recombinant protein. The reduced activity of pTet87 L chain correlated with C-terminal proteolysis of the protein upon purification. A tryptic …

Recombinant proteinMacromolecular SubstancesProteolysisMolecular Sequence DataRestriction MappingDNA RecombinantBiophysicsBiologymedicine.disease_causeImmunoglobulin light chainBiochemistryExocytosislaw.inventionNorepinephrineTetanus ToxinStructural BiologylawEscherichia coliGeneticsmedicineAnimalsAmino Acid SequenceCloning MolecularSite-directed mutagenesisMolecular BiologyEscherichia coliCells Culturedchemistry.chemical_classificationBase Sequencemedicine.diagnostic_testToxinBiological activityCell BiologyMolecular biologyRecombinant ProteinsE. coli Chromaffin cellAmino acidKineticsOligodeoxyribonucleotideschemistryBiochemistryAdrenal MedullaMutagenesis Site-DirectedRecombinant DNACalciumCattleElectrophoresis Polyacrylamide GelSite directed mutagenesisFEBS Letters
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Rotavirus stimulates release of serotonin (5-HT) from human enterochromaffin cells and activates brain structures involved in nausea and vomiting

2011

Rotavirus (RV) is the major cause of severe gastroenteritis in young children. A virus-encoded enterotoxin, NSP4 is proposed to play a major role in causing RV diarrhoea but how RV can induce emesis, a hallmark of the illness, remains unresolved. In this study we have addressed the hypothesis that RV-induced secretion of serotonin (5-hydroxytryptamine, 5-HT) by enterochromaffin (EC) cells plays a key role in the emetic reflex during RV infection resulting in activation of vagal afferent nerves connected to nucleus of the solitary tract (NTS) and area postrema in the brain stem, structures associated with nausea and vomiting. Our experiments revealed that RV can infect and replicate in human…

RotavirusViral DiseasesViral Nonstructural ProteinsMiceChildlcsh:QH301-705.5Mice Inbred BALB CArea postremaBrainNauseaVagus NerveJejunumInfectious DiseasesMEDICINChild PreschoolEnterochromaffin cellVomitingMedicineSerotonin Antagonistsmedicine.symptomProto-Oncogene Proteins c-fosResearch Articlelcsh:Immunologic diseases. Allergymedicine.medical_specialtySerotoninVomitingImmunologyBiologyMicrobiologyRotavirus InfectionsSDG 3 - Good Health and Well-beingInternal medicineCell Line TumorVirologyGeneticsmedicineEnterochromaffin CellsAnimalsHumansBiologyMolecular BiologyGlycoproteinsToxins BiologicalMEDICINEVagus nerveEndocrinologyGene Expression Regulationlcsh:Biology (General)Cell cultureParasitologyEnteric nervous systemCalciumSerotoninlcsh:RC581-607Ex vivo
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