Search results for "Chromaffin cell"

showing 7 items of 27 documents

Functional coupling of nitric oxide synthase and soluble guanylyl cyclase in controlling catecholamine secretion from bovine chromaffin cells

1997

This study was designed to evaluate whether the enzymes of the nitric oxide/cyclic-GMP pathway, nitric oxide synthase and soluble guanylyl cyclase, are functionally coupled in controlling catecholamine secretion in primary cultures of bovine chromaffin cells. In immunocytochemical studies, 80-85% of the tyrosine hydroxylase-positive chromaffin cells also possessed phenylethanolamine-N-methyltransferase, f1p4cating their capability to synthesize epinephrine. Immunoreactivity for neuronal-type nitric oxide synthase was found in over 90% of all chromaffin cells. Reverse transcription-polymerase chain reaction also demonstrated neuronal-type nitric oxide synthase messenger RNA. Immunoreactivity…

Tyrosine 3-MonooxygenaseChromaffin CellsPolymerase Chain ReactionNitric oxidechemistry.chemical_compoundCatecholaminesCytosolAdrenal GlandsmedicineAnimalsRNA MessengerCyclic GMPbiologyChemistryPhenylethanolamine N-MethyltransferaseGeneral NeuroscienceNitric oxide synthasemedicine.anatomical_structureBiochemistryGuanylate CyclaseChromaffin cellCatecholaminebiology.proteinCalciumCattleSodium nitroprussideNitric Oxide SynthaseAdrenal medullaSoluble guanylyl cyclaseAcetylcholinemedicine.drugNeuroscience
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Phosphatidylinositol phosphate kinase type I gamma regulates dynamics of large dense-core vesicle fusion.

2005

Phosphatidylinositol-4,5-bisphosphate was proposed to be an important regulator of large dense-core vesicle exocytosis from neuroendocrine tissues. Here, we have examined the kinetics of secretion in chromaffin cells from mice lacking phosphatidylinositol phosphate kinase type Iγ, the major neuronal phosphatidylinositol-4-phosphate 5-kinase. Absence of this enzyme caused a reduction of the readily releasable vesicle pool and its refilling rate, with a small increase in morphologically docked vesicles, indicating a defect in vesicle priming. Furthermore, amperometry revealed a delay in fusion pore expansion. These results provide direct genetic evidence for a key role of phosphatidylinositol…

Vesicle fusionChromaffin CellsBiologyIn Vitro TechniquesMembrane FusionExocytosisExocytosischemistry.chemical_compoundMiceAnimalsPhosphatidylinositolCells CulturedMultidisciplinaryVesicleSecretory VesiclesSNAP25Munc-18Kiss-and-run fusionBiological SciencesSecretory VesicleCell biologyKineticsMicroscopy ElectronPhosphotransferases (Alcohol Group Acceptor)chemistryCalciumProceedings of the National Academy of Sciences of the United States of America
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Activation and Inactivation of Tetanus Toxin in Chromaffin Cells

1993

Tetanus toxin is produced by Clostridium tetani as a single chain, almost non-toxic, protein with a molecular weight of approximately 150.000 representing 1315 amino acids. Bacterial proteases cleave the molecule between positions A 457 and S 458 (extracellular activation), yielding a heavy chain (MW 100.000) and a light chain (MW 50.000) tetanus toxin (HC-TeTx, LC-TeTx). Both chains remain connected to each other by a disulphur bond between positions C 439 and C 467 (Dichain-TeTx)1. The cleavage or nicking dramatically increases the biological activity2. HC-TeTx is involved in binding DC-TeTx to gangliosides lodged in the plasma membrane, which is a prerequisite for incorporation into the …

education.field_of_studyClostridium tetaniChemistryPopulationEndocytosismedicine.disease_causeExocytosisCytosolmedicine.anatomical_structureChromaffin cellmedicineExtracellularBiophysicseducationIntracellular
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Regulation of serotonin release from the intestinal mucosa.

1991

Summary In the mammalian intestine serotonin (5-hydroxytryptamine, 5-HT) is present in high concentrations in the enterochromaffin cells. The release of 5-HT from the intestinal mucosa is regulated by a complex pattern of neuronal and humoral inputs to the enterochromaffin cells. The enterochromaffin cells appear to be endowed with different inhibitory ( α 2 -adrenoceptors, GABA A - and GABA B -receptors, histamine H 3 -receptors, receptors for vasoactive intestinal polypeptide and somatostatin) as well as stimulatory receptors ( β -adrenoceptors, muscarine and nicotine receptors). The physiological significance of this complex system of receptors is suggested by experiments which demonstra…

endocrine systemmedicine.medical_specialtySerotoninVasoactive intestinal peptideBiology03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIntestinal mucosaInternal medicinemedicineAnimalsHumansIntestinal MucosaReceptor5-HT receptor030304 developmental biologyPharmacology0303 health sciencesMuscarine3. Good healthEndocrinologychemistryEnterochromaffin cellSerotonin030217 neurology & neurosurgeryAcetylcholinemedicine.drugPharmacological research
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Short- and long-term effects of vinblastine on the rat adrenal medulla

1979

The effects of a single high dose (10mg/kg) of vinblastine (vb) sulfate (“Velbe”, Lilly) on the ultrastructure, catecholamine (CA) content and activity of CA-synthesizing enzymes of the rat adrenal medulla were studied for up to 120h after intravenous injection of the drug.

medicine.medical_specialtyTime FactorsHistologyChemical PhenomenaVinblastinePathology and Forensic MedicineInternal medicineSingle high doseEnterochromaffin CellsmedicineAnimalsCells Culturedchemistry.chemical_classificationCell BiologyMolecular medicineRatsVinblastineChemistryEnzymemedicine.anatomical_structureEndocrinologychemistryAdrenal MedullaUltrastructureCatecholamineFemaleAdrenal medullamedicine.drugCell and Tissue Research
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OP0309 Intestinal sclerostin/serotonin axis is modulated by dysbiosis and regulates ilc3 expansion in as patients

2017

Background Sclerostin is an osteocyte-specific factor that binds to low-density lipoprotein receptor-related protein 5 (LRP5) inhibiting the Wnt signaling pathway and possibly contributing to the pathogenesis of Ankylosing spondylitis (AS). Subclinical gut inflammation observed in AS patients is characterized by the presence of dysbiosis and innate immune alterations. In the gut, LRP5 activation by unknown ligands inhibits serotonin production. Serotonin, by inducing glial derived neurotrophic factor (GDNF), controls ILC3 expansion, in the context of glial–ILC3–epithelial cell unit (GIECU). Sclerostin/serotonin axis has been never studied in AS. Objectives Aim of this study was to evaluate …

medicine.medical_specialtybiologybusiness.industryWnt signaling pathwayLRP5Context (language use)chemistry.chemical_compoundEndocrinologychemistryInternal medicinemedicineGlial cell line-derived neurotrophic factorbiology.proteinEnterochromaffin cellSclerostinSerotonin ProductionSerotoninbusinessOral Presentations
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Control of Gastric Acid Secretion in Somatostatin Receptor 2 Deficient Mice: Shift from Endocrine/Paracrine to Neurocrine Pathways

2007

The gastrin-enterochromaffin-like (ECL) cell-parietal cell axis is known to play an important role in the regulation of gastric acid secretion. Somatostatin, acting on somatostatin receptor type 2 (SSTR2), interferes with this axis by suppressing the activity of the gastrin cells, ECL cells, and parietal cells. Surprisingly, however, freely fed SSTR2 knockout mice seem to display normal circulating gastrin concentration and unchanged acid output. In the present study, we compared the control of acid secretion in these mutant mice with that in wild-type mice. In SSTR2 knockout mice, the number of gastrin cells was unchanged; whereas the numbers of somatostatin cells were reduced in the antru…

medicine.medical_specialtyendocrine systemCell CountGalaninBiologyHistidine DecarboxylaseArticleGastric AcidMiceEndocrinologyParietal Cells GastricInternal medicineParacrine CommunicationmedicineEnterochromaffin CellsSomatostatin receptor 2AnimalsReceptors SomatostatinEnterochromaffin-like cellGastrinMice KnockoutDelta cellSomatostatin receptorGastrin-Secreting Cellsdigestive oral and skin physiologyNeurosecretory SystemsMicroscopy ElectronEndocrinologySomatostatinGastric MucosaVesicular Monoamine Transport ProteinsG cellReceptors Galaninhormones hormone substitutes and hormone antagonists
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