Search results for "Chromatin Assembly and Disassembly"

showing 10 items of 36 documents

An essential switch in subunit composition of a chromatin remodeling complex during neural development.

2007

Summary Mammalian neural stem cells (NSCs) have the capacity to both self-renew and to generate all the neuronal and glial cell-types of the adult nervous system. Global chromatin changes accompany the transition from proliferating NSCs to committed neuronal lineages, but the mechanisms involved have been unclear. Using a proteomics approach, we show that a switch in subunit composition of neural, ATP-dependent SWI/SNF-like chromatin remodeling complexes accompanies this developmental transition. Proliferating neural stem and progenitor cells express complexes in which BAF45a, a Kruppel/PHD domain protein and the actin-related protein BAF53a are quantitatively associated with the SWI2/SNF2-…

Cellular differentiationProtein subunitNeuroscience(all)Molecular Sequence DataNeuroepithelial CellsDEVBIONerve Tissue ProteinsBiologyChromatin remodelingMOLNEUROEpigenesis Genetic03 medical and health sciencesMice0302 clinical medicineMultienzyme ComplexesAnimalsAmino Acid SequenceProgenitor cell030304 developmental biologyNeurons0303 health sciencesGeneral NeuroscienceMultipotent Stem CellsGene Expression Regulation DevelopmentalCell DifferentiationChromatin Assembly and DisassemblySTEMCELLNeural stem cellChromatinCell biologyNeuroepithelial cellProtein SubunitsNeural developmentNeuroglia030217 neurology & neurosurgeryTranscription FactorsNeuron
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Chromatin remodelling factor Mll1 is essential for neurogenesis from postnatal neural stem cells

2009

Epigenetic mechanisms that maintain neurogenesis throughout adult life remain poorly understood(1). Trithorax group (trxG) and Polycomb group (PcG) gene products are part of an evolutionarily conserved chromatin remodelling system that activate or silence gene expression, respectively(2). Although PcG member Bmi1 has been shown to be required for postnatal neural stem cell self-renewal(3,4), the role of trxG genes remains unknown. Here we show that the trxG member Mll1 (mixed-lineage leukaemia 1) is required for neurogenesis in the mouse postnatal brain. Mll1-deficient subventricular zone neural stem cells survive, proliferate and efficiently differentiate into glial lineages; however, neur…

Chromatin ImmunoprecipitationEpigenetic regulation of neurogenesisCell SurvivalNeurogenesisCellular differentiationSubventricular zoneNerve Tissue ProteinsBiologyMethylationArticleHistonesMiceBasic Helix-Loop-Helix Transcription FactorsmedicineAnimalsCell LineageCells CulturedCell ProliferationGliogenesisHomeodomain ProteinsNeuronsMultidisciplinaryStem CellsNeurogenesisCell DifferentiationHistone-Lysine N-MethyltransferaseOligodendrocyte Transcription Factor 2Chromatin Assembly and DisassemblyOlfactory BulbMolecular biologyChromatinNeural stem cellCell biologyChromatinmedicine.anatomical_structureAnimals NewbornStem cellNeurogliaMyeloid-Lymphoid Leukemia ProteinTranscription Factors
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Enhanced oxidative stress and increased mitochondrial mass during Efavirenz-induced apoptosis in human hepatic cells

2010

BACKGROUND AND PURPOSE Efavirenz (EFV) is widely used in the treatment of HIV-1 infection. Though highly efficient, there is growing concern about EFV-related side effects, the molecular basis of which remains elusive. EXPERIMENTAL APPROACH In vitro studies were performed to address the effect of clinically relevant concentrations of EFV (10, 25 and 50 mu M) on human hepatic cells. KEY RESULTS Cellular proliferation and viability were reduced in a concentration-dependent manner. Analyses of the cell cycle and several cell death parameters (chromatin condensation, phosphatidylserine exteriorization, mitochondrial proapoptotic protein translocation and caspase activation) revealed that EFV tr…

CyclopropanesMalehepatotoxicityCarcinoma HepatocellularTime FactorsAnti-HIV AgentsCell SurvivalApoptosisMitochondria LiverPhosphatidylserinesAntioxidantsSuperoxidesHumansChromansantiretroviral drugsCell Proliferationreactive oxygen speciesDose-Response Relationship DrugCell CycleLiver NeoplasmsChromatin Assembly and DisassemblyResearch PapersGlutathioneBenzoxazinesmitochondriaOxidative Stressside effectscell deathLiverAlkynesFemaleEfavirenzApoptosis Regulatory ProteinsHeLa Cells
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Transcription of genes in the biosynthetic pathway for fumonisin mycotoxins is epigenetically and differentially regulated in the fungal maize pathog…

2012

ABSTRACT When the fungal pathogen Gibberella moniliformis (anamorph, Fusarium verticillioides ) colonizes maize and maize-based products, it produces class B fumonisin (FB) mycotoxins, which are a significant threat to human and animal health. FB biosynthetic enzymes and accessory proteins are encoded by a set of clustered and cotranscribed genes collectively named FUM, whose molecular regulation is beginning to be unraveled by researchers. FB accumulation correlates with the amount of transcripts from the key FUM genes, FUM1 , FUM21 , and FUM8 . In fungi in general, gene expression is often partially controlled at the chromatin level in secondary metabolism; when this is the case, the deac…

DISRUPTIONTranscription GeneticFUM21[SDV]Life Sciences [q-bio]DIVERSITYPROTEINFusarium verticillioidesmaizeSECONDARY METABOLISMgene clusterEpigenesis GeneticHistonesFUM8FusariumGene Expression Regulation FungalASPERGILLUSPromoter Regions Genetic2. Zero hungerGenetics0303 health sciencesHistone deacetylase inhibitorhistone acetylationAcetylationArticlesGeneral MedicineChromatinChromatinGENOMEHistoneMultigene Family[SDE]Environmental SciencesTrichostatin AEpigenetics; Fusarium verticillioides; fmonisin synthesismedicine.drugCONIDIATIONChromatin Immunoprecipitationmedicine.drug_classGenes FungalChIPBiologyGFPZea maysMicrobiologyFumonisinsChromatin remodeling03 medical and health sciencesmedicineEpigeneticsMolecular Biology030304 developmental biologyepigenetics030306 microbiologyCLUSTERFumonisins; epigenetics; Fusarium verticillioides; maize; histone acetylation; histone deacetylases; ChIP; Trichostatin A; FUM1; FUM21; FUM8; GFP; gene clusterMycotoxinsChromatin Assembly and DisassemblyFUM1Histone Deacetylase InhibitorsTrichostatin AAcetylationbiology.proteinChromatin immunoprecipitationhistone deacetylases
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Chromatin remodeling regulation by small molecules and metabolites.

2010

The eukaryotic genome is a highly organized nucleoprotein structure comprising of DNA, histones, non-histone proteins, and RNAs, referred to as chromatin. The chromatin exists as a dynamic entity, shuttling between the open and closed forms at specific nuclear regions and loci based on the requirement of the cell. This dynamicity is essential for the various DNA-templated phenomena like transcription, replication, and repair and is achieved through the activity of ATP-dependent chromatin remodeling complexes and covalent modifiers of chromatin. A growing body of data indicates that chromatin enzymatic activities are finely and specifically regulated by a variety of small molecules derived f…

DNA ReplicationS-AdenosylmethionineTranscription GeneticInositol PhosphatesBiophysicsBiochemistryChromatin remodelingchemistry.chemical_compoundAdenosine TriphosphateStructural BiologyAcetyl Coenzyme AGeneticsAnimalsHumansMolecular Biologychromatin small moleculesbiologyGenome HumanDNA replicationDNAChromatin Assembly and DisassemblyNADMi-2/NuRD complexChromatinNucleoproteinChromatinHistoneBiochemistrychemistrybiology.proteinNAD+ kinaseDNABiochimica et biophysica acta
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Multiple roles for ISWI in transcription, chromosome organization and DNA replication.

2003

ISWI functions as the ATPase subunit of multiple chromatin-remodeling complexes. These complexes use the energy of ATP hydrolysis to slide nucleosomes and increase chromatin fluidity, thereby modulating the access of transcription factors and other regulatory proteins to DNA. Here we discuss recent progress toward understanding the biological functions of ISWI, with an emphasis on its roles in transcription, chromosome organization and DNA replication.

DNA ReplicationTranscriptional ActivationHMG-boxTranscription GeneticBiophysicsBiologyBiochemistryATP-dependent chromatin remodeling ISWI Transcription Replication Chromosome structureChromatin remodelingChromosomesAdenosine TriphosphateControl of chromosome duplicationStructural BiologyGeneticsNucleosomeAnimalsHumansTranscription factorGeneticsAdenosine TriphosphatasesDNA replicationChromatin Assembly and DisassemblyChromatinSettore BIO/18 - GeneticaGene Expression RegulationOrigin recognition complexTranscription FactorsBiochimica et biophysica acta
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Epigenomic k-mer dictionaries: shedding light on how sequence composition influences in vivo nucleosome positioning

2014

Abstract Motivation: Information-theoretic and compositional analysis of biological sequences, in terms of k-mer dictionaries, has a well established role in genomic and proteomic studies. Much less so in epigenomics, although the role of k-mers in chromatin organization and nucleosome positioning is particularly relevant. Fundamental questions concerning the informational content and compositional structure of nucleosome favouring and disfavoring sequences with respect to their basic building blocks still remain open. Results: We present the first analysis on the role of k-mers in the composition of nucleosome enriched and depleted genomic regions (NER and NDR for short) that is: (i) exhau…

EpigenomicsStatistics and ProbabilityGeneticsSupplementary dataSequenceGenomeSettore INF/01 - InformaticaSequence Analysis DNAComputational biologyAlgorithms and Data Structures BioinformaticsBiologyChromatin Assembly and DisassemblyBiochemistryNucleosomesComputer Science ApplicationsComputational MathematicsComputational Theory and Mathematicsk-merAnimalsHumansNucleosomeMolecular BiologyComposition (language)Epigenomics
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Curcumin and trans-resveratrol exert cell cycle-dependent radioprotective or radiosensitizing effects as elucidated by the PCC and G2-assay

2013

Curcumin and trans-resveratrol are well-known antioxidant polyphenols with radiomodulatory properties, radioprotecting non-cancerous cells while radiosensitizing tumor cells. This dual action may be the result of their radical scavenging properties and their effects on cell-cycle checkpoints that are activated in response to radiation-induced chromosomal damage. It could be also caused by their effect on regulatory pathways with impact on detoxification enzymes, the up-regulation of endogenous protective systems, and cell-cycle-dependent processes of DNA damage. This work aims to elucidate the mechanisms underlying the dual action of these polyphenols and investigates under which conditions…

G2 PhaseRadiation-Sensitizing AgentsRadiosensitizerCurcuminAntioxidantDNA damageHealth Toxicology and Mutagenesismedicine.medical_treatmentRadioprotectorCellRadiation-Protective AgentsCHO CellsBiologyRadiation ToleranceCell Fusionchemistry.chemical_compoundCricetulusCricetinaeStilbenesGeneticsmedicineAnimalsHumansRadiosensitivityMolecular BiologyCells CulturedMutagenicity TestsCell CycleCell cycleChromatin Assembly and DisassemblyRadiosensitizermedicine.anatomical_structureG2-assayBiochemistrychemistryResveratrolPeripheral blood lymphocyteCancer researchCurcumintrans-ResveratrolPremature chromosome condensationMutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
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ISWI Regulates Higher-Order Chromatin Structure and Histone H1 Assembly In Vivo

2007

Imitation SWI (ISWI) and other ATP-dependent chromatin-remodeling factors play key roles in transcription and other processes by altering the structure and positioning of nucleosomes. Recent studies have also implicated ISWI in the regulation of higher-order chromatin structure, but its role in this process remains poorly understood. To clarify the role of ISWI in vivo, we examined defects in chromosome structure and gene expression resulting from the loss of Iswi function in Drosophila. Consistent with a broad role in transcriptional regulation, the expression of a large number of genes is altered in Iswi mutant larvae. The expression of a dominant-negative form of ISWI leads to dramatic a…

Imitation SWINucleosome assemblyTranscription GeneticQH301-705.5RNA-POLYMERASE-IIPROTEINCHROMOSOME ARCHITECTUREGeneral Biochemistry Genetics and Molecular BiologyHistones03 medical and health sciencesNUCLEOSOME REMODELING FACTORHigher Order Chromatin StructureHistone H1NucleosomeAnimalsTRANSCRIPTIONBiology (General)LIVING CELLSMolecular Biology030304 developmental biologyGENE-EXPRESSIONRegulation of gene expressionGeneticsAdenosine Triphosphatases0303 health sciencesGeneral Immunology and MicrobiologybiologyGeneral Neuroscience030302 biochemistry & molecular biologyGenetics and GenomicsCell BiologyChromatin Assembly and DisassemblyChromatinChromatinCell biologyDROSOPHILAHistoneGene Expression RegulationLarvaMutationbiology.proteinLINKER HISTONEGeneral Agricultural and Biological SciencesResearch ArticleDevelopmental BiologyTranscription FactorsDOSAGE COMPENSATION
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Expression inactivation of SMARCA4 by microRNAs in lung tumors

2014

SMARCA4 is the catalytic subunit of the SWI/SNF chromatin-remodeling complex, which alters the interactions between DNA and histones and modifies the availability of the DNA for transcription. The latest deep sequencing of tumor genomes has reinforced the important and ubiquitous tumor suppressor role of the SWI/SNF complex in cancer. However, although SWI/SNF complex plays a key role in gene expression, the regulation of this complex itself is poorly understood. Significantly, an understanding of the regulation of SMARCA4 expression has gained in importance due to recent proposals incorporating it in therapeutic strategies that use synthetic lethal interactions between SMARCA4-MAX and SMAR…

Lung NeoplasmsDeep sequencingHistonesTranscription (biology)Catalytic DomainCell Line TumorGene expressionmicroRNAGeneticsHumansCloning MolecularMolecular BiologyTranscription factorGenetics (clinical)Cell ProliferationCell NucleusRegulation of gene expressionGeneticsbiologyDNA HelicasesHigh-Throughput Nucleotide SequencingNuclear ProteinsReproducibility of ResultsArticlesGeneral MedicineChromatin Assembly and DisassemblyPrognosisUp-RegulationCell biologyGene Expression Regulation NeoplasticMicroRNAsHistonebiology.proteinSMARCA4HeLa CellsTranscription FactorsHuman Molecular Genetics
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