Search results for "Claudin"

showing 10 items of 28 documents

Claudin-18 splice variant 2 is a pan-cancer target suitable for therapeutic antibody development

2008

Abstract Purpose: Antibody-based cancer therapies have emerged as the most promising therapeutics in oncology. The purpose of this study was to discover novel targets for therapeutic antibodies in solid cancer. Experimental Design: We combined data mining and wet-bench experiments to identify strictly gastrocyte lineage–specific cell surface molecules and to validate them as therapeutic antibody targets. Results: We identified isoform 2 of the tight junction molecule claudin-18 (CLDN18.2) as a highly selective cell lineage marker. Its expression in normal tissues is strictly confined to differentiated epithelial cells of the gastric mucosa, but it is absent from the gastric stem cell zone. …

Cancer ResearchPathologymedicine.medical_specialtymicemedicine.drug_classMolecular Sequence DataGene ExpressionBiologyMonoclonal antibodyMalignant transformationAntigenmedicineProtein IsoformsAnimalsHumansNeoplasms Glandular and EpithelialMembrane Proteins/geneticsAntibodies Monoclonal/immunologyProtein Isoforms/immunologyBase SequenceReverse Transcriptase Polymerase Chain ReactionNeoplasms Glandular and Epithelial/drug therapyAntibodies MonoclonalImmunotherapy ActiveMembrane ProteinsCancermedicine.diseaseFlow CytometryImmunohistochemistryImmunotherapy Active/methodsOncologyCancer cellClaudinsbiology.proteinCancer researchImmunohistochemistryAntibodyStem cell
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Comparison of Claudin 18.2 expression in primary tumors and lymph node metastases in Japanese patients with gastric adenocarcinoma.

2019

CLDN18.2 expression is highly prevalent in Japanese patients with gastric cancer, making it a targetable alteration, and supporting development of zolbetuximab as a therapeutic agent for this patient population.

Cancer Researchmedicine.medical_specialtymedicine.medical_treatmentPopulationprevalenceAdenocarcinomaGastroenterologyAsian PeopleStomach NeoplasmsInternal medicineAntineoplastic Combined Chemotherapy ProtocolsmedicineHumansRadiology Nuclear Medicine and imagingeducationLymph nodeIMAB362Chemotherapyeducation.field_of_studybiologybusiness.industrygastric cancerCancerAntibodies MonoclonalbiomarkersGeneral Medicinemedicine.diseaseImmunohistochemistryClaudinGene Expression Regulation Neoplasticmedicine.anatomical_structureOncologyLymphatic MetastasisClaudinsbiology.proteinImmunohistochemistryBiomarker (medicine)Original ArticleAntibodybusinessJapanese journal of clinical oncology
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First-in-human study of IMAB362, an anti-claudin 18.2 monoclonal antibody, in patients with advanced gastroesophageal cancer

2017

Gastroesophageal cancerOncologybusiness.industrymedicine.drug_classCancer researchMedicineIn patientHuman studyHematologyClaudinbusinessMonoclonal antibodyIMAB362Annals of Oncology
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Aberrantly activated claudin 6 and 18.2 as potential therapy targets in non-small-cell lung cancer

2014

Claudins (CLDNs) are central components of tight junctions that regulate epithelial-cell barrier function and polarity. Altered CLDN expression patterns have been demonstrated in numerous cancer types and lineage-specific CLDNs have been proposed as therapy targets. The objective of this study was to assess which fraction of patients with non-small-cell lung cancer (NSCLC) express CLDN6 and CLDN18 isoform 2 (CLDN18.2). Protein expression of CLDN6 and CLDN18.2 was examined by immunohistochemistry on a tissue microarray (n=355) and transcript levels were supportively determined based on gene expression microarray data from fresh-frozen NSCLC tissues (n=196). Both were analyzed with regard to …

Gene isoformCancer ResearchPathologymedicine.medical_specialtyTissue microarrayCancerBiologymedicine.diseaseGene expression profilingOncologymedicineCancer researchAdenocarcinomaImmunohistochemistryClaudinLung cancerInternational Journal of Cancer
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Claudin-18 gene structure, regulation, and expression is evolutionary conserved in mammals

2011

Claudin-18 isoform 2 (CLDN18.2) is one of the few members of the human claudin family of tight junction molecules with strict restriction to one cell lineage. The objective of the current study was to compare molecular structure and tissue distribution of this gastrocyte specific molecule in mammals. We show here that the CLDN18.2 protein sequence is highly conserved, in particular with regard to functionally relevant domains in mouse, rat, rabbit, dog, monkey and human and also in lizards. Moreover, promoter regions of orthologs are highly homologous, including the binding site of the transcription factor cyclic AMP-responsive element binding protein (CREB), which is known to regulate acti…

Gene isoformmiceMolecular Sequence DataGene Expressionmolecular structureMammals/geneticsBiologyphylogenyRATSConserved sequenceEvolution MolecularDogsProtein Isoforms/geneticsSequence Homology Nucleic AcidGene expressionGeneticsProtein IsoformsAnimalsTissue DistributionAmino Acid SequenceMembrane Proteins/geneticsBinding sitePromoter Regions GeneticClaudinGeneTranscription factorConserved SequenceGastric Mucosa/metabolismMammalsRegulation of gene expressionGeneticsBinding SitesBase SequenceStomachStomach/cytologyMembrane ProteinsCREB-Binding Protein/metabolismHaplorhiniGeneral MedicineCREB-Binding ProteinGene Expression RegulationGastric MucosaOrgan SpecificityMultigene FamilyClaudinsRabbitsGene
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The importance of tight junctions in the blood-brain barrier

2021

There are two barriers in the central nervous system (CNS) responsible for maintaining the homeostatic balance of the human body’s internal environment in relation to external conditions: the blood-brain barrier (BBB) and the blood-cerebrospinal fluid barrier (BCSFB). The blood-brain barrier is a physical barrier with a fixed location between the blood and the nervous tissue. Its basic structural elements include endothelial cells of the brain capillaries, astrocytes and pericytes. The blood-brain barrier is the barrier between the blood and neurons which isolates the central nervous system from the rest of the body in a unique manner. The blood-brain barrier is formed by astrocytes and per…

Inorganic Chemistrytight junctionsEcologyHealth Toxicology and Mutagenesisintercellular connectionsactive transportblood-brain barriercentral nervous systemclaudinsPollutionJournal of Elementology
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Molecular adaptations of the blood–brain barrier promote stress resilience vs. depression

2020

Significance Thirty to fifty percent of depressed individuals are unresponsive to commonly prescribed antidepressant treatments, suggesting that biological mechanisms, such as stress-induced inflammation and blood vessel dysfunction, remain untreated. The blood–brain barrier is the ultimate frontier between the brain and harmful toxins or inflammatory signals circulating in the blood. Depression and vulnerability to chronic social stress are associated with loss of this barrier integrity; however, the mechanisms involved remain poorly understood. Identification of adaptations leading to resilience under stressful conditions could help develop novel treatments. Here we combined behavioral, p…

MaleHistone Deacetylase 1InflammationFOXO1Blood–brain barrierNucleus AccumbensEpigenesis GeneticProinflammatory cytokineMice03 medical and health sciences0302 clinical medicinevascularmedicineAnimalsHumansClaudin-5030304 developmental biologyInflammationSocial stressDepressive Disorder Major0303 health sciencesantidepressantMultidisciplinaryDepressionbusiness.industrySystems BiologyBiological Sciencesmedicine.diseasemood disordersAntidepressive Agents3. Good healthMice Inbred C57BLDisease Models Animalmedicine.anatomical_structureMood disordersBlood-Brain BarrierMajor depressive disorderAntidepressantmedicine.symptombusinessNeuroscienceStress Psychologicalepigenetic030217 neurology & neurosurgerySignal TransductionProceedings of the National Academy of Sciences
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Volatile Anesthetics Influence Blood-Brain Barrier Integrity by Modulation of Tight Junction Protein Expression in Traumatic Brain Injury

2012

Disruption of the blood-brain barrier (BBB) results in cerebral edema formation, which is a major cause for high mortality after traumatic brain injury (TBI). As anesthetic care is mandatory in patients suffering from severe TBI it may be important to elucidate the effect of different anesthetics on cerebral edema formation. Tight junction proteins (TJ) such as zonula occludens-1 (ZO-1) and claudin-5 (cl5) play a central role for BBB stability. First, the influence of the volatile anesthetics sevoflurane and isoflurane on in-vitro BBB integrity was investigated by quantification of the electrical resistance (TEER) in murine brain endothelial monolayers and neurovascular co-cultures of the B…

MaleMouse610 MedizinBrain EdemaPharmacologyCardiovascularMiceAnesthesiology610 Medical sciencesEdemaMolecular Cell BiologyClaudin-5MultidisciplinaryIsofluraneQRAnimal ModelsHead Injurymedicine.anatomical_structureNeurologyBlood-Brain BarrierAnesthesiaAnesthetics InhalationMedicineCellular Typesmedicine.symptomResearch Articlemedicine.drugMethyl EthersTraumatic brain injuryCerebrovascular DiseasesScienceBrain damageBlood–brain barrierSevofluraneCell LineTight JunctionsCerebral edemaSevofluraneModel OrganismsVascular BiologymedicineAnimalsBiologybusiness.industryEndothelial Cellsmedicine.diseaseCoculture TechniquesIsofluraneBrain InjuriesAnestheticZonula Occludens-1 ProteinMolecular NeurosciencebusinessNeurosciencePLoS ONE
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Specific expression patterns of epithelial to mesenchymal transition factors in gestational molar disease.

2015

Introduction The epithelial to mesenchymal transition, a well-known and re-emerging model in pathology, has not been completely investigated in the field of gestational pathology. This study aims at improving the comprehension of this process in molar disease, even looking for new possible immunohistochemical markers. Materials and methods We have analysed the immunohistochemical expression of Twist1 and Snai2, two of the most important transcription factors involved in epithelial to mesenchymal transition, in formalin-fixed paraffin-embedded samples of 23 spontaneous abortive pregnancies, 22 molar pregnancies (10 partial and 12 complete) and 7 term placentas. Results Twist1 and Snai2 were …

MolarPathologymedicine.medical_specialtyStromal cellEpithelial-Mesenchymal TransitionBiologyPregnancymedicineHumansEpithelial–mesenchymal transitionTwistClaudinComplete mole; EMT; Molar disease; Snai2; Twist; Twist1; Obstetrics and Gynecology; Reproductive Medicine; Developmental BiologyCadherinTwist-Related Protein 1EMTObstetrics and GynecologyTrophoblastNuclear ProteinsHydatidiform MoleImmunohistochemistryComplete mole; EMT; Molar disease; Snai2; Twist; Twist1Complete moleSNAI2Molar diseasemedicine.anatomical_structureReproductive MedicineSnai2Case-Control StudiesImmunohistochemistryFemaleSnail Family Transcription FactorsBiomarkersTwist1Developmental BiologyTranscription FactorsPlacenta
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Data set of the protein expression profiles of Luminal A, Claudin-low and overexpressing HER2+ breast cancer cell lines by iTRAQ labelling and tandem…

2015

Breast cancer is the most common and the leading cause of mortality in women worldwide. There is a dire necessity of the identification of novel molecules useful in diagnosis and prognosis. In this work we determined the differentially expression profiles of four breast cancer cell lines compared to a control cell line. We identified 1020 polypeptides labelled with iTRAQ with more than 95% in confidence. We analysed the common proteins in all breast cancer cell lines through IPA software (IPA core and Biomarkers). In addition, we selected the specific overexpressed and subexpressed proteins of the different molecular classes of breast cancer cell lines, and classified them according to prot…

MultidisciplinaryQuantitative proteomicsLuminal aBiologyTandem mass spectrometryBioinformaticsClaudin-Lowmedicine.diseaselcsh:Computer applications to medicine. Medical informaticsProtein expressionBreast cancerBreast cancer cell lineLabellingCancer researchmedicinelcsh:R858-859.7lcsh:Science (General)skin and connective tissue diseaseslcsh:Q1-390Data ArticleData in Brief
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