Search results for "Clines"

showing 10 items of 63 documents

Oxidative stress response of tumor cells: microarray-based comparison between artemisinins and anthracyclines

2004

The antimalarial artemisinins also reveal profound cytotoxic activity against tumor cells. Artemisinins harbor an endoperoxide bridge whose cleavage results in the generation of reactive oxygen species (ROS) and/or artemisinin carbon-centered free radicals. Established cancer drugs such as anthracyclines also form ROS and free radicals that are responsible for the cardiotoxicity of anthracyclines. In contrast, artemisinins do not reveal cardiotoxicity. In the present investigation, we compared the cytotoxic activities of different artemisinins (artemisinin, artesunate, arteether, artemether, artemisitene, dihydroartemisinylester stereoisomers) in 60 cell lines of the National Cancer Institu…

ArtemisininsDaunorubicinAntineoplastic AgentsPharmacologyBiologymedicine.disease_causeBiochemistryAntimalarialsInhibitory Concentration 50parasitic diseasesTumor Cells CulturedmedicineAnimalsCluster AnalysisHumansIdarubicinAnthracyclinesDoxorubicinRNA MessengerArtemisininOligonucleotide Array Sequence AnalysisPharmacologyCardiotoxicityGene Expression ProfilingArtemisininsGene expression profilingOxidative StressDrug Screening Assays AntitumorOxidation-ReductionSesquiterpenesOxidative stressmedicine.drugBiochemical Pharmacology
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Arterial hypertension in cancer: The elephant in the room

2019

The great therapeutical success achieved by oncology is counterbalanced by growing evidences of cardiovascular (CV) toxicity due to many antineoplastic treatments. Cardiac adverse events may cause premature discontinuation of effective oncologic treatments or occur as late events undermining the oncologic success. Arterial hypertension is both the most common comorbidity in cancer patients and a frequent adverse effect of anticancer therapies. A pre-existing hypertension is known to increase the risk of other cardiac adverse events due to oncologic treatments, in particular heart failure. Moreover, as a strict association between cancer and CV diseases has emerged over the recent years, var…

Arterial hypertensionVascular Endothelial Growth Factor AAnthracyclines Anti VEGF agents Anti-hypertensive therapy Arterial hypertension Cancer Cardiotoxicitymedicine.medical_specialtyAnti VEGF agentmedicine.medical_treatmentAntineoplastic AgentsBlood PressureAnthracycline030204 cardiovascular system & hematologyAnthracyclines; Anti VEGF agents; Anti-hypertensive therapy; Arterial hypertension; Cancer; Cardiotoxicity; Antihypertensive Agents; Antineoplastic Agents; Blood Pressure; Humans; Hypertension; Neoplasms; Vascular Endothelial Growth Factor A03 medical and health sciences0302 clinical medicineNeoplasmsmedicineHumansAnthracyclines030212 general & internal medicineAnti-hypertensive therapyAdverse effectIntensive care medicineAntihypertensive AgentsCancerChemotherapyCardiotoxicitybusiness.industryAnti VEGF agentsCancermedicine.diseaseComorbidityCardiotoxicityDiscontinuationBlood pressureHeart failureHypertensionCardiology and Cardiovascular MedicinebusinessInternational Journal of Cardiology
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Impact of Undernutrition on the Pharmacokinetics and Pharmacodynamics of Anticancer Drugs: A Literature Review

2017

The etiology of undernourishment in cancer patients is multifactorial: tumor-related mechanisms (such as obstruction, metabolic abnormalities, and functionality changes) in addition to the influence of anticancer therapies, which can induce or worsen undernutrition. The evident role of undernutrition in cancer treatment outcomes suggests the need of considering nutritional status when evaluating anticancer drugs. In order to merge the available data and offer researchers and clinicians a global view of this phenomenon, the present manuscript reviews on a drug-by-drug basis the undernutrition-related pharmacokinetic and pharmacodynamic aspects of anticancer treatments. This review notes inte…

Cancer ResearchMedicine (miscellaneous)Antineoplastic AgentsPharmacologyBioinformatics030226 pharmacology & pharmacy03 medical and health sciences0302 clinical medicineOtotoxicityPharmacokineticsmedicineHumansAnthracyclinesDosingVinca AlkaloidsEtoposideCardiotoxicityNutrition and Dieteticsbusiness.industryMalnutritionNeurotoxicitymedicine.diseaseCancer treatmentMalnutritionMethotrexateOncology030220 oncology & carcinogenesisFluorouracilPharmacodynamic aspectsbusinessNutrition and Cancer
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Timely recognition of cardiovascular toxicity by anticancer agents: a common objective of the pharmacologist, oncologist and cardiologist.

2011

Both conventional and new anticancer drugs can frequently cause adverse cardiovascular effects, which can span from subclinical abnormalities to serious life-threatening and sometimes fatal events. This review examines the principal basic and clinical elements that may be of profit to identify, prevent and treat such toxicities. Clearly, the accomplishment of such objectives requires the strong commitment and cooperation of different professional figures including, but not limited to, pharmacologists, oncologists and cardiologists. The aspect of anticancer drug cardiotoxicity seems to be somehow underestimated, mainly due to inadequate reporting of adverse reactions from oncology drugs in t…

Cardiovascular toxicityTime FactorsSettore MED/06 - Oncologia MedicaPharmacology toxicologyCardiologyAntineoplastic AgentsPharmacologyToxicologyMedical OncologyCardiotoxinsCardiovascular SystemProfessional RolePharmacovigilanceMedicineAnimalsHumansPhysician's RoleMolecular BiologyPharmacologyCardiotoxicitybusiness.industryCardiovascular toxicity Anthracyclines Tyrosine kinase inhibitors TrastuzumabSettore MED/11 - Malattie Dell'Apparato CardiovascolareAnticancer drugLaboratory PersonnelCardiovascular DiseasesSettore BIO/14 - FarmacologiaCardiology and Cardiovascular MedicinebusinessOncology drugsCardiovascular toxicology
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Anti-inflammatory effects of chemically modified tetracyclines by the inhibition of nitric oxide and interleukin-12 synthesis in J774 cell line

2001

We investigated the effects of chemically modified tetracyclines (CMTs) on the production of nitric oxide (NO) and on the synthesis of some cytokines: tumour necrosis factor alpha (TNF-alpha), interleukin(IL)-10 and IL-12 in lipopolysaccharide (LPS)-treated J774 cell line. Furthermore, we studied the ability of these drugs to modify the viability in LPS-stimulated J774 macrophages. CMTs decreased, in a dose-dependent manner, inducible NO synthase (iNOS) activity and, consequently, nitrite formation in J774 cultures. The CMT-induced decrease in NO production is due to the inhibition of enzyme activity rather than to a direct effect on enzyme expression. The absence of the inhibition in mRNA …

Cell Survivalmedicine.medical_treatmentImmunologyNitric Oxide Synthase Type IIApoptosisEnzyme-Linked Immunosorbent AssayNitric OxideCell LineNitric oxideMicechemistry.chemical_compoundEthidiumIn Situ Nick-End LabelingmedicineAnimalsImmunology and AllergyRNA MessengerViability assayEnzyme InhibitorsFluorescent DyesPharmacologybiologyReverse Transcriptase Polymerase Chain ReactionAnti-Inflammatory Agents Non-SteroidalInterleukinBiological activityInterleukin-12Acridine OrangeCell biologyNitric oxide synthaseInterleukin 10CytokinechemistryBiochemistryTetracyclinesApoptosisbiology.proteinCytokinesElectrophoresis Polyacrylamide GelIndicators and ReagentsNitric Oxide SynthaseInternational Immunopharmacology
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Pathophysiology of anthracycline cardiotoxicity.

2016

Anthracyclines (ANTs) are powerful drugs that have reduced the mortality of cancer patients. However, their use is limited by the development of cardiotoxicity (CTX), which is dose dependent and may lead to left ventricular dysfunction and heart failure. Although various strategies have been suggested to reduce the negative effects of ANTs, CTX is still an important unresolved clinical issue. This may be due at least partly to the incomplete characterization of the molecular and cellular mechanisms of ANT-induced CTX. In addition, although various forms of cardiac damage have been demonstrated with the use of these drugs in experimental studies, it is not yet clear how these translate to th…

DrugAnthracyclinemedia_common.quotation_subjectLeftanthracyclines; cancer; cardiotoxicity; Cardiology and Cardiovascular MedicineDose dependenceAntineoplastic Agents030204 cardiovascular system & hematologyanthracyclineBioinformatics03 medical and health sciencesVentricular Dysfunction Left0302 clinical medicineAntibioticsNeoplasmsVentricular DysfunctionmedicinecancerHumansGenetic Predisposition to DiseaseAnthracyclinesmedia_commonHeart FailureCardiotoxicityAntibiotics AntineoplasticDose-Response Relationship Drugbusiness.industryanthracyclines cancer cardiotoxicityCancerHeartGeneral Medicinebiochemical phenomena metabolism and nutritionmedicine.diseaseAntineoplasticPathophysiologyCardiotoxicityCardiovascular Diseases030220 oncology & carcinogenesisHeart failureCardiology and Cardiovascular Medicinebusinessanthracyclines; cancer; cardiotoxicity; Anthracyclines; Antibiotics Antineoplastic; Cardiotoxicity; Heart Failure; Humans; Neoplasms; Ventricular Dysfunction LeftJournal of cardiovascular medicine (Hagerstown, Md.)
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Photo-induced fluorescence of magnesium derivatives of tetracycline antibiotics in wastewater samples

2009

Abstract An analytical strategy, for the determination of tetracyclines (TCs), based on a HPLC system coupled with a photo-reactor followed by post-column derivatization was developed. Higher fluorescence emission after coupling the resulting photo-fragments with magnesium ions was observed for the determination of minocycline (MC), epitetracycline (ETC), tetracycline (TC) and doxycycline (DC). The manifold included a HPLC system with a photo-reactor (PTFE tubing helically coiled around a low-pressure mercury lamp), a mixing T-piece and a fluorescence detector. The derivatization reagent was delivered at 0.5 mL min −1 by a pump. After HPLC separation using a gradient system with a mobile ph…

Environmental EngineeringLightPhotochemistrymedicine.drug_classHealth Toxicology and MutagenesisTetracycline antibioticsMagnesium Compoundschemistry.chemical_elementWaste Disposal FluidHigh-performance liquid chromatographyFluorescenceFluorescence spectroscopychemistry.chemical_compoundmedicineEnvironmental ChemistrySolid phase extractionDerivatizationWaste Management and DisposalMagnesium ionChromatography High Pressure LiquidChromatographyMagnesiumReproducibility of ResultsReference StandardsPollutionchemistryTetracyclinesReagentIndicators and ReagentsWater Pollutants ChemicalJournal of Hazardous Materials
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Reverse and normal drag along a fault

2005

An analysis of the theoretical displacement field around a single dip-slip fault at depth reveals that normal and reverse fault drag develop by perturbation flow induced by fault slip. We analytically model the heterogeneous part of the instantaneous displacement field of an isolated two-dimensional mode II fault in an infinite, homogeneous elastic body in response to fault slip. Material on both sides of the fault is displaced and ‘opposing circulation cells’ arise on opposite sides of the fault, with displacement magnitudes increasing towards the center of the fault. Both normal and reverse drag can develop at the fault center depending on the angle between the markers and the fault; norm…

Flow (psychology)Mode (statistics)Perturbation (astronomy)GeologyGeometryFault (power engineering)Displacement (vector)Physics::Fluid DynamicsComputer Science::Hardware ArchitectureDragDisplacement fieldGeotechnical engineeringRollover anticlinesComputer Science::Operating SystemsComputer Science::Distributed Parallel and Cluster ComputingGeologyJournal of Structural Geology
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Sedimentological and tectonic context of Paleoproterozoïque Franceville basin (Gabon) : fluid pressure structures, bitumen and uranium mineralization

2016

Metallogenic deposits within paleproterozoic basins depend on generation and migration of fluids. The aim of this study is to provide a better understanding of tectonic, sedimentological and diagenetic setting of the uranium deposits in the Franceville basin and to characterize hydraulic fracturing impact on fluid migration processes in sandstone reservoirs.Tectonic study define the N180-170° transfer faults, associated with Archean tectonic and the N110-120° longitudinal normal faults. These two fault directions split the Franceville basin into small sub-basins. The longitudinal normal faults are associated with footwall anticlines and hanging wall synclines. The uranium deposits of France…

Fracturation hydrauliqueBassin de FrancevilleFailles de transfertUranium mineralizationTransfer faultsHydraulic fracturingPaleoproterozoicBarrières de perméabilitéFootwall anticlinesStructures de surpression fluidesLongitudinal normal faultsFranceville basinAnticlinal de murBitumen[SDU.STU] Sciences of the Universe [physics]/Earth SciencesUraniumFluid pressure structuresPaléoprotérozoïqueBitumePermeability barriersFailles longitudinales
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Modulation of Nitric Oxide Production by Tetracyclines and Chemically Modified Tetracyclines

1999

Chemically modified tetracyclines (CMTs) dose-dependently decreased inducible nitric oxide synthase (iNOS) and, consequently, nitric oxide (NO) formation by the lipopolysaccharide (LPS)-stimulated J774 line. The inhibitory effect was due to a specific reduction in the iNOS protein content in the cells, as attested by Western blot analysis and by the inhibition of iNOS mRNA accumulation. Furthermore, CMTs cause a dose-dependent increase in cell death in the J774 line mediated by the NO-independent apoptotic mechanism.

Lipopolysaccharides0301 basic medicineLipopolysaccharideApoptosisNitric OxideDexamethasoneCell LineNitric oxideMice03 medical and health scienceschemistry.chemical_compound0302 clinical medicineWestern blotmedicineAnimalsInos proteinInhibitory effectomega-N-MethylarginineDose-Response Relationship Drugmedicine.diagnostic_testbiologyMacrophages030206 dentistryGeneral MedicineNitric oxide synthase030104 developmental biologychemistryBiochemistryTetracyclinesApoptosisInos mrnabiology.proteinNitric Oxide SynthaseAdvances in Dental Research
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