Search results for "Cognitive"

showing 10 items of 10389 documents

Relationship Between Body Mass Index, ApoE4 Status, and PET-Based Amyloid and Neurodegeneration Markers in Amyloid-Positive Subjects with Normal Cogn…

2018

Body weight loss in late-life is known to occur at a very early stage of Alzheimer's disease (AD). Apolipoprotein E4 (ApoE4) represents a major genetic risk factor for AD and is linked to an increased cortical amyloid-β (Aβ) accumulation. Since the relationship between body weight, ApoE4, and AD pathology is poorly investigated, we aimed to evaluate whether ApoE4 allelic status modifies the association of body mass index (BMI) with markers of AD pathology. A total of 368 Aβ-positive cognitively healthy or mild cognitive impaired subjects had undergone [18F]-AV45-PET, [18F]-FDG-PET, and T1w-MRI examinations. Composite cortical [18F]-AV45 uptake and [18F]-FDG uptake in posterior cingulate cor…

0301 basic medicineMaleApolipoprotein E4Body Mass Index0302 clinical medicineCognitionWeight lossCognitive declineAniline CompoundsGeneral NeuroscienceNeurodegenerationBrainCognitionNeurodegenerative DiseasesGeneral MedicinePsychiatry and Mental healthClinical PsychologyEthylene GlycolsFemalemedicine.symptommedicine.medical_specialtyAmyloidHeterozygote03 medical and health sciencesFluorodeoxyglucose F18Internal medicinemental disordersWeight LossmedicineHumansCognitive DysfunctionEffects of sleep deprivation on cognitive performanceAdaptor Proteins Signal TransducingAgedbusiness.industryZebrafish Proteinsmedicine.diseaseCortex (botany)Repressor Proteins030104 developmental biologyEndocrinologyGlucosePosterior cingulatePositron-Emission TomographyGeriatrics and GerontologyRadiopharmaceuticalsbusinessNeuroscienceBody mass index030217 neurology & neurosurgeryFollow-Up StudiesJournal of Alzheimer's disease : JAD
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Dynamic longitudinal behavior in animals exposed to chronic social defeat stress

2020

AbstractChronic social defeat (CSD) can lead to impairments in social interaction and other behaviors that are supposed to model features of major depressive disorder (MDD). Not all animals subjected to CSD, however, develop these impairments, and maintained social interaction in some animals is widely used as a model for resilience to stress-induced mental dysfunctions. So far, animals have mainly been studied shortly (24 hours and 7 days) after CSD exposure and longitudinal development of behavioral phenotypes in individual animals has been mostly neglected. We have analyzed social interaction and novel object recognition behavior of stressed mice at different time points after CSD and ha…

0301 basic medicineMaleBehavioral phenotypesTime FactorsSocial SciencesSocial defeatMice0302 clinical medicineCognitionLearning and MemoryStress (linguistics)PsychologyLongitudinal Studiesmedia_commonMammalsMultidisciplinaryAnimal BehaviorBehavior AnimalQREukaryotaResilience PsychologicalLongitudinal developmentAggressionAnimal SocialityVertebratesMedicineMajor depressive disorderPsychological resilienceDisease SusceptibilityPsychologyBehavior Observation TechniquesNetwork AnalysisClinical psychologyResearch ArticleComputer and Information SciencesSciencemedia_common.quotation_subjectRodentsNetwork Resilience03 medical and health sciencesMemorymedicineAnimalsHumansInterpersonal RelationsNovel object recognitionBehaviorDepressive Disorder MajorNetwork resilience ; Visual object recognition ; Animal performance ; Behavior ; Animal sociality ; Collective animal behavior ; Animal behavior ; MiceOrganismsCognitive PsychologyBiology and Life SciencesCollective Animal Behaviormedicine.diseaseSocial relationDisease Models Animal030104 developmental biologyCollective Human BehaviorAmniotesChronic DiseaseCognitive SciencePerceptionCollective animal behaviorVisual Object RecognitionZoology030217 neurology & neurosurgeryStress PsychologicalNeurosciencePLoS ONE
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Genome-wide association scan identifies new variants associated with a cognitive predictor of dyslexia

2019

Developmental dyslexia (DD) is one of the most prevalent learning disorders, with high impact on school and psychosocial development and high comorbidity with conditions like attention-deficit hyperactivity disorder (ADHD), depression, and anxiety. DD is characterized by deficits in different cognitive skills, including word reading, spelling, rapid naming, and phonology. To investigate the genetic basis of DD, we conducted a genome-wide association study (GWAS) of these skills within one of the largest studies available, including nine cohorts of reading-impaired and typically developing children of European ancestry (N = 2562–3468). We observed a genome-wide significant effect (p < 1 × 10…

0301 basic medicineMaleCandidate geneMultifactorial InheritanceImaging geneticsQH301 BiologyLANGUAGEGenome-wide association study3124 Neurology and psychiatryCANDIDATE GENESDyslexiaCohort StudiesREADING-DISABILITYMOLECULAR-GENETICS0302 clinical medicineCognitionAUTOMATIZED NAMING RANChildSUSCEPTIBILITY LOCUSRapid automatized namingR2CSHORT-TERM-MEMORY~DC~IMAGING-GENETICSRJ Pediatrics[SDV.NEU.SC]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Cognitive SciencesPsychiatry and Mental healthDyslexia/geneticsAnxietyFemalemedicine.symptomBDCRC0321 Neuroscience. Biological psychiatry. NeuropsychiatryClinical psychologyNeuroinformaticsAdultReading disabilityAdolescentGenotypeRJPolymorphism Single NucleotideArticlelcsh:RC321-571ENVIRONMENTAL-INFLUENCES03 medical and health sciencesCellular and Molecular NeuroscienceQH301Young AdultmedicinedysleksiaHumansGenetic Predisposition to Diseaselcsh:Neurosciences. Biological psychiatry. NeuropsychiatryBiological Psychiatrygeenitbusiness.industryDyslexiaDASmedicine.diseaseComorbiditypredictors030104 developmental biology[SDV.GEN.GH]Life Sciences [q-bio]/Genetics/Human geneticsRC0321DEVELOPMENTAL DYSLEXIAbusiness030217 neurology & neurosurgeryGenome-Wide Association Study
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Bi-allelic JAM2 Variants Lead to Early-Onset Recessive Primary Familial Brain Calcification

2020

International audience; Primary familial brain calcification (PFBC) is a rare neurodegenerative disorder characterized by a combination of neurological, psychiatric, and cognitive decline associated with calcium deposition on brain imaging. To date, mutations in five genes have been linked to PFBC. However, more than 50% of individuals affected by PFBC have no molecular diagnosis. We report four unrelated families presenting with initial learning difficulties and seizures and later psychiatric symptoms, cerebellar ataxia, extrapyramidal signs, and extensive calcifications on brain imaging. Through a combination of homozygosity mapping and exome sequencing, we mapped this phenotype to chromo…

0301 basic medicineMaleCerebellumPathology[SDV]Life Sciences [q-bio]recessive brain calcificationMice0302 clinical medicineCognitive declineAge of OnsetChildGenetics (clinical)Exome sequencingComputingMilieux_MISCELLANEOUSBrain Diseasesprimary familial brain calcificationMalalties neurodegenerativesBrainFahr diseaseCalcinosisOCLNNeurodegenerative DiseasesHuman brainMiddle AgedPedigree[SDV] Life Sciences [q-bio]medicine.anatomical_structureKnockout mouseFemalemedicine.symptomAdultmedicine.medical_specialtyAdolescentGenes RecessiveNeuropathologyBiologyCalcificacióCalcification03 medical and health sciencesBasal Ganglia DiseasesReportGeneticsmedicineAnimalsHumansAllelesSLC20A2Cerebellar ataxiaknock out mouse modelmedicine.diseaseJAM2030104 developmental biologyFahr disease; familial idiopathic basal ganglia calcification; JAM2; JAM3; knock out mouse model; MYORG; OCLN; primary familial brain calcification; recessive brain calcification; SLC20A2familial idiopathic basal ganglia calcificationJAM3MYORGXenotropic and Polytropic Retrovirus ReceptorCell Adhesion Molecules030217 neurology & neurosurgeryCalcification
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Activation of Corticothalamic Layer 6 Cells Decreases Angular Tuning in Mouse Barrel Cortex.

2019

In the mouse whisker system, the contribution of L6 corticothalamic cells (L6 CT) to cortical and thalamic processing of the whisker deflection direction was investigated. A genetically defined population of L6 CT cells project to infragranular GABAergic interneurons that hyperpolarize neurons in somatosensory barrel cortex (BC). Optogenetic activation of these neurons switched BC to an adapted mode in which excitatory cells lost their angular tuning. In contrast, however, this was not the case with a general activation of inhibitory interneurons via optogenetic activation of Gad2-expressing cells. The decrease in angular tuning, when L6 CT cells were activated, was due to changes in cortic…

0301 basic medicineMaleCognitive NeurosciencePopulationNeuroscience (miscellaneous)OptogeneticsSomatosensory systemInhibitory postsynaptic potentiallcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineThalamusexcitation inhibition balancedirection selectivitymedicineAnimalsGABAergic Neuronseducationlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal ResearchNeuronseducation.field_of_studyAfferent PathwaysNeocortexGAD2NTSR1ChemistryNeural InhibitionSomatosensory CortexBarrel cortexSensory Systemslayer 6030104 developmental biologymedicine.anatomical_structurenervous systemTouch PerceptionVibrissaeExcitatory postsynaptic potentialGABAergicFemaleNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in neural circuits
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Significance of chronic toxoplasmosis in epidemiology of road traffic accidents in Russian Federation

2017

Studies carried out in Moscow residents have revealed that the prevalence of chronic toxoplasmosis is very close to those in countries of Eastern and Central Europe. Our findings also demonstrated a statistically significant relationship between the rate of traffic accidents and the seroprevalence of chronic toxoplasmosis in drivers who were held responsible for accidents. The latter was 2.37 times higher in drivers who were involved in road accidents compared with control groups. These results suggest that the consequences of chronic toxoplasmosis (particularly a slower reaction time and decreased concentration) might contribute to the peculiarities of the epidemiology of road traffic acci…

0301 basic medicineMaleCritical Care and Emergency Medicinelcsh:MedicineAntibodies ProtozoanRussiaGeographical LocationsEnvironmental protectionEpidemiologyMedicine and Health SciencesPrevalencePublic and Occupational Healthlcsh:ScienceRoad trafficTrauma MedicineGeographic AreasMultidisciplinaryGeographyTraumatic Injury Risk FactorsAccidents Traffic030108 mycology & parasitologyEuropeGeographyRoad Traffic CollisionsFemaleSafetyTraumatic InjuryToxoplasmosisResearch ArticleUrban Areasmedicine.medical_specialtyAsiaCognitive Neuroscience03 medical and health sciencesEnvironmental healthmedicineParasitic DiseasesReaction TimeSeroprevalenceHumansBehaviorProtozoan InfectionsRoad traffic safetylcsh:RTraffic SafetyBiology and Life Sciencesmedicine.diseaseToxoplasmosis030104 developmental biologyImmunoglobulin MCase-Control StudiesImmunoglobulin GPeople and PlacesChronic DiseaseEarth SciencesCognitive Sciencelcsh:QRussian federationhuman activitiesNeurosciencePLoS ONE
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Functional hypoxia drives neuroplasticity and neurogenesis via brain erythropoietin.

2020

Erythropoietin (EPO), named after its role in hematopoiesis, is also expressed in mammalian brain. In clinical settings, recombinant EPO treatment has revealed a remarkable improvement of cognition, but underlying mechanisms have remained obscure. Here, we show with a novel line of reporter mice that cognitive challenge induces local/endogenous hypoxia in hippocampal pyramidal neurons, hence enhancing expression of EPO and EPO receptor (EPOR). High-dose EPO administration, amplifying auto/paracrine EPO/EPOR signaling, prompts the emergence of new CA1 neurons and enhanced dendritic spine densities. Single-cell sequencing reveals rapid increase in newly differentiating neurons. Importantly, i…

0301 basic medicineMaleDendritic spineGeneral Physics and AstronomyHippocampal formationVARIANTSADULT NEUROGENESIS0302 clinical medicineCognitionhemic and lymphatic diseasesReceptors ErythropoietinHypoxialcsh:ScienceNEURONSMultidisciplinaryNeuronal PlasticityPyramidal CellsNeurogenesisQBrainCell DifferentiationHEMATOPOIETIC PROGENITOR CELLSFemalemedicine.symptomProto-Oncogene Proteins c-fosmedicine.drugEXPRESSIONScienceDendritic SpinesNeurogenesisModels NeurologicalBiologyMotor ActivityGeneral Biochemistry Genetics and Molecular BiologyArticle03 medical and health sciencesParacrine signallingPhysical Conditioning AnimalNeuroplasticitymedicineAnimalsHumansErythropoietinMEMORYCognitive neuroscienceGeneral ChemistryHypoxia (medical)RECOMBINANT-HUMAN-ERYTHROPOIETINCellular neuroscienceErythropoietin receptorMice Inbred C57BLMICE030104 developmental biologyErythropoietinPhysical EnduranceIDENTITYlcsh:QTranscriptomeNeuroscience030217 neurology & neurosurgeryGene Deletion
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Maternal inflammation has a profound effect on cortical interneuron development in a stage and subtype-specific manner

2018

AbstractSevere infections during pregnancy are one of the major risk factors for cognitive impairment in the offspring. It has been suggested that maternal inflammation leads to dysfunction of cortical GABAergic interneurons that in turn underlies cognitive impairment of the affected offspring. However, the evidence comes largely from studies of adult or mature brains and how the impairment of inhibitory circuits arises upon maternal inflammation is unknown. Here we show that maternal inflammation affects multiple steps of cortical GABAergic interneuron development, i.e., proliferation of precursor cells, migration and positioning of neuroblasts, as well as neuronal maturation. Importantly,…

0301 basic medicineMaleInterneuronOffspringNeurogenesisMothersInflammationBiologyInhibitory postsynaptic potentialArticle03 medical and health sciencesCellular and Molecular NeuroscienceMice0302 clinical medicineNeuroblastCell MovementInterneuronsPregnancyPrecursor cellmedicineAnimalsCognitive DysfunctionGABAergic NeuronsMolecular BiologyCell ProliferationCerebral CortexInflammationPregnancyCell growthNeurogenesisCognitionmedicine.diseaseMice Inbred C57BLPsychiatry and Mental health030104 developmental biologymedicine.anatomical_structurenervous systemPrenatal Exposure Delayed EffectsGABAergicFemalemedicine.symptomPsychiatric disordersNeuroscience030217 neurology & neurosurgeryNeuroscience
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Sugar-sweetened and artificially-sweetened beverages and changes in cognitive function in the SUN project.

2019

Background: Sugar-sweetened beverages (SSB) and artificially-sweetened beverages (ASB) have been inconsistently associated with declines in cognitive function. Because of their low caloric content ...

0301 basic medicineMaleMedicine (miscellaneous)Neuropsychological Tests03 medical and health sciences0302 clinical medicineCognitionmedicineDementiaHumansCognitive DysfunctionFood scienceLongitudinal StudiesProspective StudiesSugarAgedSugar-Sweetened Beverages030109 nutrition & dieteticsNutrition and Dieteticsbusiness.industryGeneral Neurosciencedigestive oral and skin physiologyArtificially Sweetened Beveragesnutritional and metabolic diseasesfood and beveragesCaloric theoryCognitionGeneral MedicineMiddle Agedmedicine.diseasestomatognathic diseasesSpainbusinesshuman activities030217 neurology & neurosurgeryNutritional neuroscience
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Characterizing the Molecular Architecture of Cortical Regions Associated with High Educational Attainment in Older Individuals

2019

Neuroimaging investigations have revealed interindividual variations in anatomy, metabolism, activity, and connectivity of specific cortical association areas through which years of education (YoE), as a common proxy of cognitive reserve, may operate in the face of age- or pathology-associated brain changes. However, the associated molecular properties of YoE-related brain regions and the biological pathways involved remain poorly understood. In the present study we first identified brain areas that showed an association between cortical thickness and YoE among 122 cognitively healthy older human individuals (87 female). We subsequently characterized molecular properties of these regions by…

0301 basic medicineMaleMicroarraymetabolism [Prefrontal Cortex]Prefrontal CortexNeuroimagingBiologyGyrus CinguliBiological pathway03 medical and health sciences0302 clinical medicineNeuroimagingCognitive ReserveCortex (anatomy)immunology [Gyrus Cinguli]metabolism [Gyrus Cinguli]Gene expressionmedicineHumansddc:610diagnostic imaging [Gyrus Cinguli]Prefrontal cortexResearch ArticlesCognitive reserveAgedGeneral NeuroscienceGene Expression ProfilingMiddle AgedMental Status and Dementia Testsphysiology [Cognitive Reserve]030104 developmental biologymedicine.anatomical_structureimmunology [Prefrontal Cortex]diagnostic imaging [Prefrontal Cortex]Educational StatusFemaleNeuroscience030217 neurology & neurosurgeryIonotropic effectGenome-Wide Association Study
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