Search results for "Colcemid"

showing 4 items of 4 documents

Functional Inactivation of pRB Results in Aneuploid Mammalian Cells After Release From a Mitotic Block

2002

AbstractThe widespread chromosome instability observed in tumors and in early stage carcinomas suggests that aneuploidy could be a prerequisite for cellular transformation and tumor initiation. Defects in tumor suppressers and genes that are part of mitotic checkpoints are likely candidates for the aneuploid phenotype. By using flow cytometric, cytogenetic, immunocytochemistry techniques we investigated whether pRB deficiency could drive perpetual aneuploidy in normal human and mouse fibroblasts after mitotic checkpoint challenge by microtubule-destabilizing drugs. Both mouse and human pRB-deficient primary fibroblasts resulted, upon release from a mitotic block, in proliferating aneuploid …

DNA ReplicationCancer ResearchBrief ArticleClone (cell biology)MitosisAneuploidyCre recombinaseSpindle Apparatuslcsh:RC254-282Retinoblastoma ProteinColony-Forming Units AssayMicechemistry.chemical_compoundChromosome instabilitymedicineAnimalsHumanscentrosomesCINGenes RetinoblastomaMitosisCells CulturedIn Situ Hybridization FluorescenceCentrosomeCell cycle controlbiologyColcemidChromosome FragilityCell CycleGINDemecolcineRetinoblastoma proteinAneuploidy; Cell cycle control; Centrosomes; CIN; PRB;FibroblastsCell cyclelcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogensAneuploidyFlow Cytometrymedicine.diseaseAntineoplastic Agents PhytogenicCell biologyCell Transformation NeoplasticPRBMicroscopy Fluorescencechemistrybiology.proteinFemaleNeoplasia
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Monitoring of the action of drugs in melanoma cells by dynamic laser speckle

2014

Abstract. This work presents the development of a protocol based on the dynamic laser speckle designed tomonitor the reaction of cancer cells of line MEL-RC08 to the application of the drug Colcemid in two differentconcentrations: 0.2 and 0.4 μg∕mL. The protocol was designed using the forward scattering approach with anHe-Ne laser of 632.8 nm illuminating the samples, a control, and two variations of Colcemid, being monitoredalong 8 h. The data were analyzed numerically in the time and in the frequency domain, and the results pre-sented the ability of the technique to monitor the action of the drug, particularly Colcemid (0.4 μg∕mL). © 2014Society of Photo-Optical Instrumentation Engineers …

Diagnostic ImagingCellular activityComputer scienceBiomedical Engineeringlaw.inventionBiomaterialschemistry.chemical_compoundSpeckle patternKey pointOpticslawCell Line TumorImage Processing Computer-AssistedmedicineHumansInstrumentation (computer programming)MelanomaAnalysis of VarianceColcemidbusiness.industryLasersMelanomaCell CycleDemecolcineLasermedicine.diseaseAntineoplastic Agents PhytogenicAtomic and Molecular Physics and OpticsElectronic Optical and Magnetic MaterialschemistryFrequency domainbusinessBiomedical engineeringJournal of Biomedical Optics
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Microtubules and intermediate filaments of herpes simplex virus infected cells.

1987

The fate of microtubules and of vimentin or keratin containing intermediate filaments during infection with fusion or rounding producing strains of herpes simplex virus (HSV) was investigated. Microtubules polymerize early after fusion of cells. However, they do not reconstitute 6–7 hours post infection (p.i.) after release of a colcemid block. Keratin and vimentin are maintained around the original nucleus still inside of recruited cells in the polykaryocyte. Cells of fibroblastic and epithelial origin fuse. Inside of polykaryocytes keratin or vimentin containing fibers seem to polymerize. Keratin is to be found in invaginations in the nuclei surrounded by the inner layer of the nuclear me…

Intermediate FilamentsVimentinmacromolecular substancesmedicine.disease_causeMicrofilamentMicrotubulesEpitheliumCell LineCell Fusionchemistry.chemical_compoundCytopathogenic Effect ViralVirologyKeratinmedicineAnimalsSimplexvirusVimentinNuclear membraneIntermediate filamentCytoskeletonchemistry.chemical_classificationintegumentary systembiologyColcemidHerpes SimplexGeneral MedicineFibroblastsVirologyHerpes simplex virusmedicine.anatomical_structurechemistryCytoplasmbiology.proteinKeratinsArchives of virology
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Dis-organizing centrosomal clusters: specific cancer therapy for a generic spread?

2015

Cancer is a leading cause of mortality and the annual incidence of new cancer cases is rising worldwide. Due to the frequent development of resistance and the side effects of established anti-cancer drugs, the quest for new drugs with improved therapeutic features goes on. In contrast to cytotoxic chemotherapy of the past, the concept of targeted chemotherapy attempts to increase specificity of therapy by attacking tumor-related mechanisms. A novel emerging treatment concept represents the inhibition of centrosomal clustering. The centrosome regulates mitotic spindle formation assuring uniform separation of chromosomes to daughter cells. Many tumors contain supernumerary centrosomes, which …

PharmacologyCentrosomeBiological ProductsCell divisionColcemidOrganic ChemistryBiologyBiochemistrySpindle pole bodyVinblastineCell biologySpindle apparatusNocodazolechemistry.chemical_compoundchemistryCentrosomeNeoplasmsDrug DiscoverymedicineMolecular MedicineAnimalsHumansMultipolar spindlesmedicine.drugCurrent medicinal chemistry
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